ライフサイエンスコーパス: ear swelling

1 oduction, and neutrophil influx and prevents ear swelling.

2 R4KO mice showed a 1.3-fold increase in mean ear swelling, a 2-fold increase in epidermal thickness,

3 Ear swelling after croton oil application was similar in

4 ith MCs protected Sash mice from exacerbated ear swelling after repeated oxazolone challenge.

5 king gammadelta T cells) exhibited decreased ear swelling and downregulated expression of IL-22 and I

6 CCR2(-/-) mice had increased ear swelling and epidermal thickening, which was correla

7 mice effectively attenuated P. acnes-induced ear swelling and granulomatous inflammation.

8 use ears, thereby relieving P. acnes-induced ear swelling and granulomatous inflammation.

9 Ear swelling and inflammation persisted for more than 2

10 mice and characterized by significantly less ear swelling and inflammatory cell infiltration than in

11 e antigen challenge significantly suppressed ear swelling and markedly reduced cellular influx.

12 ation effectively decreased contact allergic ear swelling and myeloid immune cell infiltration not on

13 n Bid KO mice and observed markedly enhanced ear swelling and proliferation responses compared with w

14 itivity response, characterized by decreased ear swelling and reduced inflammatory cell infiltrates.

15 antibody to IL-1alpha significantly reduced ear swelling and suppressed the levels of MIP-2, MIP-1al

16 ion of PF6-miR-146a nanocomplexes attenuated ear-swelling and reduced the expression of pro-inflammat

17 by a standard chromium release assay and an ear swelling assay, respectively.

18 lloantigens were evaluated by a conventional ear swelling assay.

19 nal expression of beta gal and inhibited the ear-swelling assay for delayed type hypersensitivity.

20 (DTH) responsiveness was assessed using the ear-swelling assay.

21 r hapten challenge but resulted in increased ear swelling at 48 hours and delayed resolution of the i

22 athelicidin demonstrated a large increase in ear swelling, cell infiltration, and MIP-2 expression co

23 Ear swelling chronic contact hypersensitivity responses

24 However, late-phase ear swelling, due to type III hypersensitivity, was sign

25 d correlated with the immune response, i.e., ear swelling, elicited.

26 ment conditions and mouse genotypes measured ear swelling, epidermal thickness, and cytokine expressi

27 ficient (MyD88(-/-)) C57BL/6 mice had intact ear swelling, exaggerated inflammation, and higher level

28 Tolerance was measured from inhibition of ear swelling in a delayed-type hypersensitivity reaction

29 Furthermore, CP-456,773 reduces ear swelling in an imiquimod cream-induced mouse model o

30 After hapten sensitization and re-challenge, ear swelling in CCR6-/- animals was reduced 80% as compa

31 was orally efficacious in an MMP-12 induced ear-swelling inflammation model in the mouse with a good

32 uces CHS responses, as measured by decreased ear swelling, inhibition of local DETC activation, and a

33 to DNFB in wild-type mice, and DNFB-induced ear swelling is reduced by approximately 50% in TCRdelta

34 yl ester considerably attenuated TPA-induced ear swelling, leukocyte infiltration, epidermal cell pro

35 skin inflammation in both the IL-23-induced ear swelling model and the topical imiquimod model, and

36 utein demonstrated significant inhibition of ear swelling owing to UVB radiation.

37 In wild-type (WT) mice, rmIL-23 induced ear swelling (p < 0.001, all p values versus saline), ep

38 ctions into IL-17A(-/-) mice produced little ear swelling (p < 0.001, versus IL-23-injected WT mice)

39 L-22(-/-) mice resulted in relatively little ear swelling (p < 0.09) and epidermal hyperplasia (p < 0

40 e mice, ODCER transgenic mice showed reduced ear swelling, reduced neutrophil infiltration, and decre

41 esponded to DNFB challenge with a pronounced ear swelling response without previous sensitization to

42 urs before antigen challenge but provoked an ear-swelling response directly on application.

43 dingly, Myo9b(-/-) mice showed an attenuated ear-swelling response in a model of contact hypersensiti

44 igh concentrations of anti-IgE Ab induced an ear-swelling response in these strains, implying some ca

45 Ear swelling responses in major histocompatibility compl

46 let-B irradiation showed markedly suppressed ear swelling responses to dinitrofluorobenzene challenge

47 E and Ag-specific IgG1, and generated strong ear-swelling responses to intradermal administration of

48 hase of contact hypersensitivity exacerbated ear-swelling responses.

49 t of the inflammatory response, and examined ear swelling, SK activity, vascular permeability, leukoc

50 BPZE1 nasal pretreatment markedly inhibited ear swelling, skin inflammation, and production of pro-i

51 utaneous inflammation was evaluated by mouse ear swelling test (MEST), histology, serum IgE levels, a

52 and tested for immune responsiveness by the ear-swelling test for delayed-type hypersensitivity (DTH

53 Ear swelling was assessed to evaluate the anaphylactic r

54 Also, Il23-induced ear swelling was diminished in Trex2 knockout mice in co

55 In accordance, DNFB-induced ear swelling was reduced by approximately 50% in IL-17(-