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1 ension (PPHN) after prenatal ligation of the ductus arteriosus.
2 rta, suggesting little effect on tone of the ductus arteriosus.
3 neonates without clinically suspected patent ductus arteriosus.
4  predictive value for the presence of patent ductus arteriosus.
5 rteriosus, with a 92% specificity for patent ductus arteriosus.
6 phy (RVH) caused by prolonged closure of the ductus arteriosus.
7 ath or bronchopulmonary dysplasia and patent ductus arteriosus.
8 shed techniques for coil occlusion of patent ductus arteriosus.
9 essels, SM2 is precociously expressed in the ductus arteriosus.
10 , we observe a strong beta-gal signal in the ductus arteriosus.
11 ed calcium channels, and constriction of the ductus arteriosus.
12 ribute to the unique muscle phenotype of the ductus arteriosus.
13  shunting following coil occlusion of patent ductus arteriosus.
14 h those of 14 preterm infants without patent ductus arteriosus.
15 e-dimensional image of the great vessels and ductus arteriosus.
16 d as they may cause premature closure of the ductus arteriosus.
17 atal necrotizing enterocolitis or persistent ductus arteriosus.
18  a Notch ligand, die postnatally from patent ductus arteriosus.
19  the development of smooth muscle within the ductus arteriosus.
20  of indomethacin to treat symptomatic patent ductus arteriosus.
21 all groups, as was acute constriction of the ductus arteriosus.
22 ung disease, there is delayed closure of the ductus arteriosus.
23 nt, despite success in closure of the patent ductus arteriosus.
24 ous lobar pulmonary veins or a silent patent ductus arteriosus.
25 on (PPHN) following prenatal ligation of the ductus arteriosus.
26 , atrial septal defects (20/47, 43%), patent ductus arteriosus (16/47, 34%), persistent left superior
27 21, 261); superior vena cava, 137 (77, 197); ductus arteriosus, 187 (109, 265); descending aorta, 252
28  4 with atrial septal defects, 2 with patent ductus arteriosus, 2 with tetralogy of Fallot, and 1 wit
29 ransposition of the great vessels, 3 patient ductus arteriosus, 3 partial anomalous pulmonary venous
30                The narrowest diameter of the ductus arteriosus (4.3 +/- 0.4 mm vs. 6.9 +/- 2.6 mm, p
31 1 (29, 53); superior vena cava, 29 (15, 43); ductus arteriosus, 41 (25, 57); descending aorta, 55 (35
32 ngenital heart disease that results when the ductus arteriosus, a muscular artery, fails to remodel a
33 and Cx43 knockout mice an attenuation of the ductus arteriosus, a phenotype which may be indicative o
34  PGT plays a critical role in closure of the ductus arteriosus after birth by ensuring a reduction in
35 etal ductus arteriosus and in closure of the ductus arteriosus after birth.
36 normal patterning and differentiation of the ductus arteriosus and adjacent descending aorta.
37  differentiation in the vascular wall of the ductus arteriosus and adjacent descending aorta.
38               In other areas, such as patent ductus arteriosus and atrioventricular septal defect, th
39 banding with catheterization stenting of the ductus arteriosus and balloon atrial septostomy, especia
40 l relationship exists between patency of the ductus arteriosus and chronic lung disease and other mor
41 h epilepsy and other signs, including patent ductus arteriosus and coagulopathy, while hemizygous mal
42                                       Patent ductus arteriosus and coarctation of aorta occurred >3 t
43                  All patients had persistent ductus arteriosus and congenital mydriasis, and variable
44 al valve inflow and direction of flow in the ductus arteriosus and descending aorta were unrelated to
45 599+1delG) was identified in familial patent ductus arteriosus and found to disrupt normal splicing o
46 ole both in maintaining patency of the fetal ductus arteriosus and in closure of the ductus arteriosu
47 riosus to close after birth is termed patent ductus arteriosus and is one of the most common congenit
48 oth muscle differentiation in the developing ductus arteriosus and may promote precocious expression
49 cular interest because it also causes patent ductus arteriosus and moyamoya disease.
50 s describing an association between a patent ductus arteriosus and the development of morbidities, su
51 ending, transverse and descending aorta, the ductus arteriosus, and across the aortic and mitral valv
52 comitant cardiac lesion, for example, patent ductus arteriosus, and aortic cross-clamp time were dete
53 ents with interatrial communications, patent ductus arteriosus, and pulmonary artery stenosis requiri
54 ung age at angioplasty, presence of a patent ductus arteriosus, and the diameters of the aortic isthm
55 o [AOR], 0.81; 95% CI, 0.67-0.98) and patent ductus arteriosus (AOR, 0.74; 95% CI, 0.62-0.89).
