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1  We have studied the neuroanatomic extent of electroconvulsive (ECS)-responsive prepro-TRH and TRH-re
2 studies that use invasive pharmacological or electroconvulsive interventions to disrupt a putative po
3                                   The robust electroconvulsive repertoire of DLMs provides a convenie
4 gly, wild-type flies also displayed the same electroconvulsive repertoire, albeit inducible only at h
5                                      Chronic electroconvulsive seizure (chr-ECS), one of the most eff
6             Among antidepressant treatments, electroconvulsive seizure (ECS) is the most robust stimu
7                                     A single electroconvulsive seizure induces the expression of a nu
8                                              Electroconvulsive seizure therapy (ECS) is a clinically
9  Arc, but only after a very strong stimulus (electroconvulsive seizure).
10             Arc expression was induced by an electroconvulsive seizure, and the newly synthesized mRN
11 n hypothalamic tissues and upregulated after electroconvulsive seizure.
12 ine oxidase inhibitor (tranylcypromine), and electroconvulsive seizure.
13                                     Repeated electroconvulsive seizures (ECS) induced isoforms of Del
14 ats subjected to sham seizures or to 1 or 10 electroconvulsive seizures (ECS), a model of ECT.
15 pocampus and other brain regions by repeated electroconvulsive seizures (ECS), an effective antidepre
16  of Narp, an immediate early gene induced by electroconvulsive seizures (ECS), blocks its antidepress
17                   The mechanism of action of electroconvulsive seizures (ECS), one of the most effect
18 rent study examines the influence of chronic electroconvulsive seizures (ECSs), as well as chemical a
19 antipsychotic or antidepressant medications, electroconvulsive seizures, and certain lesions.
20 ral chronic perturbations, including chronic electroconvulsive seizures, psychotropic drug treatments
21 imuli, including drugs of abuse, stress, and electroconvulsive seizures.
22  and KA2 mRNAs in the rat hippocampus, using electroconvulsive shock (ECS) as a seizure paradigm not
23                                      Because electroconvulsive shock (ECS) can stimulate the producti
24        Head trauma leading to concussion and electroconvulsive shock (ECS) in humans causes amnesia f
25 lecular mechanisms underlying the effects of electroconvulsive shock (ECS) therapy, a fast-acting and
26                        In the present study, electroconvulsive shock (ECS) was used to assess maximal
27           Repeated applications (10 days) of electroconvulsive shock (ECS), but not a single treatmen
28              Adult monkeys received repeated electroconvulsive shock (ECS), which is the animal analo
29                We found that brief, repeated electroconvulsive shock (ECS)-induced seizures (three in
30                                              Electroconvulsive shock administration showed up-regulat
31 ninergic system in the monoaminergic nuclei: Electroconvulsive shock elevated galanin mRNA levels in
32 BN 50730 strongly attenuates kainic acid and electroconvulsive shock induction of prostaglandin G/H s
33                  The drug partially inhibits electroconvulsive shock induction of zif-268, but is rel
34 greater in the kainic acid model than in the electroconvulsive shock model and is much more prolonged
35 -onset antidepressant effects in the clinic: electroconvulsive shock therapy, sleep deprivation, keta
36 ilon(-/-) mice showed a higher resistance to electroconvulsive shock with shorter tonic seizures and
37 uclear orphan receptor family, is induced by electroconvulsive shock, an effective treatment for depr
38                               In response to electroconvulsive shock, heteroallelic combinations with
39               In response to kainic acid and electroconvulsive shock-induced seizures, KID-1 is induc
40 c acid (KA)-induced seizures and by a single electroconvulsive shock.
41  a reduced threshold for seizures induced by electroconvulsive shock.
42  a reduced threshold for seizures induced by electroconvulsive shock.
43 c flies following mechanical stimulation and electroconvulsive shock.
44 i such as ischemic insult, visceral pain and electroconvulsive shock.
45 resent study, we investigated the effects of electroconvulsive shocks (ECS) on the ASR, in a rat mode
46                                              Electroconvulsive stimulation (ECS) has been shown recen
47                                              Electroconvulsive stimulation (ECS), the experimental an
48                             IEG induction by electroconvulsive stimulation is virtually abolished in
49  gene expression in rat brain after rTMS and electroconvulsive stimulation, a well-established animal
50 of immediate-early gene expression than does electroconvulsive stimulation.
51                                              Electroconvulsive stimuli across the brain induced a sim
52 itive side effects of right unilateral (RUL) electroconvulsive therapy (ECT) are reported to depend o
53                                              Electroconvulsive therapy (ECT) at conventional current
54                                We have found electroconvulsive therapy (ECT) can produce life-changin
55 fficacy of right unilateral ultrabrief pulse electroconvulsive therapy (ECT) combined with venlafaxin
56 mmendations to tighten federal regulation of electroconvulsive therapy (ECT) devices have focused att
57                                              Electroconvulsive therapy (ECT) elicits a rapid and robu
58 TMS) has been reported to be as effective as electroconvulsive therapy (ECT) for major depression.
59 nd ethical controversy concerning the use of electroconvulsive therapy (ECT) for psychiatric disorder
60 and individualizing the current amplitude in electroconvulsive therapy (ECT) has been proposed as a m
61                                     Although electroconvulsive therapy (ECT) has been shown to be ext
62                                              Electroconvulsive therapy (ECT) has been successfully us
63  This may relate to the clinical efficacy of electroconvulsive therapy (ECT) in humans.
64 istance is the leading indication for use of electroconvulsive therapy (ECT) in major depression.
