ライフサイエンスコーパス: electrocardiogram

1 stenosis, and charted test results (such as electrocardiogram).

2 mble averaged relative to the R-peaks of the electrocardiogram.

3 ters were measured from the standard 12-lead electrocardiogram.

4 prodromes who had a normal heart and normal electrocardiogram.

5 ol of the duration of the QT interval of the electrocardiogram.

6 terval was measured using a standard 12-lead electrocardiogram.

7 evaluation which included echocardiogram and electrocardiogram.

8 with normal initial troponin and nonischemic electrocardiogram.

9 normal QRS duration on their last inpatient electrocardiogram.

10 d of 27 individuals, 8 with AF documented by electrocardiogram.

11 to improve the diagnostic performance of the electrocardiogram.

12 including chest pain characteristics and the electrocardiogram.

13 (speakers and observers) were monitored via electrocardiogram.

14 t rate variability measures derived from the electrocardiogram.

15 ing-state electroencephalograms and parallel electrocardiograms.

16 by pre-participation screening with 12-lead electrocardiograms.

17 ould be assessed as a routine when obtaining electrocardiograms.

18 were extracted from 10-minute resting-state electrocardiograms.

19 t mice also had normal cardiac structure and electrocardiograms.

20 or hyperthyroidism showed various changes in electrocardiograms.

21 ons, vital signs, laboratory parameters, and electrocardiograms.

22 on, event monitoring, Holter monitoring, and electrocardiograms.

23 c Health Evaluation II 28.6 +/- 7.7) had 373 electrocardiograms.

24 (eg, HCM) such as consideration for 12-lead electrocardiograms.

25 myocardial infarction was assessed by serial electrocardiograms.

26 Using repeated electrocardiograms (1986-2004), longitudinal data on PM<

27 essments, echocardiographic studies, 12-lead electrocardiograms, 24-hour Holter monitoring, blood tes

28 baseline survival time: 48 hours after first electrocardiogram; 640 patients) was 19.2% in the delaye

29 Conventional 12-lead electrocardiogram, a widely used noninvasive tool in cli

30 Electrocardiogram abnormalities are common in alternatin

31 Half the cohort (26/52) had resting 12-lead electrocardiogram abnormalities: 25/26 had repolarizatio

32 disease, a benign family history, and normal electrocardiogram accounted for 75% of indications rated

33 ponin measurements obtained within 96 hrs of electrocardiogram acquisition were used to determine the

34 In nearly all patients (27 of 30), electrocardiogram activity continued after the disappear

35 ogressive development in ambulatory external electrocardiogram (AECG) monitoring technology.

36 The HEART (History, Electrocardiogram, Age, Risk factors, and initial Tropon

37 Both the History, Electrocardiogram, Age, Risk Factors, Troponin (HEART) a

38 lthy participants from the Intercity Digital Electrocardiogram Alliance database.

39 e initial history, physical examination, and electrocardiogram alone did not confirm or exclude the d

40 m can change APD and the Q-T interval of the electrocardiogram alter APD stability, and modulate resp

41 ted QT interval and heart rate assessed from electrocardiogram among 4,588 older southern Chinese men

42 We recommand an electrocardiogram and an troponin if any cardiac symptom

43 entified that prolong the QT interval on the electrocardiogram and cause torsade de pointes arrhythmi

44 on, determined by the treating clinician and electrocardiogram and confirmed by an investigator maske

45 ac events in the subgroup with pretransplant electrocardiogram and echocardiogram (n=166 and n=112, H

46 mical and physiological measurements made by electrocardiogram and echocardiography show that affecte

47 ial electric currents that contribute to the electrocardiogram and electrically silent circular curre

48 Electrocardiogram and end-tidal capnography waveform cap

49 The electrocardiogram and finger and ear photoplethysmograms

50 The finding of no ischemic electrocardiogram and hs-TnI </= 26.2 ng/l with the TIMI

51 e ICD therapy, all with a spontaneous type 1 electrocardiogram and inducible ventricular arrhythmias.

52 questionnaires and subsequently confirmed by electrocardiogram and medical record review.

53 In addition, electrocardiogram and muscle fiber size distribution wer

54 The dynamic electrocardiogram and neurological features point to per

55 Concurrent 24-hour ambulatory electrocardiogram and personal PM(2.5) exposure informat

56 ide and ajmaline unmask the Brugada syndrome electrocardiogram and precipitate ventricular tachycardi

57 hythmia [ETA]) from short-term recordings of electrocardiogram and respiratory chest excursions, and

58 ion (N = 941) underwent 30-min recordings of electrocardiogram and respiratory chest excursions.

