ライフサイエンスコーパス: electrocardiographic

1 0% vs. 51%, p < 0.0001) and to have baseline electrocardiographic abnormalities (50% vs. 17%, p < 0.0

2 nary heart disease (CHD), diabetes, or major electrocardiographic abnormalities at baseline.

3 carotid intimal medial thickness, and major electrocardiographic abnormalities only modestly attenua

4 ed long QT syndrome (acLQTS), which produces electrocardiographic abnormalities that have been associ

5 Electrocardiographic abnormalities were 2-fold more comm

6 No clinically relevant laboratory or electrocardiographic abnormalities were reported.

7 vascular risk factors, severity of diabetes, electrocardiographic abnormalities, and coronary anatomy

8 from its main features; that is, lentigines, electrocardiographic abnormalities, ocular hypertelorism

9 differentiation syndrome, leukocytosis, and electrocardiographic abnormalities.

10 ctural heart disease, arrhythmia, or certain electrocardiographic abnormalities.

11 We performed electrocardiographic activation mapping in 33 consecutiv

12 dy sought to investigate whether noninvasive electrocardiographic activation mapping is a useful meth

13 We recorded facial electromyographic and electrocardiographic activity while participants watched

14 ears; 2898 women [55%]) without baseline AF (electrocardiographic AF or arterial flutter).

15 Using a stepwise electrocardiographic algorithm, the accuracy rates for t

16 itude spectrum area values, representing the electrocardiographic amplitude frequency spectral area c

17 chest compressions to avoid artifacts during electrocardiographic analyses and to minimize the risk o

18 Electrocardiographic analyses show that older R21C mice

19 Electrocardiographic analysis of 1,959 elite male athlet

20 Electrocardiographic and 24-h Holter monitoring findings

21 Electrocardiographic and clinical parameters of 382 LQT1

22 This study investigated the prevalence and electrocardiographic and electrophysiologic characterist

23 The electrocardiographic and electrophysiological characteri

24 The electrocardiographic and electrophysiological characteri

25 study investigated the efficacy of RFCA and electrocardiographic and electrophysiological characteri

26 This study investigated the prevalence and electrocardiographic and electrophysiological characteri

27 This study investigated the electrocardiographic and electrophysiological characteri

28 Demographic data and electrocardiographic and electrophysiological findings w

29 Electrocardiographic and mapping data were collected to

30 sk stratification, specified time points for electrocardiographic and serial troponin testing within

31 n beyond ejection fraction and commonly used electrocardiographic and serum biomarkers.

32 ctions for combining risk stratification and electrocardiographic and troponin testing in an accelera

33 Clinical, electrocardiographic, and arrhythmic outcome (composite

34 Clinical, electrocardiographic, and cardiopulmonary exercise test

35 J-point elevation, the associated clinical, electrocardiographic, and echocardiographic characterist

36 series of demographic, clinical, laboratory, electrocardiographic, and echocardiographic measures in

37 We sought to identify clinical, electrocardiographic, and electrophysiological features

38 Echocardiographic, electrocardiographic, and invasive hemodynamic data were

39 Detailed baseline clinical, angiographic, electrocardiographic, and revascularization data were co

40 Structural, histological, electrocardiographic, arrhythmic, and familial features

41 ach of classifying structural, histological, electrocardiographic, arrhythmic, and genetic features o

42 SAAP can effect ROSC from hemorrhage-induced electrocardiographic asystole in large swine.

43 A prospective continuous electrocardiographic (cECG) assessment was therefore per

44 ship between ischemia detected on continuous electrocardiographic (cECG) recording and cardiovascular

45 th persistent severe hypotension (68.2%) and electrocardiographic changes (56.8%).

46 The mechanisms of the electrocardiographic changes and arrhythmias in Brugada

47 Electrocardiographic changes and patient symptoms were m

48 No subject had chest pain or electrocardiographic changes during the stressor.

49 tive cardiac biomarkers and either ischaemic electrocardiographic changes or an atherosclerotic culpr

50 cute coronary syndromes but without ischemic electrocardiographic changes or an initial positive trop

51 003, presenting with chest pain and positive electrocardiographic changes or cardiac biomarkers consi

52 Patients with unstable angina and electrocardiographic changes or non-Q-wave myocardial in

53 on, reduced exercise capacity, nondiagnostic electrocardiographic changes, and balanced ischemia from

54 ar spasm (reproduction of symptoms, ischemic electrocardiographic changes, and no epicardial spasm).

