ライフサイエンスコーパス: elastance

1 of respiratory resistance and low-frequency elastance).

2 with increased elastic artery stiffness (or elastance).

3 om the frequency responses of resistance and elastance.

4 orrelated with body mass index or chest wall elastance.

5 cle contraction, lung resistance and dynamic elastance.

6 ere recorded to compute driving pressure and elastance.

7 ements in oxygenation and respiratory system elastance.

8 w is applied to overcome lung and chest wall elastance.

9 l pressure and the other based on chest wall elastance.

10 l pressure and the other based on chest wall elastance.

11 olic elastance (Eed), and effective arterial elastance.

12 had increased lung compliance and decreased elastance.

13 buted to increased tissue damping and tissue elastance.

14 626 +/- 153 dyn.s/cm; p = 0.714), effective elastance, (0.63 +/- 0.22 vs 0.58 +/- 0.17 mm Hg/mL; p =

15 /m(2)) and increased arterial load (arterial elastance, 1.51 mm Hg/mL) were evident.

16 Hg; p = 0.01) and greater effective arterial elastance (2.77 +/- 0.84 mm Hg .

17 , P=0.009]), and arterial function (arterial elastance [2.1%, P=0.002] and systemic arterial complian

18 post-MR versus post-PVA, P=nonsignificant), elastance (3.5+/-1.4 versus 2.9+/-1.3; post-MR versus po

19 e (27%), tissue resistance (48%), and tissue elastance (30%).

20 6+/-7 mm Hg; P<0.001) and effective arterial elastance (5.9+/-3.1 to 9.2+/-3.9 mm Hg/microl; P<0.001)

21 e-area relations were variable: end-systolic elastance, 6.5+/-3.4 to 4.3+/-2.5 mm Hg/cm2 and preload

22 Mesenchymal stem cell mitigated changes in elastance, alveolar collapse, and inflammation at days 2

23 ge compression slightly worsened static lung elastance and cardiac output (p = 0.0391).

24 at rest and improved arterial compliance and elastance and central hemodynamics during exercise.

25 rdiac function judged by reduced ventricular elastance and decreased cardiac output.

26 expiratory pressure levels minimizing global elastance and driving pressure, electrical impedance tom

27 the groups did not differ in terms of static elastance and dynamic intrinsic positive end-expiratory

28 or a potentially deleterious effect (reduced elastance and ejection fraction).

29 However, it reduced arterial elastance and improved Vo(2)max by 19% (22.8+/-3.4 versu

30 i, which is associated with decreased tissue elastance and increased quasi-static compliance of Sepn1

31 ices of force-generation, e.g., end-systolic elastance and invasive indices of diastolic properties,

32 VV sheep also displayed lower-lung elastance and mechanical heterogeneity in comparison wit

33 nonaerated lung tissue, reestablishing lung elastance and oxygenation while avoiding increased pulmo

34 Global LV systolic function (end-systolic elastance and preload recruitable stroke work) were not

35 eased contractility assessed by end-systolic elastance and provided venoarterial dilation.

36 nhanced contractility, doubling end-systolic elastance and raising fractional shortening similarly in

37 ic elastance, and left ventricular diastolic elastance and relaxation noninvasively in consecutive HF

38 ignificantly decreased perturbations in lung elastance and resistance, resulting in faster resolution

39 RV elastance and stiffness both increased (P<0.05), suggest

40 approximately 40% fall in effective arterial elastance and systemic resistance.

41 At days 1, 2, and 7, static lung elastance and the amount of alveolar collapse were simil

42 Arterial elastance and ventricular end-systolic elastance were si

43 ontractility (eg, +33+/-4.2% in end-systolic elastance) and lowered afterload (-14.2+/-2% in systemic

44 ally quasi-static lung compliance and tissue elastance) and reduced mucus production in airways.

45 ffness (total aortic compliance and arterial elastance), and maximal exercise testing.

