ライフサイエンスコーパス: eating

1 types (such as in uncontrollable gambling or eating).

2 uantitative trait loci associated with binge eating.

3 iately (in 2 to 3 seconds) evoked binge-like eating.

4 ic, subregion of the mPFC-reduced binge-like eating.

5 tified in this new mouse model of disordered eating.

6 rmacological treatment for compulsive, binge eating.

7 c food cues are robust and are suppressed by eating.

8 nd every 30 minutes, up to 240 minutes after eating.

9 Participants eating 5-7 midday MPAH had 9% lower T2D risk than those

10 ures; however, no cases of anaphylaxis after eating a Citrus unshiu, the albedo of which is rich in p

11 Five minutes after eating a commercially available boiled jellyfish product

12 h and pruritus approximately two hours after eating a frozen Citrus unshiu.

13 score, 77.7%); however, 48.6% believed that eating a healthy diet precluded the need for HCC surveil

14 ty (compared with littermate controls) after eating a high-fat diet.

15 After eating a meal corresponding to one of the two odors, par

16 ne the effects of the timing and duration of eating a variety of vegetables during breastfeeding on t

17 The study of autophagy ('self-eating'), a fundamental cell fate pathway involved in ph

18 PC1 ('binge-eating'), accounting for 38% of variation, captured temp

19 in this study only if residents demonstrated eating activities at mealtimes.

20 Most dramatically, mice eating an otherwise unhealthy high-calorie, high-sugar W

21 Recently, an increasingly popular method of eating anchovies has been to eat it in a cleaned, evisce

22 ppetitive motivation that induces compulsive eating and contributes to obesity.

23 volved protective mechanisms including binge eating and increased metabolic efficiency and fat storag

24 esults suggested that higher levels of binge eating and overeating in males at age 13 y likely cause

25 Furthermore, higher levels of binge eating and overeating may cause higher BMI in later life

26 age 7 y likely causes higher levels of binge eating and overeating, weight and shape concerns, and we

27 Behavioral factors, including timing of eating and overnight-fast duration, were also predictive

28 patterns that reduce or eliminate nighttime eating and prolong nightly fasting intervals may result

29 significant genetic factor underlying binge eating and provide a behavioral paradigm for future geno

30 fetamine, SGAs, and topiramate reduced binge eating and related psychopathology, and lisdexamfetamine

31 ard arm uses the Australian Guide to Healthy Eating and the Australian National Heart Foundation diet

32 the first time the influence of disinhibited eating and vagal tone (heart rate variability (HRV)) on

33 implications of natural OH cell signals for eating and weight in a new conditional OH cell-knockout

34 fects of OSU6162 on consummatory (binge-like eating) and appetitive (cue-controlled seeking) behavior

35 behavior traits (uncontrolled and emotional eating) and, in 3 of the 4 population groups studied, wa

36 al eating rate, 2) the effective duration of eating, and 3) the maximal food intake.

37 ing behaviors-emotional eating, uncontrolled eating, and cognitive restraint-were measured through th

38 inhibitory interplay between OH signals and eating, and demonstrate that OH cell activity is rapidly

39 ted behaviors presumably preclude concurrent eating, and loss of OH cells produces obesity, suggestin

40 n examining DA's role in reward sensitivity, eating, and obesity.

41 timulation induce drive-like effects such as eating, and short trains are rewarding.

42 High levels of cognitive control over eating appear to attenuate the genetic susceptibility to

43 nd the mechanisms contributing to compulsive eating are not yet clearly defined or understood.

44 nk these factors and contribute to unhealthy eating are unclear.

45 hen's egg was detectable in dust samples of eating area and bed.

46 actor for adverse metabolic health; yet, how eating at a later circadian time influences body composi

47 Eating at a later clock hour is a newly described risk f

48 n childhood and increased risk of disordered eating at age 13 y.

