ライフサイエンスコーパス: echocardiogram

1 n evidence-based medical therapies (baseline echocardiogram).

2 monary artery systolic pressure >40 mm Hg on echocardiogram).

3 rison group) was assessed with transthoracic echocardiogram.

4 rdiogram 30 days to 2 years from the initial echocardiogram.

5 e incremental prognostic value of a complete echocardiogram.

6 subjects would test positive and require an echocardiogram.

7 and 1 had left ventricular noncompaction on echocardiogram.

8 ll thickness and LVOTG were measured with an echocardiogram.

9 nd to develop a new tool for assessing PR by echocardiogram.

10 ortic valve regurgitation at the time of the echocardiogram.

11 uzumab and had a pre-treatment and follow-up echocardiogram.

12 opment of third-degree CHB detected by fetal echocardiogram.

13 ac vegetations identified by transesophageal echocardiogram.

14 evidence of stenosis or regurgitation on an echocardiogram.

15 did not receive contrast during their stress echocardiogram.

16 ed to become an integral part of the routine echocardiogram.

17 (28%) on 12-month follow-up transesophageal echocardiogram.

18 ) was strongly predictive of a normal stress echocardiogram.

19 association with normal cardiac enzymes and echocardiogram.

20 n relation to changes in LVEF on a follow-up echocardiogram.

21 002 and 2014, including 95 with an available echocardiogram.

22 citation myocardial function was measured by echocardiogram.

23 ocardiographic core laboratory evaluated all echocardiograms.

24 This analysis includes 2037 echocardiograms.

25 cal history, physical examination, ECGs, and echocardiograms.

26 , 130 children (2.7%) had abnormal screening echocardiograms.

27 R was higher (P=0.01) compared with pre-BAVP echocardiograms.

28 on of sinus rhythm was assessed by follow-up echocardiograms.

29 ac amyloidosis who underwent transesophageal echocardiograms.

30 nic minorities) who had technically adequate echocardiograms.

31 ysicians who request and those who interpret echocardiograms.

32 atment and had evaluable baseline and 90-day echocardiograms.

33 ive approach to the interpretation of stress echocardiograms.

34 s and were screened for cardiac function via echocardiograms.

35 ct annual health and safety reviews of these echocardiograms.

36 age, sex, initial LVEF, and interval between echocardiograms.

37 gs who were heterozygous carriers had normal echocardiograms.

38 amyloid extent than in subjects with typical echocardiograms.

39 d LVEF was measured from baseline and 5-year echocardiograms.

40 cardiovascular biomarkers, and transthoracic echocardiograms.

41 t the inclusion criteria, 741 (85%) had both echocardiograms.

42 2 months), and 27% had persistently abnormal echocardiograms.

43 Medicine Fellows performed 154 goal-directed echocardiograms, 110 with complete cardiology-reviewed t

44 -enhanced cardiovascular magnetic resonance, echocardiograms, 24-hour blood pressure monitoring, and

45 Of patients receiving Definity during the echocardiogram, 26 died within 24 h (0.42%).

46 t diagnosis and who had at least 1 follow-up echocardiogram 30 days to 2 years from the initial echoc

47 At the time of the initial echocardiogram, 323 patients (38%) were taking bisphosph

48 %-53.6%), bone density studies (6.3%-20.0%), echocardiograms (5.0%-7.8%), magnetic resonance imaging

49 vere left ventricular dysfunction on initial echocardiogram (80%) and/or the need for intravenous ino

50 Among 971 patients with evaluable echocardiograms (92%), LF (stroke volume index </=35 mL/

51 patients with AA atheroma on transesophageal echocardiogram, AA atheroma progression was associated w

52 fusing capacity, and pericardial effusion on echocardiogram all predicted mortality.

53 ass index and coronary artery calcification; echocardiograms also were obtained in 58 subjects withou

54 nts assessed for transplantation, 739 had an echocardiogram and 217 of 739 (29%) died during a mean f

55 Each patient underwent a comprehensive echocardiogram and a myocardial perfusion scintigraphy (

56 All subjects had an echocardiogram and an exercise ECG performed, followed b

57 A total of 277 preterm infants completed echocardiogram and BPD assessments at 36 weeks PMA.

58 y of Fallot (n=143; 12.5+/-3.2 years) had an echocardiogram and CMR within 3 months of each other.

