ライフサイエンスコーパス: during exercise

1 PVR and PA pressure-flow response [DeltaPQ] during exercise).

2 e potential contribution of its upregulation during exercise.

3 termined by the magnitude of fatigue accrued during exercise.

4 hmias, or attenuated blood pressure response during exercise.

5 is common in patients with ePVH at rest and during exercise.

6 r-maximum levels of neural respiratory drive during exercise.

7 patients with COPD than in control subjects during exercise.

8 bserved phosphocreatine, pH and vOX kinetics during exercise.

9 ular oscillations in minute ventilation (VE) during exercise.

10 reduction of glucagon and cortisol responses during exercise.

11 own to improve energy metabolism at rest and during exercise.

12 anterior leaflet (AL) tethering at rest and during exercise.

13 are characterized by an impaired O2 kinetic during exercise.

14 ing despite increased right-to-left shunting during exercise.

15 l muscle blood flow and vascular conductance during exercise.

16 ave key roles in governing lipid homeostasis during exercise.

17 nce of the brain in the control of breathing during exercise.

18 which have now been recorded from in humans during exercise.

19 hemodynamics, blood gases, and gas exchange during exercise.

20 d-diastolic volume index (P=0.001) decreased during exercise.

21 phic measurements were performed at rest and during exercise.

22 isplaying a marginally prolonged QT interval during exercise.

23 and right heart catheterization at rest and during exercise.

24 Skeletal muscle generates ROS at rest and during exercise.

25 tory parameters, and peripheral oxygen usage during exercise.

26 A oxidation but not glucose uptake in muscle during exercise.

27 arterial pressure were measured at rest and during exercise.

28 sed on a decreased intraventricular gradient during exercise.

29 ventriculography were performed at rest and during exercise.

30 by impaired coronary blood flow at rest and during exercise.

31 x and prevents the increase in blood lactate during exercise.

32 scle ischemia and exacerbating muscle injury during exercise.

33 port to locomotor and to respiratory muscles during exercise.

34 to increase stroke volume and cardiac output during exercise.

35 y and circulating vasoconstrictor substances during exercise.

36 reserve or 80% of the maximally predicted HR during exercise.

37 g in the suppression of alpha cell secretion during exercise.

38 res HFD-mediated inhibition of CHO oxidation during exercise.

39 lic, and respiratory variables were assessed during exercise.

40 C throughout and restored whole-body CHO use during exercise.

41 nd were unable to maintain muscle ATP levels during exercise.

42 uring rest and exercise and in pH regulation during exercise.

43 namic measurements and blood gases performed during exercise.

44 od gases were measured at 1-minute intervals during exercise.

45 mHg) to evoke muscle metaboreflex activation during exercise.

46 are not obligatory to observe sympatholysis during exercise.

47 leucine; EAA, 1.87 g leucine] were consumed during exercise.

48 mportant mediator of enhanced cardiac output during exercise.

49 distinct hemodynamic groups were identified during exercise.

50 hyperaemia in healthy subjects, particularly during exercise.

51 amic ventricular-arterial coupling responses during exercise.

52 racy to estimate pulmonary arterial pressure during exercise.

53 for abnormal pulmonary hemodynamic response during exercise.

54 glucose release from the liver are increased during exercise.

55 iance and elastance and central hemodynamics during exercise.

56 l variability in the capacity to oxidize fat during exercise.

57 and prompted a reduction in arterial glucose during exercise.

58 F and EF >/=40%, IASD treatment reduces PCWP during exercise.

59 s, electrolyte balance, and electrolyte loss during exercise.

60 t was the pulmonary capillary wedge pressure during exercise.

61 ng men relied more on endogenous fatty acids during exercise.

62 ssion was explored by administering caffeine during exercise.

63 activity and lessen oxidative perturbations during exercise.

64 tterns were, however, more likely to persist during exercise.

65 nd radionuclide ventriculography at rest and during exercise.

66 s and facilitate breathing rate acceleration during exercise.