65                                              Electroconvulsive therapy (ECT) is a robust and rapidly
66                                     Although electroconvulsive therapy (ECT) is considered the most e
67                              The efficacy of electroconvulsive therapy (ECT) is determined by stimulu
68                                              Electroconvulsive therapy (ECT) is highly effective for
69                                   The use of electroconvulsive therapy (ECT) is limited by concerns a
70                                              Electroconvulsive therapy (ECT) is one of the most effec
71                                              Electroconvulsive therapy (ECT) is regarded by many clin
72                                     Although electroconvulsive therapy (ECT) is the most effective ac
73                                     To date, electroconvulsive therapy (ECT) is the most potent treat
74 ic medication but that responded promptly to electroconvulsive therapy (ECT) on two separate occasion
75                                              Electroconvulsive therapy (ECT) remains the treatment of
76 ng to the rapid and robust clinical effects, electroconvulsive therapy (ECT) represents an optimal mo
77 persistent cognitive defects associated with electroconvulsive therapy (ECT), a highly effective and
78 mulation (ECS), the experimental analogue of electroconvulsive therapy (ECT), has been shown to produ
79            Brain stimulation, in the form of electroconvulsive therapy (ECT), has long been a gold st
80 e shock (ECS), which is the animal analog of electroconvulsive therapy (ECT), the most effective shor
81 severely depressed patients before and after electroconvulsive therapy (ECT), we measured cerebrospin
82                                              Electroconvulsive therapy (ECT), which has been in use f
83  was treated with a five-treatment course of electroconvulsive therapy (ECT), which resulted in a com
84  various antidepressant treatments including electroconvulsive therapy (ECT).
85 provement in depressed individuals receiving electroconvulsive therapy (ECT).
86 nt with antidepressants, mood stabilizers or electroconvulsive therapy (ECT).
87 of right unilateral (RUL) and bilateral (BL) electroconvulsive therapy (ECT).
88  most persistent cognitive adverse effect of electroconvulsive therapy (ECT); however, it is not know
89  therapy with ultra-brief pulse continuation electroconvulsive therapy (ECT-arm), or no add-on therap
90 categories: (1) seizure therapies, including electroconvulsive therapy and magnetic seizure therapy,
91 arbamazepine, oxcarbazepine, antipsychotics, electroconvulsive therapy and various light intervention
92 being evaluated as a possible alternative to electroconvulsive therapy for the treatment of refractor
93 cal agents were effective for depression and electroconvulsive therapy for treatment-resistant bipola
94 lar disorder, as well as antidepressants and electroconvulsive therapy have recently been shown to ac
95                                              Electroconvulsive therapy is used in those patients who
96 on, but psychotherapy, exercise therapy, and electroconvulsive therapy may also be effective.
97                                              Electroconvulsive therapy may be associated with reduced
98                                              Electroconvulsive therapy produced a marked short-term i
99                                              Electroconvulsive therapy responders received 6 months g
100                                              Electroconvulsive therapy use for severely depressed inp
101                          Pharmacotherapy and electroconvulsive therapy were found to be effective tre
102 ranscranial magnetic stimulation (instead of electroconvulsive therapy) for drug-resistant major depr
103 n (both experimentally and clinically, as in electroconvulsive therapy).
104  patients hospitalized preoperatively (e.g., electroconvulsive therapy).
105 eased in rodent brains by antidepressants or electroconvulsive therapy, but decreased in an animal mo
106 apy, ketogenic diet, emergency neurosurgery, electroconvulsive therapy, cerebrospinal fluid drainage,
107 l nerve stimulation, deep brain stimulation, electroconvulsive therapy, drainage of the cerebrospinal
108 reatment, hospital admission for depression, electroconvulsive therapy, emergency department visit fo
109 es, dopamine agonists, and/or dantrolene) or electroconvulsive therapy, if indicated.
110 ticholinergic exposure, lifetime exposure to electroconvulsive therapy, vascular illness, and incipie
111 ulation, a well-established animal model for electroconvulsive therapy.
112 effectively treated with antidepressants and electroconvulsive therapy.
113 order and resistant to other medications and electroconvulsive therapy.
114 consultation and treatment changes including electroconvulsive therapy.
115 itive or behavioural therapy), and sometimes electroconvulsive therapy.
116                                          The electroconvulsive threshold (ECT) test has been used ext
117                                          The electroconvulsive threshold (ECT) test is used commonly
118  more effective and timely interventions for electroconvulsive treatment.

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