ライフサイエンスコーパス: dysrhythmia

1 or reducing pulmonary artery distortion and dysrhythmia.

2 altered thalamic firing and thalamocortical dysrhythmia.

3 ith gating error caused by pacemaker-induced dysrhythmia.

4 uction of triggered activity and ventricular dysrhythmia.

5 a genetic susceptibility to developing this dysrhythmia.

6 One patient developed unstable cardiac dysrhythmia.

7 hyxia with that of cardiac arrest induced by dysrhythmia.

8 k of acute myocardial infarction or unstable dysrhythmia.

9 ed thalamic burst firing and thalamocortical dysrhythmia.

10 ith potassium-sensitive weakness and cardiac dysrhythmia.

11 show desensitization to nicotine-stimulated dysrhythmias.

12 increased propensity for lethal ventricular dysrhythmias.

13 syncopal episodes attributed to ventricular dysrhythmias.

14 opressin-induced emetic response and gastric dysrhythmias.

15 m circulatory diseases, specifically cardiac dysrhythmias.

16 vel mechanism for the development of cardiac dysrhythmias.

17 ufficiency, respiratory failure, and cardiac dysrhythmias.

18 andidate genes in studies of human circadian dysrhythmias.

19 lt in disruption of ICC and gastrointestinal dysrhythmias.

20 the SR can initiate various types of atrial dysrhythmias.

21 to the heart for termination of ventricular dysrhythmias.

22 .9, P<0.001), stroke (4.7 vs. 3.1, P<0.001), dysrhythmia (20.9 vs. 11.0, P<0.001), and any cardiovasc

23 Of the 724 hospitalizations for cardiac dysrhythmias, 203 were in the potentially exposed group,

24 (14.4%-20.1%), anemia (12.4%-20.4%), cardiac dysrhythmias (21.7%-29.0%), congestive heart failure (40

25 and who had no cardiovascular complications (dysrhythmias, acute myocardial infarction, or recurrent

26 rval [CI]: 0.63 to 0.86; p < 0.001), cardiac dysrhythmia (adjusted OR: 0.72; 95% CI: 0.55 to 0.94; p

27 During the observation period, there were no dysrhythmia adverse events.

28 tempt at intubation, hypotension, or cardiac dysrhythmia) after the study treatment was administered

29 ngestive heart failure; and poor (<0.36) for dysrhythmia, ambulation status, pericarditis, chronic ob

30 fibrillation (AF) is the most common cardiac dysrhythmia and a source of considerable morbidity and m

31 yopathy was associated with a propensity for dysrhythmia and characterized by overt intracellular cal

32 ncluded evidence of stronger associations of dysrhythmia and congestive heart failure visits with com

33 nce that rats exhibit stress-induced cardiac dysrhythmia and ischemia sensitivity comparable to human

34 ve pulmonary disease (COPD) was observed for dysrhythmia and peripheral and cerebrovascular disease v

35 Pediatric brady-dysrhythmias and conduction disorders are uncommon, but

36 le of preventing vasopressin-induced gastric dysrhythmias and gastric slow wave uncoupling.

37 titial cells of Cajal (ICC), which may cause dysrhythmias and impaired neural control.

38 nterval and increase the risk of ventricular dysrhythmias and sudden death.

39 n, left ventricular diastolic heart disease, dysrhythmia, and sudden death.

40 activity, a self-sustaining thalamocortical dysrhythmia, and the constant perception of pain.

41 may increase the risk of infection, cardiac dysrhythmias, and bleeding, all complications independen

42 typies, anxiety, tremor, ataxia, respiratory dysrhythmias, and seizures.

43 infarction, left-ventricular dysfunction, or dysrhythmia; and chronically-by accelerating the atheros

44 d procedures (eg, transient ischemic attack, dysrhythmia, aortic valve replacement, and femoral popli

45 Disrupted ICC networks and gastric dysrhythmias are each associated with gastroparesis.

46 Postoperative dysrhythmias are most likely to occur in patients with s

47 m slowed and ABP increased, (2) a tachypnoea/dysrhythmia area, at the level of the preBotzinger compl

48 taphylococcus as the causative organism, and dysrhythmias as a comorbid condition.

49 s have raised the possibility of ventricular dysrhythmias as the cause of these deaths.

50 or mortality, 1.5; 95% CI, 1.1-2.1), cardiac dysrhythmias (beta coefficient, 1.8; 95% CI, 1.1-2.6; OR

51 isorder termed Chronic Atrial and Intestinal Dysrhythmia (CAID) syndrome.

52 linical evidence of severe myocarditis, with dysrhythmias, cardiomegaly, and cardiogenic shock.

