ライフサイエンスコーパス: egression

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1 g(C/-) splenocytes, which impeded lymphocyte egression.

2 ant airway MMP controlling inflammatory cell egression.

3 at a soluble factor(s) regulates the rate of egression.

4 n some instances, IFN-gamma induced parasite egression.

5 ation, but the underlying mechanisms of this egression are unclear.

6 Blocking ICAM-2 inhibits egression by 50.95 +/- 10.79% (P=0.04), and blocking bot

7 and blocking both ICAM-1 and ICAM-2 inhibits egression by 69.6 +/- 5.2% (P< 0.0001).

8 t although inhibition of T-cell LFA-1 blocks egression by 75 +/- 5.6% (P<0.0001), inhibition of the L

9 We demonstrate an egression-dependent decrease in transepithelial resistan

10 ial epithelium, but we provide evidence that egression does not require epithelial injury, and can ta

11 enhance tissue damage and eventual bacterial egression from the apoplast to the leaf surface.

12 The process of egression has been relatively overlooked when considerin

13 Egression is important for immune surveillance and the r

14 site movement and IFN-gamma-induced parasite egression occur.

15 ore, both colchicine and taxol prevented the egression of A. actinomycetemcomitans from host cells in

16 We show that egression of human T cells across the bronchial epitheli

17 Egression of inflammatory cells from the lung interstiti

18 matrix metalloproteinase 2 (MMP2) for airway egression of lung eosinophils, a critical anti-inflammat

19 lishes the chemotactic gradient required for egression of lung inflammatory cells and prevention of l

20 omics approach to determine how MMPs promote egression of lung inflammatory cells through the airway.

21 Egression of MP continued throughout the 4-d period exam

22 In conclusion, we show that egression of T cells involves three distinct sequential

23 rational stages appear important for surface egression or intracellular retention of empty HLA-DR.

24 elium and into the airway lumen, is known as egression, or luminal clearance.

25 he isomers is much faster than the entry and egression rates, and that the functional groups at the r

26 The initial phase of this increased rate of egression was inhibited by antibodies to inter- cellular

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