56 ect (aORs ranging from 1.29 to 2.17), patent ductus arteriosus [aORs = 1.54, 1.63; 95% confidence int
57              Therapies designed to close the ductus arteriosus are contraindicated in some settings a
58      Much of the normoxic contraction of the ductus arteriosus at birth is related to calcium entry t
59                               Closure of the ductus arteriosus at birth, essential for postnatal adap
60 ambs (126+/-3 d; 147 d, term) we ligated the ductus arteriosus at surgery, and treated animals with e
61 on being stenting or stent redilation of the ductus arteriosus, balloon aortic valvuloplasty, and ste
62 onates subsequently diagnosed to have patent ductus arteriosus, based on clinical suspicion and echoc
63 re infants with than in those without patent ductus arteriosus because afterload is lower in the form
64  and atrioventricular septal defects, patent ductus arteriosus, bicuspid aortic valve, and coarctatio
65 ation, P62R, had a high prevalence of patent ductus arteriosus but had only mild abnormalities of fac
66 y intense beta-gal staining localizes to the ductus arteriosus but is absent or minimal in the pulmon
67 are common after coil embolization of patent ductus arteriosus, but most close spontaneously.
68 ght to establish and maintain patency of the ductus arteriosus by a new method of transvascular forma
69 ion of this pathway may contribute to patent ductus arteriosus by affecting the development of smooth
70 nificant role in control of major vessel and ductus arteriosus caliber in the rat fetus.
71 dysmorphology, osteochondrodysplasia, patent ductus arteriosus, cardiomegaly, pericardial effusion, a
72         In the vast majority of infants, the ductus arteriosus closes by 3 days of life.
73 erization of PGHS1-PGHS2 may explain how the ductus arteriosus closes normally at birth in mice expre
74                     Incidence of spontaneous ductus arteriosus closure was increased, likely contribu
75              To determine the role of PGT in ductus arteriosus closure, we used a gene-targeting stra
76     Neural crest-derived SMCs populating the ductus arteriosus (DA) and great arteries exhibited a ce
77                                      How the ductus arteriosus (DA) closes at birth remains unclear.
78                          Wild-type embryonic ductus arteriosus (DA) exhibited high levels of PRDM6, w
79                                          The ductus arteriosus (DA) is a vessel whose patency is requ
80                               Closure of the ductus arteriosus (DA) is essential for the transition f
81                                          The ductus arteriosus (DA) of newborn infants exposed in ute
82 as in the fetus, PGs maintain patency of the ductus arteriosus (DA), a vascular shunt that transmits
83  placental mammals depends on closure of the ductus arteriosus (DA), an arterial connection in the fe
84 tic change that occurs is the closure of the ductus arteriosus (DA), an arterial connection in the fe
85  occurs is the closure and remodeling of the ductus arteriosus (DA), an arterial connection in the fe
86 nd neonate, including adverse effects on the ductus arteriosus (DA).
87            Fetal aortic and pulmonary valve; ductus arteriosus (DA); and right (RPA), left (LPA), and
88 ghts into the genetic programs that underlie ductus arteriosus development and closure.
89 y artery diameter Z score <or=-2.5 or patent ductus arteriosus diameter <or=2 mm was 97% sensitive an
90 nly one infant required ligation of a patent ductus arteriosus during ECMO.
91 r morphogenetic development of the aorta and ductus arteriosus during embryonic and postnatal surviva
92 oth muscle layers of the tunica media in the ductus arteriosus exhibit positive beta-gal staining.
93 somal dominant trait characterized by patent ductus arteriosus, facial dysmorphism and hand anomalies
94 ation was observed between PM(10) and patent ductus arteriosus (for an interquartile range increase i
95 amicin clearance was decreased in the patent ductus arteriosus group vs. the control group (40.02 vs.
96                      The infants with patent ductus arteriosus had higher values for ventricular perf
97 hough transcatheter coil occlusion of patent ductus arteriosus has gained popularity, few follow-up d
98 r the prevention and treatment of the patent ductus arteriosus have not documented a decrease in the
99  transcription factor associated with patent ductus arteriosus in humans, was uniquely expressed in m
100 ms underlying O2-induced constriction of the ductus arteriosus in late-gestation fetal rabbits.
101 hysiologic flow across the atrial septum and ductus arteriosus in the human fetus.
102                              Ligation of the ductus arteriosus in utero produces pulmonary hypertensi
103 ) receptor antagonist, after ligation of the ductus arteriosus in utero.
104 in cardiopulmonary defects, including patent ductus arteriosus, in 30 to 40% of the mice.
105 the effector mechanism for O2 sensing in the ductus arteriosus involves the coordinated action of del
106                                          The ductus arteriosus is a specialized blood vessel containi
107                                          The ductus arteriosus is a vessel that connects the pulmonar
108                                          The ductus arteriosus is a vital fetal structure allowing bl
109                                          The ductus arteriosus is an arterial vessel that shunts bloo
110 opment and persists in the neonate until the ductus arteriosus is completely closed.
111             In the preterm newborn, a patent ductus arteriosus is in large part a result of the incre
112 an pulmonary artery pressure (PAP) 8 d after ductus arteriosus ligation (78+/-2, HTN vs. 70+/-4 mmHg,
113                                       Patent ductus arteriosus ligation among preterm neonates younge
114                       Since the first patent ductus arteriosus ligation by Robert Gross of Boston in
115   All 14 premature infants undergoing patent ductus arteriosus ligation in a 1-year period at our ins
116           In preterm infants, patency of the ductus arteriosus may represent a normal physiologic ada
117 istance associated with ligation of a patent ductus arteriosus might worsen ventricular performance i
118 rses of gentamicin were administered (patent ductus arteriosus, n = 106; control, n = 216).