55 ria including chest pain, a pericardial rub, electrocardiographic changes, and pericardial effusion.

56 infarction (MI) combines ischemic symptoms, electrocardiographic changes, and troponin rather than c

57 es adjusting for additional CV risk factors, electrocardiographic changes, or when only considering e

58 Hypothermia is known to result in similar electrocardiographic changes.

59 Electrocardiographic characteristics at the first clinic

60 Electrocardiographic characteristics differ depending on

61 fic injury marker, and an improvement in the electrocardiographic characteristics during the chronic

62 er pace mapping or some algorithms using the electrocardiographic characteristics less reliable.

63 urpose of this study was to evaluate whether electrocardiographic characteristics of ventricular arrh

64 There is an urgent need to identify electrocardiographic characteristics that differentiate

65 Electrocardiographic characteristics were specific for e

66 Specific electrocardiographic characteristics, including QRS morp

67 s had similar baseline echocardiographic and electrocardiographic characteristics.

68 LQTS patients relies upon a constellation of electrocardiographic, clinical, and genetic factors.

69 LEOPARD syndrome (multiple Lentigines, Electrocardiographic conduction abnormalities, Ocular hy

70 a subgroup of 113 patients with intermediate electrocardiographic criteria (QRS duration of 120-149 m

71 Several electrocardiographic criteria can help distinguish right

72 Electrocardiographic criteria used as risk predictors fo

73 from CRT, including those with intermediate electrocardiographic criteria, where CRT response is les

74 from 3 apical views add prognostic value to electrocardiographic criteria.

75 Systematic electrocardiographic data during rest, exercise, and rec

76 QRS morphology is a more important baseline electrocardiographic determinant of CRT response than QR

77 nd type qualitatively compared the automated electrocardiographic diagnostic statements generated by

78 rhythms a few days before death and, later, electrocardiographic disturbances comparable to those in

79 Electrocardiographic early repolarization (ER) pattern h

80 Electrocardiographic (ECG) abnormalities are common in o

81 ndent prognostic value of minor and/or major electrocardiographic (ECG) abnormalities in asymptomatic

82 stematic investigations on the prevalence of electrocardiographic (ECG) abnormalities in these patien

83 The goal of this study was to define electrocardiographic (ECG) and echocardiographic charact

84 This study analyzed the utility of electrocardiographic (ECG) and echocardiographic finding

85 sociation between structural progression and electrocardiographic (ECG) changes in patients with ARVD

86 This study sought to examine whether electrocardiographic (ECG) changes provide prognostic in

87 stematically study diagnostic and prognostic electrocardiographic (ECG) characteristics of arrhythmog

88 aimed to assess the diagnostic properties of electrocardiographic (ECG) criteria for right ventricula

89 Current electrocardiographic (ECG) criteria for the diagnosis of

90 This study sought to identify electrocardiographic (ECG) criteria that are associated

91 -detector row CT unit by using retrospective electrocardiographic (ECG) gating after infusion of 120-

92 reased P-wave terminal force in lead V1 , an electrocardiographic (ECG) marker of left atrial abnorma

93 nt guidelines recommend at least 24 hours of electrocardiographic (ECG) monitoring after an ischemic

94 Patients were on continuous electrocardiographic (ECG) monitoring during hospitaliza

95 Although continuous electrocardiographic (ECG) monitoring is ubiquitous in h

96 singly, this is supplemented with continuous electrocardiographic (ECG) monitoring.

97 long-term outcome and if this is additive to electrocardiographic (ECG) morphology and duration.

