ライフサイエンスコーパス: eat

1 xperiments suggest the intermediacy of Ni(0)-ate complexes, leaving some doubt that a canonical catal

2 d with early satiety (P = 0.0002, R = 0.74), eating symptoms (P = 0.007, R = 0.54), and trouble enjoy

3 ENTIFIC COMMENTARY ON THIS ARTICLE: Abnormal eating behaviour and metabolic parameters including insu

4 emporal dementia is associated with abnormal eating behaviour characterized by increased sweet prefer

5 nts and proportions of macronutrients across eating patterns on meal or dietary glycemic index (GI) a

6 After eating a meal corresponding to one of the two odors, par

7 ures; however, no cases of anaphylaxis after eating a Citrus unshiu, the albedo of which is rich in p

8 ty (compared with littermate controls) after eating a high-fat diet.

9 h and pruritus approximately two hours after eating a frozen Citrus unshiu.

10 Five minutes after eating a commercially available boiled jellyfish product

11 he felt abdominal pain and nausea only after eating fruit, along with the albedo, of Citrus unshiu.

12 uinoa could also be detected in plasma after eating quinoa, indicating that some of these unique AR c

13 o developed hypersensitivity reactions after eating enokitake.

14 Evidence suggests altered eating behavior in obesity.

15 re race-ethnic disparities in what Americans eat.

16 fin by a mechanism involving formation of an ate complex, 1,2-metalate rearrangement, and beta-elimin

17 cytosol-invading bacteria functioning as an 'eat-me' signal for xenophagy.

18 rovement of foods that are accessible to and eaten by many.

19 Both sex (P = 0.002) and eating duration (P = 0.002) were associated with the ini

20 direction of the association between BMI and eating disorders (EDs) in adults via a two-sample MR app

21 Physical functioning (PF; QLQ-C30) and eating problems (EA; QLQ-OES24) were chosen as predefine

22 related and addictive disorders, feeding and eating disorders, schizophrenia, anxiety disorder, OCD,

23 earch is needed to facilitate the liking and eating of these foods by mothers, which will, in turn, i

24 Consumption of chicken meat and eating out were the most important risk factors for Camp

25 hether the distinct and shared metabolic and eating changes represent a component of the proposed spe

26 tions were observed between gut microbes and eating behaviors, including eating frequency, early ener

27 a core feature of some forms of obesity and eating disorders, as well as of the recently proposed di

28 tric conditions such as forms of obesity and eating disorders.

29 on contributes to failed diets, obesity, and eating disorders.

30 he number of OBE days, abstinence rates, and eating disorder psychopathologic findings and may be a b

31 outcomes included energy self-regulation and eating behaviors assessed with questionnaires at 6, 12,

32 tionship between perceived food scarcity and eating behavior.

33 e propose definitions for meals, snacks, and eating occasions for use in research.

34 iation between environmental stimulation and eating performance among nursing home residents with dem

35 ich may be of importance for the storage and eating quality of the fruit.

36 when feeling full more than once a week, and eating continuously during the day, a participant who po

37 necrosis, and, by lacking the "find me" and "eat me" signals that are a feature of apoptosis, necropt

38 window by generating specific "find me" and "eat me" signals.

39 y was partially mediated by the "appetitive" eating behavior traits (uncontrolled and emotional eatin

40 are well known to feed on honeybees that are eaten by spiders, which we thus predicted as the model c

41 pect of serious clinical conditions, such as eating disorders.

42 auty differs from sensuous pleasures such as eating or sex.

43 timulation induce drive-like effects such as eating, and short trains are rewarding.

44 ropionate on the human brain or reward-based eating behavior is currently unavailable.

45 no history of jellyfish stings and had been eating commercially available jellyfish products twice y

46 on vision for capturing prey, avoiding being eaten and for spatial orientation.

47 15-36, with higher scores indicating better eating performance).

48 offered to a resident contributed to better eating performance among residents with dementia.

49 were used to examine the association between eating performance and environmental stimulation, adjust

50 ia, suggesting a complex interaction between eating behaviour, autonomic function and energy homeosta

51 Binge eating is a highly heritable trait associated with eatin

52 ks), lisdexamfetamine responders (</=1 binge eating day per week for 4 consecutive weeks and CGI-S sc

53 otypes: pathological gambling (PG) and binge eating disorder (BED).

