ライフサイエンスコーパス: egg laying

1 d pupariation occurring only at day 14 after egg laying.

2 at mediate this clozapine-specific effect on egg laying.

3 etry (GC/MS), also correlated with decreased egg laying.

4 gnal imparted to the egg during ovulation or egg laying.

5 rootworm that had lost fidelity to maize for egg laying.

6 he onset of bursting activity 17 hours after egg laying.

7 serotonergic inputs that combine to modulate egg laying.

8 he serotonergic HSN motor neurons to control egg laying.

9 l for a proper uterine-vulval connection and egg laying.

10 ndocrine cells but has no detectable role in egg laying.

11 dant heteromeric channels prevents premature egg laying.

12 ges are likely to be related to the onset of egg laying.

13 ly couple to a serotonin receptor to mediate egg laying.

14 two dominant mutations that severely inhibit egg laying.

15 e signal (attractin) that is released during egg laying.

16 s that egl-30 requires serotonin to regulate egg laying.

17 eromone attractin), which is released during egg laying.

18 neurons could also disrupt the inhibition of egg laying.

19 ing genes have well characterized defects in egg laying.

20 tations or the inhibitor aldicarb, decreased egg laying.

21 r other genes reported to lack inhibition of egg laying.

22 e the rate and alter the temporal pattern of egg laying.

23 cy of reproductive behaviors associated with egg laying.

24 vation did not, but rather yielded defective egg laying.

25 rease the value of the sucrose substrate for egg laying.

26 ufficient in the egg-laying muscles to limit egg laying.

27 ficantly in their influence on the timing of egg laying.

28 n decreased sexual receptivity and increased egg laying.

29 SP retained in the female did not influence egg laying.

30 P in females and its quantitative effects on egg laying.

31 movement and pharyngeal pumping, and delayed egg-laying.

32 ns redundantly with acetylcholine to inhibit egg-laying.

33 ng, and queens that mated once and initiated egg-laying.

34 PDZ domain 10 also inhibits 5-HT stimulated egg-laying.

35 reduce their sexual receptivity and increase egg laying [1, 2].

36 ) signaling network regulates locomotion and egg laying [1-8].

37 The egg-laying abnormal-9 (EGLN) prolyl hydroxylases have be

38 protein pheromones that are released during egg laying act in concert to stimulate mate attraction.

39 Subsequently, development of larvae into egg-laying adult hookworms (patency) coincided with a sw

40 e reproductive tract after mating and female egg laying after copulating with virgin males.

41 Like many behaviors, Caenorhabditis elegans egg laying alternates between inactive and active states

42 Birds stand out from other egg-laying amniotes by producing relatively small number

43 y to determine incubation periods in extinct egg-laying amniotes has implications for dinosaurian emb

44 showed defects in locomotion, body size, and egg laying and an inability to form dauer larvae.

45 tion mutations in clh-3 lead to misregulated egg laying and an increase in HSN excitability, indicati

46 esponsible for a dramatic increase in female egg laying and decrease in female receptivity after copu

47 s post-mating responses, including increased egg laying and decreased sexual receptivity, in the mate

48 5-HT-stimulated egg laying and egg retention correlated well with differ

49 shows a partial defect in the inhibition of egg laying and fails to respond to aldicarb.

50 embryonically gex-3 activity is required for egg laying and for proper morphogenesis of the gonad.

51 rolled by species-specific physiology, e.g., egg laying and grazing.

52 d fertility as a result of reduced levels of egg laying and hatching, and developing egg chambers dis

53 This mutation inhibits egg laying and inhibits HSN activity by decreasing its e

54 morphic behaviors of Caenorhabditis elegans, egg laying and male ventral tail curling.

55 ing effects on many behaviors in addition to egg laying and may generally act, as they do in the egg-

56 Interestingly, 5-HT inhibits egg laying and pharyngeal pumping in ser-7 null mutants

57 s essential for the 5-HT stimulation of both egg laying and pharyngeal pumping, but that other signal

58 ed to water-filled human-made containers for egg laying and production of progeny.