46 otal arterial compliance, effective arterial elastance, and aortic characteristic impedance were deri

47 mputed tomography scans, oxygenation, static elastance, and dynamic respiratory resistance and elasta

48 ontrol mice, but that lung tissue dampening, elastance, and hysteresivity were significantly elevated

49 ial elastance, left ventricular end-systolic elastance, and left ventricular diastolic elastance and

50 Throughout 4 years, blood pressure, arterial elastance, and LV mass decreased, coupled with significa

51 s of alveolar capillary barrier injury, lung elastance, and mortality than WT mice with ALI.

52 tly improve lung volumes, respiratory system elastance, and oxygenation.

53 n the restoration of normal lung compliance, elastance, and pressure-volume loops (tissue recoil).

54 eft ventricular diastolic function, arterial elastance, and ventricular-arterial coupling in hyperten

55 in ventricular diastolic function, arterial elastance, and ventricular-arterial coupling.

56 , including arterial compliance, resistance, elastance, and wave reflection.

57 tion fraction, stroke work, and end-systolic elastance are significant (P<0.01) between groups.

58 eased end-diastolic and end-systolic chamber elastance, as well as diastolic dysfunction seen at the

59 rd shift in V100 [the volume of end-systolic elastance at 100 mm Hg], 24+/-9 to 16+/-5 microL; P<0.00

60 als, and an estimated normalized ventricular elastance at arterial end diastole.

61 ervals, and estimated normalized ventricular elastance at end diastole.

62 2 hrs of treatment, left ventricular maximum elastance at end systole increased and was unchanged in

63 systolic elastance (left ventricular maximum elastance at end systole), cardiac output, circumflex ar

64 ance, and dynamic respiratory resistance and elastance at end-expiratory pressure levels of 7.5-20 cm

65 ntrol hearts reached only 42+/-4% of maximum elastance at the onset of ejection, with substantial fur

66 Esophageal pressure and chest wall elastance-based methods for estimating pleural pressure

67 Esophageal pressure and chest wall elastance-based methods for estimating pleural pressure

68 Esophageal pressure and chest wall elastance-based methods for estimating pleural pressure

69 of 0 cm H2O and targeting an end-inspiratory elastance-based transpulmonary pressure of 26 cm H2O can

70 O and the other targeting an end-inspiratory elastance-based transpulmonary pressure of 26 cm H2O.

71 O and the other targeting an end-inspiratory elastance-based transpulmonary pressure of 26 cm H2O.

72 o t/t mutants, which reached 77+/-3% of peak elastance before ejection of peak.

73 MyBP-C t/t ventricles displayed reduced peak elastance, but more strikingly a marked abbreviation of

74 e and magnitude of left ventricular systolic elastance (chamber stiffening), and assessed mechanisms

75 urve shapes allow to detect regions in which elastance changes during inflation.

76 k power, ejection fraction, and end-systolic elastance changes reduced by 32+/-34%, 66+/-64%, and 56+

77 lation improved oxygenation and reduced lung elastance compared with volume-controlled ventilation in

78 s), airspace enlargement, and decreased lung elastance compared with wild-type lungs.

79 y artery resistance (Z0), effective arterial elastance, compliance, and reflected pressure waves.

80 nt of pulmonary artery resistance, effective elastance, compliance, and reflected pressure waves.

81 RA and RV elastance (contractility) and diastolic stiffness were c

82 at is based on normalized human time-varying elastance curves [EN(tN)].

83 Time-varying elastance curves were generated from 72 PV loops (52 pat

84 regions of presumed tidal recruitment (i.e., elastance decrease during inflation, pressure-volume cur

85 d tissue damping after albuterol, but tissue elastance decreased only in the Type B group.

86 In Group I, lung elastance decreased, and blood oxygenation increased sig

87 In some, total arterial elastance decreases to maximize LV work and maintain LV

88 86 +/- 50 torr; p < 0.01) and decreased lung elastance (Delta5 +/- 5 cm H2O/L; p < 0.01).