98 rdial diffuse fibrosis and scar with surface electrocardiographic (ECG) parameters in individuals fre

99 he interpretation of normal and pathological electrocardiographic (ECG) patterns in terms of the unde

100 igate the prevalence of potentially abnormal electrocardiographic (ECG) patterns in young individuals

101 Arrhythmia or electrocardiographic (ECG) phenotypes defined by Interna

102 he purpose of this study was to establish an electrocardiographic (ECG) profile in a biracial populat

103 olonging action potential repolarisation and electrocardiographic (ECG) QT interval, associated with

104 During this time, standard vital signs, electrocardiographic (ECG) readings, and blood sample va

105 y sought to estimate the costs of a national electrocardiographic (ECG) screening of athletes in the

106 additional investigations are an obstacle to electrocardiographic (ECG) screening of young athletes f

107 (RR) and tidal volume (TV) from analysis of electrocardiographic (ECG) signals only.

108 It is unclear whether using fetal electrocardiographic (ECG) ST-segment analysis as an adj

109 igh-sensitivity C-reactive protein (CRP) and electrocardiographic (ECG) ST-segment depression (STD) h

110 this study was to validate existing 12-lead electrocardiographic (ECG) ST-segment elevation myocardi

111 yocardial contrast echocardiography with 1:4 electrocardiographic (ECG) triggering was performed at 1

112 pressure; and cardiac morbidity (defined by electrocardiographic [ECG] abnormalities).

113 t precordial leads (V1 to V3; type 1 Brugada electrocardiographic [ECG] pattern) and the presence of

114 NP), and troponin I (TnI) concentrations and electrocardiographic, echocardiographic, and clinical ch

115 Electrocardiographic, echocardiographic, and serum bioma

116 previous paper, we considered the different electrocardiographic elements of the early repolarizatio

117 eterozygous fish manifest overt cellular and electrocardiographic evidence for delayed ventricular re

118 on myocardial infarction do not present with electrocardiographic evidence of active ischemia.

119 Study subjects had electrocardiographic evidence of at least one episode of

120 cted coronary disease based on history or on electrocardiographic evidence of previous myocardial inf

121 ected coronary disease based on a history or electrocardiographic evidence of previous myocardial inf

122 as documented in 40% of study patients, with electrocardiographic evidence of Q waves corresponding t

123 frequently results in an overlap in surface electrocardiographic features of ventricular arrhythmias

124 Compared with electrocardiographic findings alone, a point score based

125 ession to electromechanical dissociation and electrocardiographic findings consistent with acute hype

126 ly stable patients with LBBB who do not have electrocardiographic findings highly specific for ST-seg

127 picture of retrosternal chest pain, aided by electrocardiographic findings of ST segment deviations a

128 ionships between PET, echocardiographic, and electrocardiographic findings were analyzed.

129 Echocardiographic and electrocardiographic findings were consistent with cardi

130 Electrocardiographic findings were unremarkable.

131 orithm includes chest pain, cardiac enzymes, electrocardiographic findings, and autopsy results.

132 There were no changes in vital signs, electrocardiographic findings, or laboratory values that

133 Clinical management issues include electrocardiographic findings, scoliosis, osteopenia, an

134 h spontaneous or drug-induced Brugada type 1 electrocardiographic findings, who underwent ICD implant

135 ng history, serum creatine phosphokinase, or electrocardiographic findings.

136 ican Heart Association encourages the use of electrocardiographic-gated single photon emission comput

137 minimized scan range, heart rate reduction, electrocardiographic-gated tube current modulation, and

138 omputed tomography and 15 studies (79%) were electrocardiographic-gated.

139 computed tomography (CT) with retrospective electrocardiographic gating (one examination per patient

140 ing in 51 consecutive patients with 16-frame electrocardiographic gating and available coronary angio

141 sessment of attenuation artifacts as well as electrocardiographic gating in enhancing the diagnosis a

142 ed the concept of attenuation correction and electrocardiographic gating in improving the diagnosis o

143 Multiple studies emphasize the importance of electrocardiographic gating in myocardial perfusion imag

144 Electrocardiographic gating is increasingly used for (82

145 The validity of electrocardiographic gating using (82)Rb for assessment

146 ion was used in 104 (97.2%), and prospective electrocardiographic gating was used in 106 (99.1%).

147 rasonography at 8000 frames per second (with electrocardiographic gating).

148 angiography of the thorax with retrospective electrocardiographic gating.