59 It also shows that egg laying and refractory period response to SP is at le

60 Male-derived Sex-peptide (SP) elicits egg laying and rejection of courting males in mated Dros

61 ch a disproportionate number were related to egg laying and were expressed in neurons and/or muscle.

62 idence of males) gene partially suppress the egg-laying and connection-of-gonad morphology defects ca

63 elayed insect development and reduced female egg-laying and egg hatching.

64 d adverse effects of imidacloprid on queens (egg-laying and locomotor activity), worker bees (foragin

65 treetlights influence timing of dawn chorus, egg-laying and male success in siring extra-pair young,

66 tic manipulations of the retina suggest that egg-laying and movement aversion of UV are both mediated

67 em to an artificial mated status, triggering egg-laying and reducing susceptibility to copulation.

68 gnificantly impairs embryonic morphogenesis, egg laying, and egg hatching even in mutants lacking the

69 is a central pathway for driving locomotion, egg laying, and growth in Caenorhabditis elegans, where

70 pha(q) signaling that drives the locomotion, egg laying, and growth of the animal.

71 , including embryogenesis, cuticle turnover, egg laying, and oocyte maturation.

72 ht activity, swarming, mating, host seeking, egg laying, and sugar feeding.

73 apoptosis in developing embryos, diminished egg-laying, and gross pattern abnormalities in dorsal lo

74 cus on the circuits that control locomotion, egg-laying, and male mating behaviors and their modulati

75 queens that mated once but did not initiate egg-laying, and queens that mated once and initiated egg

76 ells, and severer impairments in locomotion, egg-laying, and survival in Caenorhabditis elegans.

77 predominant lipoprotein found in the yolk of egg-laying animals and is involved in lipid and metal st

78 mitter(s) and cell(s) that signal to inhibit egg laying are not known.

79 eurons (HSNs), the executive neurons driving egg-laying, are tonically inhibited when CO2 is elevated

80 tors mediate avoidance of LPS in feeding and egg laying assays.

81 outputs, we propose that the interaction of egg-laying attraction and positional aversion for AA pro

82 contrast, pharyngeal taste cells mediate the egg-laying attraction to lobeline, as determined by anal

83 UV spectral preference, are dispensable for egg-laying aversion but essential for movement aversion

84 First, females exhibit egg-laying aversion of UV: they prefer to lay eggs on da

85 prove understanding of the chemical basis of egg laying behavior of Ae. aegypti, and the kairomones w

86 (HSNs) that stimulate Caenorhabditis elegans egg-laying behavior and discovered mutations in a potass

87 ablished serotonergic response in C. elegans egg-laying behavior as a paradigm, we show that action o

88 Egg-laying behavior in Caenorhabditis elegans is activat

89 Egg-laying behavior in Caenorhabditis elegans is regulat

90 ating induces changes in the receptivity and egg-laying behavior in Drosophila females, primarily due

91 sole, though they had only subtle effects on egg-laying behavior in the absence of drug.

92 Caenorhabditis elegans egg-laying behavior is inhibited by neurotransmitter sig

93 However, the neural basis of egg-laying behavior is still not well understood; in par

94 Egg-laying behavior of the Caenorhabditis elegans hermap

95 e HSN neurons play a central role in driving egg-laying behavior through direct excitation of the vul

96 lack of HSN inhibition, resulting in normal egg-laying behavior.

97 n tph-1 expression were sufficient to affect egg-laying behavior.

98 of serotonergic motor neurons that stimulate egg-laying behavior.

99 ically in the HSNs was sufficient to inhibit egg-laying behavior.

100 nervous system and in the vulval muscles for egg-laying behavior.

101 s, including EGL-47, to set the frequency of egg-laying behavior.

102 e may be multiple functions for this gene in egg-laying behavior.

103 holine released from the VC neurons inhibits egg-laying behavior.

104 to act in octopaminergic neurons to control egg-laying behavior.

105 e-specific neurons (HSNs), which control the egg-laying behavior.

106 cts cell autonomously in the HSNs to inhibit egg-laying behavior.