89 A concomitant decrease in effective arterial elastance (DeltaEa: -0.094 mm Hg/mL, P=0.004) yielded un

90 ial compliance and higher effective arterial elastance despite similar mean arterial pressures in con

91 load-recruitable stroke work and ventricular elastance did not change.

92 Contractility as end-systolic elastance did not decrease from the chronic MI to the ac

93 Static elastance did not demonstrate any significant pressure d

94 Baseline elastance distribution was estimated in 8-week-old wild

95 IVA was compared with elastance during contractility modulation by esmolol and

96 IVA was compared with elastance during contractility modulation by esmolol and

97 Group mean RV end-systolic elastance (E'es) and maximal elastance (E'max) increased

98 RV end-systolic elastance (E'es) and maximal elastance (E'max) increased with augmented dobutamine in

99 ruitable stroke work (PRSW) and end-systolic elastance (E(es)) were calculated to assess global LV sy

100 elastance (EaI) to left ventricular systolic elastance (E(LV)I), and its components, at rest and duri

101 flow; LVOT(Acc) was compared with LV maximal elastance (E(m)) acquired by conductance catheter under

102 f intraventricular pressure, volume, maximal elastance (e(max)), preload recruitable stroke work, and

103 by systemic vascular resistance and arterial elastance (Ea) and preload as determined by end-diastoli

104 temic arterial load was assessed by arterial elastance (Ea) and right ventricular afterload by pulmon

105 afterload was measured by effective arterial elastance (Ea) and systemic vascular resistance index (S

106 rterial coupling indices, effective arterial elastance (Ea) and the coupling ratio Ea/Eessb, without

107 Effective arterial elastance (Ea) end-systolic elastance (Ees) and ventricu

108 The effective arterial elastance (Ea) represents resistive and pulsatile afterl

109 that diastolic stiffness (Eed) and arterial elastance (Ea) were increased, end-systolic elastance (E

110 tance (Ees), peripheral resistance, arterial elastance (Ea), arterial compliance, aortic pulse wave v

111 ime (max -dP/dt), filling rate, and arterial elastance (Ea), respectively.

112 (CO), arterial and end-systolic ventricular elastance (Ea, Ees,) and ventriculoarterial coupling (V/

113 rial elastance/left ventricular end-systolic elastance [Ea/Ees]) after adjustment for potential confo

114 e work [Msw]), measures of RV load (arterial elastance [Ea]), and RV pulmonary artery coupling (Ees/E

115 d intercept [V0]); and VA coupling (arterial elastance [Ea]/Eessb).

116 temic arterial afterload (effective arterial elastance, Ea; total arterial compliance, Ca; and system

117 r coupling, defined by the ratio of arterial elastance (EaI) to left ventricular systolic elastance (

118 dt) by 49 +/- 8% (p < 0.001) and ventricular elastance (Ecs) by 53 +/- 16% (p < 0.03).

119 breathing, and higher values of dynamic lung elastance (EdynL) (p < 0.01) and intrinsic positive end-

120 end-systolic elastance (Ees), end-diastolic elastance (Eed), and effective arterial elastance.

121 fective arterial elastance (Ea) end-systolic elastance (Ees) and ventricular-arterial coupling (defin

122 e were significant increases in end-systolic elastance (Ees) from 0.74+/-0.11 to 0.90+/-0.16 mm Hg/ml

123 elastance (Ea) were increased, end-systolic elastance (Ees) was decreased, and arterioventricular (A

124 arterial compliance (TAC), and end-systolic elastance (Ees) were calculated at baseline and after 8

125 us peak pressure (dP/dtmax) and end-systolic elastance (Ees) were preserved in both groups compared t

126 tionship was determined to give end-systolic elastance (Ees), a load-independent measure of contracti

127 cluding ejection fraction (EF), end-systolic elastance (Ees), and preload-recruitable stroke work (PR

128 ate paired data to determine LV end-systolic elastance (Ees), end-diastolic elastance (Eed), and effe

129 y: left ventricular volumes and end-systolic elastance (Ees), peripheral resistance, arterial elastan

130 parameter of systolic function, end systolic elastance (Ees), requires invasive catheterization.

