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1 h the roles of these proteins in intravirion early gene transcription.
2 arks and the activation of viral immediately early gene transcription.
3 be due at least in part to a defect in viral early gene transcription.
4 ncer complex that stimulates viral immediate-early gene transcription.
5 e that Z interacts with CBP to enhance viral early gene transcription.
6 stimulate Elk1 phosphorylation and immediate early gene transcription.
7 in (CREB), a critical regulator of immediate early gene transcription.
8 (MAPK) phosphorylation to regulate immediate early gene transcription.
9 equired for the repression of KSHV immediate early gene transcription.
10 ssion early in development, and in immediate early gene transcription.
11 ion factor HCF-1-to activate viral immediate-early-gene transcription.
12 ed growth-factor induction of both immediate-early gene transcription and exit from G0 in an Rb-indep
13 ly proteins is important for efficient viral early gene transcription and for changes in expression o
14 ly proteins is important for efficient viral early gene transcription and for changes in expression o
15 ulture was not solely due to increased viral early gene transcription, but also to augmented initial
16 LCR) of human papillomavirus (HPV) regulates early gene transcription by interaction with several vir
17 activation of herpes simplex virus immediate-early gene transcription by the viral regulatory protein
19 pid, long-term upregulation of the immediate early gene transcription factor c-Jun mRNA and protein i
20 on of, and trans-activation by the immediate-early gene transcription factor Egr-1 in a renal epithel
21 on and immunostaining, include the immediate early gene transcription factors EGR-1, EGR-2, and c-fos
22 (Egr-1, Krox24, zif/268) family of immediate-early gene transcription factors, which includes NGFI-C,
23 ped into the following categories: immediate early genes/transcription factors (Arc, CHOP, IER5, NGFI
24 omegalovirus (CMV) promoter represses HPV-16 early gene transcription from P97 in a dose-dependent ma
25 activation of herpes-simplex-virus immediate-early gene transcription in association with the viral t
26 ed T cells (NFAT) is a critical regulator of early gene transcription in response to TCR-mediated sig
27 kinase pathway and a regulator of immediate early gene transcription is a target for inhibition by t
30 differences in the start sites of immediate-early gene transcription, or by differences in the nucle
31 had no effect on VZV replication, immediate-early gene transcription, or virion assembly in vitro.
32 in herpes simplex virus 1 (HSV-1) immediate early gene transcription, our findings suggest that EBV
36 ulatory protein VP16 activates HSV immediate-early gene transcription through formation of a multipro
37 ly increased by inclusion of an active HSV-1 Early gene transcription unit, indicating that the assoc
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