67 ,721 vs. 9,707 mm Hg/min(-1); p = 0.003) and during exercise (27,467 vs. 20,841 mm Hg/min(-1); p = 0.

68 Individuals were examined at rest and during exercise (40% peak exercise capacity) and separat

69 n were unaffected at rest, but were enhanced during exercise (-9 +/- 1%; P < 0.05 vs. before NTG).

70 ATP is released from skeletal muscle during exercise, a discovery dating back to 1969.

71 distinct hemodynamic groups were identified during exercise: a normal group, an exercise-induced pul

72 too sensor coupled to a wireless transceiver during exercise activity demonstrated its ability to con

73 ood flow (QIPAVA ) is either increased, e.g. during exercise, acute normobaric hypoxia, and the intra

74 Also during exercise, adaptation of proximal ventriculoarteri

75 and pulmonary arterial pressure at rest and during exercise after 9 weeks.

76 rial eNOS phosphorylation at S1177 increased during exercise after wortmannin treatment relative to v

77 During exercise, alpha(1)-mediated vasoconstriction was

78 During exercise, an abnormal rise in pulmonary capillary

79 cerebral and femoral circulation at rest and during exercise, an ideal model system characterized by

80 readmill running led to impaired performance during exercise and a small improvement in performance f

81 s responsible for glycogen dephosphorylation during exercise and acts during the cytosolic degradatio

82 CMR was performed on 34 subjects during exercise and free-breathing with the use of an un

83 iological evaluation can be safely performed during exercise and hyperemia in patients with severe ao

84 hors describe coronary physiological changes during exercise and hyperemia in the healthy heart and i

85 isms that promote increased IMTG utilization during exercise and improve insulin sensitivity followin

86 SIT and ET both increase net IMTG breakdown during exercise and increase in PLIN2 and PLIN5 protein

87 okine interleukin 6 is secreted from muscles during exercise and induces the release of GLP-1 that st

88 t compromised in muscle of patients with T2D during exercise and insulin stimulation.

89 to reduced CI and elevated filling pressures during exercise and may be an important surrogate for ex

90 ysis adjusted for age and sex, PASP increase during exercise and peak o2 per kilogram remained indepe

91 ently drawn from the femoral artery and vein during exercise and Q(m), a-(V(O(2))) difference and (V(

92 s = glucose + (1/2)lactate) would be similar during exercise and recovery at HA and sea level (SL).

93 In conclusion, the elevations in gCBF during exercise and recovery at HA serve to maintain CDO

94 appears to increase non-oxidative metabolism during exercise and recovery.

95 We tested this sensor on 20 volunteers during exercise and rest and measured their individual b

96 only during exercise were prescribed oxygen during exercise and sleep.

97 would predict greater dead space ventilation during exercise and that this would lead to impairment i

98 , but is reduced in older (>50 years) adults during exercise and with alveolar hypoxia, suggesting po

99 bic capacities) and IPAQ score were obtained during exercises and it was used to construction of four

100 PGI(2), also accumulate in the interstitium during exercise, and breathing 40% O(2) abolished the co

101 ems to be safe, reduces left atrial pressure during exercise, and could be a new strategy for the man

102 When detected during exercise, and especially at recovery, NSVT indica

103 pid rates of high-energy phosphate depletion during exercise, and impaired maximal oxidative capacity

104 plications for physiological fuel management during exercise, and relevance to pathophysiological con

105 Echocardiography at rest and during exercise, and selected biomarkers were assessed i

106 propose that the new SR-TT junctions formed during exercise, and that contain STIM1 and Orai1, funct

107 04); however, Ascorbate did not modulate CVC during exercise ( approximately 60% CVCmax ; both P > 0.

108 long-duration spaceflight on CBT at rest and during exercise are clearly lacking.

109 ity at rest and excessively high ventilation during exercise are common in heart failure.

110 tory adaptations, in which calories expended during exercise are counteracted by decreases in other a

111 gesting that glucoregulatory effects of Glp1 during exercise are mediated via the pancreatic Glp1r.

112 skeletal muscle oxygen consumption (V(o)(2)) during exercise are not well understood.

