ライフサイエンスコーパス: eccrine

1 he epidermal basal layer, to hair follicles, eccrine and sebaceous glands as well as to endothelium o

2 ajor sweat-producing glands present in skin; eccrine, apocrine, and apoeccrine glands.

3 In humans, eccrine appendages express DCD and CAMP, genes encoding

4 ratinocytes, melanocytes, mononuclear cells, eccrine ducts, and nerves.

5 epidermal keratinocytes (four of five), and eccrine epithelia (two of four).

6 LL-37 was localized to both the eccrine gland and sweat ductal epithelial cells, whereas

7 use Chromosome 1 that controls both hair and eccrine gland densities.

8 reciprocal determinant of hair follicle and eccrine gland density and identify a pathway that could

9 lthough reciprocal changes in hair cover and eccrine gland density are required for efficient thermor

10 confounded by a lack of knowledge about how eccrine gland fate and density are specified during deve

11 The eccrine gland is one of the major cutaneous appendages a

12 hair follicle, in myoepithelial cells of the eccrine gland, and in the basement membrane/basal lamina

13 including the dental lamina, mammary gland, eccrine gland, and primary and secondary HFs.

14 hways controlling the relative patterning of eccrine glands and hair follicles, we exploited natural

15 En1 activity directs the relative numbers of eccrine glands and hair follicles.

16 ssociated with an increased number of active eccrine glands in the Han Chinese.

17 ent study was designed to assess the role of eccrine glands in the repair of wounded human skin.

18 sarcoma (KS), and in the epithelial cells of eccrine glands within KS lesions.

19 d appendages, including the sebaceous gland, eccrine glands, and apocrine glands, as well as low leve

20 in 5 receptor localized to sebaceous glands, eccrine glands, hair follicles, and epidermis in human s

21 man and mouse epidermis, hair follicles, and eccrine glands, indicating that these channels are found

22 ected by the mutation, including mammary and eccrine glands.

23 secretory cells of sebaceous, apocrine, and eccrine glands.

24 observed in secretory cells of apocrine and eccrine glands.

25 that were present in affected porokeratotic eccrine ostial and dermal duct nevus (PEODDN) tissue but

26 Both passive (e.g., eccrine/sebaceous secretions, proteolytic) and active (e

27 LL-37 protein and mRNA was seen in the eccrine structures of normal human skin by immunohistoch

28 is transport regulator as densely as did the eccrine sweat gland when three monoclonal antibodies for

29 in receptors are also expressed on the human eccrine sweat gland, although it remains unclear whether

30 nsport in the reabsorptive duct of the human eccrine sweat gland, which most likely represents a sodi

31 y isolated human epidermal fragments and the eccrine sweat glands amplified the cystic fibrosis trans

32 Eccrine sweat glands are essential for sweating and ther

33 Eccrine sweat glands are skin-associated epithelial stru

34 ths that ultimately form new epidermis; (ii) eccrine sweat glands are the most abundant appendages in

35 vel appreciation of the unique importance of eccrine sweat glands for epidermal repair may be exploit

36 ated with mixed tumor formation arising from eccrine sweat glands found only in the foot pads of mice

37 More specifically, (i) eccrine sweat glands generate keratinocyte outgrowths th

38 The gain of eccrine sweat glands in hairy body skin has empowered hu

39 Our data demonstrate a key role for eccrine sweat glands in reconstituting the epidermis aft

40 sured in isolated nonperfused ducts of human eccrine sweat glands in vitro to investigate basolateral

41 of expansion of keratinocyte outgrowths from eccrine sweat glands parallels the rate of reepitheliali

42 increase in the density and distribution of eccrine sweat glands relative to other mammals and a con

43 es and is characterized by reduced or absent eccrine sweat glands, hair follicles and teeth, and defe

44 ), a congenital disorder of teeth, hair, and eccrine sweat glands, is usually inherited as an X-linke

45 by defective development of hair, teeth, and eccrine sweat glands, is usually inherited as an X-linke

46 well as in a subset of cells in the bone and eccrine sweat glands.

47 lated anhidrosis, but morphologically normal eccrine sweat glands.

48 dition characterised by over-activity of the eccrine sweat glands.

49 in abnormal morphogenesis of teeth, hair and eccrine sweat glands.

50 the abnormal development of teeth, hair, and eccrine sweat glands.

51 the features of hypoplastic hair, teeth, and eccrine sweat glands.

52 und, and in the duct and proximal tubules of eccrine sweat glands.

53 r cells of the sweat glands is important for eccrine sweat production.

54 Due to the high rate of secretion, eccrine sweating is a vital regulator of body temperatur