131 end-systolic elastance to effective arterial elastance [Ees/Ea]: SHF: 1.05 +/- 0.25; P = 0.002; DHF:

132 to derive contractile indexes (end-systolic elastance [Ees] and preload recruitable stroke work [Msw

133 Systolic (end-systolic elastance, Ees) and diastolic (tau) function were assess

134 h prognosis in systolic HF, but end-systolic elastance (Eessb) is not.

135 from shortening fraction, end-systolic fiber elastance (Ef(es)) measured at resting heart rates, and

136 was caused by an increase in total arterial elastance, effectively double loading the LV, contributi

137 ndexes (maximal power index and end-systolic elastance), ejection fraction, and measures of diastolic

138 tor waveform was used to measure RL and lung elastance (EL) in 21 asthmatics from approximately 0.1 t

139 Peak elastance (Emax) was determined as the slope of end-syst

140 f intraventricular pressure, volume, maximal elastance (Emax), and dP/dtmax by conductance catheteriz

141 The reference index, maximal elastance (Emax), was calculated from pressure-volume lo

142 ociated with a lowering of systemic arterial elastance (end-systolic pressure/stroke volume) and syst

143 sensitivity (resistance, tissue damping, and elastance), eosinophilic inflammation, and airway remode

144 nd load-independent measures of end-systolic elastance from pressure-area loops (r = 0.90, SEE 10.6 m

145 Chest wall and respiratory system elastances grew with increases in positive end-expirator

146 nstant, kappa, decreased (P=0.02), LV volume elastance improved (P=0.04), and the myocardial stiffnes

147 suggesting higher right ventricular systolic elastance in HFpEF.

148 and had negative effects on left ventricular elastance in the postjet ventilation period in both norm

149 h upward curvature) or overdistension (i.e., elastance increase during inflation, downward curvature)

150 distress syndrome, Z0 and effective arterial elastance increased (from 218 +/- 94 to 444 +/- 115 dyn.

151 In response, RA contractility improved (elastance increased from 0.28+/-0.12 to 0.44+/-0.13 mm H

152 Left ventricular systolic elastance increased to a greater extent in young versus

153 Chest wall and respiratory system elastances increased with increases in positive end-expi

154 ll recruitment maneuvers reduced static lung elastance independent of acute lung injury etiology.

155 MR imaging-derived elastance index correlates with ICP over a wide range of

156 The mean of the fractional SD of the elastance index in humans was 19.6%.

157 The elastance index in the five patients with intraventricul

158 The reproducibility of the elastance index measurement was established from four to

159 An elastance index was derived from the ratio of pressure t

160 The elastance index was measured and compared with invasive

161 ppearance, but a marked increase in arterial elastance, indicating increased afterload, and elevated

162 ed increase in afterload (effective arterial elastance), L-NMMA increased preload (end-diastolic dime

163 easure left ventricular maximum end-systolic elastance (left ventricular maximum elastance at end sys

164 left ventricular volume, effective arterial elastance, left ventricular end-systolic elastance, and

165 In all patients, nitroprusside reduced elastance, left ventricular filling pressures, and pulmo

166 icular-arterial coupling (effective arterial elastance/left ventricular end-systolic elastance [Ea/Ee

167 atory pressure PaO2/FIO2, respiratory system elastance, lung weight, normally aerated tissue, collaps

168 ung-distending pressure, and that chest wall elastance may vary among individuals, a physiologically

169 Finally, chest wall and respiratory system elastances may vary unpredictably with changes in positi

170 tricular (RV) systolic pressure and arterial elastance (measure of vascular resistance) more than tri

171 and 10 days, mice were anesthetized and lung elastance measured.