54 We assessed binge eating in closely related C57BL/6 mouse substrains and i

55 Compulsive, binge eating of highly palatable food constitutes a core featu

56 rmacological treatment for compulsive, binge eating.

57 ention deficit hyperactivity disorder, binge eating disorder, cocaine addiction, obesity, and type 2

58 isorders, including gambling disorder, binge eating disorder, compulsive sexual behaviour, and compul

59 The neuronal substrate for binge eating, which can at times lead to obesity, is not clear

60 volved protective mechanisms including binge eating and increased metabolic efficiency and fat storag

61 inform molecular mechanisms mediating binge eating susceptibility and establishment.

62 e in the number of days with objective binge eating episodes (OBEs) during the previous 28 days betwe

63 esults suggested that higher levels of binge eating and overeating in males at age 13 y likely cause

64 Furthermore, higher levels of binge eating and overeating may cause higher BMI in later life

65 age 7 y likely causes higher levels of binge eating and overeating, weight and shape concerns, and we

66 he criterion standard for treatment of binge eating disorder (BED), most individuals do not have acce

67 57BL/6N background that showed reduced binge eating toward a wild-type C57BL/6J-like level.

68 er criteria and had moderate to severe binge eating disorder (>/=3 binge-eating days per week for 14

69 significant genetic factor underlying binge eating and provide a behavioral paradigm for future geno

70 2 as a major genetic factor underlying binge eating in heterozygous knockout mice on a C57BL/6N backg

71 ade endocannabinoid signaling, whereas binge eating resulted in the downregulation of a gene set enri

72 uantitative trait loci associated with binge eating.

73 Binge-eating disorder (BED) is characterized by recurring epis

74 utcome variable, time to relapse (>/=2 binge-eating days per week for 2 consecutive weeks and >/=2-po

75 to severe binge eating disorder (>/=3 binge-eating days per week for 14 days before open-label basel

76 Risk of binge-eating relapse over 6 months was lower in participants c

77 owed flexibility in foraging patterns, binge-eating less and using feeders more when they experienced

78 PC1 ('binge-eating'), accounting for 38% of variation, captured temp

79 Eligible adults met DSM-IV-R binge-eating disorder criteria and had moderate to severe bing

80 cacy in adults with moderate to severe binge-eating disorder.

81 sider how to reduce short-term insecurity by eating healthy food.

82 genesis caused by dysregulation of so-called eat me ligands, which govern the edibility of cells unde

83 between fasting appetite ratings and certain eating behavior traits with daily EI.

84 er controlling for resident characteristics, eating performance was significantly associated with sti

85 The assay detects six commonly eaten meat species and provides the following benefits:

86 nges in appetite that result in compensatory eating, which may initially dissuade potential success i

87 ia experience increased risk for compromised eating performance due to intrapersonal, interpersonal,

88 Compulsive eating behavior is a transdiagnostic construct that is c

89 d the prefrontal cortex result in compulsive eating behaviors.

90 ppetitive motivation that induces compulsive eating and contributes to obesity.

91 need to improve understanding of compulsive eating through the integration of complex motivational,

92 re, we present evidence to relate compulsive eating behavior and addiction and to characterize their

93 nd the mechanisms contributing to compulsive eating are not yet clearly defined or understood.

94 The most prevalent subtype was compulsive eating, followed by punding, compulsive sexual behaviour

95 ammatory marker or adipokine concentrations, eating behaviors and changes in waist circumference duri

96 ammatory marker or adipokine concentrations, eating behaviors, comorbid psychiatric disorders or life

97 y dieting, the experiences of out-of-control eating, increased appetite, and increased fat storage ar

98 coral bleaching, and outbreaks of the coral-eating crown-of-thorns starfish (COTS), losing much of i

99 ld confirm later that all the patients could eat foods containing half of a chicken egg.

100 lancing selection on at least one case (Crab-eating monkey retrocopy 6, or CER6) in both species.

