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1 ive neurons in the vestibular portion of the eighth nerve.
2 cleus, nucleus magnocellularis (NM), require eighth nerve activation of metabotropic glutamate recept
4 se-locked and entrained responses in primary eighth nerve afferents and first-order medullary neurons
6 ular canals and otoliths, are carried by the eighth nerve and distributed to the four nuclei of the v
7 ase C betaII, as were scattered axons in the eighth nerve, and scattered axons and terminals were fou
12 itive units in the posterior division of the eighth nerve have sensory coding properties expected of
13 s magnocellularis (NM) integrates excitatory eighth nerve inputs and depolarizing GABAergic inhibitio
14 Pharmacological activity blockade in the eighth nerve mimicked afferent deprivation for only a su
15 observed in the interstitial nucleus of the eighth nerve, nucleus Y, external cuneate nucleus, and l
16 chemical and electrotonic components of the eighth nerve (NVIII) EPSP recorded in vivo in the goldfi
18 , receiving signals either directly from the eighth nerve or indirectly via projections from the VNC.
19 tory and lateral line systems, including the eighth nerve-recipient descending octaval nucleus, the a
21 hich is associated with the lateral line and eighth nerve senses, and the posterior portion, which is
24 its peripheral and central processes of the eighth nerve to be heavily labeled with protein kinase C
25 A few axons in the vestibular portion of the eighth nerve were labeled with protein kinase C gamma im
26 unilateral cochlea removal or silencing the eighth nerve with tetrodotoxin leads to a loss of 25-30%
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