107 ommunity influences Drosophila olfactory and egg-laying behaviors differently than individual members

108 at these infections significantly accelerate egg laying but do not induce cytoplasmic incompatibility

109 nts of a nicotinic receptor that can promote egg laying but is not necessary for egg-laying muscle co

110 er-7 null mutants and the 5-HT inhibition of egg laying, but not pumping, is abolished in ser-7(tm132

111 Our results suggest that EGL-47 regulates egg laying by activating Galpha(o) in the HSN motor neur

112 Caenorhabditis elegans regulates egg laying by alternating between an inactive phase and

113 hese data indicate that serotonin stimulates egg laying by directly modulating the functional state o

114 rimental outcomes than did oligidic food for egg laying by females.

115 lev-1, were essential for the stimulation of egg laying by levamisole, though they had only subtle ef

116 ity, indicating that these channels modulate egg laying by limiting HSN excitability.

117 fected host larvae were still acceptable for egg laying by parasitoids, and the parasitoids thereafte

118 to the blood, where they are acquired during egg-laying by parasitic wasps that transmit the virus.

119 nce, by decreasing mosquito survivorship and egg laying capacity.

120 ied side-by-side in three different lines of egg-laying chickens.

121 ogenetic analyses of the worm motor circuit, egg-laying circuit and mechanosensory circuits that have

122 al nature of the synaptic connections in the egg-laying circuit remain uncharacterized.

123 The VC neurons play a dual role in the egg-laying circuit, exciting the vulval muscles while fe

124 fects on specific neurons and muscles in the egg-laying circuitry have been difficult to determine.

125 sis for cholinergic neurotransmission in the egg-laying circuitry is not well understood.

126 mediate cholinergic neurotransmission in the egg-laying circuitry.

127 tion by B. megaterium was linked to impaired egg laying, corresponding to a known trade-off between f

128 The 5-HT sensitivity of egg laying could be restored by ser-5 muscle expression.

129 ists have traditionally assumed that in each egg-laying cycle mosquitoes take a single bloodmeal that

130 ogy through development of a simple model of egg-laying date evolution, parameterized using data from

131 ity territories were associated with earlier egg-laying dates and larger eggs.

132 The distributions of egg-laying dates were more constricted in the warmest (a

133 nk food-odor perception with aggregation and egg-laying decisions.

134 The egg laying defect could be rescued by the expression of

135 hibition of 5-HT metabolism also rescued the egg laying defect.

136 w genes from a screen for suppressors of the egg-laying defect associated with elevated lin-12 activi

137 dentified in a screen for suppressors of the egg-laying defect associated with hypermorphic alleles o

138 of the N'3 variant leads to paralysis and an egg-laying defect in the adult, suggesting a deficit in

139 We found that the sel-12 egg-laying defect was partially rescued by expression of

140 ic transmission and suppressed the lethargy, egg-laying defect, and deficient neurotransmitter releas

141 e stack, resulting in a blocked lumen and an egg-laying defect.

142 units OCR-1 and -4, resulting in a premature egg-laying defect.

143 notype, aph-2 mutant animals also display an egg-laying defect.

144 r defect is related to the SEL-12/presenilin egg-laying defect.

145 xylase domain protein 2 (PHD2, also known as Egg Laying Defective Nine homolog 1) is a key oxygen-sen

146 We obtained an egg-laying defective (Egl) mutant in which the AC fails

147 lin-11 mutant animals are egg-laying defective because of the abnormalities in vul

148 We isolated cog-3(ku212) as a C. elegans egg-laying defective mutant that is associated with a co

149 Previously, a screen for suppressors of the egg-laying defective phenotype caused by partial loss of

150 ake a functional connection, resulting in an egg-laying defective phenotype.

151 s die by necrosis and the mutant animals are egg-laying defective.

152 rate that grk-2 loss-of-function strains are egg laying-defective and contain low levels of serotonin

153 Here we show that the egg laying-defective mutant egl-6(n592) carries an activ

154 HD, also known as HIF prolyl hydroxylase and egg laying-defective nine protein) site specifically hyd

155 clear migration results in uncoordinated and egg-laying-defective animals.