59 cular late potentials by the signal-averaged electrocardiogram and spatial mean QRS-T angle measured

60 at hyperkalemia can be reliably diagnosed by electrocardiogram and that particular levels of hyperkal

61 rated prolongation of the QT interval on the electrocardiogram and the morphologically distinctive po

62 red for cardiac evaluation (standard 12-lead electrocardiogram and transthoracic echocardiography) to

63 Electrocardiograms and 24-h Holter monitoring showed no

64 agnostic tests were the 12-lead and exercise electrocardiograms and ajmaline provocation test.

65 tive clinical trial participants, among whom electrocardiograms and fasting blood draws were repeated

66 We performed electrocardiograms and magnetic resonance imaging for sc

67 al VT was defined by comparison with 12-lead electrocardiograms and stored implantable cardioverter-d

68 In addition, surface electrocardiograms and ventricular action potential wave

69 Blood samples were obtained, and heart rate, electrocardiogram, and blood pressure were monitored bef

70 Heart rate, electrocardiogram, and blood pressure were monitored bef

71 peroneal nerve, and arterial blood pressure, electrocardiogram, and central venous pressure were also

72 val time was set to 48 hours after the first electrocardiogram, and in all patients with recurrent MI

73 uded records of adverse events, vital signs, electrocardiogram, and laboratory tests.

74 Invasive arterial blood pressure, electrocardiogram, and oxygen saturation plethysmography

75 itant medications, laboratory abnormalities, electrocardiogram, and vital sign assessments.

76 e (HR) and heart rate variability (HRV) with electrocardiogram, and white blood cell (WBC) counts wit

77 g, immunology assessments, administration of electrocardiograms, and assessment of vital signs.

78 The clinical course, electrocardiograms, and Holter monitoring were available

79 These mice lack P waves on electrocardiograms, and isolated NCX KO SAN cells are qu

80 ined using hospital discharge records, study electrocardiograms, and Medicare claims data.

81 s with dyspnea and chest pain, nondiagnostic electrocardiograms, and no obvious diagnosis.

82 cal diagnosis was based on symptoms, initial electrocardiograms, and troponin, whereas the final diag

83 Es), clinical laboratory tests, vital signs, electrocardiograms, and validated scales.

84 Electrocardiogram, aortic blood pressure, and carotid bl

85 djacent T-wave (ST-T wave) amplitudes of the electrocardiogram are quantitative characteristics of ca

86 l action potentials, manifest on the surface electrocardiogram as QT interval prolongation.

87 aders 1 and 2 interpreted 46.4% and 30.0% of electrocardiograms as normal, and 15.3% and 12.3% as isc

88 ndpoint, biventricular pacing on the 12-lead electrocardiogram at 1 month, was achieved in 33 of 34 p

89 Incident AF cases were ascertained by electrocardiogram at ARIC follow-up visits, hospital dis

90 les, data acquisition was triggered with the electrocardiogram at specific time points in the cardiac

91 (cTnI) <99 th percentile and a nondiagnostic electrocardiogram at the time of presentation to the eme

92 Studies with ambulatory electrocardiogram-based TWA analysis with Modified Movin

93 ne instruments that recorded high-resolution electrocardiograms, behaviour and flipper accelerations

94 Electrocardiogram, blood pressure, carotid diameter and

95 longing to families with a diagnostic type 1 electrocardiogram Brugada pattern.

96 ular currents that cannot be detected by the electrocardiogram but are detectable by their magnetic f

97 The clinical examination, electrocardiogram, cardiac biomarkers, chest computed to

98 admission to pediatric intensive care unit), electrocardiogram, cardiac magnetic resonance imaging we

99 ishable from acute coronary syndrome or with electrocardiogram changes and wall motion abnormalities

100 Electrocardiogram changes occurred independently of seiz

101 other patients in the same study, including electrocardiogram changes, cardiovascular biomarkers, an

102 ynamic, beat-to-beat or electrocardiogram-to-electrocardiogram, changes were noted, suggesting the pr

103 Computerized interpretation of the electrocardiogram (CIE) was introduced to improve the co

104 a [cohort 2], one grade 3 QT prolongation on electrocardiogram [cohort 3], and one grade 3 fatigue an

105 Electrocardiogram data (67,648 hrs, mean 65 hrs/patient)

106 mmercially available 'HeRO' monitor analyzes electrocardiogram data from existing bedside monitors fo

107 al/trauma/burn ICU with available continuous electrocardiogram data.

