ライフサイエンスコーパス: eating disorder

1 limia nervosa, and adding BED as a specified eating disorder.

2 l diagnosis when evaluating a patient for an eating disorder.

3 and nutritional complaints suggestive of an eating disorder.

4 ion towards pharmacologically treating binge eating disorder.

5 cacy in adults with moderate to severe binge-eating disorder.

6 piramate reduced weight in adults with binge-eating disorder.

7 patients-in particular, depression and binge eating disorder.

8 racteristic symptomatology observed in binge-eating disorder.

9 otential pharmacological treatment for binge-eating disorder.

10 pect of serious clinical conditions, such as eating disorders.

11 ent strategies tailored for older women with eating disorders.

12 adolescent girls at high risk of obesity and eating disorders.

13 osis, and treatment for males suffering from eating disorders.

14 armacological treatment for bingeing-related eating disorders.

15 s significant to the cause and expression of eating disorders.

16 ic disorders, alcohol or substance abuse, or eating disorders.

17 e related to food avoidance commonly seen in eating disorders.

18 al (GI) symptoms are common in patients with eating disorders.

19 elopment of compulsive eating in obesity and eating disorders.

20 gical findings on the cause and treatment of eating disorders.

21 ent evidence on psychological treatments for eating disorders.

22 cess to care, thereby reducing the burden of eating disorders.

23 ion and low interoceptive awareness, such as eating disorders.

24 nd treatment of both compulsive behavior and eating disorders.

25 the upper quintile were classified as having eating disorders.

26 a notoriously difficult-to-treat symptom of eating disorders.

27 tality in a long-term study of patients with eating disorders.

28 ent implications of cognitive flexibility in eating disorders.

29 pears to be dysregulated in individuals with eating disorders.

30 at reported mortality rates in patients with eating disorders.

31 on contributes to failed diets, obesity, and eating disorders.

32 fect-driven feeding and loss of restraint in eating disorders.

33 g of these signaling molecules may result in eating disorders.

34 or antisocial, addictive, mood, anxiety, and eating disorders.

35 circuits might participate in obesity or in eating disorders.

36 tive disorders, and 3.5 (95% CI=1.6-7.3) for eating disorders.

37 nding of the etiology and treatment of binge eating disorders.

38 contribute to the recent rise of obesity and eating disorders.

39 rgets for the treatment of cachexia or other eating disorders.

40 control systems in the pathogenesis of these eating disorders.

41 cteristics and risk factors of patients with eating disorders.

42 tice guideline for patients with restrictive eating disorders.

43 major influence on the later development of eating disorders.

44 tric conditions such as forms of obesity and eating disorders.

45 of therapeutic targets for the treatment of eating disorders.

46 cide attempts are common in individuals with eating disorders.

47 a potential therapeutic for alcohol use and eating disorders.

48 y, obsessive compulsive disorder, autism and eating disorders.

49 e after adjusting for the index individuals' eating disorders.

50 t deposition may be particularly relevant to eating disorders.

51 0 million of people in the world suffer from eating disorders.

52 its associated with addiction and binge-type eating disorders.

53 t are associated with increased incidence of eating disorders.

54 d-habit system, which could differ among the eating disorders.

55 ising therapeutic strategies for obesity and eating disorders.

56 ned more sex-neutral diagnostic criteria for eating disorders.

57 dition (2.3%; 95% CI, 1.7% to 2.9%), and any eating disorder (0%).

58 For eating disorders, 0.8% had partial- or full-criteria bul

59 depression (19% [95% CI, 14%-25%]) and binge eating disorder (17% [95% CI, 13%-21%]).

60 ) and 991 males (0.09% of all males) had any eating disorder, 7680 females (0.70%) and 453 males (0.0

61 And with regards to eating disorders, a closer examination of both full diag

62 e of the shared risk factors for obesity and eating disorders, a targeted prevention of both conditio

63 culture in both the emergence and spread of eating disorders across the globe.

