ライフサイエンスコーパス: ectasia

1 uality of vision without the risk of corneal ectasia.

2 There was no ectasia.

3 ication, ulceration, neovascularization, and ectasia.

4 agen cross-linking in postrefractive surgery ectasia.

5 sler-Krumeich classification for severity of ectasia.

6 toconus and postlaser in-situ keratomileusis ectasia.

7 ts (ICRS) for treating patients with corneal ectasia.

8 treatment of both keratoconus and post-LASIK ectasia.

9 potentially decreased risk of postprocedure ectasia.

10 merged as a novel approach for management of ectasia.

11 on of keratoconus and postrefractive surgery ectasia.

12 AV disease with and without ascending aortic ectasia.

13 oing PTK, in an effort to prevent iatrogenic ectasia.

14 to stabilize and sphericize the cornea with ectasia.

15 tive and structural abnormalities of corneal ectasias.

16 K value, CDVA, and UDVA in eyes with corneal ectasia 1 year after treatment, with an excellent safety

17 ce of Chiari I malformation (6.4%) and dural ectasia (42.6%); and physical examination findings of a

18 ), carcinoma (13), colitis (5), and vascular ectasia (5).

19 More eyes with stage 4 ectasia achieved 20/25 visual acuity after PROSE than af

20 ost recent management strategies for corneal ectasia after keratorefractive surgery.

21 Management options for ectasia after laser in situ keratomileusis include intra

22 gation has resulted in increased interest in ectasia after laser in situ keratomileusis.

23 , and future research directions for corneal ectasia after laser in situ keratomileusis.

24 facilitates recognition of risk factors for ectasia after laser in-situ keratomileusis.

25 sslinking (CXL) for the treatment of corneal ectasia after laser refractive surgery.

26 ients (18 male, 8 female) with postoperative ectasia after LASIK (23 eyes) and PRK (3 eyes) were incl

27 cido-based corneal topography that developed ectasia after LASIK (ectasia group) and 174 eyes from 88

28 Ectasia after LASIK and PRK was arrested by CXL with sta

29 ve patients treated with CXL for progressive ectasia after LASIK or PRK at the Institute for Refracti

30 xteen eyes of 14 patients with postoperative ectasia after LASIK were enrolled.

31 e hundred seventy-nine subjects with corneal ectasia after previous refractive surgery.

32 Patients with progressive keratoconus or ectasia after refractive surgery (n = 510).

33 comparable representation of keratoconus and ectasia after refractive surgery in the 2 treatment arms

34 coronary disease, vascular atherosclerosis, ectasia and aneurysm, collateral vessel development and

35 he most frequent vascular abnormalities were ectasia and aneurysm.

36 esions, such as optic pathway gliomas, dural ectasia and aqueduct stenosis.

37 The biliary ductual ectasia and hepatic portal fibrosis associated with ARPK

38 s been documented to stop the progression of ectasia and in some cases may cause regression.

39 Mucinous ductal ectasia and IPN differed histopathologically only in deg

40 Mucinous ductal ectasia and IPNs represent newly recognized categories o

41 able lenses for the visual rehabilitation of ectasia and irregular astigmatism, and an update on scle

42 lens design, predominantly in patients with ectasia and postkeratoplasty astigmatism.

43 ad arteriopathy, the combination of arterial ectasia and stenosis and, importantly, absence of the ty

44 Decreased lesional vascular ectasia and tortuosity were also observed and were accom

45 III antibodies with gastric antral vascular ectasia, and a temporal association between SSc onset an

46 days, including jeopardy score and coronary ectasia, and at 1 year, including previous percutaneous

47 y cases can improve visual acuity, stabilize ectasia, and delay or even prevent the need for more inv

48 on of tumor-associated macrophages, vascular ectasia, and hemorrhage.

49 onary intervention, jeopardy score, coronary ectasia, and increasing number of diseased vessels.

50 romising results for keratoconus, post-LASIK ectasia, and pellucid marginal degeneration.

51 osis, diffuse or focal; segmental dilatation/ectasia; and tortuosity.

52 Findings included aneurysms, ectasia, arteriovenous fistulas, and anomalous origins.

53 considered in patients with advanced corneal ectasia before proceeding to keratoplasty, especially if

54 odality for both angiodysplasia and vascular ectasia bleeding.

55 greater for angiodysplasia than for vascular ectasia bleeding.

56 otentially reducing the incidence of corneal ectasia but seems to be associated with an increased inc

57 Mucinous ductal ectasia, but not IPN, was characteristically mucin-hyper

58 t the progression of keratoconus and corneal ectasia by inducing corneal stiffening.

59 The presence of coronary artery ectasia (CAE) is influenced by genetic factors and relat

60 Ectasia can also rarely occur in patients without curren

61 Eyes with advanced corneal ectasia can be successfully fitted with the PROSE device

62 egments, and photorefractive keratectomy for ectasia, corneal edema, and infectious keratitis.

63 Coronary artery aneurysms or ectasia develop in approximately 15 to 25% of untreated

64 When ectasia develops, early recognition and proper managemen

65 egarding the increasing incidence of corneal ectasia following laser in situ keratomileusis procedure

66 topathology, topographic pseudokeratectasia, ectasia from transient raised intraocular pressure, poor

67 ectasias, including gastric antral vascular ectasia (GAVE) and angiodysplasia, are increasingly reco

68 (GI) bleeding due to gastric antral vascular ectasia (GAVE).