49 bserved positive associations of problematic eating attitudes at age 11.5 y with new-onset obesity (O

50 However, the link between problematic eating attitudes in early adolescence, which can lead to

51 Problematic eating attitudes in midchildhood seem to be related to t

52 assessed whether variations in midchildhood eating attitudes influence the future development of ove

53 or body mass index at age 6.5 y, problematic eating attitudes remained positively associated with new

54 response rate) who completed the Children's Eating Attitudes Test (ChEAT) at age 11.5 y and in whom

55 /=85th percentile (indicative of problematic eating attitudes) compared with scores <85th percentile

56 Necrotizing fasciitis (NF) caused by flesh-eating bacteria is associated with high case fatality.

57 multi-faceted approach to estimate how fruit-eating bats (Uroderma bilobatum) manage a high-energy li

58 re, we present evidence to relate compulsive eating behavior and addiction and to characterize their

59 results show that OSU6162 reduces binge-like eating behavior and attenuates the impact of cues on see

60 utrients to the brain, initiating changes in eating behavior and energy expenditure, to maintain ener

61 in the support of self-regulatory aspects of eating behavior and inhibitory control.

62 ion, we tested for interactions between each eating behavior and the BMI-GRS on BMI.The association b

63 eostatic central nervous networks regulating eating behavior as well as decreased hunger feelings and

64 The disturbed eating behavior can be restored by substitution with the

65 d with sham tDCS) has an immediate effect on eating behavior during ad libitum food intake, resulting

66 lations, and can be applied to determine how eating behavior factors influence total food intake.

67 Evidence suggests altered eating behavior in obesity.

68 Compulsive eating behavior is a transdiagnostic construct that is c

69 ropionate on the human brain or reward-based eating behavior is currently unavailable.

70 The mediating effect of each eating behavior on the association between the BMI-GRS a

71 fruit) and the FF scale from the Children's Eating Behavior Questionnaire.

72 Children's Eating Behavior Questionnaires were also completed by ca

73 ing appetite ratings (visual analog scales), eating behavior traits (Three-Factor Eating Questionnair

74 y was partially mediated by the "appetitive" eating behavior traits (uncontrolled and emotional eatin

75 between fasting appetite ratings and certain eating behavior traits with daily EI.

76 Alzheimer disease) were recruited, and their eating behavior was compared with that of 25 healthy con

77 e duodenum also suppressed appetite ratings, eating behavior was not altered.

78 tionship between perceived food scarcity and eating behavior.

79 tributes to metabolic control by suppressing eating behavior.

80 ic, neuroendocrine, and visual modulation of eating behavior.

81 (CVD), can gain health benefits from healthy eating behaviors and appropriate physical activity.

82 ammatory marker or adipokine concentrations, eating behaviors and changes in waist circumference duri

83 outcomes included energy self-regulation and eating behaviors assessed with questionnaires at 6, 12,

84 1; 2001-2003) was associated with disordered eating behaviors at time 2 (2004-2006), as well as weigh

85 which, in turn, may contribute to unhealthy eating behaviors later in life.

86 est to public health.Here we explore whether eating behaviors mediate or modify genetic susceptibilit

87 ly adolescence, which can lead to disordered eating behaviors, and future cardiometabolic health is,

88 Potentially modifiable mechanisms, including eating behaviors, are of particular interest to public h

89 ammatory marker or adipokine concentrations, eating behaviors, comorbid psychiatric disorders or life

90 tions were observed between gut microbes and eating behaviors, including eating frequency, early ener

91 The eating behaviors-emotional eating, uncontrolled eating,

92 fast life-history strategy, and dysregulated-eating behaviors.

93 d the prefrontal cortex result in compulsive eating behaviors.

94 changes across groups and their relations to eating behaviors.

95 dy, the microbial community itself, or human eating behaviors.

96 ENTIFIC COMMENTARY ON THIS ARTICLE: Abnormal eating behaviour and metabolic parameters including insu

97 emporal dementia is associated with abnormal eating behaviour characterized by increased sweet prefer

98 l correlates overlap the network involved in eating behaviour in frontotemporal dementia, suggesting

99 ratings of child EOE and EUE using the Child Eating Behaviour Questionnaire.

100 ia, suggesting a complex interaction between eating behaviour, autonomic function and energy homeosta

101 Given its critical role in eating behaviour, the primary region of interest was the

102 tal studies emphasises a range of changes in eating behaviours and metabolism in amyotrophic lateral

103 These include psychological determinants of eating behaviours, the decline in physical activity lead

104 Autophagy is the process of cellular self-eating by a double-membrane organelle, the autophagosome

105 gh humans are not typical prey, habitual man-eating by lions is well documented.