64 porting pharmacological treatments for binge eating disorder, advances in treatment for adults have b

65 the prevalence of concerns with physique and eating disorders among males and their relation to subse

66 5 y of follow-up, 134 individuals with binge-eating disorder and 134 individuals with no history of e

67 ss longitudinally the relation between binge-eating disorder and components of the metabolic syndrome

68 mia nervosa and more likely to exhibit binge eating disorder and eating disorder not otherwise specif

69 and obesity, emphasizing binge eating, binge eating disorder and food addiction as useful conceptual

70 ondary outcomes included OBEs at follow-ups, eating disorder and general psychopathologic findings, b

71 ted in a specialist inpatient unit and their eating disorder and psychological distress features.

72 tory processes may contribute to symptoms in eating disorders and addictive disorders, but little is

73 PT may also be effective in the treatment of eating disorders and anxiety disorders and has shown pro

74 d highly idealized images has been linked to eating disorders and body image dissatisfaction in men,

75 We examined the association between eating disorders and death by suicide separately, but th

76 velop much needed new therapeutics to tackle eating disorders and obesity.

77 evelop better pharmacological treatments for eating disorders and obesity.

78 havior may inspire new treatment options for eating disorders and obesity.

79 iology, features, and potential treatment of eating disorders and related body-image concerns in midd

80 not surprising that the comorbidity between eating disorders and substance abuse disorders is high.

81 whether a shared neurobiology contributes to eating disorders and substance abuse, this review focuse

82 Prevalence and correlates of eating disorders and subthreshold conditions.

83 Eating disorders and subthreshold eating conditions are

84 l cortex, insula, and striatum is altered in eating disorders and suggests altered brain circuitry th

85 milial liability for the association between eating disorders and suicide.

86 uicide attempts in individuals with lifetime eating disorders and their relatives.

87 re: anorexia nervosa, bulimia nervosa, binge eating disorder, and eating disorder not otherwise speci

88 sociated with posttraumatic stress disorder, eating disorders, and anxiety disorders other than speci

89 me, obsessive-compulsive spectrum disorders, eating disorders, and autism.

90 e is the most common period for the onset of eating disorders, and early intervention is critical.

91 role, such as mood, substance-use disorders, eating disorders, and obesity.

92 uding post-traumatic stress disorder [PTSD], eating disorders, and psychoses).

93 disorders were captured by 3 variables (any eating disorder, anorexia nervosa, and bulimia nervosa)

94 rity of individuals seeking treatment for an eating disorder are classified as eating disorder not ot

95 The best treatment options for binge-eating disorder are unclear.

96 Anorexia nervosa (AN) and related eating disorders are complex, multifactorial neuropsychi

97 Incidence rates of eating disorders are higher in schools characterised by

98 and mental disorders, but findings regarding eating disorders are inconsistent and inconclusive.

99 strialized Western Europe and North America, eating disorders are increasingly documented in diverse

100 Eating disorders are lethal and heritable; however, the

101 review first identifies diseases with which eating disorders are often confused and then explores fe

102 Eating disorders are serious psychiatric conditions requ

103 Eating disorders are severe conditions, but little is kn

104 that pathological eating behaviors and frank eating disorders are surprisingly common in older women,

105 rbidity and mortality rates in patients with eating disorders are thought to be high, but exact rates

106 bulimia nervosa; and the inclusion of binge eating disorder as a formal diagnosis.

107 esearch also supports the inclusion of binge eating disorder as a formal diagnosis.

108 a core feature of some forms of obesity and eating disorders, as well as of the recently proposed di

109 or anxiety, somatoform, neurocognitive, and eating disorders, as well as prolonged grief and autism.

110 Outcome was any diagnosed eating disorder at 16-20 years, as defined by an ICD (9

111 n, attention-deficit/hyperactivity disorder, eating disorders, autism spectrum disorder, substance us

112 rls deemed at high risk of adult obesity and eating disorders because of a body mass index (BMI) betw

113 Binge-eating disorder (BED) is characterized by recurring epis

114 ey, the average lifetime prevalence of binge eating disorder (BED) was 2%.