59 Preprocedure transesophageal echocardiogram and computed tomography/magnetic resonanc

60 ately after a standardized dobutamine stress echocardiogram and decreased after 1 hour.

61 , and a cardiology evaluation which included echocardiogram and electrocardiogram.

62 ction, in addition to simple two-dimensional echocardiogram and radionucleotide angiography, has also

63 n at a referral PH clinic with transthoracic echocardiogram and right heart catheterization within 1

64 zation immediately followed by transthoracic echocardiogram and TAPSE measurement.

65 dex (LAVI) is a predictor of a normal stress echocardiogram and thus a predictor of low ischemic risk

66 Median time difference between goal-directed echocardiogram and transthoracic echocardiography was 21

67 All had unobstructed neo-coronary ostia by echocardiogram and were asymptomatic.

68 ar [LV] DD) with normal systolic function by echocardiogram and without severe mitral or tricuspid in

69 nt's angiographic report, electrocardiogram, echocardiogram and, if available, nuclear stress test.

70 Echocardiograms and 2-dimensional strain analysis were o

71 A total of 984 participants with evaluable echocardiograms and baseline LF AS (LVSVI </=35 mL/m2) w

72 Echocardiograms and blood samples were obtained from 43

73 ir cancer therapy (total of 15 months) using echocardiograms and blood samples.

74 l cavopulmonary anastomosis and had complete echocardiograms and catheterizations within three months

75 this study, 162 subjects with CKD underwent echocardiograms and computed tomography scans to assess

76 Echocardiograms and computed tomography scans were revie

77 Two hundred twenty-five echocardiograms and concomitant high-sensitivity troponi

78 dicine Fellow's performance of goal-directed echocardiograms and intensivists' interpretations for ev

79 Patients had echocardiograms and measures of B-type natriuretic pepti

80 mmunity-based Framingham Heart Study who had echocardiograms and provided DNA samples but did not hav

81 Serial echocardiograms and pulmonary function tests were perfor

82 VAD were prospectively evaluated with serial echocardiograms and right heart catheterizations.

83 Echocardiograms and serial serum measurements of cardiac

84 red in two established technologies: resting echocardiograms and stress tests with nuclear imaging.

85 ients undergoing CRT with available baseline echocardiograms and subsequent clinical and echocardiogr

86 ts who underwent CRT with available baseline echocardiograms and subsequent clinical and echocardiogr

87 Nonetheless, physicians who order echocardiograms and those who provide them must work tog

88 Transesophageal long-axis echocardiograms and ventricular pressure by micromanomet

89 sment of LVM (either by electrocardiogram or echocardiogram), and at follow-up a measurable LVEF.

90 ac vegetations identified by transesophageal echocardiogram, and all underwent percutaneous lead extr

91 physical examination, laboratory evaluation, echocardiogram, and ECG.

92 valuated by 12-lead electrocardiogram (ECG), echocardiogram, and laboratory studies.

93 tiology of MR was determined on preoperative echocardiogram, and patients were stratified into no/mil

94 tolic function was assessed by transthoracic echocardiogram, and systolic dysfunction was defined as

95 Clinical data, transthoracic echocardiograms, and brain imaging of 53 consecutive pat

96 Plasma, echocardiograms, and clinical outcomes were collected at

97 Patients were followed up with serial echocardiograms, and clinical validations were made with

98 Clinical assessment (including vital signs, echocardiograms, and electrocardiographs) and testing of

99 We analysed all CT scans, echocardiograms, and neurological events in a masked fas

100 After the first abnormal echocardiogram, angiotensin-converting enzyme inhibitor

101 The components of a fetal echocardiogram are described in detail, including descri

102 inical entity is discussed, and illustrative echocardiograms are provided.

103 tom questionnaire, physical examination, and echocardiogram as follows: stage 0, healthy; stage A, HF

104 performed and documented their goal-directed echocardiogram as normal or abnormal for right ventricul

105 125 consented to a follow-up transesophageal echocardiogram at 12 months.