106 nic tone, which resulted in a faster rate of eating carrot-flavored cereal than that in infants who w

107 hether the distinct and shared metabolic and eating changes represent a component of the proposed spe

108 A minor change in the dietary habits of eating cleaned anchovies may lead to a reduction in micr

109 no history of jellyfish stings and had been eating commercially available jellyfish products twice y

110 topped eating when feeling full, and stopped eating continuously during the day after surgery would b

111 when feeling full more than once a week, and eating continuously during the day, a participant who po

112 coral bleaching, and outbreaks of the coral-eating crown-of-thorns starfish (COTS), losing much of i

113 the INTERMAP UK cohort (n=225) and a healthy-eating Danish cohort (n=66).

114 ks), lisdexamfetamine responders (</=1 binge eating day per week for 4 consecutive weeks and CGI-S sc

115 to severe binge eating disorder (>/=3 binge-eating days per week for 14 days before open-label basel

116 utcome variable, time to relapse (>/=2 binge-eating days per week for 2 consecutive weeks and >/=2-po

117 Change from baseline in binge eating days/week at weeks 11-12 (primary efficacy endpoi

118 was superior to placebo in decreasing binge eating days/week from baseline and improving binge eatin

119 gestation) infants are at increased risk of eating difficulties compared with their term-born peers.

120 scores >90th percentile were classified with eating difficulties in each domain.

121 The aims were to assess the prevalence of eating difficulties in infants born LMPT at 2 y correcte

122 49, 3.48) were independently associated with eating difficulties in multivariable analyses.

123 Large plant-eating dinosaurs are usually presumed to have been stric

124 er criteria and had moderate to severe binge eating disorder (>/=3 binge-eating days per week for 14

125 depression (19% [95% CI, 14%-25%]) and binge eating disorder (17% [95% CI, 13%-21%]).

126 Binge-eating disorder (BED) is characterized by recurring epis

127 he criterion standard for treatment of binge eating disorder (BED), most individuals do not have acce

128 otypes: pathological gambling (PG) and binge eating disorder (BED).

129 The Internet and Binge Eating Disorder (INTERBED) study is a prospective, multi

130 ondary outcomes included OBEs at follow-ups, eating disorder and general psychopathologic findings, b

131 Anorexia nervosa (AN) is a serious eating disorder characterized by self-starvation and ext

132 Eligible adults met DSM-IV-R binge-eating disorder criteria and had moderate to severe bing

133 nderstanding the underpinning biology of the eating disorder is important, as well as potential co-oc

134 he number of OBE days, abstinence rates, and eating disorder psychopathologic findings and may be a b

135 Reductions in eating disorder psychopathologic findings were significa

136 Neither depression nor binge eating disorder was consistently associated with differe

137 ention deficit hyperactivity disorder, binge eating disorder, cocaine addiction, obesity, and type 2

138 isorders, including gambling disorder, binge eating disorder, compulsive sexual behaviour, and compul

139 cacy in adults with moderate to severe binge-eating disorder.

140 piramate reduced weight in adults with binge-eating disorder.

141 patients-in particular, depression and binge eating disorder.

142 While eating disorders (EDs) are thought to result from a comb

143 direction of the association between BMI and eating disorders (EDs) in adults via a two-sample MR app

144 PT may also be effective in the treatment of eating disorders and anxiety disorders and has shown pro

145 havior may inspire new treatment options for eating disorders and obesity.

146 Eating disorders are lethal and heritable; however, the

147 hasize the need for further investigation of eating disorders in military service members.

148 Clinically diagnosed eating disorders may have adverse cardiometabolic conseq

149 tion to maladaptive feeding behavior seen in eating disorders or obesity may arise from dysregulation

150 However, eating disorders remain understudied in military samples

151 as a therapeutic target for the treatment of eating disorders such as anorexia nervosa.

