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1 zed that CS exposure modulates expression of Dual oxidase 1 (Duox-1), a NADPH oxidases known to gener
2 indicating involvement of the NADPH oxidase dual oxidase 1 (DUOX1) in epithelial wound responses, we
4 itical for hBD-3 induction, while ADAM10 and dual oxidase 1 (Duox1) were not required for hBD-3 induc
8 metry, we identified the local production of dual oxidase 1 (Duox1)-derived H(2)O(2) by Tnfa- and Tnf
10 a novel TLR-4-->protein kinase C alphabeta-->dual oxidase 1-->reactive oxygen species-->TACE-->TGF-al
16 ditis elegans (Ce), and are termed Duox for "dual oxidase" because they have both a peroxidase homolo
18 o light new processes and mediators, such as dual oxidases, defense against radiation injuries, and n
19 idence supports a role for the NADPH oxidase dual oxidase (Duox) as an important source for regulated
21 tor of Chagas disease, we show that an ovary dual oxidase (Duox), a NADPH oxidase, is the source of t
22 on, we show that this gradient is created by dual oxidase (Duox), and that it is required for rapid r
23 dityrosine network (DTN) is dependent upon a dual oxidase (Duox), which is a member of the NADPH oxid
25 ore of this enzyme, were described recently; dual oxidase (Duox)1/thyroid oxidase 1 and Duox2/thyroid
31 uggests a potentially conserved role for the dual oxidase family in hydrogen peroxide production and
32 ecreted by the Anopheles gambiae midgut, and dual oxidase form a dityrosine network that decreases gu
36 ination with BLI-3, an H2O2-generating NADPH dual oxidase, MLT-7 is essential for post-embryonic deve
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