106 diography before CRT and underwent follow-up echocardiograms at 1 year.

107 Post-procedural echocardiograms at 48 h, 1 month, and 2 months demonstra

108 Echocardiograms at 7 days of age may be a useful tool to

109 CD rate in 139 participants with quantitated echocardiograms at all time points.

110 Patients were assigned to CRT-D with paired echocardiograms at baseline and at 12 months (n = 752).

111 MADIT-CRT) trial who survived and had paired echocardiograms at enrollment and at 12 months (n=752) w

112 Only those who had follow-up echocardiograms at least a year apart were included.

113 1995 to December 2003 had > or =2 follow-up echocardiograms at our institution and were included.

114 Included patients had echocardiograms at the time of ablation and at 1-year cl

115 Serial echocardiograms (at 1, 2, 3, 4, 6, 9, and 12 months) and

116 Of the 24 patients who had echocardiograms available for reread, there was a respon

117 easible for 90 of the 96 patients (94%) with echocardiograms available.

118 Time from presentation to the echocardiogram before left ventricular fractional shorte

119 ams of consecutive patients who had baseline echocardiograms between January 1, 2005, and December 31

120 t baseline cardiopulmonary exercise testing, echocardiogram, biomarker assessment, and rhythm status

121 Results of audiology testing, echocardiogram, brain magnetic resonance imaging, renal

122 esented to our institution with a diagnostic echocardiogram but a normal ECG.

123 ac vegetations identified on transesophageal echocardiogram can safely undergo complete device extrac

124 Trends regarding use of echocardiograms, changing drug therapy for Marfan syndro

125 f the findings of right heart dysfunction on echocardiograms, computed tomography angiography, or car

126 A comprehensive echocardiogram consisting of 2-dimensional (2D) and 3-di

127 f 45), or mild POPH (n = 11 of 18) or normal echocardiograms (controls, n = 86 of 122) (P = .77).

128 randomized subjects, 624 had paired Doppler echocardiogram data for >/=1 analyses at 6, 12, 18, or 2

129 Five-year echocardiogram data were available for 424 patients afte

130 On the basis of echocardiogram data, pHTN was defined as right ventricul

131 Echocardiogram demonstrated significant functional impro

132 e albumin/creatinine >30 mg/g) and available echocardiogram-derived pulmonary artery systolic pressur

133 ture and measurement of heart function using echocardiogram/Doppler parameters.

134 lide (RN) stress test or a dobutamine stress echocardiogram (DSE).

135 y physical examinations, laboratory results, echocardiograms, electrocardiograms, and in subjects wit

136 ants with birthweights 500-1,250 g underwent echocardiogram evaluations at 7 days of age (early) and

137 coronary artery disease risk scores with an echocardiogram, exercise stress test, computerized tomog

138 onist clenbuterol and were regularly tested (echocardiograms, exercise tests, catheterizations) with

139 Patients underwent a 3-dimensional echocardiogram focused on the tricuspid apparatus.

140 ram changes and wall motion abnormalities on echocardiogram following neurologic injury.

141 Despite standard guidelines that recommend echocardiogram for screening before transplantation, fou

142 of the NBA mandates annual preseason stress echocardiograms for each player.

143 Available postoperative echocardiograms for Group II (n = 37) demonstrated impro

144 ograms from 74 HAART-exposed children to 860 echocardiograms from 140 HAART-unexposed but HIV-infecte

145 d Children) study prospectively compared 148 echocardiograms from 74 HAART-exposed children to 860 ec

146 ars with cardiovascular risk factor data and echocardiograms from CARDIA year 5 and 25 examinations.