152 is a highly heritable trait associated with eating disorders that is comorbid with mood and substanc

153 sociated with posttraumatic stress disorder, eating disorders, and anxiety disorders other than speci

154 a core feature of some forms of obesity and eating disorders, as well as of the recently proposed di

155 n, attention-deficit/hyperactivity disorder, eating disorders, autism spectrum disorder, substance us

156 related and addictive disorders, feeding and eating disorders, schizophrenia, anxiety disorder, OCD,

157 The fourth pattern, seen in eating disorders, was directly opposite of that observed

158 ly controlled research of brain structure in eating disorders, which will ultimately help predict the

159 a notoriously difficult-to-treat symptom of eating disorders.

160 on contributes to failed diets, obesity, and eating disorders.

161 pect of serious clinical conditions, such as eating disorders.

162 rgets for the treatment of cachexia or other eating disorders.

163 tric conditions such as forms of obesity and eating disorders.

164 nds, such as those generated by other people eating, drinking, chewing, and breathing [1-8].

165 sus arctos) hibernate for 5-7 months without eating, drinking, urinating, and defecating at a metabol

166 Both sex (P = 0.002) and eating duration (P = 0.002) were associated with the ini

167 (P = 0.03 and P = 0.012) with the effective eating duration and maximum food intake (adjusted R(2) =

168 at diurnal size changes in the liver require eating during a mouse's normal awake time and that these

169 strated that perceived hunger, importance of eating, eating frequencies, and liking ratings of food p

170 I-GRS and BMI was mediated by both emotional eating (EDEN: P-Sobel = 0.01; Fenland: P-Sobel = 0.02) a

171 1; Fenland: P-Sobel = 0.02) and uncontrolled eating (EDEN: P-Sobel = 0.04; Fenland: P-Sobel = 0.0006)

172 o developed hypersensitivity reactions after eating enokitake.

173 e in the number of days with objective binge eating episodes (OBEs) during the previous 28 days betwe

174 regulated weight-management behaviors (i.e., eating even in the absence of hunger), which were predic

175 hibition promotes growth of cells reliant on eating extracellular protein.

176 The most prevalent subtype was compulsive eating, followed by punding, compulsive sexual behaviour

177 that perceived hunger, importance of eating, eating frequencies, and liking ratings of food pictures

178 gut microbes and eating behaviors, including eating frequency, early energy consumption, and overnigh

179 Eating frequency, percentage of energy consumed before 1

180 time of day or behavioral factors, including eating frequency, percentage of energy consumed early in

181 he felt abdominal pain and nausea only after eating fruit, along with the albedo, of Citrus unshiu.

182 rences in morphology and diet of the termite-eating gecko Gymnodactylus amarali between five such new

183 marshes experiencing a severe drought, plant-eating grazers eliminated drought-stressed vegetation th

184 variance in concordance with the WHO healthy eating guidelines that aim to prevent non-communicable d

185 This may be because of barriers to healthy eating hampering adherence, but whether these barriers h

186 Picky eating has been associated with lower weight status and

187 pically focused on the long-term benefits of eating healthy food.

188 sider how to reduce short-term insecurity by eating healthy food.

189 tion of causality between BMI and disordered eating in childhood, adolescence, and adulthood.MR analy

190 We assessed binge eating in closely related C57BL/6 mouse substrains and i

191 2 as a major genetic factor underlying binge eating in heterozygous knockout mice on a C57BL/6N backg

192 sults show that RO5256390 blocked binge-like eating in rats responding 1 h per day for a highly palat

193 valence of self-reported barriers to healthy eating in Switzerland overall and according to sex, age,

194 y dieting, the experiences of out-of-control eating, increased appetite, and increased fat storage ar

195 ting performance as measured by the Level of Eating Independence scale (range: 15-36, with higher sco

196 anean diet (aMED), and the Alternate Healthy Eating Index (aHEI) diet-quality scores with cardiometab

197 (HEI)-2005, HEI-2010, and Alternate Healthy Eating Index (AHEI)-2010-in relation to ovarian cancer r

198 ation of 4 diet quality indexes [the Healthy Eating Index (HEI) 2010, the Alternate Mediterranean Die

199 uated 3 index-based dietary patterns-Healthy Eating Index (HEI)-2005, HEI-2010, and Alternate Healthy

200 Diet quality index scores on Healthy Eating Index 2010 (HEI-2010), Alternative HEI-2010, alte

201 ity was measured with the use of the Healthy Eating Index 2010 (HEI-2010).

