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1 s of drugs of abuse and, possibly, increased drug seeking behavior.
2 heroin dependence and their association with drug seeking behavior.
3 avior and may have a role in the etiology of drug-seeking behavior.
4 aminergic neurons involved in motivation and drug-seeking behavior.
5 t/orx has been implicated in food reward and drug-seeking behavior.
6 accumbens (NAc) exerts powerful control over drug-seeking behavior.
7 lves, which is an effect that could underlie drug-seeking behavior.
8 symptoms of cocaine addiction is compulsive drug-seeking behavior.
9 and glutamatergic systems may play a role in drug-seeking behavior.
10 e ventral prefrontal cortex had no effect on drug-seeking behavior.
11 including physical dependence and compulsive drug-seeking behavior.
12 rtant for maintaining prolonged sequences of drug-seeking behavior.
13 ns underlies cocaine-primed reinstatement of drug-seeking behavior.
14 ere inactivation of only the cBLA attenuated drug-seeking behavior.
15 geneous than commonly thought for regulating drug-seeking behavior.
16 ired CSs intact and able to continue driving drug-seeking behavior.
17 rding effects of cocaine and cocaine-induced drug-seeking behavior.
18 actions between these factors for motivating drug-seeking behavior.
19 series circuit that mediates cocaine-induced drug-seeking behavior.
20 t also play a role in drug reinforcement and drug-seeking behavior.
21 by inducing a craving response that prompts drug-seeking behavior.
22 e transmission in cocaine-induced relapse to drug-seeking behavior.
23 It also failed to induce reinstatement of drug-seeking behavior.
24 mediator of cocaine-induced reinstatement of drug-seeking behavior.
25 hin the DG that could directly contribute to drug-seeking behavior.
26 ivational states that instigate and maintain drug-seeking behavior.
27 d heroin self-administration and cue-induced drug-seeking behavior.
28 ian memories that can precipitate relapse to drug-seeking behavior.
29 Addiction involves an inability to control drug-seeking behavior.
30 discover new therapeutic candidates to treat drug-seeking behavior.
31 drug users, and are thought to facilitate a drug-seeking behavior.
32 d effects on an addict's emotional state and drug-seeking behavior.
33 ortex (mPFC) is implicated in the relapse of drug-seeking behavior.
34 muli, such cues can instigate and invigorate drug-seeking behavior.
35 C) that promote behavioral sensitization and drug-seeking behavior.
36 e memory processes involved in extinction of drug-seeking behavior.
37 ecific HDAC is involved in the extinction of drug-seeking behavior.
38 The ventral pallidum (VP) is necessary for drug-seeking behavior.
39 craving") that instigates and/or invigorates drug-seeking behavior.
40 ological responses, craving, withdrawal, and drug-seeking behavior.
41 the study of anxiogenesis and stress-induced drug-seeking behavior.
42 th the ability of a drug-paired cue to drive drug-seeking behavior.
43 ility of drug-predictive cues to precipitate drug-seeking behavior.
44 utamate signaling plays an essential role in drug-seeking behavior.
45 sducing stimuli into salient cues that drive drug-seeking behavior.
46 tic disorders or predispose an individual to drug-seeking behavior.
47 activation in drug-induced reinstatement of drug-seeking behavior.
48 y contribute to cue-induced drug craving and drug-seeking behavior.
49 indings that have important implications for drug-seeking behavior.
50 this crucial circuitry to promote compulsive drug-seeking behavior.
51 icantly enhanced resistance to extinction of drug-seeking behavior.
52 inhibition on sensitized behavior to foster drug-seeking behavior.
53 tal cellular phenomenon driving pathological drug-seeking behavior.
54 fic behavioral modifications associated with drug seeking behaviors.
55 considerations when designing experiments on drug-seeking behaviors.
56 in dopamine-mediated craving and relapse to drug-seeking behaviors.
57 med in regions involved in the extinction of drug-seeking behaviors.
58 differences in extinction and incubation of drug-seeking behaviors.
59 g modulator that potentially plays a role in drug-seeking behaviors.
60 at regulates orexin neuronal activity during drug-seeking behaviors.
61 modulating gene expression that may subserve drug-seeking behaviors.
62 onergic state underlying depression-like and drug-seeking behaviors.
63 l raphe nucleus that mediates depressive and drug-seeking behaviors.
64 tress modulation, depression, and relapse to drug-seeking behaviors.
