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1  been proposed as another cause of suspected drug-induced liver disease.
2 sment remain an important aspect in defining drug-induced liver disease and proposed new upper limits
3 ulties inherent in establishing causality of drug-induced liver disease and the potential negative co
4 emains very involved in the process by which drug-induced liver disease can be detected early in drug
5 orms of liver injury, and several others had drug-induced liver disease confirmed by additional repor
6                                A workshop on drug-induced liver disease cosponsored by the Food and D
7  drug withdrawals and new general reviews of drug-induced liver disease (DILD), including a review of
8 and first-time reports of various aspects of drug-induced liver disease for several classes of compou
9 se remains low, a large case-series of acute drug-induced liver disease from Spain and Sweden support
10  epidemiological and mechanistic features of drug-induced liver disease in 2005.
11 logical, and epidemiological descriptions of drug-induced liver disease in the calendar year 2004.
12  hold the key to treatment and prevention of drug-induced liver disease in the future and discusses t
13 nisms of injury to predict and avert serious drug-induced liver disease in the future from drugs unde
14                                     Although drug-induced liver disease is less common in children, s
15                                              Drug-induced liver disease remains an important cause of
16                                              Drug-induced liver disease remains an important topic of
17                       While the frequency of drug-induced liver disease remains low, a large case-ser
18  any prescription drugs withdrawn because of drug-induced liver disease, the US Food and Drug Adminis
19 ies, more than half the instances of alleged drug-induced liver disease were found to have other caus
20 as the most commonly reported agents causing drug-induced liver disease, with acetaminophen continuin

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