69 rent hemorrhage from gastric antral vascular ectasias (GAVE).

70 pography that developed ectasia after LASIK (ectasia group) and 174 eyes from 88 consecutive patients

71 In the ectasia group, percent tissue altered >/=40 was the most

72 nsive gastropathy (PHG) and gastric vascular ectasia (GVE), to transjugular intrahepatic portosystemi

73 families with a combination of SRNS, tubular ectasia, haematuria and facultative neurological involve

74 Gastric antral vascular ectasias have strongly been associated with the presence

75 The 46 patients with UGI vascular ectasia hemorrhage included 27 patients with angiodyspla

76 th stenosis in 19%, and segmental dilatation/ectasia in 56%.

77 cluding arterial stenoses, interruptions and ectasia in 7.6%.

78 XL stabilized primary and iatrogenic corneal ectasia in 89% of the patients.

79 in slowing, halting or reversing progressive ectasia in both keratoconus and progressive post-LASIK k

80 rrated eyes, including treatment for corneal ectasia in conjunction with collagen cross-linking (CXL)

81 a safe and effective new technique to reduce ectasia in eyes with advanced keratoconus, potentially a

82 wman layer graft is a new approach to reduce ectasia in eyes with advanced keratoconus.

83 ificantly associated with the development of ectasia in eyes with normal preoperative topography and

84 r generating topographic features of corneal ectasia in human tissue is demonstrated.

85 llucid marginal degeneration, and post-LASIK ectasia, in addition to potentially decreasing or delayi

86 portal gastropathy but not gastric vascular ectasias.In the area of gastric cancer, management revol

87 Management options for postoperative ectasia include conservative management with various typ

88 Vascular ectasias, including gastric antral vascular ectasia (GAV

89 g all of the individual indices, the maximum ectasia index for epithelium had the highest ability to

90 The discriminant function containing maximum ectasia indices of epithelium and Bowman's layer further

91 All Pentacam ectasia indices significantly differed between Groups 1

92 se in having erythrocytic sickling, vascular ectasia, intravascular hemolysis, exuberant hematopoiesi

93 Gastric antral vascular ectasia is a vascular manifestation, and bleeding may be

94 roceeding to keratoplasty, especially if the ectasia is deemed stable.

95 Early management of ectasia is essential to prevent its progression and to p

96 Our knowledge about ectasia is still in evolution.

97 clude scoliosis, chest wall deformity, dural ectasia, joint hypermobility, and acetabular protrusion.

98 ous drainage (kappa = 1), presence of venous ectasia (kappa = 1), and final Cognard classification of

99 ith more distal ductal obstruction or ductal ectasia may benefit from pancreaticojejunostomy.

100 Because ectasia may occur in the absence of risk factors, there

101 nstitutional experience with mucinous ductal ectasia (MDE) and intraductal papillary neoplasms (IPNs)

102 Coronary artery aneurysms (CAAs) and ectasia occur in 0.2% to 5.3% of patients referred for a

103 nking was a safe and effective treatment for ectasia occurring after LASIK.

104 same-day corneal collagen cross-linking for ectasia occurring after LASIK.

105 cterized by symmetrically large kidneys with ectasia of collecting ducts.

106 by thinning of the posterior cornea without ectasia of the anterior cornea.

107 ed in a patient with gastric antral vascular ectasia or watermelon stomach, a disorder that is increa

108 1) and when including only eyes with stage 4 ectasia (P < .001).

109 erosclerosis: stenosis-producing plaques and ectasia-producing abdominal aortic aneurysm (AAA).

110 scharge, with fibrocystic disease and ductal ectasia providing the next most common causes.

111 , residual stromal bed </=300 mum (57%), and ectasia risk score >/= 3 (43%) (P < .001 for all).

112 by residual stromal bed </= 300 mum (74) and ectasia risk score >/= 4 (8).

113 ral corneal thickness, residual stromal bed, Ectasia Risk Score System scores, and percent tissue alt

114 by providing a good sensitivity in detecting ectasia-susceptible corneas.

115 es in the keratoplasty group had more severe ectasia than eyes in the PROSE group (P = .038).

116 n being less in patients with postrefractive ectasia than keratoconus.

117 charts of consecutive patients with corneal ectasia that were evaluated for PROSE or underwent kerat

118 hown to be an effective modality for corneal ectasia, the regression being less in patients with post

119 nase for generating an experimental model of ectasia to evaluate the topographic effects of CXL inter

120 ines, the AUC of the Belin/Ambrosio enhanced ectasia total derivation (BAD-D) and the inferior-superi

121 ed progressive primary or iatrogenic corneal ectasia underwent CXL following the Siena protocol.

122 tion, reduction and stabilization of corneal ectasia was achieved in eyes with progressive, advanced

123 e, and the visual acuity outcome for stage 4 ectasia was better and more rapid compared to keratoplas

124 n excluding eyes with early signs of corneal ectasia when screening patients for excimer laser surger

125 in expansive vascular remodeling and luminal ectasia, whereas Th1 immune responses cause intimal hype

126 ients underwent PROSE evaluation for corneal ectasia while 37 patients underwent keratoplasty for the

127 esults in halting the progression of corneal ectasia, with significant improvement in CDVA and long-t