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1 cells, which undergo a binary choice during early differentiation.
2 ly regulated activation of NF-kappaB but not early differentiation.
3 for a specific task related to stemness and early differentiation.
4 or cell cycle progression, pluripotency, and early differentiation.
5 sion of developmental genes and induction of early differentiation.
6 numbers of inactivated X chromosomes during early differentiation.
7 nvolved in the LESC maintenance and/or their early differentiation.
8 ature of Earth's heterogeneous accretion and early differentiation.
9 vitro, along with upregulation of markers of early differentiation.
10 ell fusion, none of the treatments prevented early differentiation.
11 kely because of an ASC-sensitive block(s) in early differentiation.
12 competence is acquired only gradually during early differentiation.
13 t are repressed in ESCs and derepressed upon early differentiation.
14 xpressed gene in the vascular lineage during early differentiation.
15 of involucrin, consistent with promotion of early differentiation.
16 etylation also alters gene expression during early differentiation.
17 yonic mammalian hair cells but not for their early differentiation.
19 th-defying phenotype of KCs does not require early differentiation; 5) NF-kappaB is one regulator of
20 to identify 69 genes altered by IRF-8 during early differentiation (62 up-regulated and 7 down-regula
21 -regulated opposition to pluripotency during early differentiation, a process highly distinct from st
22 ast, contains cells that are maturing toward early differentiation and are likely transit-amplifying
24 express the transcription factor MafB during early differentiation and maintain calbindin expression
27 ses define a specific role for both genes in early differentiation and survival of the placodally der
28 ling promoted osteoprogenitor proliferation, early differentiation, and commitment to the osteoblasti
29 e-specific matrix genes that are silenced in early differentiation are expressed during the terminal
31 immature T-ALLs are characterized by a very early differentiation arrest and show unique genetic and
33 nsable for osteoblast lineage commitment and early differentiation but also blocks osteoblast maturat
34 rvoirs showed that strong coupling supported early differentiation but required a higher number of ba
35 ed intrinsically in the germ cells for their early differentiation, but is required in the germ cells
36 ars is thought to hold clues to the planet's early differentiation, but until now no meteoritic regol
37 promoting ESC attachment, proliferation, and early differentiation, compared to native EB and decellu
39 se of only approximately 15% decrease during early differentiation despite global changes in gene exp
40 aging and gene expression profiling to study early differentiation dynamics spontaneously occurring w
43 n through the cell cycle and that these very early differentiation events do not require the accumula
44 ce promotes proliferation of osteoblasts and early differentiation events like production of alkaline
46 tive surfaces; conidia germinate and undergo early differentiation events, but appressorium maturatio
49 6-cell cysts, bridging the expression of the early differentiation factor Bam with late markers such
50 n, probably by inhibiting the translation of early differentiation factors, whereas niche signals pre
51 f adult hippocampal newborn cells die during early differentiation from intermediate progenitors (IPC
52 involved in epidermal differentiation, with early differentiation genes (KRT1, KRT10, DSC1, DSG1) be
53 that distinguish tonsil BCs from tonsil PCs (early differentiation genes [EDGs]), and tonsil PCs from
62 d of endothelial cells expressing markers of early differentiation, including VEGFR2 (Flk1), Tal1/SCL
63 embryonic stem cells (hESCs) in response to early differentiation, induced by retinoic acid, versus
66 rom stem cell self-renewal to overt signs of early differentiation is a poorly understood but importa
67 bitors (p15, p16, p21, and p27), a marker of early differentiation (keratin 1), mediators of apoptosi
68 inal differentiation program, stimulating an early differentiation marker (keratin 1) and suppressing
69 suggests that 9-O-acetylation appears as an early differentiation marker as cells mature from the DP
71 tors unexpectedly increased the synthesis of early differentiation marker protein delta-crystallin in
75 Induction of p21WAF1/Cip1 expression and early differentiation markers occur through two differen
76 NOTCH3 (N3) transcription, inducing HES5 and early differentiation markers such as involucrin (IVL) a
77 rogram characterized by normal expression of early differentiation markers such as myogenin and p21,
78 s encoding general cell growth functions and early differentiation markers was not affected, suggesti
79 D34(+) cells were equivalent with respect to early differentiation markers, and following culture, th
81 Activated Notch1 also induces expression of 'early' differentiation markers, while suppressing the la
82 derlying the switch between self-renewal and early differentiation may be acting in these two pools.
