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1 infected individuals who were all started on early therapy.
2 han 12 months) may also receive benefit from early therapy.
3 n ganciclovir is given for prophylaxis or as early therapy.
4 ttle is known about the virologic effects of early therapy.
7 e on the basis of recent trials showing that early therapy can be potentially beneficial to patients.
8 day 7 PET follow-up may be exploited toward early therapy change, especially for the 15% of patients
10 nce interval, -1.83 to 4.32; P=0.42) for the early therapy group and 0.55 (95% confidence interval, -
11 d cellular infiltration and bone loss in the early therapy group, and reduced T cell proliferation in
12 single dose after collagen boost on day 21 (early therapy group, or as a single dose upon acquisitio
13 sease severity in the prophylactic group and early therapy group, reduced cellular infiltration and b
14 nts in the delayed-therapy group than in the early-therapy group (30 percent vs. 60 percent) had plas
15 linked transmissions, only 1 occurred in the early-therapy group (hazard ratio, 0.04; 95% CI, 0.01 to
16 f death of 69%, as compared with that in the early-therapy group (relative risk in the deferred-thera
17 bove each of the two thresholds of interest (early-therapy group) with that of patients who deferred
20 nt to contain HCV replication, and also that early therapy is effective independent of such responses
21 ute to increased mortality, but nonselective early therapy may result in excess costs and drug resist
23 commodate immune escape, we hypothesize that early therapy of primary infection may be beneficial des
26 e antiretroviral therapy either immediately (early therapy) or after a decline in the CD4 count or th
27 y according to age--three months or younger (early therapy) or older than three months (delayed thera
30 ition, sPLA(2) may be useful for instituting early therapies to prevent or reduce the clinical morbid
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