1 hour, consistent with rapid distribution and drug elimination.

2 s hepatic expression and function in hepatic drug elimination.

3 ysis revealed a three-compartmental model of drug elimination and a long terminal half-life (154 +/-

4 s detailed pharmacokinetic phenomena such as drug elimination and accumulation over multiple doses.

5 rug targets, downstream activation pathways, drug elimination, and toxicity activation.

6 y play a role in organic solute transport or drug elimination by the kidney.

7 h ketamine and CP-101,606 augment AEPs after drug elimination, consistent with synaptic potentiation

8 ic self-resistance mechanisms, which include drug elimination, drug modification, target modification

9 wer volume of distribution (17.4mL/kg).Total drug elimination from the circulation after the administ

10 s have identified variants in genes encoding drug elimination or drug target pathways that in some ca

11 mors versus the intestinal mucosa during the drug elimination period, compared with the ratio during

12 duce fetal exposure through a cholestyramine drug elimination procedure.

13 r filtration and a major mechanism for renal drug elimination, suggesting important clinical conseque

14                        The principal mode of drug elimination was renal.

15  association of oxidative-stress defense and drug elimination with treatment failure in DLBCL and ide