ライフサイエンスコーパス: ejaculation

1 al stimulation (e.g., mounts, intromissions, ejaculations).

2 of defined spinal segments in the control of ejaculation.

3 olinergic circuits used for intromission and ejaculation.

4 gy is coupled to cellular events involved in ejaculation.

5 o become receptive within 30 min and mate to ejaculation.

6 y and length of intromission, and latency of ejaculation.

7 on T2-weighted images, both before and after ejaculation.

8 the medial preoptic area (MPOA), facilitates ejaculation.

9 ocus must result from events following sperm ejaculation.

10 R imaging while masturbating to the point of ejaculation.

11 he vas deferens during the emission phase of ejaculation.

12 ng copulation, and especially at the time of ejaculation.

13 eurons that express Fos after mating reflect ejaculation.

14 ndent predictor for failure of PVS to induce ejaculation.

15 rugs are approved for treatment of premature ejaculation.

16 nt for men with moderate-to-severe premature ejaculation.

17 dapoxetine in patients with severe premature ejaculation.

18 ehavior, but failed to rescue the deficit in ejaculation.

19 current understanding of central control of ejaculation.

20 n evoked as unconditional responses (URs) to ejaculation.

21 The latter could help to trigger ejaculation.

22 neurons signals information associated with ejaculation.

23 rons is triggered by stimuli associated with ejaculation.

24 nt (ADC) values was applied before and after ejaculation.

25 achieved less intromissions and virtually no ejaculations.

26 g the amount of sperm in a similar number of ejaculations.

27 d intromissions, peaked 2-3 min after male's ejaculation (0.2-0.4 degrees C), and abruptly dropped un

28 dial preoptic area (MPOA) of male rats after ejaculation; (2) whether increasing 5-HT in these sites,

29 , oxymetholone generally failed to stimulate ejaculation above the levels of the oil group.

30 hese percentages are combined with those for ejaculation-activated cells involved in the PdPN and lat

31 Ejaculation-activated cells participate in the PdPN and

32 To identify other potential targets of ejaculation-activated cells, we traced PdPN and lateral

33 Ejaculation-activated PdPN cells project to the AVPv (43

34 Upon ejaculation and during their transit through the female

35 f the bulbospongiosus muscle, which mediates ejaculation and ejaculatory behavior, is markedly dimini

36 eurons were activated in males by display of ejaculation and in females by vaginocervical stimulation

37 fective methods of treatment of disorders of ejaculation and orgasm in men.

38 adulthood, resulting in increased latency to ejaculation and postejaculatory intromission and longer

39 However, the neural substrates mediating ejaculation and processing ejaculation-related signals r

40 In mammals, sperm cells become motile during ejaculation and swim up the female reproductive tract.

41 ivation of PdPN and lateral MeApd neurons at ejaculation and that NOS in PdPN and MPNm cells is regul

42 nsignificantly increased risk for retrograde ejaculation and urogenital problems.

43 uprapubic discomfort or pain during or after ejaculation and voiding complaints such as irritative an

44 return latencies following intromissions and ejaculations and increase withdrawal from the male follo

45 creases during copulation, especially during ejaculation, and increased glutamate facilitates copulat

46 cause its levels in the preoptic area affect ejaculation, and it could synchronize clustered neurons

47 LHAA; no 5-HT increases were observed before ejaculation, and levels were decreased toward basal valu

48 reuptake inhibitors (SSRIs) impair erection, ejaculation, and libido.

49 hieve and maintain an erection, frequency of ejaculation, and orgasm frequency than did patients rece

50 y large increase in samples collected during ejaculation (approximately 300% of BL).

51 ain cells that are activated specifically at ejaculation as assessed by Fos expression.

52 Testosterone cypionate sustained ejaculation at all doses tested.

53 Stanozolol failed to maintain ejaculation at any dose tested.

54 s induction was specifically associated with ejaculation, because mounts or intromissions did not tri

55 ar trends for reductions in intromission and ejaculation behavior.

56 Mammalian spermatozoa become motile at ejaculation, but before they can fertilize the egg, they

57 ular neuron damage; these males copulated to ejaculation, but they had lower intromission ratios and

58 oes not occur by augmenting the frequency of ejaculations, but by increasing the amount of sperm in a

59 on) and consummatory (mounts, intromissions, ejaculations) components of male sexual behavior were me

60 Sexual dysfunction like ejaculation discomfort is described as a symptom of CP/C

61 l contact and others were not, revealed that ejaculation duration was the key factor in the induction

62 that alteration of NTPDase1 activity affects ejaculation efficacy and male fertility.

63 Successful dynamic MR imaging of ejaculation events and the ability to visualize internal

64 ial (first mount or intromission) and final (ejaculation) events of each copulatory cycle, suggesting

65 similarly to the microinjection, increasing ejaculation frequency and decreasing ejaculation latency

66 blocked 8-OH-DPAT's facilitative effects on ejaculation frequency and latency, while the 5-HT1A anta

67 No association was observed between ejaculation frequency in early adulthood and LUTS.

68 Our results suggest that ejaculation frequency is not related to increased risk o

69 Ejaculation frequency was assessed by asking participant

70 Ejaculation frequency was not statistically significantl

71 However, high ejaculation frequency was related to decreased risk of t

72 Most categories of ejaculation frequency were unrelated to risk of prostate

73 ieve erection, ability to maintain erection, ejaculation frequency, orgasm frequency, and sexual desi

74 exually transmitted infections, prostatitis, ejaculation frequency, surgery for an enlarged prostate,

75 A spinal ejaculation generator (SEG) has been identified in the r

76 ulatory behavior and may be part of a spinal ejaculation generator.