101 ting disease in experimentally infected crab-eating macaques, while simian hemorrhagic fever virus (S

102 browsers for three species (brown kiwi, crab-eating macaque and Malayan flying lemur); eight updated

103 nonhuman primate cell cultures and then crab-eating macaques with either simian hemorrhagic fever vir

104 s of wakefulness and ingestion events, daily eating pattern offers a new potential target for lifesty

105 ent fasting, meal frequency (number of daily eating occasions), and timing of eating occasions.

106 in this study only if residents demonstrated eating activities at mealtimes.

107 Clinically diagnosed eating disorders may have adverse cardiometabolic conseq

108 the first time the influence of disinhibited eating and vagal tone (heart rate variability (HRV)) on

109 light novel mechanisms by which disinhibited eating leads to obesity and insulin resistance.

110 n, attention-deficit/hyperactivity disorder, eating disorders, autism spectrum disorder, substance us

111 sociated with posttraumatic stress disorder, eating disorders, and anxiety disorders other than speci

112 tion of causality between BMI and disordered eating in childhood, adolescence, and adulthood.MR analy

113 with increased risk of developing disordered eating patterns.

114 n childhood and increased risk of disordered eating at age 13 y.

115 tified in this new mouse model of disordered eating.

116 ausal effect of BMI at age 7 y on disordered eating patterns at age 13 y with the use of data from th

117 s, which could inform and prevent disordered eating problems in adolescence.

118 o estimate the effect of the same disordered eating patterns at age 13 y on BMI at age 17 y via a spl

119 ly adolescence, which can lead to disordered eating behaviors, and future cardiometabolic health is,

120 1; 2001-2003) was associated with disordered eating behaviors at time 2 (2004-2006), as well as weigh

121 Using clock hour to document eating times may be misleading owing to individual diffe

122 fast life-history strategy, and dysregulated-eating behaviors.

123 regulated weight-management behaviors (i.e., eating even in the absence of hunger), which were predic

124 ion, we tested for interactions between each eating behavior and the BMI-GRS on BMI.The association b

125 The mediating effect of each eating behavior on the association between the BMI-GRS a

126 that perceived hunger, importance of eating, eating frequencies, and liking ratings of food pictures

127 (P = 0.03 and P = 0.012) with the effective eating duration and maximum food intake (adjusted R(2) =

128 It is either eaten raw or processed, and also as a functional ingredi

129 behavior traits (uncontrolled and emotional eating) and, in 3 of the 4 population groups studied, wa

130 The eating behaviors-emotional eating, uncontrolled eating, and cognitive restraint-wer

131 I-GRS and BMI was mediated by both emotional eating (EDEN: P-Sobel = 0.01; Fenland: P-Sobel = 0.02) a

132 ating this reaction is two-fold: it enables "ate" complex formation and overcomes catalytic inhibitio

133 tion trial findings suggested that a low-fat eating pattern may reduce breast cancers with greater mo

134 established neurotoxicant of concern to fish-eating organisms.

135 to patients in all groups and data on foods eaten and compliance were collected.

136 ary GI and GL values that are calculated for eating patterns, which may alter the interpretation of t

137 havior may inspire new treatment options for eating disorders and obesity.

138 tion of average dietary GI and GL values for eating patterns.

139 multi-faceted approach to estimate how fruit-eating bats (Uroderma bilobatum) manage a high-energy li

140 Large, fruit-eating vertebrates have been lost from many of the world

141 BACKGROUND & AIMS: Healthy eating patterns assessed by diet quality indexes (DQIs)

142 (CVD), can gain health benefits from healthy eating behaviors and appropriate physical activity.

143 ing is a prominent policy to promote healthy eating.We aimed to evaluate the effects of 2 interpretiv

144 rends in prevalence of 6 barriers to healthy eating (taste, price, daily habits, time, lack of willpo

145 This may be because of barriers to healthy eating hampering adherence, but whether these barriers h

146 valence of self-reported barriers to healthy eating in Switzerland overall and according to sex, age,

147 Diet quality estimated adherence to healthy eating recommendations and was assessed by using the 201

148 e.Between 1997 and 2012, barriers to healthy eating remained highly prevalent (>/=20%) in the Swiss p

149 variance in concordance with the WHO healthy eating guidelines that aim to prevent non-communicable d

150 the INTERMAP UK cohort (n=225) and a healthy-eating Danish cohort (n=66).

