ライフサイエンスコーパス: ectopy

1 re more likely to have mature cervixes (less ectopy).

2 ection was not independently associated with ectopy.

3 recovery time; and (c) inducible ventricular ectopy.

4 ntified sites initiating pulmonary vein (PV) ectopy.

5 ous dissociated PV rhythm and ADO-induced PV ectopy.

6 yopathy with frequent or complex ventricular ectopy.

7 tricular tachycardia or frequent ventricular ectopy.

8 rsus 14.5% (p=0.002) for complex ventricular ectopy.

9 sponse, heart rate recovery, and ventricular ectopy.

10 ith low atrial voltage and nonpulmonary vein ectopies.

11 tricular arrhythmia observed (0 indicates no ectopy; 1, isolated premature ventricular beats; 2, bige

12 in trials requiring evidence of ventricular ectopy (25%) than in the remaining trials (10%).

13 r than patients without adenosine-induced PV ectopy (63% versus 76% at 1 year; log rank, 0.014).

14 Increased atrial ectopy (AE) increases the risk of atrial fibrillation (A

15 pose of the study was to determine if atrial ectopy (AE) or atrial arrhythmias during exercise are pr

16 s remained associated with lower ventricular ectopy after cardiovascular comorbidities were controlle

17 We hypothesized that ventricular ectopy after exercise (i.e., during the recovery phase)

18 iency virus (HIV) infection, associated with ectopy among adolescent girls aged 12-20 years who were

19 acids may be associated with low ventricular ectopy among AMI patients.

20 n n-3 fatty acid consumption and ventricular ectopy among AMI patients.

21 profile of ventricular and supraventricular ectopy and bradyarrhythmia on ambulatory 24-h Holter ECG

22 nd is associated with reversible ventricular ectopy and DCM.

23 ainide has been shown to depress ventricular ectopy and improve exercise capacity in patients with An

24 We examined the extent of ectopy and metaplastic activity as risks for HPV16 acqui

25 analysis of MKLT and describe the effects of ectopy and slow changes in cardiac cycles on the disturb

26 t of ablation was absence of frequent atrial ectopy and spontaneous AF during isoproterenol infusion

27 nts and produced pause-dependent ventricular ectopy and sustained ventricular tachycardia after acute

28 -25(-/-) mice exhibited more frequent atrial ectopy and were also more susceptible to pacing-induced

29 ts of amount of exposed columnar epithelium (ectopy) and age on the presence of alpha9 or alpha3/alph

30 es and ARIs during sinus rhythm, ventricular ectopy, and premature stimulation (r=0.72, slope=-0.76,

31 s, abnormal heart rate recovery, ventricular ectopy, and ST-segment abnormalities.

32 wall motion abnormalities; RV outflow tract ectopy; and exercise-induced T-wave pseudonormalization.

33 c metaplasia rather than the sheer extent of ectopy appears to increase risk for incident HPV16 in he

34 he acute phase of myocardial ischemia, focal ectopies arising from this location, and including both

35 ocardial infarction (MI) may be triggered by ectopy arising from Purkinje fibers.

36 n of ventricular tachycardia and ventricular ectopy arising in a PAP has a high success rate.

37 All patients had frequent ectopy at baseline with a median PVC count of 7275 (rang

38 After adjustment for ventricular ectopy at rest and during exercise, peak oxygen uptake,

39 acid) was associated with lower ventricular ectopy (beta = -0.35, P = 0.011), and this effect remain

40 ulated metaplastic rate as the difference in ectopy between visits.

41 An increase in ventricular ectopy burden was associated with progressively lower ev

42 ot be restricted to the generation of atrial ectopy but extends to the development of atrial remodeli

43 ll appreciated is whether simple ventricular ectopy can result in cardiomyopathy.

44 In this study, we describe murine thymic ectopy, cervical thymic tissue that possesses the same g

45 Ventricular ectopy could be induced by isoproterenol-challenge in is

46 Ectopy data for 189 and 92 HIV-positive and -negative ad

47 ta-blocker significantly reduced ventricular ectopy during exercise compared with placebo plus beta-b

48 1 to 1.9; P=0.003), but frequent ventricular ectopy during exercise did not (adjusted hazard ratio, 1

49 Frequent ventricular ectopy during exercise predicted an increased risk of de

50 10 to 1.97; p = 0.0089), whereas ventricular ectopy during exercise was not predictive of death in th

51 ue to patient factors--excessive ventricular ectopy during exercise, unsustained MTWA, or failure to

52 reased risk of death better than ventricular ectopy during exercise.

53 among patients without frequent ventricular ectopy during exercise; hazard ratio, 1.8; 95 percent co

54 Frequent ventricular ectopy during recovery after exercise is a better predic

55 Severe ventricular ectopy during recovery after exercise is predictive of i

56 Although ventricular ectopy during recovery after exercise predicts death in

57 confounding variables, frequent ventricular ectopy during recovery predicted an increased risk of de

58 er potential confounders, severe ventricular ectopy during recovery remained predictive of death (adj

59 5 to 2.1; P<0.001), but frequent ventricular ectopy during recovery was a stronger predictor (11 perc

60 Severe ventricular ectopy during recovery was associated with an increased

61 23 patients, 140 (7%) had severe ventricular ectopy during recovery.

62 ormal QT interval, and displayed ventricular ectopy during stress testing consistent with CPVT.

63 ADO induced PV ectopy during the early phase of ADO effect only in 12 P

64 ify the implications of adenosine-induced PV ectopy for atrial fibrillation (AF) recurrence after PV

65 elayed the development of spontaneous atrial ectopy, fully prevented sAF, suppressed atrial dilation,

66 1 partner in the past 6 months, and cervical ectopy greater than 25%.