115 etamine dimesylate (LDX) vs placebo in binge eating disorder (BED) was evaluated in two multicenter,

116 bese individuals seeking treatment for binge eating disorder (BED) were compared with 19 non-BED obes

117 cts with (n = 30) and without (n = 30) binge eating disorder (BED) were compared with matched healthy

118 Binge-eating disorder (BED), a public health problem associate

119 he criterion standard for treatment of binge eating disorder (BED), most individuals do not have acce

120 ood, in alcohol use disorders (AUD) or binge-eating disorder (BED), suggest a disturbance in explore-

121 otypes: pathological gambling (PG) and binge eating disorder (BED).

122 e United States on the epidemiology of binge eating disorder (BED).

123 s an effective specialty treatment for binge eating disorder (BED).

124 Finally, some eating disordered behaviors, which have until now receiv

125 OGCT malignancies, anxiety, hearing loss, or eating disorders between groups.

126 A 4.4% variation in incidence of eating disorders between schools was seen; after taking

127 and 2.9% had partial or full-criteria binge eating disorder but no association with the outcomes of

128 disinhibition are observed in patients with eating disorders, but neural correlates of inhibitory co

129 IPT had significant effects on eating disorders, but the effects are probably slightly

130 The changing face of eating disorders calls for a new conceptualization of cu

131 Eating disorders can be associated with profound and pro

132 ng disorders that appear in the 'Feeding and Eating Disorders' chapter of the Diagnostic and Statisti

133 exia nervosa (AN) is a complex and heritable eating disorder characterized by dangerously low body we

134 Anorexia nervosa (AN) is an eating disorder characterized by extreme hypophagia, hyp

135 Anorexia nervosa (AN) is a serious eating disorder characterized by self-starvation and ext

136 Anorexia nervosa (AN) is a serious eating disorder characterized by self-starvation, extrem

137 inferred from an appointment at a specialist eating disorder clinic.

138 who presented for treatment at a specialized eating disorders clinic in an academic medical center.

139 ention deficit hyperactivity disorder, binge eating disorder, cocaine addiction, obesity, and type 2

140 isorders, including gambling disorder, binge eating disorder, compulsive sexual behaviour, and compul

141 Eligible adults met DSM-IV-R binge-eating disorder criteria and had moderate to severe bing

142 numerous clinical conditions such as autism, eating disorders, depression, and chronic pain.

143 nstrated an increased risk of suicide across eating disorder diagnoses.

144 Treatments have been evaluated within each eating disorder diagnosis and across diagnoses.

145 pecified' (EDNOS) was the most common DSM-IV eating disorder diagnosis in both clinical and community

146 , 1,019 males and 15,395 females received an eating disorder diagnosis.

147 he recent publication of revised feeding and eating disorder diagnostic criteria in DSM-5.

148 e, 6 wk, 6 mo, 12 mo, and 24 mo by using the Eating Disorder Diagnostic Interview, the Multidimension

149 lar affective disorder, depressive disorder, eating disorder, drug dependency, or alcohol dependency,

150 d and motivation, ranging from depression to eating disorders, drug addiction, and related compulsive

151 To summarize the recent literature examining eating disorders, eating behavior, and body image in mid

152 People with eating disorders (ED) frequently present with inflexible

153 have been widely identified in persons with eating disorders (EDs) and have been implicated in their

154 Eating disorders (EDs) are severe, life-threatening ment

155 While eating disorders (EDs) are thought to result from a comb

156 direction of the association between BMI and eating disorders (EDs) in adults via a two-sample MR app

157 sa and bulimia nervosa are common and severe eating disorders (EDs) of unknown etiology.

158 atus (SES) is associated with higher risk of eating disorders (EDs).