151 lations, and can be applied to determine how eating behavior factors influence total food intake.

152 actor for adverse metabolic health; yet, how eating at a later circadian time influences body composi

153 dy, the microbial community itself, or human eating behaviors.

154 wel disease (IBD) is, "Doctor, what should I eat?" Findings from epidemiology studies have indicated

155 utrients to the brain, initiating changes in eating behavior and energy expenditure, to maintain ener

156 l correlates overlap the network involved in eating behaviour in frontotemporal dementia, suggesting

157 Reductions in eating disorder psychopathologic findings were significa

158 Given its critical role in eating behaviour, the primary region of interest was the

159 The fourth pattern, seen in eating disorders, was directly opposite of that observed

160 ly controlled research of brain structure in eating disorders, which will ultimately help predict the

161 gut microbes and eating behaviors, including eating frequency, early energy consumption, and overnigh

162 time of day or behavioral factors, including eating frequency, percentage of energy consumed early in

163 Potentially modifiable mechanisms, including eating behaviors, are of particular interest to public h

164 onmental stimulation influences individuals' eating performance at mealtimes.

165 (P = 0.002) were associated with the initial eating rate (adjusted R(2) = 0.23).

166 ntake using 3 key parameters: 1) the initial eating rate, 2) the effective duration of eating, and 3)

167 cal and practical importance - since insects eat 20% of crops - no exhaustive analysis of gene repert

168 Intentional eating with mindful attention to the timing and frequenc

169 Finally, data suggest that irregular eating patterns appear less favorable for achieving a he

170 technique of low amylose/waxy paddy that is eaten after soaking in water and requires no cooking.

171 results show that OSU6162 reduces binge-like eating behavior and attenuates the impact of cues on see

172 sults show that RO5256390 blocked binge-like eating in rats responding 1 h per day for a highly palat

173 fects of OSU6162 on consummatory (binge-like eating) and appetitive (cue-controlled seeking) behavior

174 ic, subregion of the mPFC-reduced binge-like eating.

175 iately (in 2 to 3 seconds) evoked binge-like eating.

176 R1 may have a role in compulsive, binge-like eating; we tested this hypothesis by assessing the effec

177 ore, RO5256390 fully blocked compulsive-like eating when the palatable diet was offered in an aversiv

178 Man-eating was likely a viable alternative to hunting and/or

179 gh humans are not typical prey, habitual man-eating by lions is well documented.

180 teeth to assess whether these notorious man-eating lions scavenged carcasses during their depredatio

181 Most dramatically, mice eating an otherwise unhealthy high-calorie, high-sugar W

182 assessed whether variations in midchildhood eating attitudes influence the future development of ove

183 What lactating mothers eat flavors breast milk and, in turn, modifies their inf

184 Therefore, she never ate enokitake since that time.

185 patterns that reduce or eliminate nighttime eating and prolong nightly fasting intervals may result

186 Among infants aged 12 months who did not eat fish or seafood, the geometric mean total urinary As

187 97 microg/L) compared with those who did not eat rice or rice products (2.85 microg/L; all P < .01).

188 in the support of self-regulatory aspects of eating behavior and inhibitory control.

189 pically focused on the long-term benefits of eating healthy food.

190 These include psychological determinants of eating behaviours, the decline in physical activity lead

191 ne the effects of the timing and duration of eating a variety of vegetables during breastfeeding on t

192 al eating rate, 2) the effective duration of eating, and 3) the maximal food intake.

193 ng meal significantly modified the effect of eating litchis on illness (odds ratio [OR] 7.8 [95% CI 3

194 ful attention to the timing and frequency of eating occasions could lead to healthier lifestyle and c

195 strated that perceived hunger, importance of eating, eating frequencies, and liking ratings of food p

196 hasize the need for further investigation of eating disorders in military service members.

197 participants demonstrated moderate levels of eating performance (M=27.08, SD=5.16).

198 Recently, an increasingly popular method of eating anchovies has been to eat it in a cleaned, evisce

199 The aims were to assess the prevalence of eating difficulties in infants born LMPT at 2 y correcte

200 habits, including the number and quality of eating occasions, are potential targets for primary prev

201 nic tone, which resulted in a faster rate of eating carrot-flavored cereal than that in infants who w

202 hen's egg was detectable in dust samples of eating area and bed.