67 Ectopy (>/= 1000 premature ventricular complexes/24 hour

68 Although thymic ectopy has long been recognized in humans, the functiona

69 ort the prognostic importance of ventricular ectopy immediately after exercise, when reactivation of

70 220 electrically silent PVs, ADO induced PV ectopy in 28 (13%) veins.

71 Adenosine can also induce ectopy in electrically silent PVs after isolation, possi

72 However, ADO can induce PV ectopy in electrically silent PVs in a manner not closel

73 rognostic importance of frequent ventricular ectopy in recovery after exercise among patients with sy

74 eart rate recovery, and frequent ventricular ectopy in recovery.

75 Repetitive monomorphic ventricular ectopy (in the absence of sustained ventricular tachycar

76 ars) with repetitive monomorphic ventricular ectopy, including 8 patients (30%) with depressed ventri

77 Different patterns of ectopy, including isolated PVCs, bigeminy, trigeminy, an

78 sity to CDA rendered the atria vulnerable to ectopy-induced arrhythmia.

79 Patients with ectopy-induced cardiomyopathy are significantly older th

80 In this milieu, spontaneous ectopy initiated AF.

81 r tachyarrhythmias and other benign forms of ectopy: inpatient versus outpatient.

82 Adenosine-induced PV ectopy is a predictor of recurrent AF after PV isolation

83 Very frequent ventricular ectopy may also result in a cardiomyopathy in a minority

84 high-resolution photograph of the cervix for ectopy measurement were collected.

85 tive use to be associated with the amount of ectopy, multivariate logistic regression analysis showed

86 ricular tachycardia, and complex ventricular ectopy (nonsustained ventricular tachycardia or bigeminy

87 Frequent ventricular ectopy occurred only during exercise in 945 patients (3

88 an increased risk of death than ventricular ectopy occurring only during exercise.

89 tor, with more partners associated with less ectopy (odds ratio, 0.47; 95% confidence interval, 0.22-

90 ar cell models suggested that rate-dependent ectopy of Purkinje fiber origin is the predominant ventr

91 Lown grade 4 ventricular ectopy on ambulatory ECG was present in three patients i

92 (RCA) perfusion area and showed ventricular ectopy on electrocardiogram (ECG) at rest that diminishe

93 cardiomyopathy or heart failure, ventricular ectopy or nonsustained ventricular tachycardia, and lowe

94 g arrhythmia patients, 6 had frequent atrial ectopy or tachycardia, 86 had paroxysmal AF, 39 had pers

95 s not different than in patients with atrial ectopy or tachycardia.

96 almost twice the odds of complex ventricular ectopy (OR, 1.74; 95% CI, 1.11-2.74).

97 In 2 patients, posterior ectopy organized to subsequently initiate isthmus-depend

98 n abnormalities in regions where ventricular ectopy originates.

99 p between repetitive monomorphic ventricular ectopy originating from the right ventricular outflow tr

100 Exercise-induced ventricular ectopy predicts an increased risk of death in population

101 ar tachycardia (VT) and frequent ventricular ectopy (premature ventricular contractions [PVCs] >10/h)

102 -perfused hearts, we demonstrated that focal ectopies require, in the normal mouse heart, the simulta

103 ablation of the focal source of ventricular ectopy results in normalization of left ventricular func

104 displayed 3-fold higher rates of ventricular ectopy than Casq2+/+ mice (n=31; P<0.05).

105 inus rhythm, pacing, and AF initiation by PV ectopy that are determined largely by the functional pro

106 ing isoproterenol infusion identified atrial ectopy that initiated AF and the presence of inducible A

107 Although the most common sites of atrial ectopy that trigger atrial fibrillation (AF) are in or a

108 Of the several measures of ventricular ectopy that were univariate predictors, the frequency of

109 ion of extrasystoles and the factors causing ectopies to be arrhythmia triggers during myocardial isc

110 of extrasystoles, and the properties leading ectopies to become arrhythmia triggers (topology), in th

111 other and fetus; these may range from benign ectopy to life-threatening arrhythmias.

112 occur during pregnancy, ranging from benign ectopy to life-threatening arrhythmias.

113 ependent progression from spontaneous atrial ectopy to paroxysmal and eventually long-lasting AF.

114 lt T-wave alternans (MTWA) test, when due to ectopy, unsustained MTWA, or low exercise heart rate (HR

115 with bileaflet prolapse, complex ventricular ectopy (VE), and abnormal T waves comprise the recently

116 uent association of more serious ventricular ectopy, VT, with lower [K+]e concentrations supports the

117 Severe ventricular ectopy was defined as the presence of ventricular triple

118 Frequent ventricular ectopy was defined by the presence of seven or more vent

119 multivariate analysis, adenosine-induced PV ectopy was found to be the only independent predictor of

120 However, ectopy was not significant, whether measured before or c

121 Ventricular ectopy was present only in MyBP-C(t/t) mice during ambul

122 The burden of ectopy was quantified through 24-hour Holter monitoring.

123 Ectopy was quantitatively measured on colpophotographs.

124 For each isolated PV, dissociated ectopy was recorded and ADO was administered.

125 Adenosine-induced PV ectopy was seen in 45 (30%) patients, and dormant conduc

126 However, the burden of ventricular ectopy was similar in patients with (17,859+/-13,488 ect

127 solation of 270 PVs, 50 PVs with dissociated ectopy were identified.

128 Older age and decreasing cervical ectopy were independently positively associated with hav

129 of adenosine-induced PV reconnection and PV ectopy were recorded.

130 AF among patients with adenosine-induced PV ectopy were significantly lower than patients without ad

131 Prospective Randomized Evaluation of Cardiac Ectopy with Dobutamine or Nesiritide Therapy) trial were

132 The timing of ADO-induced PV ectopy with respect to ADO effects on heart rate varied.