113 lling skeletal muscle oxygen consumption ( ) during exercise are not well understood.

114 age-related reductions in cerebral perfusion during exercise are partly associated with a lower P aC

115 ight ventricular-pulmonary vascular function during exercise as evidenced by lower right ventricular

116 racting muscle and improved muscle perfusion during exercise as measured by Doppler and microsphere c

117 o assess the role of RV measures at rest and during exercise as predictors of prognosis in asymptomat

118 f echocardiographic measurements at rest and during exercise as predictors of valve surgery in asympt

119 ddress the dynamic component of secondary MR during exercise as well.

120 decreases in OCI (by ~1.5) and (by ~10 mmHg) during exercise at 75% W(max).

121 ed research on the topic of oxidative stress during exercise at altitude.

122 imultaneous expired gas analysis at rest and during exercise before and after treatment with inhaled

123 imultaneous expired gas analysis at rest and during exercise, before and 15 min after treatment with

124 dynamic process, with increasing AL opening during exercise being associated with higher exercise EO

125 1 +/- 3% vs.; P < 0.05) and remained blunted during exercise (blockade: -15 +/- 5 vs. CONTROL: -14 +/

126 During exercise breathing cold and dry air, PFO+ subject

127 ally-mediated sweating alterations in humans during exercise brought about by warm and cool fluid ing

128 availability by increasing glucose synthesis during exercise but rather adapted by altering whole bod

129 AC5 phosphorylation in mouse skeletal muscle during exercise, but resulted in a compensatory increase

130 or were unchanged; ePAD/EDV ratio increased during exercise, but the increase was independent of a c

131 ct relative carbohydrate and fat utilization during exercise, but the older men had higher uptake of

132 The aorta is exposed to hemodynamic stress during exercise, but whether or not the aorta is larger

133 ng IL-6 is thought to maintain energy status during exercise by acting as an energy sensor for contra

134 extend our understanding of vascular control during exercise by identifying fibre-type-selective peri

135 ion for the age-related reduction in P aC O2 during exercise by the provision of supplementary CO2 is

136 uptake ( max) and cardiac output at rest and during exercise (C2H2 rebreathing) were measured at the

137 RV dilation during exercise can predict adverse ventricular-vascular

138 We here show that during exercise CBT rises higher and faster in space tha

139 During exercise, central command and reflexes from exerc

140 During exercise, CFIp declined within 1 minute from 0.56

141 During exercise, cognitive task performance was impaired

142 t would have higher rates of gluconeogenesis during exercise compared to those who follow a mixed mac

143 ker significantly reduced ventricular ectopy during exercise compared with placebo plus beta-blocker

144 r had a coefficient of variation (CV) of 27% during exercise, compared with 8% at rest.

145 Peak oxygen consumption during exercise correlated best with RVOT ejection fract

146 Stroke volume reserve during exercise correlates with exercise training respon

147 Measurement of EMGdi%max during exercise could prove useful in identifying patien

148 essure (PCWP) develop in patients with HFpEF during exercise coupled with impaired nitric oxide (NO)

149 exercise-induced arterial hypoxaemia (EIAH) during exercise decreases the severity of quadriceps fat

150 Women have a higher work of breathing during exercise, dedicate a greater fraction of whole-bo

151 Women have a greater work of breathing during exercise, dedicate a greater fraction of whole-bo

152 symptoms were difficulties in walking, pain during exercise, delayed motor milestones and learning d

153 those who developed the greatest hypoxaemia during exercise demonstrated the most attenuation of qua

154 In RMA patients, EOA increases during exercise despite fixed annular size.

155 maintained in hypoxic conditions at rest and during exercise, despite attenuated oxygen delivery foll

156 ed with wheeze in the past 12 months, wheeze during exercise, doctor and/or emergency room visits for

157 Increased exposure to air pollution during exercise does not outweigh beneficial effects of

158 or quiet at rest that, when activated, e.g., during exercise, drives active expiration.

159 ients with hypertrophic cardiomyopathy (HCM) during exercise echocardiography.