147 We performed speckle-tracking analysis of echocardiograms from participants in the Hypertension Ge

148 index) and 60 randomly selected preoperative echocardiograms from patients with presumed balanced AVS

149 We studied serial echocardiograms from presentation until the last follow-

150 , 82 were enrolled on the basis of screening echocardiogram, fulfillment of inclusion criteria, and i

151 ants were divided into 3 categories based on echocardiograms: HF-REF if EF was <50%, HF-PEF if EF was

152 oma was detected on baseline transesophageal echocardiogram in 167 consecutive patients who had preva

153 We examined absolute GLS on the baseline echocardiogram in relation to changes in LVEF on a follo

154 n the cohort was restricted to those with an echocardiogram in the prior 30 days and no intervening e

155 We performed echocardiograms in 43 patients who met the following inc

156 Clinical examination and blinded readings of echocardiograms in 457 losartan-treated and 459 atenolol

157 The percentage of abnormal stress echocardiograms in each LAVI category was 5.7%, 21.9%, 3

158 We used serial routine echocardiograms in participants of the Framingham Heart

159 Increasing provision of echocardiograms in physicians' offices contributed to in

160 re evaluated longitudinally with a series of echocardiograms in the first trimester, in the third tri

161 =18 years of age at 19 centers with a normal echocardiogram included age, sex, race, ethnicity, heigh

162 re scheduled yearly until the first abnormal echocardiogram indicative of DCM and quarterly thereafte

163 Echocardiogram, invasive hemodynamic pressure-volume ana

164 Transesophageal echocardiogram is a readily available diagnostic tool th

165 enario for which the indication for a stress echocardiogram is uncertain.

166 myocardial infarction when the transthoracic echocardiogram may not be adequate.

167 evated pulmonary artery systolic pressure on echocardiogram, may identify an at-risk population that

168 se, and had acceptable quality 3-dimensional echocardiograms (mean age, 76+/-5 years; 59% women).

169 of cardiac or pulmonary compression on CT or echocardiogram, mitral valve prolapse, arrhythmia, or re

170 elin-1 levels and tricuspid regurgitation on echocardiogram (n = 3223) at the time of first examinati

171 oup with pretransplant electrocardiogram and echocardiogram (n=166 and n=112, HR 4.75, 95% CI 2.07-10

172 Serial echocardiograms (N = 499) were obtained from 115 doxorub

173 Fetal echocardiograms, neonatal ECG, and genetic testing were

174 jority were symptomatic at the time of index echocardiogram (New York Heart Association [NYHA] functi

175 r than 20 years of age, had a normal ECG and echocardiogram, no personal history of heart failure, an

176 who had systolic blood pressure (SBP) and an echocardiogram obtained 30 days after TAVR.

177 Transthoracic echocardiograms obtained during evaluation for transplan

178 ea (EDA) was calculated from transesophageal echocardiograms obtained during initiation and weaning o

179 Echocardiograms, obtained in 203 athletes (10%), did not

180 trial, SPWMD was measured from the baseline echocardiogram of 79 heart failure patients (ejection fr

181 reviewed the intraoperative transesophageal echocardiograms of 13,092 patients without prior diagnos

182 We reviewed the clinical records and echocardiograms of 20 affected patients encountered in o

183 We studied 200 serial echocardiograms of 48 children with DCM (7.0+/-6.0 years

184 this cohort study, first- and last-available echocardiograms of 85 patients with ARVD/C fulfilling 20

185 We reviewed echocardiograms of all cardiac donors in the United Netw

186 analyzed core laboratory-generated data from echocardiograms of all patients enrolled in the Placemen

187 tudied all transthoracic and transesophageal echocardiograms of cardiac amyloid patients at the Mayo

188 We reviewed clinical records and echocardiograms of consecutive patients who had baseline

189 nal cohort of complete AVSD, 52 preoperative echocardiograms of patients with presumed right dominant

190 Six hundred three patients had echocardiograms of sufficient quality for quantitative a

191 ained; using a sequential sampling strategy, echocardiograms on family members were performed.

192 tained a three-generation family history and echocardiograms on first-degree relatives.

193 ulum followed by performing 20 transthoracic echocardiograms on patients receiving invasive mechanica

194 ection fraction (EF) </=45% as determined by echocardiogram or left ventriculogram within 12 hours of

195 iduals for 9 cardiac phenotypes (assessed by echocardiogram or magnetic resonance imaging) to 2.5 mil

196 icular ejection fraction of at least 55% (by echocardiogram or multiple-gated acquisition scan).