203 a notoriously difficult-to-treat symptom of eating disorders.

204 Behavioral factors, including timing of eating and overnight-fast duration, were also predictive

205 er of daily eating occasions), and timing of eating occasions.

206 d with sham tDCS) has an immediate effect on eating behavior during ad libitum food intake, resulting

207 d that parenting practices have an impact on eating styles which determine obesity levels in children

208 hibition promotes growth of cells reliant on eating extracellular protein.

209 However, EI at one eating occasion (EO) is also dependent on EI at surround

210 types (such as in uncontrollable gambling or eating).

211 rgets for the treatment of cachexia or other eating disorders.

212 High levels of cognitive control over eating appear to attenuate the genetic susceptibility to

213 Patients ate turshi at 2 locations on different days over 1 month

214 nds, such as those generated by other people eating, drinking, chewing, and breathing [1-8].

215 versible formation of a pentavalent peroxide ate complex, prior to rate-limiting aryl migration, has

216 Picky eating has been associated with lower weight status and

217 marshes experiencing a severe drought, plant-eating grazers eliminated drought-stressed vegetation th

218 Large plant-eating dinosaurs are usually presumed to have been stric

219 Problematic eating attitudes in midchildhood seem to be related to t

220 However, the link between problematic eating attitudes in early adolescence, which can lead to

221 bserved positive associations of problematic eating attitudes at age 11.5 y with new-onset obesity (O

222 /=85th percentile (indicative of problematic eating attitudes) compared with scores <85th percentile

223 or body mass index at age 6.5 y, problematic eating attitudes remained positively associated with new

224 In total, 11.1% of children never or rarely ate the food after a negative OFC.

225 consumed fish, compared to those who rarely ate fish (all p < 0.05).

226 rs - even mildly defended mimics were rarely eaten by a community of invertebrate and vertebrate pred

227 e duodenum also suppressed appetite ratings, eating behavior was not altered.

228 males prefer the type they have not recently eaten.

229 ty, the least fattening of widely recognised eating patterns needs to be measured and supported.

230 at diurnal size changes in the liver require eating during a mouse's normal awake time and that these

231 ing appetite ratings (visual analog scales), eating behavior traits (Three-Factor Eating Questionnair

232 Autophagy is the process of cellular self-eating by a double-membrane organelle, the autophagosome

233 fter autophagy) is a catabolic cellular self-eating process by which unwanted organelles or proteins

234 n examining DA's role in reward sensitivity, eating, and obesity.

235 Anorexia nervosa (AN) is a serious eating disorder characterized by self-starvation and ext

236 When she was 4 years old, she ate enokitake with a hot-pot dish.

237 o this end, apoptotic cells express specific eat-me signals, such as externalized phosphatidylserine

238 e cardiometabolic health effects of specific eating patterns: skipping breakfast, intermittent fastin

239 1 point increase in stimulation specificity, eating performance increased by 8.78 points (95% CI=0.59

240 topped eating when feeling full, and stopped eating continuously during the day after surgery would b

241 stoperatively started to self-weigh, stopped eating when feeling full, and stopped eating continuousl

242 Such eating styles can have various effects on cardiometaboli

243 One such "eat me" signal observed during apoptosis is the exposure

244 tributes to metabolic control by suppressing eating behavior.

245 ency preserved the expression of CD47 (don't eat me signal) on cardiac MSCs after both TLR4 stimulati

246 ic immune checkpoint that engages the "don't eat me" signal CD47 expressed on tumors and normal tissu

247 uces anti-phagocytic signals from the 'don't eat me' CD47 ligand through negative Shp1 signaling, the

248 oplastic cells generate this dominant 'don't eat me' signal, we analyse the CD47 regulatory genomic l

249 posed of what has become known as the 'don't-eat me' signal CD47, which is a protein broadly expresse

250 Temporal eating patterns, based on the distribution of EOs across

251 isreporting on the relation between temporal eating patterns and adiposity.

