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1 mally helps protect against the formation of ectopic beats.
2 ectopic beats compared with those with <0.1% ectopic beats.
3 hythmias were mainly single supraventricular ectopic beats.
4    In the pre-implantation Holter recording, ectopic beats accounted for a mean 3.2 +/- 5.5% of all b
5 50; p < 0.001) in patients with 0.1% to 1.5% ectopic beats and 13-fold (odds ratio: 13.42; 95% confid
6 ed with late interval count decreases due to ectopic beats and 9% with erratic count changes due to a
7 dings showed no change in mean heart rate or ectopic beats and no arrhythmias.
8                            Adenosine reduced ectopic beats and the incidence of ventricular tachycard
9 olume, and dP/dtmax were obtained during all ectopic beats and ventricular pacing.
10 an evoke graded depolarizations, propagating ectopic beats, and if timed appropriately, spiral reentr
11 hm, constant pacing, spontaneous ventricular ectopic beats, and premature stimulation at intermediate
12                     It is often initiated by ectopic beats arising from the pulmonary veins and atriu
13  to 25.66; p < 0.001) in patients with >1.5% ectopic beats compared with those with <0.1% ectopic bea
14                Relatively low frequencies of ectopic beats (&gt;/=0.1%) dramatically increase the probab
15 n sleep-disordered breathing and ventricular ectopic beats/h (p<0.0003) considered as a continuous ou
16                        Patients with >/=0.1% ectopic beats had significantly less reverse remodeling
17 hancing dispersion of refractoriness so that ectopic beats have a high probability of inducing reentr
18 : 3.13 and 1.84, respectively) and for >1.5% ectopic beats (hazard ratio: 2.38 and 2.74, respectively
19 sed significantly in those with 0.1% to 1.5% ectopic beats (hazard ratio: 3.13 and 1.84, respectively
20 th negligible early afterdepolarizations and ectopic beats in untreated controls.
21                     Epicardial origin of the ectopic beats increases transmural dispersion of repolar
22 nderlies the degradation of a pulmonary vein ectopic beat into AF.
23 lecular mechanisms alters the probability of ectopic beats is not understood.
24  or who are predisposed to magnesium loss or ectopic beats may require more dietary magnesium than wo
25 rrhythmias ranging from frequent ventricular ectopic beats, nonsustained and sustained ventricular ta
26 ardiograms of Kir2.1 (-/-) neonates, neither ectopic beats nor re-entry arrhythmias were observed.
27  models were used to assess the influence of ectopic beats on the outcomes of heart failure (HF) or d
28 ic regression, we estimated the influence of ectopic beats on the percentage of biventricular pacing.
29 nization therapy (CRT), but the influence of ectopic beats on the success of biventricular pacing has
30 as similar in patients with (17,859+/-13,488 ectopic beats per 24 hours) and without (17,541+/-11,479
31 s per 24 hours) and without (17,541+/-11,479 ectopic beats per 24 hours; P=0.800) preserved ventricul
32 n LVESV 31 +/- 15%) than patients with <0.1% ectopic beats (percent reduction in LVESV 39 +/- 14%; p
33  such as conduction velocity restitution and ectopic beats, promote spatially discordant alternans.
34  This study sought to determine if increased ectopic beats reduce the chance of high biventricular pa
35         Without removing any outliers due to ectopic beats, the method is able to detect a degradatio
36 rning heart rate variability parameters, and ectopic beats throughout the recording (20%).
37 nimal models, early afterdepolarizations and ectopic beats were observed in 33% and 40% of embryoid b

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