119                                       Patent ductus arteriosus occlusion can be accomplished by umbre
120 ersal of the physiologic shunting across the ductus arteriosus or atrial septum in utero (i.e., left
121                     Patients with persistent ductus arteriosus or congenital mydriasis with a label o
122                  Impaired development of the ductus arteriosus or disruption of signaling pathways th
123 n with secundum atrial septal defect, patent ductus arteriosus, or pulmonic stenosis have had infecti
124 olume of distribution was greater for patent ductus arteriosus patients (0.61 L/kg) than for controls
125  of buttoned device (BD) occlusion of patent ductus arteriosus (PDA) in a large number of patients wi
126 us for the screening and treatment of patent ductus arteriosus (PDA) in extremely preterm infants.
127                          Nonsyndromic patent ductus arteriosus (PDA) is a common congenital heart def
128                                       Patent ductus arteriosus (PDA) is a common congenital heart dis
129                                       Patent ductus arteriosus (PDA) is a relatively common form of c
130                                       Patent ductus arteriosus (PDA) is associated with increased mor
131 Observational studies have associated patent ductus arteriosus (PDA) ligation among preterm infants w
132                                       Patent ductus arteriosus (PDA) poses a diagnostic and therapeut
133 rformed to determine the frequency of patent ductus arteriosus (PDA) reopening and the factors that m
134  may undergo palliation with either a patent ductus arteriosus (PDA) stent or a modified Blalock-Taus
135 theter closure of a moderate to large patent ductus arteriosus (PDA) using conventional techniques is
136                                   The patent ductus arteriosus (PDA) was the first congenital heart l
137 ne atrial septal defect (ASD) and two patent ductus arteriosus (PDA) were missed by DSCT.
138                         We found that patent ductus arteriosus (PDA), a common congenital heart disea
139 il occlude the moderate to large size patent ductus arteriosus (PDA).
140 ), coarctation of the aorta (CoA) and patent ductus arteriosus (PDA).
141 eceiving indomethacin (INDO) to close patent ductus arteriosus (PDA).
142 neurysmal dilatation of the aorta and patent ductus arteriosus (PDA).
143 ut died prior to postnatal day 3 from patent ductus arteriosus (PDA).
144 locus at 16p for TAAD associated with patent ductus arteriosus (PDA).
145 eptal defect (ASD); device closure of patent ductus arteriosus (PDA); pulmonary valvuloplasty; aortic
146 use of Gianturco coils to close large patent ductus arteriosus (PDAs) (> or = 3.5 mm) and describes t
147                   Maintaining patency of the ductus arteriosus postnatally is necessary with many for
148  and atrioventricular septal defects; patent ductus arteriosus; pulmonary stenosis; aortic stenosis;
149  right and left pulmonary arteries (QP), and ductus arteriosus (QDA) were calculated in 63 normal fet
150 r leukomalacia, chronic lung disease, patent ductus arteriosus requiring surgery, retinopathy of prem
151                 Although a persistent patent ductus arteriosus results in diminished cardiac function
152 s been controversy as to whether or when the ductus arteriosus should be closed by either pharmacolog
153 ts who were delivered alive from the reverse ductus arteriosus shunt group and 4 of 12 from the rever
154                                     Reversed ductus arteriosus shunting was found with severe right h
155  fetal development, a specialized vessel the ductus arteriosus, shunts blood from the pulmonary arter
156 septal defects, patent foramen ovale, patent ductus arteriosus, stent placement for pulmonary artery
157 sure can result in persistent patency of the ductus arteriosus, the third most common congenital hear
158 increase in oxygen causes contraction of the ductus arteriosus, thus diverting blood flow to the lung
159                               Failure of the ductus arteriosus to close after birth is termed patent
160 ng should be altered in neonates with patent ductus arteriosus to reflect the impact of higher volume
161 ion, hypoplastic left heart syndrome, patent ductus arteriosus, valvar pulmonary stenosis, tetralogy
162 t transvascular formalin infiltration of the ductus arteriosus wall has been applied successfully to
163                                          The ductus arteriosus wall was successfully infiltrated and
164 f transvascular formalin infiltration of the ductus arteriosus wall.
165 d cases regardless of whether preterm patent ductus arteriosus was included.
166 ts who required surgical closure of a patent ductus arteriosus was lower in the budesonide group than
167 ficant constriction of the great vessels and ductus arteriosus was observed with L-NIL, whereas both
168                        A functionally closed ductus arteriosus was traversed with a specially designe
169 asthma, smoking in the household, and patent ductus arteriosus were predictive of wheezing in the pre
170 rsistent left superior vena cava, and patent ductus arteriosus, were present in 32% of patients.
171 opment of better therapy to close the patent ductus arteriosus, which constitutes approximately 10% o
172                     Necropsy revealed patent ductus arteriosus with normal intimal thickening but dil
173 n was a useful marker for presence of patent ductus arteriosus, with a 92% specificity for patent duc

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