202 dex 2010 [HEI-2010], the Alternative Healthy Eating Index 2010 [AHEI-2010], the alternate Mediterrane

203 sociations between 4 DQI scores (the Healthy Eating Index 2010 [HEI-2010], the Alternative Healthy Ea

204 west tertile of modified Alternative Healthy Eating Index [mAHEI]; 23.2%, 18.2-28.9 for lowest two te

205 trients, dietary patterns (Alternate Healthy Eating Index and Alternate Mediterranean Diet Score), an

206 ciation of 3 diet-quality indicators-Healthy Eating Index-2010 (HEI), Whole Plant Foods Density (WPF)

207 micronutrients, 2) dietary quality [Healthy Eating Index-2010 (HEI)], and 3) beverage categories.Par

208 Binge eating is a highly heritable trait associated with eatin

209 Eating is a learned process.

210 light novel mechanisms by which disinhibited eating leads to obesity and insulin resistance.

211 owed flexibility in foraging patterns, binge-eating less and using feeders more when they experienced

212 teeth to assess whether these notorious man-eating lions scavenged carcasses during their depredatio

213 ng meal significantly modified the effect of eating litchis on illness (odds ratio [OR] 7.8 [95% CI 3

214 browsers for three species (brown kiwi, crab-eating macaque and Malayan flying lemur); eight updated

215 nonhuman primate cell cultures and then crab-eating macaques with either simian hemorrhagic fever vir

216 ting disease in experimentally infected crab-eating macaques, while simian hemorrhagic fever virus (S

217 nergy intake was measured with the universal eating monitor in a test meal.In mice, AITC administrati

218 lancing selection on at least one case (Crab-eating monkey retrocopy 6, or CER6) in both species.

219 However, EI at one eating occasion (EO) is also dependent on EI at surround

220 ful attention to the timing and frequency of eating occasions could lead to healthier lifestyle and c

221 e propose definitions for meals, snacks, and eating occasions for use in research.

222 ent fasting, meal frequency (number of daily eating occasions), and timing of eating occasions.

223 habits, including the number and quality of eating occasions, are potential targets for primary prev

224 er of daily eating occasions), and timing of eating occasions.

225 Compulsive, binge eating of highly palatable food constitutes a core featu

226 earch is needed to facilitate the liking and eating of these foods by mothers, which will, in turn, i

227 auty differs from sensuous pleasures such as eating or sex.

228 established neurotoxicant of concern to fish-eating organisms.

229 Consumption of chicken meat and eating out were the most important risk factors for Camp

230 his was accompanied by increased light phase eating, particularly increased meal size.

231 tion trial findings suggested that a low-fat eating pattern may reduce breast cancers with greater mo

232 s of wakefulness and ingestion events, daily eating pattern offers a new potential target for lifesty

233 I.This study found that a "grazing" temporal eating pattern was modestly but significantly associated

234 isreporting on the relation between temporal eating patterns and adiposity.

235 Finally, data suggest that irregular eating patterns appear less favorable for achieving a he

236 Eating patterns are increasingly varied.

237 BACKGROUND & AIMS: Healthy eating patterns assessed by diet quality indexes (DQIs)

238 o estimate the effect of the same disordered eating patterns at age 13 y on BMI at age 17 y via a spl

239 ausal effect of BMI at age 7 y on disordered eating patterns at age 13 y with the use of data from th

240 ty, the least fattening of widely recognised eating patterns needs to be measured and supported.

241 nts and proportions of macronutrients across eating patterns on meal or dietary glycemic index (GI) a

242 of evidence also support the hypothesis that eating patterns that reduce or eliminate nighttime eatin

243 Temporal eating patterns, based on the distribution of EOs across

244 odels assessed associations between temporal eating patterns, nutrient intakes, diet quality, and adi

245 was to examine associations between temporal eating patterns, nutrient intakes, diet quality, and mea

246 ary GI and GL values that are calculated for eating patterns, which may alter the interpretation of t

247 with increased risk of developing disordered eating patterns.

248 tion of average dietary GI and GL values for eating patterns.

249 e cardiometabolic health effects of specific eating patterns: skipping breakfast, intermittent fastin

250 participants demonstrated moderate levels of eating performance (M=27.08, SD=5.16).

