ライフサイエンスコーパス: dyslipidemic

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1 ats, were obese (621 vs. 461 g; P < 0.0001), dyslipidemic (133 vs. 67 mg/dl; P < 0.001), and glucose

2 ipid levels were determined at enrollment in dyslipidemic adult patients on stable lipid-lowering the

3 Dyslipidemic and inflammatory environments attenuate end

4 s, i.e. inhibition of caspase-1 in ECs under dyslipidemic and inflammatory environments attenuates EC

5 Under these dyslipidemic and inflammatory environments, EC-caspase-1

6 development of hypertensive complications in dyslipidemic and/or insulin-resistant patients.

7 Peritoneal macrophages from dyslipidemic animals were primed for more robust TLR res

8 ke of L5, an electronegative LDL abundant in dyslipidemic but not normolipidemic human plasma.

9 Mean HDL cholesterol levels are lower in dyslipidemic children from households with smokers than

10 ians screening for dyslipidemia or following dyslipidemic children should be aware of the powerful ef

11 The clinical use of niacin to treat dyslipidemic conditions is limited by noxious side effec

12 between positive selection and the obese and dyslipidemic conditions.

13 put and hypertension) or metabolic (that is, dyslipidemic) derangements associated with obesity may p

14 n APOA5, elevated plasma triacylglycerol and dyslipidemic disease.

15 promotes unregulated platelet activation in dyslipidemic disorders.

16 tic CB1 receptor resulted in a rescue of the dyslipidemic effects of glucocorticoid exposure, while n

17 th region-wide significance for low HDL-C in dyslipidemic families of Mexican and European descent an

18 ance, we tested this gene for association in dyslipidemic families originating from two distinct popu

19 trait in 2 multigenerational French Canadian dyslipidemic families.

20 entral fat mass (Delta+40 g), accompanied by dyslipidemic (>30% elevated, P < 0.05) serum triglycerid

21 sequences of these genes were analyzed in 80 dyslipidemic individuals.

22 The term 'HIV/HAART associated dyslipidemic lipodystrophy (HADL)' describes this syndro

23 e effects of diabetes are masked in severely dyslipidemic mice, suggesting that the effects of glucos

24 shown to inhibit atherosclerosis in several dyslipidemic mouse models.

25 ain levels and may be a treatment option for dyslipidemic patients who cannot tolerate statin therapy

26 istration with other lipid-altering drugs in dyslipidemic patients, including a large (n=2121) 96-wee

27 s surrogate endpoints for clinical trials in dyslipidemic patients.

28 ffspring with prenatal famine exposure had a dyslipidemic pattern characterized by elevated total cho

29 Features of the dyslipidemic pattern reported with the use of antiretrov

30 of n-3 HUFAs in the diet of hypertensive and dyslipidemic persons may have substantial benefits in re

31 studies have suggested that there is a novel dyslipidemic profile consisting of isolated low high-den

32 patibility complex-mismatched, diabetic, and dyslipidemic rats, immunologic and metabolic mechanisms

33 d hepatic lipids when orally administered to dyslipidemic rodent models.

34 ecretory degradation may be relevant in some dyslipidemic states.

35 added to LDL-C-lowering therapy, but a small dyslipidemic subgroup may benefit.

36 mized, observer-blinded study, we treated 60 dyslipidemic subjects without cardiovascular disease wit

37 al fluency, particularly in hypertensive and dyslipidemic subjects.

38 can Caribbeans were less centrally obese and dyslipidemic than Europeans.

39 Among available dyslipidemic therapies, although statins remain the main

40 Combination dyslipidemic therapy affords the most efficacious approa

41 The importance of combination dyslipidemic therapy, such as a statin plus niacin, in t

42 was reduced to optimal levels with intensive dyslipidemic therapy.

43 a large proportion of psoriatic patients are dyslipidemic, this finding is of clinical significance a

44 ely insulin resistant, hyperinsulinemic, and dyslipidemic, with evidence of endothelial dysfunction,

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