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1 were evaluated for generalization to cocaine drug discrimination after i.p. administration, and twelv
2 urement of stimulated locomotor activity and drug discrimination against cocaine (10 mg/kg, ip) with
4 nd in vitro, 6b, was tested in the two-lever drug discrimination assay in rats trained to discriminat
5 For example, 7b substituted for LSD in the drug discrimination assay with an ED50 of 61 nmol/kg and
10 and sedative side effects of ketamine in the drug discrimination, conditioned place preference, pre-p
12 ayed initially for activity in the two-lever drug discrimination (DD) paradigm in rats trained to dis
13 d along with 1 for activity in the two-lever drug discrimination (DD) paradigm in rats trained to dis
17 ike behavioral activity in the rat two lever drug discrimination model that was slightly more potent
18 ys, engender nicotine-like responding in rat drug discrimination, or alter current amplitude in alpha
19 and evaluated for activity in the two-lever drug discrimination paradigm in rats trained to discrimi
20 like activity was evaluated in the two-lever drug discrimination paradigm using LSD- and DOI-trained
21 en-like activity, evaluated in the two-lever drug discrimination paradigm using LSD-trained rats, was
26 or nicotine-like interoceptive effects under drug-discrimination procedures, and neither of the FAAH
27 , and locomotor activity effects in mice and drug discrimination results in cocaine-trained rats of t
28 he new analogue substituted fully for LSD in drug discrimination studies and was 5-fold more potent t
31 omotor effects or substituted for cocaine in drug discrimination studies, suggesting that the mu-opio
37 ere shown to fully substitute for cocaine in drug discrimination tests in rats, and one had a very lo
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