252 odels assessed associations between temporal eating patterns, nutrient intakes, diet quality, and adi

253 was to examine associations between temporal eating patterns, nutrient intakes, diet quality, and mea

254 I.This study found that a "grazing" temporal eating pattern was modestly but significantly associated

255 rences in morphology and diet of the termite-eating gecko Gymnodactylus amarali between five such new

256 er on a treadmill than did control rats that ate an isocaloric diet that was supplemented with either

257 score, 77.7%); however, 48.6% believed that eating a healthy diet precluded the need for HCC surveil

258 of evidence also support the hypothesis that eating patterns that reduce or eliminate nighttime eatin

259 The eating behaviors-emotional eating, uncontrolled eating,

260 ulum stress with consequent exposure of the "eat-me" signal calreticulin on the cell surface, thereby

261 Alzheimer disease) were recruited, and their eating behavior was compared with that of 25 healthy con

262 If proven to be efficacious, these eating regimens offer promising nonpharmacological appro

263 For predators able to eat only a few species (i.e., specialists), low phenotyp

264 kg/m(2)): 27-35; age: 18-45 y] were asked to eat either a main meal at lunch (LM) or a main meal at d

265 pular method of eating anchovies has been to eat it in a cleaned, eviscerated form.

266 ass, ie, weekly self-weighing, continuing to eat when feeling full more than once a week, and eating

267 In participants directed to eat as much as they could compared with as much as they

268 fects of increasing the homeostatic drive to eat on feeding behavior during appetite suppressing cond

269 ic disorder associated with reduced drive to eat.

270 es in cortex for enhancing the motivation to eat are discriminable from those for hedonic impact.

271 eed.SIGNIFICANCE STATEMENT The motivation to eat depends on the relative balance of activity in disti

272 ons about where, when, what, and how much to eat are not merely reflexive responses to food-relevant

273 se persuasive appeals to encourage people to eat healthier, purchase a particular product, or vote fo

274 can be treated by simply advising people to eat less and move more.

275 After adjustments, pressure to eat and overt control at 4 y of age were associated with

276 The tendency to eat more or less in response to emotion is learned rathe

277 roup, VAS ratings for hunger and the urge to eat declined significantly more (P = 0.01 and P = 0.05,

278 ts with obesity should have the willpower to eat less.

279 changes across groups and their relations to eating behaviors.

280 Ready-to-eat baby porridge was the only category in which all mea

281 During processing of ready-to-eat fresh fruits, large amounts of peel and seeds are di

282 ncluding the vesicular glutamate transporter eat-4/VGLUT, induction of neuropeptide expression, chang

283 1; Fenland: P-Sobel = 0.02) and uncontrolled eating (EDEN: P-Sobel = 0.04; Fenland: P-Sobel = 0.0006)

284 ing behaviors-emotional eating, uncontrolled eating, and cognitive restraint-were measured through th

285 nk these factors and contribute to unhealthy eating are unclear.

286 which, in turn, may contribute to unhealthy eating behaviors later in life.

287 nergy intake was measured with the universal eating monitor in a test meal.In mice, AITC administrati

288 ondary outcomes included OBEs at follow-ups, eating disorder and general psychopathologic findings, b

289 The dependent variable was eating performance as measured by the Level of Eating In

290 te that, in a limited sense, "we are what we eat" and provide a perspective on a nutrition-driven mod

291 acial expressions of distaste initially when eating the carrot cereal.

292 ers were estimated in a study (n = 49) where eating rates were deliberately changed.

293 est to public health.Here we explore whether eating behaviors mediate or modify genetic susceptibilit

294 e, glp-1 mutation only delays p death, while eat-2 mutation reduces P death.

295 While eating disorders (EDs) are thought to result from a comb

296 r, most of the questionnaire respondents who ate untreated meals knew how to prevent Anisakis infecti

297 s concentrations were higher among those who ate infant rice cereal (9.53 microg/L) or rice snacks (4

298 49, 3.48) were independently associated with eating difficulties in multivariable analyses.

299 is a highly heritable trait associated with eating disorders that is comorbid with mood and substanc

300 scores >90th percentile were classified with eating difficulties in each domain.

301 sus arctos) hibernate for 5-7 months without eating, drinking, urinating, and defecating at a metabol