159 s have confirmed that the DSM-5 criteria for eating disorders effectively reduce the proportion of ED

160 characteristics are associated with rates of eating disorders, even after accounting for characterist

161 The primary outcome measure was the Eating Disorder Examination (EDE) Global score.

162 Self-esteem (P < .05) and global Eating Disorder Examination (P < .05) scores were modera

163 re and after weight restoration by using the Eating Disorder Examination interview and the Global Sev

164 The main outcome measure was the Eating Disorder Examination interview, which was adminis

165 ticipants in FBT also had greater changes in Eating Disorder Examination score at EOT than those in A

166 The odds ratios for low and high global Eating Disorder Examination scores were 2.8 for BWL, 2.9

167 Binge eating as measured by the Eating Disorder Examination was assessed at baseline, at

168 asures were bulimic symptoms assessed by the Eating Disorder Examination-Questionnaire.

169 personality disorders (factors 2 and 4), and eating disorders (factors 1 and 4).

170 both treatments, substantial improvements in eating disorder features and general psychopathology wer

171 rapy and was generally faster in alleviating eating disorder features and general psychopathology.

172 f studies about psychotherapy approaches for eating disorders focus on adult women, there is a growin

173 This issue provides a clinical overview of eating disorders focusing on prevention, diagnosis, trea

174 tion of both full diagnostic and less common eating disorders following bariatric surgery would be pr

175 irls than boys had an increased incidence of eating disorders: for each 10% increase in the proportio

176 The eating disorder groups also showed reduced white matter

177 All eating disorder groups exhibited increased gray matter v

178 predicted sensitivity to reward in all three eating disorder groups.

179 er criteria and had moderate to severe binge eating disorder (>/=3 binge-eating days per week for 14

180 Individuals with any eating disorder had an increased risk (reported as odds

181 uals (index) who had a full sibling with any eating disorder had an increased risk of suicide attempt

182 which is sometimes viewed as a "less severe" eating disorder, had elevated mortality risks, similar t

183 he effectiveness of family interventions for eating disorders has increased over the past 5 years.

184 The conceptual framework of the cause of eating disorders has undergone great changes in the past

185 The causal factors underpinning eating disorders have been clarified by understanding ab

186 rweight and higher BMIs at various stages of eating disorders have been confirmed repeatedly.

187 aracteristics, assessment, and mortality for eating disorders have been reported independently for ma

188 Individuals with eating disorders have significantly elevated mortality r

189 ison of individuals with and without a binge-eating disorder in analyses adjusted for age, sex, basel

190 The risk was attenuated for any eating disorder in more-distant relatives (maternal half

191 findings support a distinct subtype of binge eating disorder in obesity with similarities in risk-tak

192 We aimed to investigate whether rates of eating disorders in 16-20-year-old girls vary between up

193 out the epidemiology, course, and outcome of eating disorders in accordance with the fifth edition of

194 a risk factor for both increased weight and eating disorders in adolescents.

195 orthern European groups; increasing rates of eating disorders in Asia; increasing rates of eating dis

196 ce base for the use of family treatments for eating disorders in children and adolescents.

197 The area of feeding and eating disorders in children remains relatively under-re

198 in journal articles relating to feeding and eating disorders in children, making a succinct overview

199 ure about recent trends in the occurrence of eating disorders in different cultures.

200 Research in eating disorders in males has been active lately compare

201 arental education showed no association with eating disorders in males, but twin or triplet status an

202 verview of the recently published studies of eating disorders in males.

203 hasize the need for further investigation of eating disorders in military service members.

