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1 ive neurons in the vestibular portion of the eighth nerve.
2 cleus, nucleus magnocellularis (NM), require eighth nerve activation of metabotropic glutamate recept
3                               Elimination of eighth-nerve activity results in the death of 30% of the
4 se-locked and entrained responses in primary eighth nerve afferents and first-order medullary neurons
5                   Iso-intensity responses of eighth nerve afferents from the sacculus, the main audit
6 ular canals and otoliths, are carried by the eighth nerve and distributed to the four nuclei of the v
7 ase C betaII, as were scattered axons in the eighth nerve, and scattered axons and terminals were fou
8                                          The eighth nerve carries vestibular information to four brai
9 e enhanced temporal fidelity seen across the eighth nerve/cochlear nucleus synapse.
10 amined for loss of spiral ganglion cells and eighth nerve fibers.
11      Our findings not only confirm that each eighth nerve has afferents that drive ipsiversive excita
12 itive units in the posterior division of the eighth nerve have sensory coding properties expected of
13 s magnocellularis (NM) integrates excitatory eighth nerve inputs and depolarizing GABAergic inhibitio
14     Pharmacological activity blockade in the eighth nerve mimicked afferent deprivation for only a su
15  observed in the interstitial nucleus of the eighth nerve, nucleus Y, external cuneate nucleus, and l
16  chemical and electrotonic components of the eighth nerve (NVIII) EPSP recorded in vivo in the goldfi
17 nd primarily dependent on lesions within the eighth nerve or brainstem.
18 , receiving signals either directly from the eighth nerve or indirectly via projections from the VNC.
19 tory and lateral line systems, including the eighth nerve-recipient descending octaval nucleus, the a
20                The surface of neurons in the eighth nerve root and in neighboring nodes of Ranvier st
21 hich is associated with the lateral line and eighth nerve senses, and the posterior portion, which is
22 caudal lobe associated with lateral line and eighth nerve senses.
23                 We present evidence that the eighth nerve tetanus that induces LTP does not act by tr
24  its peripheral and central processes of the eighth nerve to be heavily labeled with protein kinase C
25 A few axons in the vestibular portion of the eighth nerve were labeled with protein kinase C gamma im
26  unilateral cochlea removal or silencing the eighth nerve with tetrodotoxin leads to a loss of 25-30%

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