160 n, knowledge on dynamic mitral regurgitation during exercise, effectiveness of therapy and appropriat

161 vasive lactate sensing in human perspiration during exercise events using a flexible printed temporar

162 ed in rates of GNG between groups at rest or during exercise (Exercise: LCHF, 2.8 +/- 0.4 mg kg(-1) m

163 Endurance capacity during exercise exhaustion test was impaired in apoA-I k

164 a1 -adrenergic vasoconstriction is augmented during exercise following inhibition of inwardly rectify

165 l challenge, and no episodes of hypoglycemia during exercise for >4 years after gene transfer.

166 found in all three acute exercise paradigms: during exercise (g=0.101; 95% confidence interval [CI];

167 During exercise, glucose levels were equivalently clampe

168 During exercise, glycolytic intermediates, TCA cycle int

169 illary wedge pressure at rest (>15 mm Hg) or during exercise (>25 mm Hg).

170 tify abnormal pulmonary hemodynamic response during exercise (>3.0 mm Hg/L per minute increase), with

171 d pressure, although its mechanism of action during exercise has not been investigated.

172 During exercise (high heart rate), there is elevated ste

173 right-to-left shunting through PFO increases during exercise impairing exercise performance.

174 n in resting skeletal muscle that is blunted during exercise in an intensity-dependent manner.

175 e inhibitor, prolongs the time to chest pain during exercise in angina.

176 We characterized QT adaptation during exercise in anorexia.

177 Rates of GLY and GNG increase during exercise in athletes following a mixed macronutri

178 r blockade increased FBF and FVC at rest and during exercise in both groups, although the increase in

179 ntilation were higher (P < 0.05) at rest and during exercise in both patients with ILD and patients w

180 bility of LV strains and torsional mechanics during exercise in HCM patients.

181 s and pulmonary artery pressures at rest and during exercise in HFpEF.

182 aneous cardiac baroreflex sensitivity (cBRS) during exercise in humans.

183 f intravascular ATP in blood flow regulation during exercise in humans.

184 tation reduced muscle metabolic perturbation during exercise in hypoxia and restored exercise toleran

185 derangements of cardiac failure that develop during exercise in individuals with HFpEF.

186 luding a general impairment of substrate use during exercise in LKB1 MKO mice.

187 was a shift to a higher and more stable CHOx during exercise in men but not in women.

188 the leading medical cause of death and death during exercise in NCAA student-athletes.

189 Thus, if impairments in vascular control during exercise in older adults involve vasoactive ATP,

190 This increase in FBF and FVC during exercise in older adults was associated with impr

191 aired muscle blood flow and vascular control during exercise in older adults.

192 L in both OL and OO, SSL area only increased during exercise in OO.

193 Shunting significantly increased during exercise in patients with COPD with PFO.

194 hat sildenafil would reduce filling pressure during exercise in patients with diastolic dysfunction a

195 PCWP was higher at rest and during exercise in patients with LA volume index >/=35 m

196 hysical fitness and cardiovascular responses during exercise in patients with POTS.

197 did not decrease filling pressure at rest or during exercise in post-myocardial infarction patients w

198 increases rates of whole-body fat oxidation during exercise in race walkers over a range of exercise

199 take (V'O2) slope were significantly greater during exercise in subjects with asthma or misdiagnosed

200 ed indices of arterial stiffness at rest and during exercise in subjects with HFpEF and hypertensive

201 ies were prospectively conducted at rest and during exercise in subjects with invasively proven HFpEF

202 ced (P < 0.05) blood flow and VC at rest and during exercise in the kidneys, adrenals and liver.

203 rtery area and blood flow failed to increase during exercise in the mildly diseased vessel, but both

204 wever, when SNA was experimentally increased during exercise in the normotensives, sympatholysis was

205 he high heart rate (HR) observed at rest and during exercise in these patients is due to this low SV.

206 ot reduce muscle glucose uptake or oxidation during exercise in vivo, excluding a general impairment

207 nd uptake of lactate and glucose are similar during exercise in young and older individuals.