108 he impedance cardiogram was recorded through electrocardiogram/defibrillator pads in standard cardiac

109 Electrocardiograms demonstrate that 3-day postnatal Post

110 Electrocardiograms demonstrated that MetS-VLDL induced p

111 Although 13 of the electrocardiogram derived metrics demonstrated simple (i

112 sly assess affective haptic perception using electrocardiogram-derived information exclusively.

113 Eligible patients had a nonischemic electrocardiogram determined and high-sensitivity tropon

114 Electrocardiogram displayed abnormalities in 64% of pati

115 graphy, and/or ischemic ST-segment change on electrocardiogram during 1 or more of the 3 mental stres

116 Electrocardiograms during atrial arrhythmia episodes wer

117 hen supplemented with patient history and an electrocardiogram (ECG) (the extended algorithm) for pre

118 We developed a 12-lead smartphone-based electrocardiogram (ECG) acquisition and monitoring syste

119 HR was measured from electrocardiogram (ECG) and echocardiograph (Echo) Doppl

120 lectrooculogram (EOG), Electromyogram (EMG), Electrocardiogram (ECG) and parameters along with other

121 ts with a spontaneous or drug-induced type I electrocardiogram (ECG) and without history of cardiac a

122 ed studies have assessed the resting 12-lead electrocardiogram (ECG) as a screening test in intermedi

123 We also evaluated AF recurrence by 12-lead electrocardiogram (ECG) at 3, 6, and 12 months.

124 e origin of atrial ectopic activity from the electrocardiogram (ECG) can help to diagnose the early o

125 potentials on the heart surface based on the electrocardiogram (ECG) data from the distributed sensor

126 lue and controversies of including a 12-lead electrocardiogram (ECG) in addition to a comprehensive p

127 eart rate-corrected QT (QTc) interval on the electrocardiogram (ECG) is associated with the onset of

128 nisms whereby variants affect AF risk, using electrocardiogram (ECG) measurements.

129 urine samples at baseline with parameters of electrocardiogram (ECG) performed during 2005-2010, 5.9

130 The standard 12-lead electrocardiogram (ECG) provides a method for non-invasi

131 Continuous Holter electrocardiogram (ECG) recordings were made before and

132 Electrocardiogram (ECG) screening increases the sensitiv

133 etection (<0.005 microg/L) and a nonischemic electrocardiogram (ECG) to rule out AMI in adults presen

134 A 12-lead electrocardiogram (ECG) was obtained at these times and

135 We assessed whether the electrocardiogram (ECG) was useful in predicting sudden

136 ed during exercise while heart rate (HR) and electrocardiogram (ECG) were monitored.

137 f tachyarrhythmias on 24-h ambulatory Holter electrocardiogram (ECG) with regard to delayed enhanceme

138 ty cardiac troponin T (hs-cTnT) level and an electrocardiogram (ECG) without signs of ischemia can ru

139 The features of the surface electrocardiogram (ECG), a measure of the electrical act

140 xic effects can be acute, such as changes in electrocardiogram (ECG), arrhythmias, ischemia, and peri

141 to improve the correct interpretation of the electrocardiogram (ECG), facilitating health care decisi

142 aracterized by a prolonged QT interval in an electrocardiogram (ECG), leading to higher risk of sudde

143 assium concentration ([K(+)]) influences the electrocardiogram (ECG), particularly T-wave morphology.

144 f MPI, hybrid systems obtain a low-dose, non-electrocardiogram (ECG)-gated CT scan that is used to pe

145 unction and broad QRS complex in the surface electrocardiogram (ECG).

146 ce these cases are not readily identified by electrocardiogram (ECG).

147 the benefit of most testing except exercise electrocardiogram (ECG).

148 antation must include an interpreted 12-lead electrocardiogram (ECG).

149 ope and the presence of a spontaneous type 1 electrocardiogram (ECG).

150 rdiomyopathy (HCM) who present with a normal electrocardiogram (ECG).

151 gested by abnormalities on a resting 12-lead electrocardiogram (ECG).