172 ed after surgery (p < 0.02), whereas dynamic elastance of the chest wall did not change.

173 Dynamic elastance of the lung (Edyn,L) decreased after surgery (

174 The failure group had a higher dynamic elastance of the lung than the success group (p < 0.01),

175 d-expiratory pressure resulted in the lowest elastance of the respiratory system (18.6 +/- 6.1 cm H2O

176 In 60 patients, elastances of lung and chest wall were computed, and lun

177 chest wall components or in terms of dynamic elastances of the respiratory system and chest wall.

178 teral annular diameter, but not end-systolic elastance or regional myocardial function.

179 increased respiratory system resistance and elastance (p < 0.05).

180 ction to exercise due to pathologic arterial elastance (p = 0.04).

181 of a significant decrease in total arterial elastance (P<0.01 versus Group I).

182 atter inversely correlated with end-systolic elastance (P=0.0001).

183 ompliance (P=0.03), and decrease in arterial elastance (P=0.02).

184 utrophilic infiltration, tissue damping, and elastance parameters, in association will less peribronc

185 Ventilation at lowest elastance positive end-expiratory pressure preceded by a

186 preload adjusted maximal power, end-systolic elastance, preload recruitable stroke work) and produced

187 s also correlated significantly with maximal elastance (r = .85 +/- .04) from pressure-volume relatio

188 hemodynamics, stroke work, and end-systolic elastance return to preinfarction values 1 week after in

189 Measurements of lung resistance and elastance (RL and EL) from 0.1 to 8 Hz reflect both the

190 m Hg (+28.1+/-5.3%; P=0.02), and ventricular elastance rose from 6.0+/-1.6 to 10.5+/-2.2 mm Hg/mm (P=

191 RV afterload assessed by effective arterial elastance rose similarly in both groups; thus, ventricul

192 gest that left ventricular (LV) and arterial elastance (stiffness) increase with age, but data examin

193 stolic (Ees), and ventricular diastolic (Ed) elastance (stiffness) may contribute to the pathogenesis

194 End-systolic elastance (stiffness) was higher in patients with HF-nlE

195 systolic properties, namely EF, end systolic elastance, stroke work, and preload recruitable stroke w

196 re pronounced effects of weight loss on lung elastance suggest that the distal lung is inherently mor

197 ic data, left ventricular ejection fraction, elastance, tau (relaxation constant), left ventricular s

198 The abbreviated elastance time course and lower peak were consistent wit

199 kingly a marked abbreviation of the systolic elastance time course, which peaked earlier (27.6+/-2.1

200 g was worse in Cpc-PH patients (end-systolic elastance to effective arterial elastance [Ees/Ea]: SHF:

201 tion (i.e., pressure change/volume change or elastance), transmural left ventricular end-systolic pre

202 e of myocytes, but it depresses end-systolic elastance; under conditions of exercise, the beneficial

203 toring of dynamic respiratory resistance and elastance ventilator settings can be used to optimize ve

204 ationship held across a wide range of atrial elastance, ventricular relaxation and systolic function,

205 Effective arterial elastance was also higher (2.6+/-0.5 versus 1.9+/-0.5 mm

206 After jet ventilation, left ventricular elastance was decreased 36 +/- 8% in normal hearts and 3

207 End-systolic elastance was determined using blood pressure, stroke vo

208 atio of ventricular end-systolic to arterial elastances) was approximately 0.25 at baseline and doubl

209 red compliance, and increased resistance and elastance were observed in subjects with HFpEF.

210 erial elastance and ventricular end-systolic elastance were similarly increased in hypertensive contr

211 re levels, chest wall and respiratory system elastances were calculated at each positive end-expirato

212 e, collapsed tissue, and lung and chest wall elastances were similar between the two groups.

213 ic input impedance, compliance, and arterial elastance), were significantly modified by TAVR, exhibit

214 did not augment contractility (end-systolic elastance) whereas IPAH did (P<0.001).