197 nce of PPM was identified from postoperative echocardiograms or preoperative manufacturer-provided ch

198 (Tras), or both (Dox+Tras), we obtained 1249 echocardiograms over a median follow-up of 2.0 (interqua

199 ly improved in patients with normal baseline echocardiograms (p=0.005) or electrocardiographs (p=0.05

200 On follow-up echocardiogram, patients in continuous sinus rhythm had

201 013 and who had baseline and post-transplant echocardiograms; patients with simultaneous heart transp

202 aminations, and 19th and 20th examinations), echocardiograms performed by trainees were compared with

203 The protocol included fetal echocardiograms performed weekly from 16 to 26 weeks' ge

204 coronary artery bypass grafting underwent an echocardiogram, plasma biomarker determination, and intr

205 strategies, intra-operative transesophageal echocardiogram, pulmonary artery catheterization, cardio

206 ons, brain magnetic resonance imaging (MRI), echocardiograms, pulmonary function tests, and physical

207 In a longitudinal analysis of 799 echocardiograms, R-N fusion also was associated with a g

208 Of this cohort, 22.3% had abnormal echocardiogram: reduced left ventricular ejection fracti

209 of 122 consecutive patients referred for an echocardiogram regardless of the admitting diagnosis.

210 -up, the maximum pulmonary valve gradient by echocardiogram remained significantly reduced relative t

211 Between January 2011 and January 2014, 2093 echocardiograms reported moderate or greater aortic sten

212 Clinical characteristics and echocardiogram reports were abstracted.

213 ECG and echocardiogram results were classified by blinded core l

214 Evaluation of the most recent outpatient echocardiogram revealed an average peak instantaneous gr

215 rfarin therapy if the 45-day Transesophageal echocardiogram revealed either minimal or no peri-device

216 An echocardiogram revealed severe left ventricular dysfunct

217 Echocardiograms revealed LV dilation, as well as decreas

218 On echocardiogram, RV function was assessed by (1) Doppler

219 ssed with exercise stress test (EST), stress echocardiogram (SE), and stress myocardial perfusion sca

220 The echocardiogram showed an abrupt increase in the severity

221 rformed on schedule unless the goal-directed echocardiogram showed critical findings.

222 olic and systolic time-velocity integrals on echocardiogram showed moderate correlation with RF on CM

223 At the time of referral, Doppler echocardiograms showed major mechanical dyssynchrony at

224 In 36 of these patients, follow-up echocardiograms showed resolution of the constrictive he

225 detected at a median of 6 months after prior echocardiograms showing mild or less gradients.

226 After a rapid bedside echocardiogram suggesting pulmonary embolus, thrombolyti

227 a prospective multisite cohort study design, echocardiograms taken between birth and 24 months were c

228 require a routine screening transesophageal echocardiogram (TEE) before pulmonary vein isolation (PV

229 Transesophageal echocardiogram (TEE) is considered the gold standard mod

230 elative contraindications to transesophageal echocardiogram (TEE), revealed in a hemophilic patient,

231 Thirty-eight patients had echocardiograms that demonstrated normal left ventricula

232 From the 14-month to pre-Fontan echocardiogram, the MBTS group had stable indexed RV vol

233 On the most recent echocardiogram, the median left ventricular end-diastoli

234 ntext of a patient's age and the rest of the echocardiogram to describe diastolic function and guide

235 to compare measures of PR and RV function on echocardiogram to those on cardiac magnetic resonance (C

236 y mitral regurgitation (MR) in transthoracic echocardiogram (TTE) due to relative contraindications t

237 se criteria (AUC) have defined transthoracic echocardiogram (TTE) indications for which there is a cl

238 or hearts with LVSD on initial transthoracic echocardiogram (TTE) that resolved during donor manageme

239 tolic pulmonary artery pressure >35 mm Hg on echocardiogram underwent a right heart catheterization.

240 Right ventricular dysfunction on initial echocardiogram was 61% sensitive (95% confidence interva

241 (RVSP) (mean +/- SD) on the two-dimensional echocardiogram was 68 +/- 21 mm Hg.

242 Abnormal echocardiogram was independently associated with all cau

243 The stress echocardiogram was interpreted as abnormal if wall motio

244 ation functional class was reassessed and an echocardiogram was obtained and compared with pre-CRT.

