ライフサイエンスコーパス: dysarthria

1 h impaired gait coordination, dysmetria, and dysarthria.

2 ing that is distinguishable from aphasia and dysarthria.

3 gait ataxia, upper limb incoordination, and dysarthria.

4 toxicity evidenced by ataxia, confusion, and dysarthria.

5 , mild ataxia, mild cognitive deficiency and dysarthria.

6 pathy, 53%; cerebellar ataxia, dysmetria, or dysarthria, 38%; and encephalopathy, 27%.

7 ted signs and symptoms were dysphagia (53%), dysarthria (39%), and generalized weakness (37%).

8 axia, sensory loss, reduced tendon reflexes, dysarthria, absent lower limb reflexes, and loss of posi

9 rred from an outside facility with worsening dysarthria and confusion after having presented 4 weeks

10 ed 2 Omani families with HSP, short stature, dysarthria and developmental delay-core features of Troy

11 Dysarthria and disequilibrium were common in patients wi

12 of somatosensory sensations, motor symptoms, dysarthria and multimodal responses were significantly a

13 The sensory ataxic neuropathy with dysarthria and ophthalmoparesis (SANDO) syndrome is a su

14 Alpers syndrome, sensory ataxia, neuropathy, dysarthria and ophthalmoparesis (SANDO), Parkinsonism, a

15 ome to adult-onset sensory ataxic neuropathy dysarthria and ophthalmoparesis.

16 and communicative, but with choreoathetosis, dysarthria, and an unsteady gait.

17 s a subacute encephalopathy, with confusion, dysarthria, and dysphagia, and that progresses to severe

18 al speech therapy can lead to improvement of dysarthria, and intensive programmes have had substantia

19 cally characterized by gait and limb ataxia, dysarthria, and nystagmus.

20 t over hours to days of dystonia, dysphagia, dysarthria, and parkinsonism.

21 had an atypical clinical triad of epilepsy, dysarthria, and paroxysmal kinesigenic dystonia, and a h

22 parate occasions developed transient ataxia, dysarthria, and weakness within 3 days of returning from

23 right eyelid with narrow palpebral fissure, dysarthria, anisocoria (narrower pupil on the right side

24 (sensory/motor) or negative (speech arrest, dysarthria, anomia, phonological and semantic paraphasia

25 Blurred vision, dysphagia, and dysarthria are common initial complaints.

26 She had dysarthria but was able to speak in full sentences.

27 ter small focal lesions (ataxic hemiparesis, dysarthria-clumsy hand syndrome, dysarthria-dysmetria an

28 ed brainstem symptoms (eg, isolated vertigo, dysarthria, diplopia) are not consistently classified as

29 remor severity correlated with the degree of dysarthria, dysmetria and dysdiadochokinesia but not wit

30 Physical examination revealed dysarthria, dysmetria, and ataxia.

31 ads to moderate disability with gait ataxia, dysarthria, dysmetria, mild oculomotor abnormalities, an

32 emiparesis, dysarthria-clumsy hand syndrome, dysarthria-dysmetria and dysarthria-facial paresis).

33 had a rapid progression with disequilibrium, dysarthria, dysphagia, and central hypoventilation, and

34 was preceded by gait instability followed by dysarthria, dysphagia, ataxia, or chorea.

35 Patients had dystonia, chorea, parkinsonism, dysarthria, dysphagia, seizures, cognitive abnormalities

36 s associated with AS (ataxia, action tremor, dysarthria, dysphagia, sialorrhea and excessive chewing/

37 lessly progressive choreoathetoid movements, dysarthria, dysphagia, spastic paralysis, and behavioral

38 Both patients exhibited dysarthria, dysphagia, tongue atrophy, neck extensor wea

39 h prominent bulbar palsies such as diplopia, dysarthria, dysphonia, and dysphagia that would typicall

40 characterized by gait abnormalities, ataxia, dysarthria, dystonia, vertical gaze palsy, and cognitive

41 umsy hand syndrome, dysarthria-dysmetria and dysarthria-facial paresis).

42 the subthalamic nucleus was associated with dysarthria, fatigue, paraesthesias, and oedema, whereas

43 on after having presented 4 weeks prior with dysarthria, gait ataxia, and bilateral upper extremity w

44 ond presented at 3.5 years with gait ataxia, dysarthria, gross motor regression, hypotonia, ptosis an

45 ctive reports of a worsening in pre-existing dysarthria, hypophonia and hypersalivation/drooling foll

46 Bulbar signs and symptoms, including dysarthria in 10 and dysphagia in 8 patients, also were

47 Ocular signs were observed in 84% and dysarthria in 66%.

48 Dysarthria in Parkinson's disease can be characterised b

49 Dysarthria (in four patients assigned to neurostimulatio

50 Treatment studies for poststroke dysarthria indicate that speech supplementation strategi

51 ehabilitation and strategies for aphasia and dysarthria is scarce.

52 ffects on parkinsonian speech, management of dysarthria is still challenging for the clinician and sh

53 Common accompanying features included dysarthria, labile emotions, and unsteady gait.

54 gnitive dysexecutive slowing, forgetfulness, dysarthria, mood changes, urinary symptoms, and short-st

55 At presentation, dysarthria (Motor Speech Examination median [IQR] score:

56 ): hemiparesis, vertigo/dizziness, diplopia, dysarthria, nystagmus, nausea/vomiting, head pain, hemia

57 nvolvement by weakness, wasting, spasticity, dysarthria or dysphagia of one central nervous system re

58 in movement "vigor," leading to "hypokinetic dysarthria" or "hypophonia." This is an additional examp

59 tients, the initial symptoms included falls, dysarthria, or clumsiness followed by a complete cerebel

60 nt of our hospital with a 2-month history of dysarthria, progressively worsening vertigo, and difficu

61 enting with nfvPPA, presence of early severe dysarthria, relatively selective white matter atrophy at

62 phic lateral sclerosis--including dysphagia, dysarthria, respiratory distress, pain, and psychologica

63 ment was correlated with overall symptom and dysarthria severity.

64 disorder, characterized by spells of ataxia, dysarthria, vertigo, and migraines, associated with muta

65 Dysphagia and dysarthria were the most commonly reported cranial nerve

66 re variably effective for the alleviation of dysarthria, whereas bilateral procedures typically lead

67 ogic illness defined initially by ataxia and dysarthria, which fluctuated in relation to each subsequ

68 ospital with severe non-systemic vertigo and dysarthria, which had lasted for a couple of weeks.

69 g to acquired aphasia, apraxia of speech and dysarthria with special attention to clinically signific