251 iation between environmental stimulation and eating performance among nursing home residents with dem

252 offered to a resident contributed to better eating performance among residents with dementia.

253 were used to examine the association between eating performance and environmental stimulation, adjust

254 The dependent variable was eating performance as measured by the Level of Eating In

255 onmental stimulation influences individuals' eating performance at mealtimes.

256 ia experience increased risk for compromised eating performance due to intrapersonal, interpersonal,

257 1 point increase in stimulation specificity, eating performance increased by 8.78 points (95% CI=0.59

258 er controlling for resident characteristics, eating performance was significantly associated with sti

259 Eating performance was significantly lower among older r

260 15-36, with higher scores indicating better eating performance).

261 a (1.9% versus 0.7%, P < 0.001) and in those eating pork liver sausage (1.4% versus 0.7%, P < 0.01),

262 sociated with increasing age (P < 0.001) and eating pork meat (P = 0.03), pork liver sausages (P < 0.

263 Physical functioning (PF; QLQ-C30) and eating problems (EA; QLQ-OES24) were chosen as predefine

264 s, which could inform and prevent disordered eating problems in adolescence.

265 fter autophagy) is a catabolic cellular self-eating process by which unwanted organelles or proteins

266 cal properties of swede and indicated a good eating quality of swedes grown at low temperatures.

267 ich may be of importance for the storage and eating quality of the fruit.

268 cales), eating behavior traits (Three-Factor Eating Questionnaire), and EI (food buffet or menu).Fat-

269 uinoa could also be detected in plasma after eating quinoa, indicating that some of these unique AR c

270 (P = 0.002) were associated with the initial eating rate (adjusted R(2) = 0.23).

271 ntake using 3 key parameters: 1) the initial eating rate, 2) the effective duration of eating, and 3)

272 ers were estimated in a study (n = 49) where eating rates were deliberately changed.

273 Diet quality estimated adherence to healthy eating recommendations and was assessed by using the 201

274 If proven to be efficacious, these eating regimens offer promising nonpharmacological appro

275 Risk of binge-eating relapse over 6 months was lower in participants c

276 days/week from baseline and improving binge eating-related key secondary endpoints.

277 e.Between 1997 and 2012, barriers to healthy eating remained highly prevalent (>/=20%) in the Swiss p

278 ade endocannabinoid signaling, whereas binge eating resulted in the downregulation of a gene set enri

279 Such eating styles can have various effects on cardiometaboli

280 d that parenting practices have an impact on eating styles which determine obesity levels in children

281 y to perform lignocellulose digestion ('wood eating'), suggesting this ability may predate the divers

282 diet (HFD)-induced obesity increase daytime eating, suggesting an alteration in circadian food intak

283 inform molecular mechanisms mediating binge eating susceptibility and establishment.

284 d with early satiety (P = 0.0002, R = 0.74), eating symptoms (P = 0.007, R = 0.54), and trouble enjoy

285 rends in prevalence of 6 barriers to healthy eating (taste, price, daily habits, time, lack of willpo

286 acial expressions of distaste initially when eating the carrot cereal.

287 s decreased in LCR rats after only 3 days of eating the HFD.

288 need to improve understanding of compulsive eating through the integration of complex motivational,

289 Using clock hour to document eating times may be misleading owing to individual diffe

290 57BL/6N background that showed reduced binge eating toward a wild-type C57BL/6J-like level.

291 The eating behaviors-emotional eating, uncontrolled eating, and cognitive restraint-wer

292 Large, fruit-eating vertebrates have been lost from many of the world

293 Man-eating was likely a viable alternative to hunting and/or

294 ing is a prominent policy to promote healthy eating.We aimed to evaluate the effects of 2 interpretiv

295 R1 may have a role in compulsive, binge-like eating; we tested this hypothesis by assessing the effec

296 stoperatively started to self-weigh, stopped eating when feeling full, and stopped eating continuousl

297 ore, RO5256390 fully blocked compulsive-like eating when the palatable diet was offered in an aversiv

298 The neuronal substrate for binge eating, which can at times lead to obesity, is not clear

299 nges in appetite that result in compensatory eating, which may initially dissuade potential success i

300 Intentional eating with mindful attention to the timing and frequenc