152 e and connect its results to features of the electrocardiogram (ECG).

153 itude and duration of the QRS complex on the electrocardiogram (ECG).

154 Twelve-lead electrocardiograms (ECG) of spontaneous VT often are not

155 Electrocardiograms (ECGs) and echocardiograms were norma

156 to determine the association of pre-hospital electrocardiograms (ECGs) and the timing of reperfusion

157 Interpretation of electrocardiograms (ECGs) is a complex task involving vi

158 rolonged heart rate-corrected QT interval on electrocardiograms (ECGs) is associated with increased r

159 Clinical histories, electrocardiograms (ECGs), and coronary 64-section multi

160 tely 16 h predose to 24 h postdose); 12-lead electrocardiograms (ECGs); clinical chemistry, hematolog

161 Electrocardiograms, echocardiographic images, and videos

162 serum markers for cardiomyocyte cell death, electrocardiograms, echocardiography, and cardiac angiog

163 ically inactive receiving antenna) including electrocardiogram (EKG) lead, nonactive "bovie" pencil,

164 In addition to clinical and laboratory data, electrocardiograms (EKGs), chest radiographs, and pulmon

165 ities in vital signs, laboratory results, or electrocardiogram findings were identified.

166 ally significant vital signs, laboratory, or electrocardiogram findings were recorded.

167 The most useful electrocardiogram findings were ST-segment depression (s

168 If there are concerns on the basis of electrocardiogram findings, medical history or family hi

169 Our recordings included the electrocardiogram, finger photoplethysmographic arterial

170 Measurements included the electrocardiogram, finger photoplethysmographic arterial

171 We recorded the electrocardiogram, finger photoplethysmographic arterial

172 ial nerve muscle sympathetic activities; the electrocardiogram; finger photoplethysmographic arterial

173 NDATION 3 (SCREENING): Obtain a pretreatment electrocardiogram for all patients to measure the QTc in

174 lactate) and an electrophysiological signal (electrocardiogram), for more comprehensive fitness monit

175 a, left bundle branch block, signal-averaged electrocardiogram, fragmented QRS, QRS-T angle, and T-wa

176 We analyzed 3,011 electrocardiograms from 113 patients with non-APL acute

177 eft ventricular (LV) mass and volume require electrocardiogram-gated PET data.

178 Longitudinal dynamic electrocardiogram-gated small-animal PET/CT studies were

179 y the cardiac cycle are discussed, including electrocardiogram gating, subject-specific acquisition w

180 4.3 MBq of (18)F-FDG and imaged for 2 h with electrocardiogram gating.

181 rated from dynamic (11)C-acetate PET without electrocardiogram gating.

182 nterpretable baseline and 60-minute post-PCI electrocardiograms had at least 1 mm of baseline ST-segm

183 S <12 years of age with a spontaneous type I electrocardiogram have a higher risk of arrhythmic event

184 Automated analysis of continuous electrocardiogram heart rate dynamics detects new-onset

185 ty of RR and QT intervals from standard 24-h electrocardiogram Holter recordings, could modulate the

186 detected by either scheduled or unscheduled electrocardiogram, Holter, transtelephonic monitor, or r

187 Atrial fibrillation (adjudicated using electrocardiograms, hospital discharge codes, and death

188 ystematically ascertained using clinic visit electrocardiograms, hospital discharge diagnosis codes,

189 Electrocardiogram, impedance cardiograph, and neuroendoc

190 ower spectral analysis on 24-hour ambulatory electrocardiogram in 459 middle-aged veteran male twins.

191 diotransmitters to record the SGNA, VNA, and electrocardiogram in 9 ambulatory dogs.

192 nt AF was ascertained for hospital and study electrocardiograms in 8,265 participants of the PREVEND

193 measured HRV and arrhythmia with ambulatory electrocardiograms in a cohort panel study for up to 235

194 ransients that were synchronized to the host electrocardiogram, indicating electromechanical coupling

195 with septic shock, inter-rater agreement of electrocardiogram interpretation for myocardial ischemia

196 The reliability of electrocardiogram interpretation to diagnose myocardial

197 assessed intra- and inter-rater agreement of electrocardiogram interpretation, and the effect of know

198 temperatures and changes in heart rates and electrocardiogram intervals for 28 consecutive days with

199 and skeletal muscle by > 90%, yet survival, electrocardiogram intervals, cardiac ejection fraction a

200 Prolongation of the QT interval on the electrocardiogram is also a risk factor for arrhythmias

201 The morphology of the QRS complexes on electrocardiogram is an excellent tool to identify the s

202 The electrocardiogram is useful for differentiating the site

203 reflecting myocardial repolarization on the electrocardiogram, is a heritable risk factor for sudden

204 ted with the same baseline risk factors plus electrocardiogram left ventricular hypertrophy.