245 An echocardiogram was obtained as part of an evaluation for

246 diagnosed as having PAH, then a new Doppler echocardiogram was obtained to measure cardiac parameter

247 days before ablation, and a transesophageal echocardiogram was performed to rule out clot.

248 f 138 SSc patients with technically adequate echocardiograms was studied and compared with 40 age- an

249 l of 10 471 individual patient transthoracic echocardiograms, we identified moderate or severe PH in

250 our systemic markers (cardiac involvement by echocardiogram, weight loss > 10 pounds, orthostatic int

251 gnosis of HF before or within 30 days of the echocardiogram were excluded.

252 2013, and had a postoperative transthoracic echocardiogram were included.

253 as asymptomatic, and the cardiac enzymes and echocardiogram were normal; therefore, electrophysiologi

254 Severely abnormal hemodynamics on echocardiograms were also infrequent and not associated

255 Echocardiograms were analyzed according to established g

256 ECGs and echocardiograms were analyzed independently, blinded to

257 Acceptable echocardiograms were available at both time points in 24

258 Follow-up echocardiograms were available in 17 patients.

259 Echocardiograms were collected using a common protocol,

260 Their baseline and 12-month echocardiograms were compared between the group with and

261 Echocardiograms were digitized, and endocardial borders

262 Most recent echocardiograms were evaluated for right ventricular out

263 Electrocardiograms (ECGs) and echocardiograms were normal in 16 and 17 patients, respe

264 Echocardiograms were obtained 1 year after study entry a

265 Serial Doppler echocardiograms were obtained at baseline and 6 and 12 m

266 Echocardiograms were obtained at baseline, discharge, 30

267 Echocardiograms were obtained at baseline, discharge, 30

268 Centrally interpreted echocardiograms were obtained at KD diagnosis and 1 and

269 Doppler echocardiograms were obtained at randomization (after 30

270 ood samples, electrocardiographs (ECGs), and echocardiograms were obtained before, during, and after

271 Echocardiograms were obtained from 935 patients with HFp

272 METHODS AND In 8 centers, echocardiograms were obtained on 169 patients prospectiv

273 Echocardiograms were obtained on 2383 participants (1993

274 ficient, and 2-dimensional and 3-dimensional echocardiograms were obtained post-MR and post-PVA injec

275 Follow-up echocardiograms were obtained to determine freedom from

276 Echocardiograms were performed 2 days after myocardial i

277 Echocardiograms were performed at least every 3 weeks fr

278 Preoperative transthoracic echocardiograms were performed for assessment of LAV, le

279 Detailed echocardiograms were performed in 29 probands and compar

280 Echocardiograms were performed on 1,013 participants usi

281 Echocardiograms were performed to assess systolic and di

282 uring the study period, approximately 50,660 echocardiograms were performed.

283 All echocardiograms were re-reviewed by experts (2012 World

284 Doppler echocardiograms were recorded at baseline, before hospit

285 Echocardiograms were reported in a standardized, blinded

286 Fetal echocardiograms were reviewed from 43 fetuses diagnosed

287 Comprehensive echocardiograms were reviewed in blinded fashion.

288 Catheterization and ECMO records and echocardiograms were reviewed, as were the clinical cour

289 functional parameters (measured from recent echocardiograms) were compared with biochemical paramete

290 Twenty-three studies (1,638 echocardiograms) were included.

291 ed systolic function, as assessed on a study echocardiogram when the participants were 23 to 35 years

292 patients with a reduced LVEF <50% (screening echocardiogram), whose LVEF had increased by at least 10

293 sence of PFO was determined by transthoracic echocardiogram with second harmonic imaging and transcra

294 clinicians, including the integration of the echocardiogram with the history and physical examination

295 rtension Genetic Epidemiology Network) study echocardiograms with available urinary sodium data (N =

296 patients were evaluated with the use of rest echocardiograms with partial LVAD support and cardiopulm

297 severe traumatic brain injury; transthoracic echocardiogram within 1 day after mild traumatic brain i

298 Transthoracic echocardiogram within 1 day and over the first week afte

299 >/=55% and who subsequently had a follow-up echocardiogram within 6 to 24 months.

300 myopathy can occur within 1 week of a normal echocardiogram without initial first-degree block.