53 pulmonary fibrosis, pulmonary hypertension, dysrhythmias, cardiomyopathy, hypercalcemia, and renal f

54 uency, meaning that the varying amplitude of dysrhythmia could predispose patients to recurrent attac

55 C accumulation may contribute to ventricular dysrhythmias during ischemia.

56 Atrial fibrillation is the most common dysrhythmia encountered in clinical practice.

57 lity attributable to ischemic heart disease, dysrhythmias, heart failure, and cardiac arrest.

58 psychosis, lethargy, seizures, tachycardia, dysrhythmias, hypertension, and hyperthermia.

59 This current may contribute to dysrhythmias, hypertrophy, and altered contractile funct

60 orrection algorithm on the pacemaker-induced dysrhythmia image set to see whether it repaired this se

61 hm accurately repaired the pacemaker-induced dysrhythmia image set; when it was applied to the 64-pat

62 e occurred in 6 subjects (0.65%), because of dysrhythmia in 2, device embolization in 1, and cardiac

63 fibrillation (AF) is the most common cardiac dysrhythmia in the United States.

64 cortex and hippocampus in vitro and cortical dysrhythmia in vivo.

65 duction abnormalities and sudden ventricular dysrhythmias in pediatric patients taking psychotropic d

66 Dysrhythmias included abnormalities of initiation (stabl

67 T-channels may contribute to thalamocortical dysrhythmia, including absence epilepsy.

68 cardiovascular conditions in 10%, including dysrhythmia, left pulmonary artery thrombosis, and chron

69 iorespiratory arrest from underlying cardiac dysrhythmia may be a cause.

70 Severe myocardial dysfunction and dysrhythmias may accompany respiratory syncytial virus i

71 with sepsis are prone to developing cardiac dysrhythmias, most commonly atrial fibrillation.

72 ntolerance, hypertrophic cardiomyopathy with dysrhythmia, myotonic myopathy and hypotension, all dist

73 f superficial wound complications and atrial dysrhythmias, obesity is not a significant multivariate

74 in these translational models is that human dysrhythmia occurs while SCN circuitry is genetically an

75 al pathways leading to epilepsy, an episodic dysrhythmia of the cerebral cortex marked by abnormal ne

76 upport a model that attributes tinnitus to a dysrhythmia of the thalamocortical loop, leading to hypo

77 Dysrhythmias of breathing occur in several clinical diso

78 ein-losing enteropathy, thromboembolism, and dysrhythmias often lead to significant morbidity and mor

79 s but evokes prostaglandin-dependent gastric dysrhythmias only in nonsmokers.

80 benefits when selected for the treatment of dysrhythmias or chronic pain in patients with coexisting

81 Electrogastrography may identify dysrhythmias or failure of signal power to increase post

82 disease, stroke, peripheral artery disease, dysrhythmias, or heart failure), ascertained by Internat

83 ions (P = .005; odds ratio, 1.8), and atrial dysrhythmias (P = .04; odds ratio, 1.2).

84 Atrial dysrhythmia patients have exaggerated intra-atrial condu

85 ital SND characterized by bradycardia, sinus dysrhythmia, prolonged sinoatrial node recovery time, in

86 was an intrinsic property of ICC and whether dysrhythmias result from disruptions of ICC networks.

87 that clinically important cardiomyopathy and dysrhythmia secondary variants can be identified in unse

88 7 cycles/min), and tachygastric frequencies; dysrhythmias showed velocity anisotropy (mean, 3.3 mm/s

89 c disorders characterized by thalamocortical dysrhythmia, such as CAE.

90 dentify CAID syndrome as a novel generalized dysrhythmia, suggesting a new role for SGOL1 and the coh

91 aracterized by Chronic Atrial and Intestinal Dysrhythmia, termed CAID syndrome, in 16 French Canadian

92 Vasopressin induced gastric dysrhythmias, uncoupling of slow waves, and vomiting and

93 er, we found an increased risk of other CVD (dysrhythmia, valvular dysfunction, and pericarditis) (ad

94 Rates of dysrhythmia were higher.

95 lence and associated factors for ventricular dysrhythmias were defined.

96 he recovery period was shorter, symptoms and dysrhythmias were fewer, hypertension and hypotension we

97 Slow-wave dysrhythmias were identified in all 9 subjects with CUNV

98 Gastric dysrhythmias were more frequent on postoperative day 3 (

99 days, damage to ICC networks and electrical dysrhythmias were observed.

100 of relatives, whereas other supraventricular dysrhythmias were present in 16%.

101 field, respectively; P < .05), but slow-wave dysrhythmias were similar between groups.

102 and chronic pain may reflect thalamocortical dysrhythmia, which results from abnormal theta-range res

103 a indicate the presence of a thalamocortical dysrhythmia, which we propose is responsible for all the

104 fibrillation after cardiac surgery remains a dysrhythmia with significant implications.

105 ce in the treatment of patients with cardiac dysrhythmias with the introduction of radiofrequency abl