205 is, pericardial effusion, low voltage on the electrocardiogram, marked elevation of serum enzymes, an

206 Twelve-lead electrocardiogram matched in 15 of 19 VTs between days 8

207 vernight urine collection, a 12-lead resting electrocardiogram, measurement of carotid intima-media t

208 Abnormal signal-averaged electrocardiogram measures did not associate with mortal

209 d removal from the aquatic habitat for micro-electrocardiogram (microECG) measurements, we developed

210 Finally, ambulatory electrocardiogram monitoring captured the abrupt onset o

211 Ambulatory electrocardiogram monitoring demonstrated reduced ventri

212 ent) ST-segment depression during ambulatory electrocardiogram monitoring occurs more often than symp

213 Patients were on continuous electrocardiogram monitoring until hospital discharge, a

214 ntinuous interstitial glucose and ambulatory electrocardiogram monitoring.

215 med in Sprague-Dawley rats with simultaneous electrocardiogram monitoring.

216 es with left bundle branch block (LBBB)-like electrocardiogram morphology (left ventricular ejection

217 CAD was defined as angina, ischemic electrocardiogram, myocardial infarction confirmed by Q-

218 died healthy supine astronauts on Earth with electrocardiogram, non-invasive arterial pressure, respi

219 No on-treatment electrocardiogram occurrences of corrected QT interval m

220 ptomatic male patient who develops a Brugada electrocardiogram on flecainide is diagnosed with "asymp

221 scars, of which 114 (78%) were undetected by electrocardiogram or by clinical adjudication.

222 yocardial infarction confirmed by Q-waves on electrocardiogram or hospital records, angiographic sten

223 nstable angina and frequently had a positive electrocardiogram or marker evidence of myocardial ische

224 =94% (OR: 3.0), and Q-wave on the presenting electrocardiogram (OR: 2.8).

225 mg bisoprolol (within 30 min after the first electrocardiogram) or 24 hours after acute myocardial in

226 inically significant changes in vital signs, electrocardiogram, or laboratory values were observed.

227 linically meaningful changes in vital signs, electrocardiogram, or laboratory values.

228 ithout noticeable changes in vital signs, on electrocardiograms, or in laboratory values.

229 e (finger photoplethysmography), heart rate (electrocardiogram), oxygen saturation (pulse oximetry),

230 no significant effects on blood pressure and electrocardiogram parameters in telemetrized cynomolgus

231 evant changes in vital signs, laboratory, or electrocardiogram parameters.

232 A spontaneous type 1 electrocardiogram pattern at diagnosis was present in 50

233 rehospital factors such as postresuscitation electrocardiogram pattern or clinical features are poorl

234 ensive care unit patients (2.99%) who had an electrocardiogram performed.

235 L was identified from baseline and follow-up electrocardiograms performed biannually.

236 aboratory tests, three radiologic tests, and electrocardiograms performed in each ICU.

237 the first case of lidocaine-induced Brugada electrocardiogram phenotype.

238 ified packets, including physician notes and electrocardiograms, procedures, and clinical laboratory

239 , including physician notes and results from electrocardiograms, procedures, and laboratory tests.

240 on for BrS is asymptomatic but has a Brugada electrocardiogram provoked by a drug.

241 e pulmonary embolism (seven [3%]), prolonged electrocardiogram QT (five [2%]), decreased neutrophil c

242 ory of AF, in sinus rhythm on their baseline electrocardiogram, randomly assigned to losartan- or ate

243 All had 12-lead electrocardiogram recordings available for cardiac axis,

244 ilable, historical and prolonged single-lead electrocardiogram recordings during electrocardiogram-vi

245 Electrocardiogram recordings from 40 mechanically ventil

246 We gathered 21,912 hours of routine electrocardiogram recordings from a heterogenous group o

247 Telemetric electrocardiogram recordings in Mybphl mice revealed car

248 try was used to investigate effects of CO on electrocardiogram recordings in vivo.