156 Analysis of egg-laying-defective mutants has provided insight into a

157 conserved prolyl residues by members of the egg-laying-defective nine (EGLN) family.

158 The eggshell and egg-laying defects of cuff mutants are suppressed by a m

159 ations that enhance the excretory system and egg-laying defects of hypomorphic lin-45 raf mutants.

160 The egg-laying defects of unc-4, cha-1, and unc-17 were resc

161 ct NAD(+), whereas the uv1 cell necrosis and egg-laying defects result from accumulation of the subst

162 tion of the egl-47 gene caused no detectable egg-laying defects, suggesting that EGL-47 functions red

163 hat are defective in HLH-17 via RNAi display egg-laying defects, while those carrying null mutations

164 earance, while hsf-1 and sup-45 mutants have egg-laying defects.

165 oduced a pleiotropic phenotype that included egg-laying defects.

166 ex muscles led to uncoordinated movement and egg-laying defects.

167 This study suggests that egg-laying demand can temporarily convert UV into an ave

168 nal tracking of single females suggests that egg-laying demand increases their tendency to turn away

169 ies exhibit UV aversion in response to their egg-laying demand.

170 Activation of egg laying depends on the serotonergic hermaphrodite-spe

171 irst established at approximately 23 h after egg laying during Drosophila testis morphogenesis.

172 In a field study, egg-laying E. solidaginis females discriminated against

173 TGF-beta Dauer pathway that is masked by an egg-laying (Egl) phenotype; mutants in the pathway displ

174 the sucrose substrate frequently before most egg-laying events, suggesting that they actively suppres

175 st, the 3rd instar larvae (90-94 hours after egg laying) expressing a mutated form of human alpha-Syn

176 How egg-laying females respond to UV light is not known, how

177 Mutations that alter egg-laying frequency have identified genes encoding a nu

178 Male birds of egg-laying hen strains have no commercial value and are

179 focuses on sex-specific neurons such as the egg laying hermaphrodite-specific neurons (HSNs), VCs, a

180 in pairs and were associated with successful egg-laying, highlighting a potential fitness relevance o

181 at is associated with prolonged secretion of egg-laying hormone (ELH) from peptidergic neurons in int

182 The egg-laying hormone ovulin (Acp26Aa) is among the most ra

183 ollect and concentrate peptide release, with egg-laying hormones and acidic peptide detected.

184 rs such as nicotine and levamisole stimulate egg laying; however, the genetic and molecular basis for

185 dies reveal that 5-HIAA functions to inhibit egg laying in a manner dependent on the 5-HT receptor SE

186 Clozapine stimulated egg laying in C. elegans in a dose-dependent manner.

187 Egg laying in Caenorhabditis elegans has been well studi

188 5-HT) stimulates both pharyngeal pumping and egg laying in Caenorhabditis elegans.

189 Loss of ser-1 function leads to decreased egg laying in hermaphrodites and defects in the turning

190 With a model on female egg laying in insects, we demonstrate how variation in b

191 choose a plain (sucrose-free) substrate for egg laying in our sucrose versus plain decision assay.

192 For example, 5-HT did not stimulate egg laying in ser-1, ser-7, or ser-7 ser-1 null animals,

193 In contrast, 5-HT-stimulated egg laying in ser-4;mod-1 animals was greater than in wi

194 5-HT also fails to stimulate egg laying in ser-7(tm1325), ser-1(ok345), and ser-7(tm1

195 l fluid proteins stimulate sperm storage and egg laying in the mated female but also cause a reductio

196 Serotonin (5-HT) stimulation of egg-laying in Caenorhabditis elegans is abolished in ser

197 n, vitellogenin (Vg), which is necessary for egg-laying in queens, brood food production in workers,

198 mpz-1 RNAi has no effect on 5-HT stimulated egg-laying in ser-1 mutants rescued by expression of a t

199 Ns), a homologous pair of cells required for egg-laying in the hermaphrodite.

200 mpz-1 RNAi reduces 5-HT stimulated egg-laying in wild type animals and in ser-1 mutants res

201 Indeed, body touch appears to inhibit egg laying, in part by interfering with HSN calcium osci

202 gh the vulva and release tyramine to inhibit egg laying, in part via the LGC-55 tyramine-gated Cl(-)

203 rent downstream targets to control different egg-laying-induced behavioral modifications.

