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1                                              ego-1 interacts genetically with him-17, another regulat
2                                              ego-2 activity also promotes aspects of development not
3 kers to derive continuous measurements about ego motion from ground contacts.
4 ocytes, the astrocyte even takes on an alter ego of the stem cell itself (S. Goldman, this issue of T
5 ntinuum between the fibroblast and its alter ego, the myofibroblast.
6 t CSR-1 directly binds to histone mRNA in an ego-1-dependent manner using biotinylated 2'-O-methyl RN
7               Here, we show that atx-2 is an ego gene.
8 es of such changes on the distribution of an ego's network ties are not well understood.
9  "I" accounting for individual behavior, and ego psychologists' concepts of the organizing functions
10 ttern of pregnancy denial, dissociation, and ego disorganization.
11 resources by the pressures of reputation and ego might interfere with the thought processes needed to
12 ent a complex phenotypic interaction between ego-2 and alx-1, consistent with their relationship bein
13  symptoms, global functioning, self-concept, ego defenses, work and social functioning, and readmissi
14 le in the recovery of object movement during ego movement.
15 ary psychological/physical resources (i.e., "ego depletion").
16 bit system may play a key role in explaining ego-dystonic or self-destructive behaviour.
17 se of young children and the case of fragile egos.
18   For a number of germ-line-expressed genes, ego-1 mutants were resistant to a form of PTGS called RN
19                         Notably, for a given ego, these social signatures tend to persist over time,
20 of small RNAs and the rise of mRNA levels in ego-1(-) animals.
21 nd centrality measures as well as individual ego measures and further constructed sociograms.
22 uct problems, oppositional behavior, and low ego control; withdrawal or passivity (12 studies), inclu
23            We first sample a set of multiple ego networks of varying orders that form a patch, or a n
24 -1 gene structure and the molecular basis of ego-1 alleles.
25 cost model offers an ultimate explanation of ego depletion that helps to move the field beyond biolog
26 sentation of object-centered and not just of ego-centered space.
27 re, we provide more insight into the role of ego-1 in germ-line development.
28             Here, we investigate the role of ego-1 in germline proliferation.
29 x (RSC) correlated strongly with ratings of "ego-dissolution" and "altered meaning," implying the imp
30 r LSD correlated with subjective reports of "ego dissolution." The present results provide the first
31 network maintained by a focal individual, or ego, is intrinsically dynamic and typically exhibits som
32 ar circuit for the maintenance of "self" or "ego" and its processing of "meaning." Strong relationshi
33  mortality produce larger numbers of kin per ego and decrease the inequality of the distribution of k
34 onism, neuroticism, highly sensitive person, ego resiliency, need for structure, and negative emotion
35                                     Putative ego-1 null mutants had multiple, previously unreported d
36                                      Putting ego aside, the 'astrocyte' is also (and perhaps more imp
37 r-1, the RNA-dependent RNA polymerase (RdRP) ego-1, or the dicer-related helicase drh-3, leads to def
38             We previously identified several ego (enhancer of glp-1) genes that promote germline prol
39                           Here, we show that ego-2 positively regulates signaling in various tissues
40                                          The ego-1 gene is also required for a robust response to RNA
41                                          The ego-1 gene is the first example of a gene encoding an Rd
42                                          The ego-1 gene was originally identified on the basis of gen
43                                          The ego-1 transcript was found predominantly in the germ lin
44                            Stimulated by the ego-dystonic nature of obsessive-compulsive disorder (OC
45                       We have determined the ego-1 gene structure and the molecular basis of ego-1 al
46                 We originally identified the ego-1 gene on the basis of a genetic interaction with gl
47                 We previously identified the ego-2 (enhancer of glp-1) gene as a positive regulator o
48 le turnover in the identity of alters in the ego network.
49 alls and survey data to track changes in the ego networks and communication patterns of students maki
50 n which they transcend the boundaries of the ego and the confines of time and space.
51 cal systems and that his descriptions of the ego are consistent with the functions of the default-mod
52 cells and for the nature and severity of the ego- mutant defect.
53  concepts of the organizing functions of the ego.
54 ownstream neurons driving motor responses to ego-rotation receive inputs primarily from a small subse

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