1             The gonads most at risk are both dysgenetic and intra-abdominal, and early gonadectomies

2      DBP exposure in pregnancy induces focal dysgenetic areas in fetal testes that appear between e19

3 treatment produces more extensive and severe dysgenetic areas, with more ectopic SC and germ cells (G

4 ilure (p <.05) included age < or =24 months; dysgenetic condition; syndromic condition; chronic respi

5               These regions included focally dysgenetic cortex, the cortex lining schizencephalic cle

6                             Lastly, OGCTs in dysgenetic gonads arise in a diploid rather than a tetra

7 umors, and mixed GCTs)], and GCTs arising in dysgenetic gonads of 46,XY individuals.

8 ires knowledge of the risk factors including dysgenetic gonads, a Y chromosome with intra-abdominal g

9 , a transcription factor gene mutated in the dysgenetic lens mouse.

10 r gene Foxe3 was shown to be involved in the dysgenetic lens phenotype in mice, which is characterize

11 Y chromosome with intra-abdominal gonads and dysgenetic syndromes with WT1 gene splice variants.