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1 ures that cause measurable changes in tissue echogenicity.
2  and ultrasound assessment of Rectus Femoris echogenicity.
3 of the plaque, which was normalized to lumen echogenicity.
4 C concentrations that is necessary for blood echogenicity.
5 e mass contained a central area of increased echogenicity.
6 pered by the absence of a means of measuring echogenicity.
7 aracterise the changes in macroscopic muscle echogenicity and fascial characteristics that occur earl
8 rse correlation between normalized LP plaque echogenicity and gray-scale median score.
9 afting reduced the pathological increases of echogenicity and neointima formation in rats.
10       The aim of this work was to assess the echogenicity and the size of perirenal haematomas in pat
11 ney cross-sections and enabled measuring the echogenicity and thickness of the abnormalities at the s
12                                   Initial US echogenicity and vascularization influence the ablation
13 nts (thickness, width, cross-sectional area, echogenicity) and 3.0-T MR imaging measurements (thickne
14 hic images with regard to origin, insertion, echogenicity, and location.
15  of propagation speed, attenuation, relative echogenicity, and mass density are reported for all tiss
16 ups differed regarding bowel wall thickness, echogenicity, and perfusion in sonograhy and color Doppl
17    US parameters were renal length, relative echogenicity, and resistive index (RI).
18 sis of parameters such as x-ray attenuation, echogenicity, and sound attenuation.
19                                              Echogenicity at the site of the pathologic finding in th
20 t of nonviable tumor determined as decreased echogenicity at ultrasonography (US) and lack of enhance
21      After cryoablation, there was increased echogenicity at US and increased density at mammography;
22           US images were graded to determine echogenicity changes, CT attenuation was measured (in Ho
23                                       Lesion echogenicity (class I to IV), degree of stenosis, and ma
24       Correlation of neovascularization with echogenicity, degree of stenosis, and maximal lesion thi
25 c, "eyeglass" shape), grade II (intermediate echogenicity, "dumbbell" outline), and grade III (hypere
26 n to the chest wall, border characteristics, echogenicity, homogeneity, enhancement or shadowing, and
27 Each tumor was evaluated for size, location, echogenicity, homogeneity, shadowing, hypoechoic rim, an
28  hyperplasia, which correlated with the high echogenicity in HFU images and the large mechanical stre
29 IRE ablation produced greater alterations to echogenicity in normal tissues than in tumors.
30 r lesion shape (kappa=0.14), substantial for echogenicity (kappa=0.61), and moderate for posterior fe
31 onths to years and the pattern of lymph node echogenicity may suggest the diagnosis of ALPS.
32  = 4) on CT scans; and homogeneous, moderate echogenicity (n = 3) on US scans.
33                                          The echogenicity of lipomas ranged from hypoechoic to hypere
34 statistically significant, difference in the echogenicity of perirenal haematomas compared to the rou
35        Raw linear data were used to quantify echogenicity of the plaque, which was normalized to lume
36                       If the vascularity and echogenicity of the scrotal mass is similar with the nor
37 e small intestine loops, and increase in the echogenicity of the surrounding mesenteric fat tissue.
38 sented hypoechogenic thrombus, whereas mixed echogenicity of thrombus appeared on 11 patients.
39 c resonance imaging, 92% had increased liver echogenicity on ultrasonography, and 65% had splenomegal
40 reased attenuation on CT scans and increased echogenicity on US scans of renal adenomatous tumors are
41 of the following: persistent periventricular echogenicity or echolucency on neuroimaging, chronic lun
42 fibrin-targeted contrast exhibited increased echogenicity (P < .05); control thrombi remained acousti
43 r platelet count (P = 0.007); abnormal liver echogenicity (P < 0.001); and splenomegaly (P = 0.001) a
44 tion using a high-frequency ultrasonic (HFU) echogenicity platform and estimated the endothelium yiel
45                                  Sonographic echogenicity ranged from hypoechoic to hyperechoic relat
46 under receiver operator curve for ultrasound echogenicity's prediction of myofiber necrosis was 0.74
47                  Lesion location, diameters, echogenicity, shape, and posterior features were recorde
48  sonographic examination, the lesions showed echogenicity similar to, or slightly lower than, the tes
49 he masses were evaluated for size, location, echogenicity, sound attenuation, and vascularity.
50 for tendon nonvisualization, abnormal tendon echogenicity, tendon thinning, greater tuberosity cortic
51 rfluorocarbon emulsion that has low inherent echogenicity unless bound to a surface or itself.
52                                        Blood echogenicity was examined with the use of quantitative v
53                             Change in muscle echogenicity was greater in patients who developed muscl
54                         Normalized LP plaque echogenicity was greater in the symptomatic group (0.39;
55   In a total of 293 atherosclerotic lesions, echogenicity was inversely correlated with grade of intr
56 ation, a significantly greater difference in echogenicity was observed and reached 31 dB.
57 evised an ultrasonic grading system in which echogenicity was quantified by linear gain reduction and
58 , plaque texture, plaque surface, and plaque echogenicity were analyzed.

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