ライフサイエンスコーパス: effective refractory period

1 eriod; the inner limit, however, was not its effective refractory period.

2 namic measurements or changes in ventricular effective refractory period.

3 ) 50 micromol/L had no significant effect on effective refractory period.

4 n=30) by an S2 at intervals shorter than the effective refractory period.

5 properties, and atrial, AV, and ventricular effective refractory periods.

6 Patients with DWR had a shorter effective refractory period (138.8+/-13.4 versus 163.8+/

7 , and rate-adaptive shortening of the atrial effective refractory periods (14+/-13 versus 12+/-14 ms;

8 10% increases in noninfarct zone ventricular effective refractory period, 3% to 5% increases in infar

9 1 +/- 28 ms; p = 0.05) and ventriculo-atrial effective refractory periods (AC(VI): 97 +/- 21 ms; cont

10 Vagal stimulation shortens the atrial effective refractory period (AERP) and maintains atrial

11 In pace/controls, atrial effective refractory period (AERP) at a drive cycle leng

12 nt efficacy in the preclinical rabbit atrial effective refractory period (AERP) model.

13 brillation (AF)-induced shortening of atrial effective refractory period (AERP), we examined the pote

14 mil has rate-dependent effects on the atrial effective refractory period (AERP).

15 Atrial effective refractory period, AF inducibility, and durati

16 ardioversion to sinus rhythm included atrial effective refractory periods, AF cycle lengths, left atr

17 tential duration and conduction time and the effective refractory period after delivery of the basic

18 ially excitable EBZ, pinacidil shortened the effective refractory period and abolished conduction blo

19 Effective refractory period and APD are closely related

20 he effects of AP14145 and vernakalant on the effective refractory periods and acute burst pacing-indu

21 matic children had similar accessory pathway effective refractory periods and supraventricular tachyc

22 to 5% increases in infarct zone ventricular effective refractory period, and 4% to 6% increases in Q

23 The changes in AVNW-CL, AV nodal effective refractory period, and ventricular response du

24 oupling interval, from 2 to 45 ms beyond the effective refractory period, and was associated with uni

25 he atria to investigate conduction patterns, effective refractory periods, and inducibility of AF.

26 the pacing site and the other MAPs, and PRR (effective refractory period-APD90=PRR) and related to th

27 urately reproduced AP shortening and reduced effective refractory period associated with altered IKs

28 with organized atrial electrograms and long effective refractory periods associated with disorganize

29 rial effective refractory period, with short effective refractory periods associated with organized a

30 The effective refractory period at the high right atrium rem

31 ed, an effect not related to a change in the effective refractory period at the site of block.

32 /-554 versus 376 +/- 466 ms; P=0.86), atrial effective refractory periods at 90 bpm (250+/-32 versus

33 The difference in the effective refractory period between the high right atriu

34 siological changes in heart rates and atrial effective refractory period, but both significantly incr

35 ne was associated with a prolongation of the effective refractory period by 18 +/- 2 ms (P < .05), an

36 Current treatments extend the atrial effective refractory period by nonselective blockade of

37 by 17%, and APD(-61 mV) (reflecting cellular effective refractory period) by 22% (P < 0.05 for each).

38 ore normal areas of the EBZ, nor was the EBZ effective refractory period changed.

39 ogy study for 45 minutes to determine atrial effective refractory periods, conduction velocity, condu

40 electrograms (type I) and the longest atrial effective refractory period corresponding to disorganize

41 refractory period, with the shortest atrial effective refractory period corresponding to organized a

42 The effective refractory period data were used to determine

43 Concurrently, the right effective refractory period decreased.

44 dent prolonged action potential duration and effective refractory period, decreased LSG function were

45 The effective refractory period difference between the sites

46 applied to the site with the shortest atrial effective refractory period, disorganized atrial electro

47 rolonged the antegrade atrioventricular node effective refractory period (ERP) (from 252+/-60 to 303+

48 heterogeneity (p < 0.001); no change in the effective refractory period (ERP) (p > 0.8) or ERP heter

49 t ventricular (RV) and left ventricular (LV) effective refractory period (ERP) and absolute refractor

50 30 minutes, and their effects on ventricular effective refractory period (ERP) and arrhythmia develop

51 otential duration (APD90), right ventricular effective refractory period (ERP) and blood pressure mea

52 Atrial fibrillation (AF) shortens the atrial effective refractory period (ERP) and predisposes to fur

53 Atrial fibrillation (AF) shortens the atrial effective refractory period (ERP) and predisposes to fur

54 Standard restitution, effective refractory period (ERP) and VF threshold (VFT)

55 rdings provide a surrogate for measuring the effective refractory period (ERP) in human ventricle.

56 ct of atrial fibrillation (AF) on the atrial effective refractory period (ERP) in humans is unknown.