204 herapy approaches recommended for teens with eating disorders in order to effectively refer patients

205 ating disorders in Asia; increasing rates of eating disorders in the Arab region; and increasing rate

206 high prevalence and incidence of obesity and eating disorders in US adolescent girls are serious heal

207 that most commonly mimic the presentation of eating disorders including Crohn disease (CrD), celiac d

208 disease-specific roles in the development of eating disorders, including via perinatal variation with

209 The Internet and Binge Eating Disorder (INTERBED) study is a prospective, multi

210 Binge eating disorder is an addiction-like disorder characteri

211 Binge-eating disorder is characterized by excessive, uncontrol

212 nderstanding the underpinning biology of the eating disorder is important, as well as potential co-oc

213 mproved understanding of the neural basis of eating disorders is a timely challenge because these dis

214 ence; the present lifetime prevalence of all eating disorders is about 5%.

215 of the timing and predictors of mortality in eating disorders is limited.

216 but whether this is true for other types of eating disorders is unclear.

217 oup cognitive-behavioral treatment for binge eating disorder led to higher binge eating abstinence ra

218 ng disorder symptom data collected using the Eating Disorder Longitudinal Interval Follow-Up Examinat

219 ted of 5 factors: internalizing (anxiety and eating disorders, major depressive episode, and cluster

220 Binge-eating disorder may confer a risk of components of the m

221 Binge-eating disorder may represent a risk factor for the meta

222 Clinically diagnosed eating disorders may have adverse cardiometabolic conseq

223 A broader range of eating disorders needs to be defined to diagnose these i

224 nervosa (N=177), bulimia nervosa (N=906), or eating disorder not otherwise specified (N=802) who pres

225 ether anorexia nervosa, bulimia nervosa, and eating disorder not otherwise specified are associated w

226 ent for an eating disorder are classified as eating disorder not otherwise specified based on DSM-IV

227 m for anorexia nervosa, bulimia nervosa, and eating disorder not otherwise specified until the end of

228 The residual category 'eating disorder not otherwise specified' (EDNOS) was the

229 Individuals with eating disorder not otherwise specified, which is someti

230 gned to the heterogeneous residual category, eating disorder not otherwise specified, which provides

231 likely to exhibit binge eating disorder and eating disorder not otherwise specified.

232 vosa, 3.9% for bulimia nervosa, and 5.2% for eating disorder not otherwise specified.

233 ratios were elevated for bulimia nervosa and eating disorder not otherwise specified.

234 bulimia nervosa, binge eating disorder, and eating disorder not otherwise specified.

235 gnificantly elevated for bulimia nervosa and eating disorder not otherwise specified; suicide standar

236 considerable potential for the treatment of eating disorders, obesity, and/or diabetes.

237 xt Revision (DSM-IV-TR) section 'Feeding and Eating Disorders of Infancy or Early Childhood'; clarifi

238 tion to maladaptive feeding behavior seen in eating disorders or obesity may arise from dysregulation

239 No studies were identified on eating disorders or puerperal psychosis.

240 ether these GI symptom factors (clusters) in eating disorder patients hold true to the Rome II classi

241 The GI symptoms common in eating disorder patients very likely represent the same

242 onal constipation, however, are prominent in eating disorder patients.

243 in high status clothes) or non-aspirational (eating disordered patients in grey leotards), or to comb

244 ers, schizophrenia, mood disorders, anxiety, eating disorders, personality disorders, mental retardat

245 he number of OBE days, abstinence rates, and eating disorder psychopathologic findings and may be a b

246 Reductions in eating disorder psychopathologic findings were significa

247 Eating disorder psychopathology and psychological distre

248 vosa (AN), its influence in women with AN on eating disorder psychopathology and psychological distre

249 shown between body-composition variables and eating disorder psychopathology in the AN group, and the

250 does not, however, seem to influence either eating disorder psychopathology or psychological distres

251 d for patients with low self-esteem and high eating disorder psychopathology.

