ライフサイエンスコーパス: electrocorticography

1 reo-electroencephalography or intraoperative electrocorticography.

2 cy local field potentials (LFPs) recorded on electrocorticography.

3 preresection evaluations and intraoperative electrocorticography.

4 iduals were excluded because of poor-quality electrocorticography.

5 t these short EEEs are undetectable by scalp electrocorticography.

6 ivity using quantified behavioral rating and electrocorticography.

7 cortical resection guided by intraoperative electrocorticography.

8 The electrocorticography amplitude changes of high-gamma (70

9 On electrocorticography analysis, high-gamma augmentation i

10 del, a circadian probability, and a combined electrocorticography and circadian model.

11 dband and arrhythmic, or scale-free, macaque electrocorticography and human magnetoencephalography ac

12 Simultaneous epidural-electrocorticography and scalp-electroencephalography re

13 clinical practice of observing epileptiform electrocorticography and simultaneous ictal behaviour, a

14 used simultaneous, colocalized recordings of electrocorticography and tissue oxygen pressure (p(ti)O(

15 enile swine using subdural electrode strips (electrocorticography) and intraparenchymal neuromonitori

16 on, direct cortical electrical interference, electrocorticography, and a variation of the technique o

17 From high-resolution electrocorticography arrays implanted on motor and senso

18 sured high gamma-range field potentials from electrocorticography arrays implanted over a large porti

19 We compared the results of an electrocorticography-based logistic regression model, a

20 Finally, we present four human examples of electrocorticography capturing short (<2 s), stereotyped

21 We report on a quantitative analysis of electrocorticography data from a study that acquired con

22 Recent electrocorticography data have demonstrated excessive co

23 We used electrocorticography data obtained from frontal and temp

24 f multiple neurochemicals and direct-current electrocorticography (DC-ECoG).

25 hat the slow cortical potentials recorded by electrocorticography demonstrate a correlation structure

26 sholds, were determined against intracranial electrocorticography-determined seizure-onset region and

27 acted to doxycycline exposure by spontaneous electrocorticography-documented nonconvulsive seizures,

28 Frontal electrocorticography [duration: 54 h (34, 66)] from subd

29 recorded cortical physiology using subdural electrocorticography during a spatial-attention task to

30 from hand sensorimotor cortex using subdural electrocorticography during a visually cued, incentivize

31 Using high-density EEG and intracranial electrocorticography during gradual induction of propofo

32 Spreading depolarisations were monitored by electrocorticography during intensive care and were clas

33 seizures were assessed by 5-electrode video-electrocorticography (ECoG) 2 to 16 weeks postinjury.

34 We used a combination of electrocorticography (ECoG) and electrical brain stimula

35 human participants (both males and females): electrocorticography (ECoG) and fMRI.

36 calp EEG and across much of the cortex using electrocorticography (ECOG) and localized transcortical

37 ween electric field potentials measured with electrocorticography (ECoG) and the blood oxygen level-d

38 ctroencephalography (EEG) and intraoperative electrocorticography (ECoG) are routinely used in the ev

39 In this study subjects implanted with electrocorticography (ECoG) arrays for long-term epileps

40 Intraoperative electrocorticography (ECoG) can be used to delineate the

41 from electrical stimulation and from resting electrocorticography (ECoG) correlations showed similar

42 are tools supporting the analysis of complex Electrocorticography (ECoG) data.

43 Chronic electrocorticography (ECoG) demonstrated spontaneous chr

44 Seven subjects were implanted with electrocorticography (ECoG) electrodes and had multiple

45 reactivity relative to conventional clinical electrocorticography (ECoG) electrodes.

46 tection task, we provide evidence from human electrocorticography (ECoG) for an inverted-U brain-beha

47 Purpose To develop an electrocorticography (ECoG) grid by using deposition of

48 Although acute electrocorticography (ECoG) is routinely performed durin

49 activity directly from the cortical surface, electrocorticography (ECoG) provides a powerful method t

50 asures obtained from extraoperative subdural electrocorticography (ECoG) recording could predict long

51 derwent corpus callosal transection prior to electrocorticography (ECoG) recordings and ICH injury.

52 on a fine spatiotemporal scale by recording electrocorticography (ECoG) signals measured directly fr

53 High-resolution electrocorticography (ECoG) signals were recorded direct

54 n this paper, we present a portable wireless electrocorticography (ECoG) system.

55 Here, we used a multimodal approach of electrocorticography (ECoG), high-resolution functional

56 Our study investigated the feasibility of an electrocorticography (ECoG)-based BCI system in an indiv

57 as high-frequency local field potentials and electrocorticography (ECoG).

58 tudied with magnetoencephalography (MEG) and electrocorticography (ECoG).

59 atic brain injury were monitored by invasive electrocorticography (ECoG; subdural electrodes) and non

60 ocal, recurrent and spontaneous epileptiform electrocorticography events (EEEs) that are never observ

61 erent groups of human subjects: intracranial electrocorticography from 15 participants over a 38 year

62 The analyses used long-term, continuous electrocorticography from nine subjects, recorded for an

63 ing functional magnetic resonance imaging or electrocorticography have produced inconsistent results.

64 gery, we use the novel technique of subdural electrocorticography in combination with subthalamic nuc

65 netetrazole-induced seizures was assessed by electrocorticography in head-restrained nonanesthetized

66 hypothesis using magnetoencephalography and electrocorticography in humans to record changes in neur

67 tial and temporal resolution of intracranial electrocorticography in humans.

68 Electrocorticography indices of cortical activation at t

69 al electrodes, similar ultra-flexible, micro-electrocorticography (mu-ECoG) arrays with platinum (Pt)

70 went two-stage epilepsy surgery with chronic electrocorticography recording were studied.

71 on and the interaction between them, we used electrocorticography recordings from 16 neurosurgical su

72 Here we addressed these issues using electrocorticography recordings in epileptic patients.

73 rrelation of MR findings with intraoperative electrocorticography results indicated that the MR study

74 ehavioural analysis performed blinded to the electrocorticography revealed that (i) brief EEEs lastin

75 stimulation and event-related modulation of electrocorticography signals.

76 Here we examined this issue in a large-scale electrocorticography study in patients performing a dema

77 In this electrocorticography study of human patients, we examine

78 Recently we showed with invasive electrocorticography that one robust measure of this syn

79 Here we use electrocorticography to address this uncertainty in thre

80 Here, we use human resting-state fMRI and electrocorticography to demonstrate that delta-band acti

81 rd steady-state visual evoked potentials via electrocorticography to directly assess the responses to

82 Here, we leveraged electrocorticography to examine the temporal profile of

83 y in rats following status epilepticus, late electrocorticography to identify epileptic animals and p

84 ed temporal resolution of human intracranial electrocorticography to investigate the mechanisms by wh

85 We used high-density electrocorticography to record neural population activit

86 To address this, we utilized subdural electrocorticography to study cortical oscillations in t

87 iovisual) stimuli with a button press, while electrocorticography was recorded over auditory and moto

88 Spectral content and bandwidth of vascular electrocorticography were comparable to those of recordi

89 the patients' epileptogenic zone, defined by electrocorticography, were resected neurosurgically from