249 completed both baseline 5-minute and 12-lead electrocardiogram recordings on a nondialysis day.

250 rt rate and HRV measures obtained from 2-min electrocardiogram recordings performed at baseline (1987

251 long or short photoperiods and then analyzed electrocardiogram recordings.

252 of the Q wave to the end of the S wave on an electrocardiogram, reflects ventricular depolarization a

253 initial nonshockable rhythm, and unspecific electrocardiogram repolarization abnormalities were inde

254 ns, both readers initially reviewed 25 trial electrocardiograms representing normal to abnormal.

255 Using 1.6 million electrocardiogram results from 380,000 patients in our i

256 dverse events, clinical laboratory data, and electrocardiogram results were assessed.

257 amperometric lactate biosensor and a bipolar electrocardiogram sensor, are co-fabricated on a flexibl

258 Among 486 patients whose electrocardiogram showed a PR interval greater than 200

259 using a combination of real recorded patient electrocardiogram signals and a simulated patient experi

260 Telemetric measurement of electrocardiogram signals demonstrated autonomic disturb

261 QRS-T angle but not abnormal signal-averaged electrocardiogram significantly associates with cardiova

262 enital or acquired disorders with diagnostic electrocardiograms (ST-segment elevation and prolonged Q

263 istory and physical, basic laboratories, and electrocardiogram stress testing to include CPET.

264 ardiac arrest, with normal postresuscitation electrocardiogram, sufficient hemodynamic conditions, an

265 ng QT (interval between the Q and T waves in electrocardiogram) syndrome that predisposes afflicted i

266 In hearts where electrocardiogram T waves involve a well-defined repolar

267 action potential (AP) oscillations and cause electrocardiogram T-wave alternans (TWA).

268 Furthermore electrocardiogram telemetry revealed that mice with miR-

269 prodromes who had a normal heart and normal electrocardiogram (the study group) with those of 31 pat

270 24-hour Holter electrocardiograms to assess PAC prevalence and frequenc

271 Dynamic, beat-to-beat or electrocardiogram-to-electrocardiogram, changes were not

272 Crohn's disease, multiple sclerosis, and an electrocardiogram trait, without prior knowledge of phys

273 gle-lead electrocardiogram recordings during electrocardiogram-videotelemetry were analysed.

274 Clinical laboratory, electrocardiograms, vital signs, and adverse event monit

275 rse events, clinical laboratory assessments, electrocardiograms, vital signs, and physical examinatio

276 status (past history of AF or AF on baseline electrocardiogram vs. no AF) using adjusted Cox models a

277 His most recent electrocardiogram was profoundly changed from previous t

278 ital signs were within normal limits, and an electrocardiogram was unchanged from baseline.

279 A 2-minute electrocardiogram was used to measure HRV.

280 between the Q and the T waves on the cardiac electrocardiogram), was investigated after recombinant e

281 racic echocardiography, biological data, and electrocardiogram were obtained serially on ICU admissio

282 Electrocardiograms were analyzed for group 1 (training-r

283 coronary heart disease) from whom continuous electrocardiograms were available.

284 Electrocardiograms were manually measured and visually c

285 erse events, clinical laboratory values, and electrocardiograms were monitored.

286 nts, vital signs, laboratory parameters, and electrocardiograms were monitored.

287 Twelve-lead electrocardiograms were normal in 10 (53%) of the genoty

288 Electrocardiograms were obtained at birth.

289 gh-sensitivity cardiac troponin T levels and electrocardiograms were obtained every 12 and 24 h, resp

290 During this time, vital signs and electrocardiograms were recorded at regular intervals.

291 Electrocardiograms were recorded at years 0 (Y0), 7 (Y7)

292 Twelve-lead electrocardiograms were recorded before study drug, and

293 After 4 months, HF-parameters were assessed, electrocardiograms were recorded, and blood and ventricu

294 Electrocardiograms were systematically collected to incl

295 The electrocardiograms were then further evaluated by a blin

296 n was significant for atrial fibrillation on electrocardiogram with subsequent echocardiography revea

297 In the multivariable analysis, type 1 electrocardiogram with syncope (hazard ratio: 4.96; 95%

298 diac conduction system and apparently normal electrocardiograms with normal QRS intervals.

299 to measure the QTc interval and a follow-up electrocardiogram within 30 days and annually.

300 baseline heart rate documented by a 12-lead electrocardiogram without pacing or atrial fibrillation