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1 were evaluated for generalization to cocaine drug discrimination after i.p. administration, and twelv
2 urement of stimulated locomotor activity and drug discrimination against cocaine (10 mg/kg, ip) with
3 roduced dose-related effects in food intake, drug-discrimination and neurotoxicity studies.
4 nd in vitro, 6b, was tested in the two-lever drug discrimination assay in rats trained to discriminat
5   For example, 7b substituted for LSD in the drug discrimination assay with an ED50 of 61 nmol/kg and
6 tive compounds yet discovered in the in vivo drug discrimination assay.
7                                    Two-lever drug discrimination assays in rats trained to discrimina
8        Falling into this latter category was drug discrimination behavior, where dual FAAH/MAGL block
9 ne, do not lead to locomotor stimulation and drug discrimination behaviors in animal models.
10 and sedative side effects of ketamine in the drug discrimination, conditioned place preference, pre-p
11                                          The drug discrimination data in both LSD- and DOI-trained ra
12 ayed initially for activity in the two-lever drug discrimination (DD) paradigm in rats trained to dis
13 d along with 1 for activity in the two-lever drug discrimination (DD) paradigm in rats trained to dis
14                                        Using drug discrimination learning, rats were trained to discr
15                   Indeed, through the use of drug discrimination methods, rats show decreased sensiti
16 pound was selected for testing in an in vivo drug discrimination model of hallucinogenesis.
17 ike behavioral activity in the rat two lever drug discrimination model that was slightly more potent
18 ys, engender nicotine-like responding in rat drug discrimination, or alter current amplitude in alpha
19  and evaluated for activity in the two-lever drug discrimination paradigm in rats trained to discrimi
20 like activity was evaluated in the two-lever drug discrimination paradigm using LSD- and DOI-trained
21 en-like activity, evaluated in the two-lever drug discrimination paradigm using LSD-trained rats, was
22             The ED(50) of 6 in the two-lever drug discrimination paradigm using rats trained to discr
23 a, catalepsy, locomotor activity, and in the drug discrimination paradigm.
24 ompared to cocaine in locomotor activity and drug discrimination paradigms in mice.
25  from vehicle in a two-lever food-reinforced drug discrimination procedure.
26 or nicotine-like interoceptive effects under drug-discrimination procedures, and neither of the FAAH
27 , and locomotor activity effects in mice and drug discrimination results in cocaine-trained rats of t
28 he new analogue substituted fully for LSD in drug discrimination studies and was 5-fold more potent t
29                                              Drug discrimination studies employing a training dose of
30 10 mg/kg) does not substitute for cocaine in drug discrimination studies in rats.
31 omotor effects or substituted for cocaine in drug discrimination studies, suggesting that the mu-opio
32                                              Drug-discrimination studies failed to demonstrate a sign
33                                           In drug-discrimination studies these selected compounds inc
34                                         In a drug discrimination study procedure, none of these three
35 a contextual CS in that appetitive Pavlovian drug discrimination task.
36                                      In vivo drug discrimination techniques and in vitro receptor/tra
37 ere shown to fully substitute for cocaine in drug discrimination tests in rats, and one had a very lo
38 d have potency similar to that of cocaine in drug discrimination tests.
39 eir ability to substitute for cocaine in rat drug discrimination tests.

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