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1 zed that CS exposure modulates expression of Dual oxidase 1 (Duox-1), a NADPH oxidases known to gener
2  indicating involvement of the NADPH oxidase dual oxidase 1 (DUOX1) in epithelial wound responses, we
3                                              Dual oxidase 1 (Duox1) is the NADPH oxidase responsible
4 itical for hBD-3 induction, while ADAM10 and dual oxidase 1 (Duox1) were not required for hBD-3 induc
5      Activation of the NADPH oxidase homolog dual oxidase 1 (DUOX1) within the airway epithelium repr
6                                              Dual oxidase 1 (Duox1), a homologue of glycoprotein p91(
7          Here, we report a critical role for dual oxidase 1 (Duox1), a newly identified NADPH oxidase
8 metry, we identified the local production of dual oxidase 1 (Duox1)-derived H(2)O(2) by Tnfa- and Tnf
9 rs, and importantly small interfering RNA of dual oxidase 1 inhibited LPS-induced wound repair.
10 a novel TLR-4-->protein kinase C alphabeta-->dual oxidase 1-->reactive oxygen species-->TACE-->TGF-al
11                                              Dual oxidase 2 (DUOX2), a hydrogen-peroxide generator at
12                                              Dual oxidase 2 (DUOX2), an NADPH:O(2) oxidoreductase fla
13 ion of Panx1 was due to the up-regulation of dual oxidase 2 (Duox2).
14                                              Dual oxidase 2 is a member of the NADPH oxidase (Nox) ge
15                     One pathway involves the dual oxidase 2-mediated generation of reactive oxygen sp
16 ditis elegans (Ce), and are termed Duox for "dual oxidase" because they have both a peroxidase homolo
17       Specifically, reducing expression of a dual oxidase, Ce-Duox1/BLI-3, causes a decrease in ROS p
18 o light new processes and mediators, such as dual oxidases, defense against radiation injuries, and n
19 idence supports a role for the NADPH oxidase dual oxidase (Duox) as an important source for regulated
20                                              Dual oxidase (DUOX) enzymes support a wide variety of es
21 tor of Chagas disease, we show that an ovary dual oxidase (Duox), a NADPH oxidase, is the source of t
22 on, we show that this gradient is created by dual oxidase (Duox), and that it is required for rapid r
23 dityrosine network (DTN) is dependent upon a dual oxidase (Duox), which is a member of the NADPH oxid
24                          Here, we identify a dual oxidase (Duox)-regulating pathway that contributes
25 ore of this enzyme, were described recently; dual oxidase (Duox)1/thyroid oxidase 1 and Duox2/thyroid
26                We recently demonstrated that dual oxidase (Duox)2, an NADPH oxidase essential for rea
27                                              Dual oxidases (DUOX) are conserved reduced nicotinamide
28                                              Dual oxidases (Duox1 and Duox2) are plasma membrane-targ
29        An abnormal increase of antimicrobial dual oxidase (DUOX2) expression was detected in associat
30                                              Dual oxidase enzyme of airway epithelial cells generated
31 uggests a potentially conserved role for the dual oxidase family in hydrogen peroxide production and
32 ecreted by the Anopheles gambiae midgut, and dual oxidase form a dityrosine network that decreases gu
33           Finally, we show that a sea urchin dual oxidase homolog, Udx1, is responsible for generatin
34                             By contrast, the dual oxidase isoforms produce ROS that provide vital pro
35                             Two members, the dual oxidase isozymes DUOX1 and DUOX2, share a structura
36 ination with BLI-3, an H2O2-generating NADPH dual oxidase, MLT-7 is essential for post-embryonic deve
37                      Although the N-terminal dual oxidase motif has been proposed to directly convert
38                                          The dual oxidase-thiocyanate-lactoperoxidase (Duox/SCN(-)/LP
39  oxygen to hydrogen peroxide by a sea urchin dual oxidase, Udx1.

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