57 ced blockade of membrane currents on APD and effective refractory period (ERP) in rat endocardial and

58 ing cycle length, obese patients had shorter effective refractory period (ERP) in the left atrium (25

59 tion of the action potential duration and/or effective refractory period (ERP) is thought to decrease

60 n AP profile, AP duration (APD) restitution, effective refractory period (ERP) restitution, and condu

61 The effect of effective refractory period (ERP) shortening on the vuln

62 imals had repeat attempts at inducing AF and effective refractory period (ERP) testing.

63 shorter action potential duration (APD) and effective refractory period (ERP) than a noninducing sit

64 LAA effective refractory period (ERP) was measured before an

65 maintaining AF and the width, area, weight, effective refractory period (ERP), and wavelength in atr

66 Atrial effective refractory period (ERP), conduction velocity,

67 Action potential durations (APD(50,75,90)), effective refractory period (ERP), post repolarization r

68 While approaching the effective refractory period (ERP), the tissue response i

69 AP duration (APD), conduction velocity (CV), effective refractory period (ERP), tissue excitation thr

70 atrial fibrillation interval (AFI) and local effective refractory period (ERP).

71 as associated with attenuation of the atrial effective refractory period (ERP).

72 I, we constructed restitution curves for the effective refractory period (ERP).

73 n potentials (APs) at 90% repolarization and effective refractory periods (ERPs) (60 +/- 1 ms vs. 44

74 Effective refractory periods (ERPs) were determined at 5

75 The effective refractory periods (ERPs) were measured in the

76 /kg) and propranolol (0.1 mg/kg), and atrial effective refractory periods (ERPs) were obtained at bas

77 r limit of the AF vulnerability zone and the effective refractory period for a BCL, decreased as BCL

78 ular action potentials, resulting in shorter effective refractory periods, greater beat-to-beat varia

79 ular action potentials, resulting in shorter effective refractory periods, greater beat-to-beat varia

80 dial APD90, endocardial APD90 or ventricular effective refractory period in Scn5a+/Delta and WT heart

81 VIP shortens atrial effective refractory periods in dogs.

82 n5a+/Delta hearts, and prolonged ventricular effective refractory periods in initially non-arrhythmog

83 At the first-degree AV block dose, AVN effective refractory period increased from 186+/-37 to 2

84 Effective refractory periods increased from 149+/-16 to

85 ned by measuring prolongation of ventricular effective refractory period induced by bilateral vagal s

86 Effective refractory period is most closely reflected by

87 Given its association with a reduced effective refractory period, it may contribute to the su

88 type I ECG, history of syncope, ventricular effective refractory period <200 ms, and QRS fragmentati

89 n/rapid atrial pacing</=250 ms (or antegrade effective refractory period</=250 ms if shortest preexci

90 dicting VF identified an optimal anterograde effective refractory period of the accessory pathway cut

91 ersus 432 +/- 104 ms, P < .0001), as did the effective refractory period of the AV node (279 +/- 60 v

92 The relative and effective refractory period of the His-Purkinje system i

93 ersistent AF had shorter left atrial APD and effective refractory period (p = 0.01).

94 is demonstrated that short accessory-pathway effective refractory period (P<0.001) and atrioventricul

95 arrhythmias showed shorter accessory-pathway effective refractory period (P<0.001) and more often exh

96 pressure (P<0.0003), and reduction in atrial effective refractory periods (P<0.0001) compared with co

97 on potential upstroke, a prolongation of the effective refractory period secondary to the development

98 The atrial effective refractory period shortened in ATR and CAF gro

99 Atrial effective refractory period shortened progressively from

100 After establishing chronic AF, atrial effective refractory period shortening, increases in spo

101 Using vagally induced atrial effective refractory period shortening, slowing of spont

102 reentrant circuit, the resulting changes in effective refractory periods tend to stabilize reentry i

103 with programmed extra stimuli at 10 ms above effective refractory period than with stable pacing (13.

104 d to the site of shortest and longest atrial effective refractory periods until atrial fibrillation i

105 We also measured ventricular effective refractory period (V-ERP) and QT interval in s

106 g ventricular fibrillation (VF), ventricular effective refractory period (VERP) and defibrillation th

107 tivity via a prolongation of the ventricular effective refractory period (VERP) in the models, althou

108 c Scn5a+/- hearts, and prolonged ventricular effective refractory periods (VERPs) in non-arrhythmogen

109 with programmed extra stimuli at 10 ms above effective refractory period versus 66.1 +/- 22.9 ms with

110 n of atrial electrogram type with the atrial effective refractory period was further demonstrated by

111 wave duration, but not differences in atrial effective refractory periods, was associated with the de

112 that AV nodal function and right ventricular effective refractory period were impaired in the mutant

113 rogram of atrial fibrillation and the atrial effective refractory period were obtained from multiple

114 Resetting response curves and atrial effective refractory periods were determined with single

115 Effective refractory periods were increased homogeneousl

116 re observed at sites with the longest atrial effective refractory period, whereas 1:1 atrial capture

117 specific location are related to the atrial effective refractory period, with short effective refrac

118 am types closely followed that of the atrial effective refractory period, with the shortest atrial ef