204 Egg-laying involves a simple motor program involving a s

205 Egg laying is inhibited by the G protein Galphao and act

206 SER-1-dependent withdrawal response in which egg laying is significantly decreased.

207 e genomes of two species of mongooses and an egg-laying mammal called an echidna show that a virus cu

208 the embryonic pattern in modern monotremes (egg-laying mammals) and placental mammals, but is a paed

209 ting states," with two extremes (virgins and egg-laying, mated queens) that differ dramatically in th

210 e absence of synaptic input, suggesting that egg laying may be controlled through modulation of auton

211 that both genes are required maternally for egg laying, mitotic progression in early embryos, and em

212 is also present in the duckbill platypus, an egg-laying monotreme, consistent with TCRmu being ancien

213 The egg-laying motor circuit comprises a simple three-compon

214 in-coupled receptor that inhibits C. elegans egg-laying motor neurons in a G(o)-dependent manner.

215 its to it, but by actively suppressing their egg-laying motor program during their visits.

216 e found that the spontaneous activity of the egg-laying motorneurons was silenced by serotonin.

217 rgic VC motor neurons slow locomotion during egg-laying muscle contraction and egg release.

218 promote egg laying but is not necessary for egg-laying muscle contraction.

219 annels localize to postsynaptic sites in the egg-laying muscle, and mutants lacking ERG have more fre

220 ns, which synapse onto both the HSNs and the egg-laying muscles and are thus the third cell type comp

221 t calcium transients and contractions of the egg-laying muscles even during the inactive phase.

222 speed video recording and calcium imaging of egg-laying muscles in behaving animals, we found that th

223 function is necessary and sufficient in the egg-laying muscles to limit egg laying.

224 e-specific neurons (HSNs) that innervate the egg-laying muscles to stimulate contraction.

225 kdown results in a defect in the function of egg-laying muscles.

226 ate behavior by limiting excitability of the egg-laying muscles.

227 kes calcium transients or contraction of the egg-laying muscles.

228 drives twitching or full contraction of the egg-laying muscles.

229 egulating serotonin response pathways in the egg-laying neuromusculature.

230 red the responses of both vulval muscles and egg-laying neurons to serotonin; specifically, mutations

231 the schistosome larvae and markedly impaired egg laying of adult worm pairs maintained in culture.

232 lity" of the sucrose-containing medium as an egg-laying option to the reproductive system depends on

233 that the value of a sucrose substrate- as an egg-laying option-can be adjusted by the activities of a

234 whether male SP expression influences female egg laying or if any effect of SP is mediated by SP rete

235 ions in parallel with egl-30 with respect to egg laying or is not the major effector of EGL-30.

236 iety of species, mismatches in the timing of egg laying or numbers of offspring have had relatively l

237 , but found no additional Acps that affected egg laying or receptivity upon ectopic expression.

238 tween humidity and gathering nest materials, egg laying, or changes in rates of vocalization.

239 f either unmated queens (n = 8) with induced egg laying, or queens (n = 12) mated in isolation with d

240 lation between male SP expression levels and egg laying, or the amount of SP in the female reproducti

241 In birds, maternal effects on egg-laying order and offspring growth, two proximate det

242 f female mammals, with much less known about egg-laying organisms or paternally-mediated effects.

243 gy from 280 plant and insect species and the egg-laying phenology of 21 British songbird species, we

244 Here we describe the egg-laying phenotypes of eight levamisole resistance gen

245 strinae): a helper's chance of inheriting an egg-laying position, and the workforce available to rear

246 neurons compete to either enhance or inhibit egg-laying preference for ethanol-containing food.

247 However, our observations reveal that this egg-laying preference is a complex process, as it direct

248 Egg-laying preference provided a context-dependent fitne

249 rning and decision center, affected females' egg-laying preferences in our sucrose versus plain assay

250 n (Acp26Aa), stimulates an early step in the egg-laying process, the release of oocytes by the ovary.