77 ncrease in research with regard to premature ejaculation has led to a significant number of new paper

78 dence-based definition of lifelong premature ejaculation has set a model in the evaluation and treatm

79 rm from copulating for several minutes after ejaculation have been identified.

80 ndings in the physiology and neurobiology of ejaculation have expanded our understanding of male sexu

81 cies and intervals between intromissions and ejaculation, higher lordosis quotients and ratings, more

82 areas of the gerbil brain are activated with ejaculation, i.e., the posterodorsal preoptic nucleus (P

83 Activating these Crz INs caused rapid ejaculation in isolated males, a phenotype mimicked by i

84 sal medial amygdala (MeApd) express Fos with ejaculation in male gerbils.

85 sal medial amygdala (MeApd)-are activated at ejaculation in male rats and gerbils as seen with Fos im

86 othesis that these cells are activated after ejaculation in male rats and vaginocervical stimulation

87 ng mounts and pelvic thrusts in females, and ejaculation in males.

88 Men with moderate-to-severe premature ejaculation in stable, heterosexual relationships took p

89 g the gender-specific outcome of copulation: ejaculation in the male and sperm transport in the femal

90 imately 280% of BL), increased the number of ejaculations in the 40 min test, decreased ejaculation l

91 ring subsequent retention testing, and after ejaculation) in experimental rats [that received electro

92 Sexual activity, and especially ejaculation, increased levels of glutamate in the MPOA.

93 In particular, ejaculation-induced Fos expression is expressed in the m

94 nd mating-specific (such as for erection and ejaculation) inputs.

95 Ejaculation is an integral part of normal sexual functio

96 ons playing an pivotal role in expression of ejaculation is reviewed.

97 as been reported for silodosin, but abnormal ejaculation is the most commonly reported adverse effect

98 Ejaculation is the most reinforcing component of sexual

99 es found in the caput are destroyed prior to ejaculation, is a newly discovered function for the epid

100 produce a change in mount, intromission, and ejaculation latency or in mount and intromission frequen

101 f ejaculations in the 40 min test, decreased ejaculation latency, and decreased the postejaculatory l

102 reasing ejaculation frequency and decreasing ejaculation latency, postejaculatory interval and mount

103 us, every PdPN and MeApd cell activated with ejaculation may participate in one of these projections.

104 has been suggested that 5-HT released after ejaculation may promote the sexual quiescence of the pos

105 Similarly, ejaculation of more sperm when males are paired with fem

106 males were removed after the first or second ejaculation or left in the test chamber.

107 Erectile function, arousal, ejaculation, orgasm, and overall satisfaction domain mea

108 CREB activity, however, has no influence on ejaculation parameters.

109 per month compared with men reporting 4 to 7 ejaculations per month at ages 20 to 29 years were 0.89

110 relative risks for men reporting 21 or more ejaculations per month compared with men reporting 4 to

111 In females, only ejaculation preceded by multiple intromissions induced a

112 ncrease in Fos-IR; multiple intromissions or ejaculation preceded by only 0-1 intromission did not af

113 Thus, we determined whether the ejaculation-related cells produce NO by assessing Fos co

114 s minimal in gerbils, involving few, if any, ejaculation-related cells.

115 These cells are positioned to relay ejaculation-related signals from reproductive organs to

116 strates mediating ejaculation and processing ejaculation-related signals remain poorly understood.

117 males tested immediately after receiving two ejaculations showed analgesia.

118 e spinothalamic pathway involved in relay of ejaculation-specific signals is discussed.

119 ed for sexual desire, arousal, and orgasm or ejaculation stages of sexual responding.

120 s arousal, orgasm and the expulsion phase of ejaculation such as functional MRI, dynamic pelvic ultra

121 ired more intromissions at a faster pace per ejaculation than did bLR males.

122 turn to the male after an intromission or an ejaculation, thereby decreasing the percentage of time s

123 ar 5-HT in the MPOA, yet both can facilitate ejaculation, these data suggest that moderate changes in

124 participants to report the average number of ejaculations they had per month during the ages of 20 to

125 apable of fertilizing eggs immediately after ejaculation; they acquire fertilization capacity after r

126 nable to fertilize the egg immediately after ejaculation; they acquire this capacity during migration

127 animals, male seminal fluid coagulates upon ejaculation to form a hardened structure known as a copu

128 Ejaculation was correlated with enhanced 5-HT release fr

129 No changes before and after ejaculation were observed in T1 values or in T2 and ADC

130 s, young-onset prostatitis, and frequency of ejaculation, were investigated in relation to lower urin

131 penile vibratory stimulation (PVS) to elicit ejaculation when the concerned spinal segments were inju

132 howed reduced levels of intromissions and no ejaculations whereas simple mounting behavior was not af

133 ibute to a multifactorial model of premature ejaculation with some neurobiological vulnerability.

134 nd lateral MeApd cells that express Fos with ejaculation would be retrogradely labeled.