149 Patients underwent 48-h electrocardiographic Holter monitoring quarterly to dete

150 ients with a broad spectrum of disease using electrocardiographic imaging (a method for noninvasive c

151 sequences of AF in humans using noninvasive electrocardiographic imaging (ECGI).

152 Electrocardiographic imaging accurately identifies areas

153 The results suggest a potential role for electrocardiographic imaging and late gadolinium enhance

154 invasive mapping of cardiac arrhythmias with electrocardiographic imaging and noninvasive delivery of

155 Electrocardiographic imaging colocalizes the epicardial

156 Electrocardiographic imaging constructs epicardial elect

157 Electrocardiographic imaging depicted salient features o

158 ombining anatomical imaging with noninvasive electrocardiographic imaging during ventricular tachycar

159 Electrocardiographic imaging is a noninvasive method for

160 Nevertheless, raw electrocardiographic imaging phase maps presented reentr

161 AND EGM, body surface potential mapping, and electrocardiographic imaging phase maps were obtained fr

162 Electrocardiographic imaging reveals electrophysiologica

163 m (EGM), body surface potential mapping, and electrocardiographic imaging signals.

164 This review summarizes results from electrocardiographic imaging studies of arrhythmogenic s

165 Noninvasive electrocardiographic imaging was used before and after 1

166 Here, we use a noninvasive imaging modality (electrocardiographic imaging) to study normal activation

167 Electrocardiographic imaging, a noninvasive functional E

168 In electrocardiographic imaging, HDF filtering allowed to i

169 enerated phase maps and activation maps from electrocardiographic imaging-reconstructed epicardial un

170 The times to angina and to electrocardiographic ischemia also increased in the rano

171 Reduction of electrocardiographic left ventricular hypertrophy (LVH)

172 Although the presence and severity of electrocardiographic left ventricular hypertrophy (LVH)

173 Electrocardiographic left ventricular hypertrophy (LVH)

174 evaluated in 8831 hypertensive patients with electrocardiographic left ventricular hypertrophy with n

175 index, smoking, valvular disease, diabetes, electrocardiographic left ventricular hypertrophy, hyper

176 to 80 years with essential hypertension and electrocardiographic LV hypertrophy had LV mass measured

177 nts with essential hypertension and baseline electrocardiographic LV hypertrophy, lower LV mass durin

178 Change in Cornell product electrocardiographic LVH between baseline and in-study e

179 Less-severe electrocardiographic LVH by Cornell product and Sokolow-

180 n, aged 55-80 years (median, 67 years), with electrocardiographic LVH by Cornell voltage-duration pro

181 dictive value of changes in the magnitude of electrocardiographic LVH criteria during antihypertensiv

182 New-onset AF in relation to electrocardiographic LVH determined at baseline and subs

183 Reduction in Cornell product electrocardiographic LVH during antihypertensive therapy

184 Lower Cornell product electrocardiographic LVH during antihypertensive therapy

185 ict development of AF, whether regression of electrocardiographic LVH is associated with a decreased

186 However, whether reduction of electrocardiographic LVH is associated with decreased he

187 rapy targeted at regression or prevention of electrocardiographic LVH may improve prognosis.

188 rapy targeted at regression or prevention of electrocardiographic LVH may reduce the incidence of new

189 Use of electrocardiographic LVH to select patients may have inc

190 Electrocardiographic LVH was measured using sex-adjusted

191 ent blood pressure, and baseline severity of electrocardiographic LVH, in-treatment decrease of Corne

192 ent blood pressure, and baseline severity of electrocardiographic LVH, lower in-treatment Cornell pro

193 as similar in both groups, and regression of electrocardiographic-LVH was greater with losartan.

194 The QT interval is the electrocardiographic manifestation of ventricular repola

195 In order to more clearly understand the electrocardiographic manifestations of early transmural

196 Using an electrocardiographic mapping (ECM) system in 27 patients

197 l, noninvasive, beat-by-beat mapping system, Electrocardiographic Mapping (ECM), in facilitating the

198 entricular electrical uncoupling measured by electrocardiographic mapping predicted clinical CRT resp

199 patients using a noninvasive multielectrode electrocardiographic mapping technique.

