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1 y induce TLR4-mediated neurodegeneration and dysmotility.
2 g disease characterized by severe intestinal dysmotility.
3 ) analogs may develop enteric neuropathy and dysmotility.
4 infertility, resulting from cilia and sperm dysmotility.
5 om defecation disorders and advanced colonic dysmotility.
6 to the brush border due to small intestinal dysmotility.
7 ecrease the inflammation that contributes to dysmotility.
8 phages within the muscularis associated with dysmotility.
9 scle groups associated with gastrointestinal dysmotility.
10 ticipates in mediating early TLR4-transduced dysmotility.
11 ession/signaling in causing gastrointestinal dysmotility.
12 ct to driving enteric neuropathy and colonic dysmotility.
13 t exhibit WD-induced myenteric cell loss and dysmotility.
14 ferent forms of experimental antigen-induced dysmotility.
15 d induced mast cell degranulation in mid-gut dysmotility.
16 mice did not exhibit myenteric cell loss or dysmotility.
17 ce of urinary retention and gastrointestinal dysmotility.
18 or, could lead to new therapies for pain and dysmotility.
19 and often confirms the clinical suspicion of dysmotility.
20 ith esophageal atresia and severe esophageal dysmotility.
21 ese cells may contribute to gastrointestinal dysmotilities.
22 h potential impact on global health included dysmotility (59%), hypertension (37%), osteoporosis (22%
23 ecture and neurochemical profile, intestinal dysmotility, abnormal mucosal secretion, reduced levels
27 m of clinical features (including severe gut dysmotility and a movement disorder) and electrographic
28 al-Mylk-knockout mouse model with severe gut dysmotility and abnormal function of the bladder support
30 mutations in CCDC114 are a cause of ciliary dysmotility and PCD and further demonstrate the utility
33 stinal surgery protects against postsurgical dysmotility and reduces the severity of postoperative il
34 nutrient malabsorption, bowel dilatation and dysmotility, and changes in bacterial flora influence th
35 nes interleukin-4 and -13 in antigen-induced dysmotility, and interleukin-5 in the pathogenesis of mu
37 sed woman developed chronic gastrointestinal dysmotility as a consequence of acute cytomegalovirus in
41 stinal transplantation contributing to graft dysmotility, bacterial translocation, and possibly, acut
42 on is believed to underlie ageing-associated dysmotilities but the mechanisms have not been fully elu
44 e external ophthalmoplegia; gastrointestinal dysmotility; cachexia; peripheral neuropathy; and leucoe
45 efined clinically by severe gastrointestinal dysmotility; cachexia; ptosis, ophthalmoparesis, or both
46 inherited disorder of ciliary and flagellar dysmotility characterized by chronic upper and lower res
49 r isolated or syndromic ocular and/or facial dysmotility disorders, but who did not have the combined
50 l smooth muscle that are responsible for the dysmotility following small bowel transplantation (SBTX)
51 acological approach preventing postoperative dysmotility for clinical intestinal transplantation.
55 ce implicating mucosal allergic responses in dysmotility has been extended to include disorders consi
56 ute CMV infection can cause gastrointestinal dysmotility in nonimmunocompromised individuals and that
61 mechanisms that appear to be involved in the dysmotility, including defects in neurons, smooth muscle
66 e external ophthalmoplegia, gastrointestinal dysmotility, leukoencephalopathy, thin body habitus, and
67 +/- 15 years old, 75% were female, 70% with dysmotility-like FD, and 30% with ulcer-like FD) were ra
69 indings need confirmation, they suggest that dysmotility may result subsequent to these infections.
70 requiring dilatation, persistent esophageal dysmotility (mid esophageal hematoma), and vocal cord pa
71 as the presence of a DeMeester score >14 or dysmotility more severe than "mild nonspecific disorder"
72 y might be involved in the prodromal gastric dysmotility observed in patients with early-stage Parkin
73 re, but is underrecognized as a cause for GI dysmotilities of varying anatomic extent, severity, and
77 nt lymphoproliferative disorder (n=1), graft dysmotility or dysfunction (n=3), ACR with severe infect
78 stinal mucosal inflammation (associated with dysmotility or short bowel) were significantly shorter t
79 cardia syndrome, idiopathic gastrointestinal dysmotility, or diabetic autonomic neuropathy (9 percent
80 five family members with distinctive ocular dysmotility patterns that co-segregated with a novel hyp
81 abnormalities, poor growth, gastrointestinal dysmotility, renal tubular acidosis, seizures, and episo
83 ffuse SSc, calcinosis, Raynaud's, esophageal dysmotility, sclerodactyly, and telangiectasias syndrome
84 inosis cutis, Raynaud's syndrome, esophageal dysmotility, sclerodactyly, and telangiectasias) syndrom
85 (calcinosis, Raynaud phenomenon, esophageal dysmotility, sclerodactyly, telangiectasia) syndrome.
86 erance, secretomotor, upper gastrointestinal dysmotility, sleep dysfunction, urinary, and autonomic d
87 sinophil infiltration in proximal and distal dysmotility syndromes (oesophageal, gastric and colorect
88 were intestinal gastroschisis and intestinal dysmotility syndromes in children, and mesenteric thromb
89 (CIPO) is characterized by severe intestinal dysmotility that mimics a mechanical subocclusion with n
90 ient mice develop transient gastrointestinal dysmotility, urinary retention, dilated pupils, reduced
93 a (PCD), a disorder characterized by ciliary dysmotility; yet, radial spoke functions remain unclear.
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