252 ar adds to the previous Lancet Seminar about eating disorders, published in 2003, with an emphasis on

253 The changes to eating disorders, recommended by the Eating Disorders Wo

254 However, eating disorders remain understudied in military samples

255 Binge eating disorder represents a public health problem at le

256 ered eating and body image, older women with eating disorder resemble younger women with similar cond

257 related and addictive disorders, feeding and eating disorders, schizophrenia, anxiety disorder, OCD,

258 th conditions, including substance abuse and eating disorders, seem to be exacerbated or triggered in

259 anticipation of rewards, subjects with binge eating disorder show greater risk-taking, similar to sub

260 Although the majority of adolescents with an eating disorder sought some form of treatment, only a mi

261 c, stress-related, and somatoform disorders; eating disorders; specific personality disorders; and a

262 eflect the changing landscape of culture and eating disorders: stabilization of the incidence of anor

263 Important differences were observed between eating disorder subtypes concerning sociodemographic cor

264 reliminary evidence that during adolescence, eating disorder subtypes may be distinguishable in terms

265 These results may have implications for eating disorders such as anorexia nervosa (AN), in which

266 as a therapeutic target for the treatment of eating disorders such as anorexia nervosa.

267 ia nervosa were followed for 7 years; weekly eating disorder symptom data collected using the Eating

268 Yet, males tended to score lower on eating disorder symptom measures than females.

269 cues) rBN subjects had a greater increase in eating disorder symptoms during CD compared with healthy

270 associated with vulnerability for developing eating disorder symptoms in response to reduced catechol

271 reatment groups for the primary outcome, for eating disorder symptoms or comorbid psychiatric disorde

272 ry to earlier suggestions, no differences in eating disorder symptoms such as binging, vomiting, or l

273 ian of 9.5 years to obtain weekly ratings of eating disorder symptoms, comorbidity, treatment partici

274 This article reviews the modifications to eating disorders that appear in the 'Feeding and Eating

275 is a highly heritable trait associated with eating disorders that is comorbid with mood and substanc

276 ietary habits during pregnancy of women with eating disorders that may lie in the causal pathway of a

277 ith a lack of control over eating, and binge eating disorder, the Diagnostic and Statistical Manual-5

278 o compare three types of treatment for binge eating disorder to determine the relative efficacy of se

279 of topics relevant to childhood feeding and eating disorders, to include: presentation, diagnosis an

280 However, widespread access to effective eating disorder treatments remains limited.

281 Inpatients in a specialised eating disorder unit completed the Rome II questionnaire

282 first study to investigate whether rates of eating disorders vary between schools; however, use of r

283 Clinical impression is that rates of eating disorders vary between schools; we are not aware

284 Neither height nor history of an eating disorder was associated with menopausal age.

285 Neither depression nor binge eating disorder was consistently associated with differe

286 ion of girls at a school, the odds ratio for eating disorders was 1.07 (95% CI 1.01-1.13, p=0.017).

287 post-secondary education, the odds ratio for eating disorders was 1.14 (1.09-1.19, p<0.0001).

288 The fourth pattern, seen in eating disorders, was directly opposite of that observed

289 anorexia nervosa, bulimia nervosa, and binge-eating disorder were 0.3%, 0.9%, and 1.6%, respectively.

290 A total of 259 adults diagnosed with binge eating disorder were randomly assigned to 20 weeks of th

291 motor-disturbances, night-time behavior and eating disorders were also worse in the institutionalize

292 Eating disorders were captured by 3 variables (any eatin

293 While historically, eating disorders were conceptualized as primarily afflic

294 disorder, bipolar disorder, or psychotic or eating disorders were excluded.

295 ly controlled research of brain structure in eating disorders, which will ultimately help predict the

296 order and 134 individuals with no history of eating disorders, who were frequency-matched for age, se

297 pped to discuss psychotherapy approaches for eating disorders with adolescents and their families and

298 uding drug abuse, behavioral addictions, and eating disorders with binge features.

299 hed treatments for bulimia nervosa and binge eating disorder, with stepped-care approaches showing pr

300 nges to eating disorders, recommended by the Eating Disorders Work Group, aim to clarify existing cri

301 l and social/interpersonal issues underlying eating disorders would increase treatment efficacy.