251 lie the transition from a virgin to a mated, egg-laying queen.

252 queens than those in workers and virgin, non-egg-laying queens, even if methoprene or precocene treat

253 than mortality of bee larvae or reduction in egg laying rate.

254 r of ovarioles per ovary strongly influences egg-laying rate and fecundity.

255 ostmating response (PMR) comprises increased egg-laying rate and reduced sexual receptivity and is co

256 We show that peptidoglycan regulates egg-laying rate by activating NF-kappaB signaling pathwa

257 ve workers, and foragers, as well as reduced egg-laying rate, could impact colony dynamics over multi

258 as developmental progression, body size, and egg-laying rate.

259 ion to AA-containing food for the purpose of egg-laying relies on the gustatory system, while positio

260 ractoriness to further mating and triggering egg-laying, remains elusive.

261 This inhibition of egg laying requires MOD-1, a 5-HT-gated chloride channel

262 we found that clozapine-induced increase in egg laying requires tyramine biosynthesis.

263 between repulsive positional and attractive egg-laying responses toward the bitter-tasting compound

264 We found that selection of egg-laying site in Drosophila melanogaster is a suitable

265 These findings suggest that selection of egg-laying site involves a simple decision-making proces

266 Drosophila melanogaster egg-laying site selection offers a genetic model to stud

267 tive about the chemosensory quality of their egg-laying sites, an important trait that promotes the s

268 ence of food, potential mates, or attractive egg-laying sites.

269 wing that inbreeding cost is reduced in some egg-laying species by postcopulatory mechanisms that fav

270 This constraint is relaxed in egg-laying species, which are sensitive to hormones duri

271 nce of the two commonest wasp species at the egg-laying stage and favours the pollinators.

272 no significant effect upon blood feeding or egg laying success.

273 ive queue and that PQs take over the role of egg-laying, successively, without overt conflict, as the

274 lar mechanisms of presynaptic development in egg-laying synapses of Caenorhabditis elegans, demonstra

275 The egg-laying system of Caenorhabditis elegans hermaphrodit

276 CO2 modulates the egg-laying system partly through the AWC olfactory neuro

277 A functional egg-laying system requires cell fate specification event

278 ays multiple roles in the development of the egg-laying system, acting to both promote cell fate and

279 ing and may generally act, as they do in the egg-laying system, to integrate multiple signals and con

280 n coordinating development of the C. elegans egg-laying system.

281 are thus the third cell type comprising the egg-laying system.

282 gulation of lin-11 during development of the egg-laying system.

283 ent modules and together specify a wild-type egg-laying system.

284 indgut, but not in other tissues such as the egg-laying system.

285 Three weeks after the onset of egg laying, T-cell responsiveness began to increase and

286 eromones are predicted to be released during egg laying that act in concert with albumen gland attrac

287 also reveals an inhibitory effect of 5-HT on egg laying that is normally masked by SER-1-dependent st

288 ls and exhibited significant 5-HT-stimulated egg laying that was dependent on a previously uncharacte

289 e plays a specific role in the inhibition of egg laying, the modulation of reversal behavior, and the

290 Among the best studied behaviors is egg-laying, the process by which hermaphrodites deposit

291 EGL-47 functions redundantly, or it inhibits egg laying under specific circumstances as yet unidentif

292 mote release of neurotransmitters that block egg laying until eggs filling the uterus deform the neur

293 or indirectly to the risk of Se toxicity to egg-laying vertebrates (fish and piscivorous birds) in S

294 mes available to toxicologically susceptible egg-laying vertebrates.

295 Further, fluoxetine can stimulate egg laying via the Gq protein EGL-30, independent of SER

296 5-HT-stimulated egg laying was absent in ser-5;ser-4;mod-1;ser-7 ser-1 a

297 s that contributed to the 5-HT modulation of egg laying were identified in Caenorhabditis elegans.

298 vior of the nematode Caenorhabditis elegans, egg laying, which is driven by a pair of serotonergic ne

299 ned the behavior and physiology of queenless egg-laying workers.

300 single egg layer (queen) and a number of non-egg-laying workers.