65 ed memories.SIGNIFICANCE STATEMENT Continued drug-seeking behavior, a defining characteristic of coca
66 ished efficacy of the cocaine S(D) to elicit drug-seeking behavior after 4 months of abstinence paral
67 e with the ability of multiple cues to drive drug-seeking behavior after just one reactivation and tr
68 with OCT3 mediates corticosterone effects on drug-seeking behavior and establish OCT3 function as an
69 e neuroadaptations underlying stress-induced drug-seeking behavior and may be useful in the treatment
70 tagonist, on cocaine-primed reinstatement of drug-seeking behavior and on cocaine-induced changes in
71 al justice system often results from illegal drug-seeking behavior and participation in illegal activ
72 cleus accumbens (NAc) facilitate conditioned drug-seeking behavior and primarily originate from media
74 opeptide released into the VTA that promotes drug-seeking behaviors and potentiates excitatory synapt
75 ession, is well known to increase relapse of drug seeking behavior, and can adversely impact health.
77 iously drug-associated stimuli on subsequent drug-seeking behavior, and the NMDA subtype of glutamate
82 ketamine doses used were capable of inducing drug-seeking behaviors as measured by place preference c
83 footshock stress did not by itself reinstate drug-seeking behavior but potentiated reinstatement in r
84 hippocampal neurogenesis and drug-taking or drug-seeking behaviors, but the lack of a causative link
85 xciting new possibility is the extinction of drug-seeking behavior by manipulation of epigenetic mech
88 In the present study, the extent to which drug-seeking behavior could be subject to these differen
89 e-associated cue was sufficient to reinstate drug-seeking behavior, despite the continued presence of
92 tly extinguished and spontaneous recovery of drug-seeking behavior following presentation of previous
93 n about the role that astrocytes may play in drug-seeking behavior for commonly abused substances.
96 a mean of preventing the "high" and reducing drug-seeking behavior has become a major strategy in med
98 established, the neurophysiological bases of drug-seeking behavior have yet to be fully elucidated.
99 s been shown to facilitate the extinction of drug-seeking behavior in a manner resistant to reinstate
100 (2) priming- or cue-induced reinstatement of drug-seeking behavior in abstinent subjects (models of r
104 potent of the analogues successfully reduced drug-seeking behavior in an animal model of drug-relapse
105 hanges in reward circuit activity that drive drug-seeking behavior in animal models of addiction.
107 ntal cues, suggesting that cues that trigger drug-seeking behavior in nonhuman primates do not cause
110 hat this disconnection selectively decreased drug-seeking behavior in rats extensively trained under
111 ions on the maintenance and reinstatement of drug-seeking behavior in rats trained to self-administer
114 we tested whether an estrogen could augment drug-seeking behavior in response to an ordinarily subth
115 ions and in the mediation of drug-taking and drug-seeking behaviors in animal models of addiction.
117 on of only the rBLA blocked reinstatement of drug-seeking behavior induced by cocaine-associated cues
118 BLA and cBLA attenuated the reinstatement of drug-seeking behavior induced by cocaine-associated cues
121 ms by which stress triggers reinstatement of drug-seeking behaviors is particularly pertinent to nico
122 oned behaviors, such as conditioned fear and drug-seeking behavior, is a process of active learning,
123 e primary targets for cocaine, the continued drug-seeking behavior of transporter knock-out mice sugg
125 session, a drug-priming injection reinstated drug-seeking behavior only in rats that in the past had
126 ears to contribute both to the initiation of drug-seeking behavior (pre-lever press phasic neuronal r
127 kg, i.p.) alone were sufficient to reinstate drug-seeking behavior, pretreatment with E2 potentiated
128 comotor-activating effects of cocaine and on drug-seeking behavior, rats receiving methyl supplementa
129 tly reduced previously acquired instrumental drug-seeking behavior that depends on drug-associated cu
130 pproach to facilitate learned suppression of drug-seeking behavior that may aid drug abstinence.
131 sions profoundly impaired the acquisition of drug-seeking behavior that was maintained by drug-associ
132 Pavlovian and operant conditioning influence drug-seeking behavior, the role of rapid dopamine signal
133 ssential for cocaine self-administration and drug-seeking behavior, there is limited knowledge of the
137 cellular dopamine during extended periods of drug-seeking behavior triggered by a visual cue was dete
138 pear to selectively and dissociably regulate drug-seeking behavior under conditions of cocaine abstin
139 Drugs of abuse are able to elicit compulsive drug-seeking behaviors upon repeated administration, whi
140 cannabis, on heroin self-administration and drug-seeking behavior using an experimental rat model.
141 of GRIA1, a glutamatergic gene implicated in drug-seeking behavior, verified the increased enrichment
143 During subsequent reinstatement sessions, drug-seeking behavior was assessed after noncontingent p
146 ration, and cocaine-induced reinstatement of drug-seeking behavior, whereas R-MOD inhibited cocaine-i
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