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1 BN were pain, loss of abdominal domain, and early satiety.
2 ic target to modulate in the ES patient with early satiety.
3 more chest pain (2.7 vs. 1.0, P = 0.03) and early satiety (2.8 vs. 1.3, P = 0.02) and worse physical
6 tional dyspepsia, including epigastric pain, early satiety, and postprandial nausea, are nonspecific,
7 tburn, regurgitation, dysphagia, chest pain, early satiety, and postprandial pain) and improved QOL (
9 s included nausea, abdominal pain, bloating, early satiety, as well as the composite score of these 4
11 ell as nausea, abdominal pain, bloating, and early satiety compared with placebo (composite score, P
12 Questions focus on fatigue, concentration, early satiety, inactivity, night sweats, itching, bone p
13 l symptoms improved significantly except for early satiety (mean [SD] score, 3.18 [1.88] at baseline
14 , but greater than half of the patients with early satiety, night sweats, fatigue, pruritus, and coug
15 of leukocytosis or anemia and no symptoms of early satiety, night sweats, pruritus, or erythromelalgi
16 ue functional dyspepsia was epigastric pain, early satiety or postprandial fullness, and no organic G
17 P-1 area under the curve was associated with early satiety (P = 0.0002, R = 0.74), eating symptoms (P
21 classic symptoms of gastroparesis (including early satiety, postprandial fullness, bloating, abdomina
22 ient's nausea, vomiting, abdominal pain, and early satiety resolved after a short course of treatment
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