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1 mber (reduction) and whole-plant morphology (dwarfing).
2 otypes, including spontaneous cell death and dwarfing.
3 y cranial capacity cannot result from normal dwarfing.
4 ID1c silencing corresponded to the degree of dwarfing.
5 ade avoidance, causing proximity-conditional dwarfing.
6 Conversely, LOH2 overexpression resulted in dwarfing.
7 ized to understand the mechanisms underlying dwarfing.
8 sponse to gibberellin is conferred by mutant dwarfing alleles at one of two Reduced height-1 (Rht-B1
11 ion lines of M. truncatula results in severe dwarfing, altered development, reduction in lignin conte
13 D EPIDERMAL FLUORESCENCE 4 (REF4) that cause dwarfing and decreased accumulation of phenylpropanoids.
15 ession of ABI3 or ABI5 does not suppress the dwarfing and Glc dependence caused by abi8 but partially
16 tive transcriptional process responsible for dwarfing and inhibition of lignin biosynthesis, and sugg
17 4 mutant alleles suppress the ssi2-conferred dwarfing and lesion development, the NPR1-independent ex
19 For example, the M.7 EMLA rootstock is semi-dwarfing and reduces the susceptibility of the scion to
20 protease, the overexpression of which causes dwarfing and resistance to virulent Pseudomonas syringae
21 effects, including but not limited to severe dwarfing appearance, chlorosis, nearly complete reductio
22 ains than would be predicted from a model of dwarfing based on the intraspecific scaling of the mainl
24 cause pseudoachondroplasia (PSACH), a severe dwarfing condition that has a growth plate chondrocyte p
25 SEMD) and mapped a gene associated with this dwarfing condition to chromosome 10q23-24, a region synt
26 oachondroplasia (PSACH), a severe short-limb dwarfing condition, results from mutations that cause mi
29 sembly lines can reach almost 5 megadaltons, dwarfing even the ribosome (approximately 2.6 megadalton
30 against tissue rupture was unaffected by the dwarfing gene but was consistently lower (-26%, p<0.01)
35 rachydactyly type B (BDB1) and the mesomelic dwarfing in mice homozygous for a lacZ and/or a neo inse
36 rt that activation of RPP5 locus R genes and dwarfing in the bal variant are caused by a 55-kb duplic
37 st rapid and well documented cases of island dwarfing known thus far took place over thousands of yea
38 pic effects that include facial dysmorphism, dwarfing, male sterility, anemia, and cystic choroid ple
39 tropic effects including facial dysmorphism, dwarfing, male sterility, anemia, and progressive polycy
45 ression of ACT1 in vegetative tissues causes dwarfing of plants and altered morphology of most organs
46 n, ACT1, in vegetative tissues causes severe dwarfing of plants with aberrant organization of most pl
51 s, reduced stolon formation, together with a dwarfing phenotype and a lack of flowering in the most s
53 aling pathways of gibberellin (GA) can cause dwarfing phenotypes in plants, and the use of such mutat
54 owth, we performed a comparative analysis of dwarfing phenotypes manifested in mouse mutants lacking
57 xotic lines that have been introgressed with dwarfing quantitative trait loci (QTL) from a common par
58 life span and flowering duration, sterility, dwarfing, reduced seed yield and shorter root length.
59 omes involving craniosynostosis, whereas the dwarfing syndromes are largely associated with FGFR3 mut
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