83 reas the transition between self-renewal and early differentiation modes of the network underlies the
84 ates the role of Akt and Clk1 kinases in the early differentiation of 3T3-L1 cells to adipocytes.
85 ess exposure increased the proliferation and early differentiation of adult neural progenitor cells.
86 correlates temporally and spatially with the early differentiation of angioblasts into the endothelia
88 search is needed to develop tools that allow early differentiation of bvFTD from primary psychiatric
90 o create a precise genetic definition of the early differentiation of cap mesenchyme progenitors.
92 ide evidence to show that MAIT cells promote early differentiation of CCR2-dependent monocytes into m
95 host dendritic cells (DCs) and that there is early differentiation of donor-derived DCs, even after t
98 e conclude that stat92E is necessary for the early differentiation of follicle cells and for proper g
100 expression of Wnt3a in the neocortex induced early differentiation of IPs into neurons and the accumu
105 1), which is localized in the nucleus during early differentiation of odontoblasts, is able to bind s
107 en postulated to play a critical role in the early differentiation of oligodendrocytes (OLs) in addit
108 est that proliferation, cell cycle exit, and early differentiation of primary lens fiber cells are re
109 a mutation in valentino (val), which blocks early differentiation of rhombomeres 5 and 6 in the hind
110 the nuclear localization of SOX9 during the early differentiation of Sertoli cells and the determina
113 promoter based upon the role of keratin 4 in early differentiation of the esophageal squamous epithel
114 veal a novel role for mesodermal Fgf8 on the early differentiation of the NC along the parasympatheti
115 hese two genes has specific consequences for early differentiation of the primary olfactory pathway-w
116 studies imply that Sox9 is required for the early differentiation of the prostate bud epithelia.
119 in the steps of cell fate determination and early differentiation of various retinal cell types.
123 l a developmental clock characteristic of an early differentiation program common to all EBs, further
124 uggesting that TopIIbeta is not required for early differentiation programming but is specifically re
125 in the regulation of keratinocyte growth and early differentiation, rather than terminal differentiat
126 Microarray analysis at various times during early differentiation reveal that mesoderm- and endoderm
127 ion, tubular organization and elongation and early differentiation) revealed signaling pathways poten
128 lterations in their generation, survival, or early differentiation, since all occurred normally in in
130 ive ascending (inside-out) placement, common early differentiation stage (regardless of size, locatio
131 ells by delivering IL-1 signaling during the early differentiation stage and integrating IL-23 signal
132 eukemic cell lines corresponding to the same early differentiation stages express abundant NELL2 mRNA
135 xpression is a general characteristic of the early differentiation stages of rodent trophoblast, give
136 ast, forced expression of polysialic acid in early differentiation stages reduces myotube formation a
137 genesis, while Six genes appear to act in an early differentiation step during thymus/parathyroid mor
138 if treated with patterning molecules at very early differentiation steps before neural induction.
140 ation, while P2X5 and P2X7 receptors control early differentiation, terminal differentiation and deat
143 in each head segment, we have examined their early differentiation using Alcian labeling of cartilage
145 induction of scd1 expression by cAMP during early differentiation was distinct from that observed du
146 s suggest that TLE activity is essential for early differentiation where it acts to suppress the plur
147 regulated independent of pluripotency during early differentiation, which is distinct from what occur
148 myogenic regulatory proteins shifted toward early differentiation with increased erythropoietin rece
149 gulatory network to balance self-renewal and early differentiation within the "mitotic region," which
150 AT12 protein was found expressed in the root early differentiation zone, where its abundance was modu
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