200 ive imaging techniques based on body surface electrocardiographic mapping to elucidate the mechanisms

201 Electrocardiographic maps revealed homogeneous patterns

202 No electrocardiographic measure is specific enough to provi

203 The QT interval, an electrocardiographic measure reflecting myocardial repol

204 The V(2) transition ratio is a novel electrocardiographic measure that reliably distinguishes

205 ations between usual dietary fish intake and electrocardiographic measures of heart rate, atrioventri

206 Although electrocardiographic measures of repolarization abnormal

207 ation pattern were reviewed to delineate the electrocardiographic measures to be used when defining t

208 phenotypes relating to metabolic traits and electrocardiographic measures, along with another 8 prev

209 cardiac mode at four pitch values and three electrocardiographic modulation temporal windows.

210 be initiated or reinitiated with continuous electrocardiographic monitoring and in the presence of t

211 ommendations on indications and duration for electrocardiographic monitoring in accordance with the A

212 SCAF is frequently detected by continuous electrocardiographic monitoring in older patients withou

213 Thus, electrocardiographic monitoring is required to minimize

214 Continuous electrocardiographic monitoring of cardiac activity demo

215 commendations for Indication and Duration of Electrocardiographic Monitoring presented by patient pop

216 Long-term continuous electrocardiographic monitoring shows a substantial prev

217 (AT), verified by monthly visits, ambulatory electrocardiographic monitoring, and implantable loop re

218 echocardiography, optical mapping, telemetry electrocardiographic monitoring, and inducibility studie

219 5 days after surgery, as assessed by Holter electrocardiographic monitoring, and myocardial injury w

220 on formulas should be considered for routine electrocardiographic monitoring.

221 We implanted subcutaneous electrocardiographic monitors (St. Jude CONFIRM-AF) in p

222 ate, ST segment behavior, and arrhythmias in electrocardiographic monitors may lead to inappropriate

223 Patients were followed with implanted electrocardiographic monitors when possible (85.2% of FI

224 We compared the electrocardiographic morphology of ventricular tachycard

225 In 18 patients, the 12-lead electrocardiographic morphology was left bundle branch b

226 were ICU mortality, readmission to ICU, new electrocardiographic or cardiac enzyme changes suggestiv

227 r fibrillation (VF) and sudden death without electrocardiographic or echocardiographic abnormalities

228 f abnormalities were observed in laboratory, electrocardiographic, or Holter monitoring assessments.

229 Electrocardiographic P-wave duration agreed with the dur

230 h baseline echocardiographic (p < 0.001) and electrocardiographic (p < 0.001) LVM remained associated

231 ardiogram was recorded through defibrillator/electrocardiographic pads oriented in the standard cardi

232 tive prognostic value to routine clinical or electrocardiographic parameters (P<0.001).

233 ks models evaluated associations between GEH electrocardiographic parameters and SCD.

234 is study sought to analyze the usefulness of electrocardiographic parameters as markers of sudden car

235 ween dietary fish intake and several cardiac electrocardiographic parameters in humans relevant to ar

236 Electrocardiographic parameters of sympathetic nervous s

237 Electrocardiographic parameters were measured from the s

238 sence of diabetes mellitus, body mass index, electrocardiographic parameters, B-type natriuretic pept

239 deled the effects of ion channel activity on electrocardiographic parameters, estimating the change i

240 ciation studies conducted in recent years on electrocardiographic parameters, highlighting their pote

241 Electrocardiographic parameters, including QT intervals,

242 analysis of blood chemistry, urinalysis, and electrocardiographic parameters; these methods were anal

243 oung patients with the Wolff-Parkinson-White electrocardiographic pattern are scarce.

244 by using the Valsalva maneuver to reveal the electrocardiographic pattern in family members.

