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1 ures that cause measurable changes in tissue echogenicity.
2 and ultrasound assessment of Rectus Femoris echogenicity.
3 of the plaque, which was normalized to lumen echogenicity.
4 C concentrations that is necessary for blood echogenicity.
5 e mass contained a central area of increased echogenicity.
6 pered by the absence of a means of measuring echogenicity.
7 aracterise the changes in macroscopic muscle echogenicity and fascial characteristics that occur earl
11 ney cross-sections and enabled measuring the echogenicity and thickness of the abnormalities at the s
13 nts (thickness, width, cross-sectional area, echogenicity) and 3.0-T MR imaging measurements (thickne
15 of propagation speed, attenuation, relative echogenicity, and mass density are reported for all tiss
16 ups differed regarding bowel wall thickness, echogenicity, and perfusion in sonograhy and color Doppl
20 t of nonviable tumor determined as decreased echogenicity at ultrasonography (US) and lack of enhance
25 c, "eyeglass" shape), grade II (intermediate echogenicity, "dumbbell" outline), and grade III (hypere
26 n to the chest wall, border characteristics, echogenicity, homogeneity, enhancement or shadowing, and
27 Each tumor was evaluated for size, location, echogenicity, homogeneity, shadowing, hypoechoic rim, an
28 hyperplasia, which correlated with the high echogenicity in HFU images and the large mechanical stre
30 r lesion shape (kappa=0.14), substantial for echogenicity (kappa=0.61), and moderate for posterior fe
34 statistically significant, difference in the echogenicity of perirenal haematomas compared to the rou
37 e small intestine loops, and increase in the echogenicity of the surrounding mesenteric fat tissue.
39 c resonance imaging, 92% had increased liver echogenicity on ultrasonography, and 65% had splenomegal
40 reased attenuation on CT scans and increased echogenicity on US scans of renal adenomatous tumors are
41 of the following: persistent periventricular echogenicity or echolucency on neuroimaging, chronic lun
42 fibrin-targeted contrast exhibited increased echogenicity (P < .05); control thrombi remained acousti
43 r platelet count (P = 0.007); abnormal liver echogenicity (P < 0.001); and splenomegaly (P = 0.001) a
44 tion using a high-frequency ultrasonic (HFU) echogenicity platform and estimated the endothelium yiel
46 under receiver operator curve for ultrasound echogenicity's prediction of myofiber necrosis was 0.74
48 sonographic examination, the lesions showed echogenicity similar to, or slightly lower than, the tes
50 for tendon nonvisualization, abnormal tendon echogenicity, tendon thinning, greater tuberosity cortic
55 In a total of 293 atherosclerotic lesions, echogenicity was inversely correlated with grade of intr
57 evised an ultrasonic grading system in which echogenicity was quantified by linear gain reduction and
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