ライフサイエンスコーパス: ejection time

1 ic fluid content index, and left ventricular ejection time).

2 and 0.390+/-0.051 (P<0.023 versus beta of LV ejection time).

3 e plus isovolumic relaxation time divided by ejection time).

4 ia due to excessive prolongation of systolic ejection time.

5 of the pre-ejection period/right ventricular ejection time.

6 determinations of left ventricular systolic ejection times.

7 1.2 versus 4.9 +/- 1.2 cm(2)/m(2)), systolic ejection times (214.0 +/- 29.4 versus 231.3 +/- 28.6 ms)

8 e mean differences were as follows: systolic ejection time 25 ms (95% CI 18-32, p<0.0001), stroke vol

9 inute(-)(1) (-13, -3; P=0.003) and increased ejection time 26 ms (2, 50; P=0.03).

10 In both groups, systolic ejection time and ejection fraction increased after OM (

11 novel inotropic agent that prolongs systolic ejection time and increases ejection fraction through my

12 beta values of the relation between LV ejection time and LV end-diastolic volume and mass were

13 ymptomatic AS, metoprolol increases systolic ejection time and reduces aortic valve gradients, global

14 erally well tolerated, it increased systolic ejection time, and it may have improved dyspnea in the h

15 een MA peak velocity, MA displacement and LV ejection time, and LV end-diastolic volume (and mass) we

16 ed less severe abnormalities of the systolic ejection times, and the patients without infarction were

17 n rate and the ratio of acceleration time to ejection time (AT/ET).

18 Right ventricular ejection time correlated with hemodynamics and survival

19 The LV dimensions, corrected ejection time (ETc), percent fractional shortening (%FS)

20 HF myocardium, which may extend the systolic ejection time in vivo.

21 ect measurement of left ventricular systolic ejection time is a valuable adjunct in the bedside asses

22 concentration-related increases in systolic ejection time (mean increase from baseline at maximum to

23 or bacteriophage lambda, we establish a mean ejection time of roughly 5 min with significant cell-to-

24 Correspondingly, the left ventricular rapid-ejection time of the transgenic mouse hearts was signifi

25 lected-wave transit time to left ventricular ejection time (P < .001), which contributed to early and

26 lated increases in left ventricular systolic ejection time (p < 0.0001) and decreases in end-systolic

27 a high pre-ejection period/right ventricular ejection time ratio (p < .0001).

28 en the pre-ejection period/right ventricular ejection time ratio and the slope of the right ventricul

29 The right ventricular preejection period to ejection time ratio was normal in all subjects.

30 on the pre-ejection period/right ventricular ejection time ratio, the slope of the right ventricular

31 op area was determined by cardiac output, LV ejection time, tau, and early transmitral flow.

32 ing sodium) reduces the pressure, increasing ejection time to 8-11 s.

33 total IVT (s/min; calculated as: 60 - [total ejection time + total filling time] ) and CO were measur

34 rdiac output, left ventricular filling time, ejection time, total isovolumic time, mitral regurgitati

35 tion-dependent increases in left ventricular ejection time (up to an 80 ms increase from baseline) an

36 Controlling the ion trap ejection time was found to result in efficient removal o

37 Right ventricular ejection time was measured from the rapid upstroke of th

38 RV ejection time was prolonged and correspondingly filling

39 tion and relaxation times were prolonged and ejection time was shortened (p < 0.001) in patients with

40 and isovolumetric relaxation time divided by ejection time, was measured from left ventricular outflo

41 ectromechanical systole and left ventricular ejection time were shortened in acute myocardial infarct

42 mal-mean area, opening and closing rates and ejection times were obtained and compared with Doppler-d

43 Left ventricular systolic ejection times were significantly higher and inversely r

44 ocity, peak velocity, acceleration time, and ejection time, were measured in each patient by investig

45 n without excessive prolongation of systolic ejection time, which may compromise diastolic filling.

46 n equation (stroke volume = left ventricular ejection time x volume of electrically participating tis