245 a defibrillator (CRT-D) in patients with an electrocardiographic pattern showing left bundle-branch

246 This electrocardiographic pattern was considered benign until

247 PP1 did not change infarct size, electrocardiographic pattern, or cardiac function.

248 It discusses the electrocardiographic phenomenon of T-wave alternans (TWA

249 cell, 2D, and 3D) accurately reproduced the electrocardiographic phenotype of the proband, including

250 Early repolarization (ER), a common electrocardiographic phenotype, has been associated with

251 The electrocardiographic PR interval increases with aging, d

252 notypes: higher resting heart rate (HR), the electrocardiographic PR interval, atrial fibrillation an

253 Prolongation of the electrocardiographic PR interval, known as first-degree

254 ividuals from a primary care population with electrocardiographic preexcitation.

255 Increased electrocardiographic QRS duration (>/=120 ms) is a marke

256 We evaluated the relations of maximal electrocardiographic QRS duration to echocardiographic L

257 of this study was to assess the relations of electrocardiographic QRS duration to left ventricular (L

258 of individuals free of HF and MI, increasing electrocardiographic QRS duration was positively related

259 The electrocardiographic QRS duration, a measure of ventricu

260 Prolongation of the electrocardiographic QT interval is a risk factor for su

261 Prolongation of the electrocardiographic QT interval, a measure of cardiac r

262 Extremes of the electrocardiographic QT interval, a measure of cardiac r

263 eterminant of arrhythmia, as measured by the electrocardiographic QT interval.

264 cal hypokalaemia is associated with acquired electrocardiographic QT prolongation and arrhythmic acti

265 resulted in a significant shortening of the electrocardiographic QTc interval and reduction of left

266 nitrogen and creatinine levels and a longer electrocardiographic QTc interval than did the sham grou

267 Hypoglycemia produces electrocardiographic QTc lengthening, a predictor of arr

268 e intensive care unit is a relatively common electrocardiographic reading both by standard interpreta

269 33a levels increased QT intervals in surface electrocardiographic recordings and action potential dur

270 Continuous electrocardiographic recordings and serial blood samplin

271 iability was calculated in 5-min sections of electrocardiographic recordings at baseline and 4 hrs af

272 Heart period variability was analyzed from electrocardiographic recordings collected from 159 preho

273 (10-15 mg/dL) clamps for 3 h with continuous electrocardiographic recordings.

274 changes in heart rate variability (HRV) and electrocardiographic repolarization changes measured bef

275 value over traditional risk factors, stress electrocardiographic, rest echocardiographic, and SE var

276 ardiopulmonary arrests regardless of initial electrocardiographic rhythm with return of spontaneous c

277 Electrocardiographic sampling was done for two days befo

278 g debate about the role of pre-participation electrocardiographic screening for the prevention of sud

279 Adding electrocardiographic screening is being considered by so

280 Echocardiographic and electrocardiographic screening of first-degree relatives

281 ive prognostic value to routine clinical and electrocardiographic selection criteria for cardiac resy

282 uce, from digital files, the original analog electrocardiographic signals of previously instrumented

283 onin-based criteria were less likely to have electrocardiographic ST-segment elevation and had better

284 the final study population of 46 cases with electrocardiographic ST-segment elevation myocardial inf

285 it patients who present with chest pain, the electrocardiographic ST-segment elevation myocardial inf

286 The genesis of the electrocardiographic T wave is incompletely understood a

287 'Cardiac memory' describes an electrocardiographic T wave vector change, recorded duri

288 on studies have found an association between electrocardiographic T-wave morphology parameters and ca

289 We assessed the predictive value of electrocardiographic T-wave morphology parameters and TP

290 Electrocardiographic T-wave morphology parameters descri

291 up studies assessing the association between electrocardiographic T-wave peak to T-wave end interval

292 A) reflects beat-to-beat fluctuations in the electrocardiographic T-wave, and is associated with disp

293 rried out a genome-wide association study of electrocardiographic time intervals in 6,543 Indian Asia

294 llumina HumanCVD Beadchip and 4 quantitative electrocardiographic traits (PR interval, QRS axis, QRS

295 Electrocardiographic traits are important, substantially

296 s genome-wide association study findings for electrocardiographic traits, while the expression analys

297 hythmia suppression is essential for optimal electrocardiographic triggering and image acquisition.

298 for ARVC when applied to athletes exhibiting electrocardiographic TWI and to identify discriminators

299 The mother had electrocardiographic U-wave changes consistent with Ande

300 Electrocardiographic variables that significantly increa