208 e of EOV in HF, among measurements performed during exercise, included higher right atrial pressure a

209 al mechanisms governing cutaneous blood flow during exercise-induced heat stress and provide directio

210 de a cascade of pathophysiological responses during exercise-induced ischemia and reperfusion at rest

211 skeletal muscle to enhance lipid utilization during exercise is a form of metabolic plasticity essent

212 istribution of blood flow to skeletal muscle during exercise is altered with advancing age.

213 junctional alpha-adrenergic vasoconstriction during exercise is impaired with age, whereas the sympat

214 the hypotheses that (a) circulatory control during exercise is normal in POTS; and (b) that physical

215 portance of this size variation to diffusion during exercise is reinforced by functional links betwee

216 Elevated left atrial pressure, particularly during exercise, is a key contributor to morbidity and m

217 o an increased perception of dyspnea, which, during exercise, is mainly associated with systemic infl

218 hanisms, in particular the dynamic component during exercise, is of timely importance.

219 in the dynamic deterioration of secondary MR during exercise, its functional and prognostic impact, a

220 Limited heart rate (HR) rise (HRR) during exercise, known as chronotropic incompetence (CI)

221 is indicative of high dead space ventilation during exercise, leading to excessive and inefficient ve

222 to compensate for the further increase in Ea during exercise led to deterioration in Ees/Ea.

223 During exercise, leg substrate utilization (respiratory

224 is hypothesized to influence the BP response during exercise, limited data exist on the association o

225 Compensatory increases in minute ventilation during exercise maintained alveolar ventilation and arte

226 d is closely related to RV systolic function during exercise, maximal exercise capacity, and survival

227 lustrate how ROS released from muscle fibres during exercise may help maintain the integrity of axons

228 hat can result from its synthesis by muscles during exercise, may play a role in the mobilization of

229 Abnormal pulsatile aortic loading during exercise occurs in HFpEF independent of hypertens

230 ng men demonstrated net leg glycerol release during exercise, older men showed net glycerol uptake.

231 for maintaining systemic energy homeostasis during exercise or nutrient deprivation.

232 r change in ventricular function that occurs during exercise or pharmacological stress (typically wit

233 hm of the heartbeat in normal situations and during exercise or stress are initiated by a small numbe

234 leep or at rest were more common than deaths during exercise or with emotional stress: 82% versus 16%

235 systemic vascular resistance index decreased during exercise (P<0.0001).

236 ced increases observed in femoral blood flow during exercise (P<0.05 versus rest) in proportion to th

237 xia (P<0.05 versus normoxia), and especially during exercise (P<0.05 versus rest), with the most pron

238 ndex was reduced at rest and reduced further during exercise (P=0.008-interaction P=0.029).

239 5.5 mm Hg to 30.2 +/- 14.3 mm Hg, p < 0.001) during exercise (paradoxical response to exercise [PRE])

240 eous vasodilatation is reportedly diminished during exercise performed at a high (700 W) relative to

241 During exercise, postprandial EE was lower after EGCG th

242 l and elevates skeletal muscle O(2) delivery during exercise predominantly in fast-twitch type II mus

243 ot enhance alpha1 -mediated vasoconstriction during exercise (Protocol 1: -27 +/- 3%; P = 0.2 vs. con

244 ndependent predictors of the Borg-V'O2 slope during exercise (r(2) = 0.853, P < .001).

245 related to elevated systemic blood pressure during exercise (r=0.49, P=0.0005).

246 edge pressure at rest (r=0.63, P<0.0001) and during exercise (r=0.57, P<0.0001).

247 he consumption of whole eggs with egg whites during exercise recovery in young men.In crossover trial

248 ed venous o2 content difference, [C(a-v)o2]) during exercise significantly contributes to impaired ex

249 increasing endothelium-dependent signalling during exercise significantly enhanced the ability of co

250 breakdown of IMTG in type I fibres occurred during exercise (SIT 27 +/- 13%, ET 43 +/- 6%; P < 0.05)

251 CV Ex9 did not enhance glucose levels or HGP during exercise, suggesting that glucoregulatory effects

252 During exercise, sympathetic nervous system activity inc

253 on peak metabolic equivalents (MET) achieved during exercise test and eight categories based on fitne

254 We aimed to study whether low HR at rest or during exercise testing was a predictor of AF in initial

255 d >85% of their maximum-predicted heart rate during exercise testing.

256 lumes and arterial pressure both at rest and during exercise than HCM patients in whom the gradient i

257 ests) showed a slower increase in heart rate during exercise than in the placebo-treated group.

258 anxiety were more likely to exhibit ischemia during exercise than women without anxiety (odds ratio,

259 eft ventricular (LV) end-diastolic pressures during exercise that contribute to dyspnea.

260 gists can discuss hair management strategies during exercise that facilitate routinely performing exe

261 During exercise there is a balance between vasoactive fa

262 During exercise, there is a decline in CFIp that indicat

263 At rest and during exercise, there were no significant differences i

264 athways are not obligatory for sympatholysis during exercise; therefore, we tested the hypothesis tha

265 by reducing ventilatory response and dyspnea during exercise; these effects were possibly mediated th

266 During exercise, this results in ventricular-pulmonary v

267 During exercise, this sensor reveals clearly the onset a

268 pression and the rate-pressure product (RPP) during exercise to determine whether ranolazine's mechan

269 isproportionate increase in filling pressure during exercise to maintain stroke volume.

270 During exercise, total hindlimb blood flow (control: 108

271 ic response of individual proteins in humans during exercise training.

272 Exercise-induced abnormalities during exercise treadmill testing (ETT) were initially c

273 normalized time to peak filling) at rest and during exercise using radionuclide ventriculography, pea

274 nce as interactions at distinct body changes during exercise, using complex network models.

275 During exercise ventilation increased by 6.64 +/- 0.84 i

276 in the endogenous rate of glucose appearance during exercise was blunted in the KO mice because of a

277 primary end point of ventricular arrhythmias during exercise was compared between the flecainide and

278 PCWP during exercise was compared between treatment groups us

279 imilarly, alpha(2)-mediated vasoconstriction during exercise was significantly blunted in both young

280 The median ventricular arrhythmia score during exercise was significantly reduced by flecainide

281 etween dyspnea intensity and EMGdi/EMGdi,max during exercise was similar in all three groups.

282 tty acids) and endogenous glucose production during exercise were also reduced, and glucose infusion

283 Measures of metabolites during exercise were less reliable: exercising PCr had a

284 Changes in the QT interval during exercise were measured, and QT/RR-interval slopes

285 our oxygen, and those with desaturation only during exercise were prescribed oxygen during exercise a

286 Respiratory symptoms during exercise were reported by 57 (88%) players.

287 ic BP at rest, Bruce stage 2, and maximal BP during exercise were significantly associated with CVD d

288 Rates of EGP and GLY both at rest and during exercise were significantly lower in the LCHF gro

289 Work rate and mean arterial pressure during exercise were similar in controls and SIPE-suscep

290 t times of greater need for NO signaling, as during exercise when left ventricular filling pressures

291 related to the magnitude of fatigue accrued during exercise, which may explain the reported consiste

292 ater to elicit the cardiac response observed during exercise while heart rate (HR) and electrocardiog

293 in POTS at any given oxygen uptake (V(O(2))) during exercise while the cardiac output (Q(c))-V(O(2))

294 patients, sildenafil improved cardiac index during exercise with a decrease in total pulmonary resis

295 HFpEF was associated during exercise with a major increase in Ep (+155 +/- 19

296 se (control, n = 10) were studied before and during exercise with characterization of cardiovascular

297 s in the maximal rate of fat oxidation (MFO) during exercise with potential implications for metaboli

298 nd Vt in response to dead space (DS) loading during exercise would indicate true ventilatory limitati

299 ic failure, we hypothesized that OR blockade during exercise would prevent exercise-associated autono

300 rom pathological changes in cardiac function during exercise, yet imaging modalities have seldom been