ライフサイエンスコーパス: electroencephalogram

1 eneralized spike and wave discharges seen on electroencephalogram.

2 fter cardiac arrest and preceded isoelectric electroencephalogram.

3 e performed a spectral analysis of the sleep electroencephalogram.

4 he theta rhythm, as shown on the hippocampal electroencephalogram.

5 signal strength and map dissimilarity of the electroencephalogram.

6 early visual cortex as measured by the human electroencephalogram.

7 oach to measure mnemonic hidden states in an electroencephalogram.

8 eration of a local field potential (LFP) and electroencephalogram.

9 t timescale interaction with the hippocampal electroencephalogram.

10 n reported using the median frequency of the electroencephalogram.

11 re burden and a relatively normal background electroencephalogram.

12 numeric scale from 0 to 100 derived from the electroencephalogram.

13 y obvious signs of seizure activity on scalp electroencephalogram.

14 ings with foramen ovale electrodes and scalp electroencephalogram.

15 ed evoked responses relative to pre-stimulus electroencephalogram.

16 in functional magnetic resonance imaging and electroencephalogram.

17 mes of brain probed by clinical intracranial electroencephalograms.

18 n electrographic signatures in intracerebral electroencephalograms.

19 monitor that allows continuous recording of electroencephalogram activity at the bedside and to dete

20 Lack of npas2 affected electroencephalogram activity of thalamocortical origin;

21 The second finding is that oscillatory electroencephalogram activity recorded over right tempor

22 ation again, this time applied to observers' electroencephalogram activity, we established where and

23 ical blood flow, in response to synchronized electroencephalogram activity.

24 Isoelectric or low-voltage electroencephalograms after 24 hrs predicted poor outcom

25 Electroencephalogram allows reliable prediction of both

26 Electroencephalogram alpha power at age 8 significantly

27 ir social skills, and the children's resting electroencephalogram alpha power was recorded.

28 Quantitative electroencephalogram analysis revealed anomalous increas

29 aemic encephalopathy, an abnormal background electroencephalogram and a large seizure burden, and hav

30 tinct brain state characterized by activated electroencephalogram and complete skeletal muscle paraly

31 In all patients, continuous electroencephalogram and daily somatosensory evoked pote

32 CI device integrates wearable, wireless, dry electroencephalogram and electrooculogram systems and a

33 were monitored for seizures by serial video-electroencephalogram and for long-term survival.

34 lunteers for the acquisition of simultaneous electroencephalogram and functional magnetic resonance i

35 Simultaneous recording of electroencephalogram and functional MRI is being increas

36 ls debated the issues on the accuracy of the electroencephalogram and its place, the absolute need fo

37 amma during fast stopping and recorded scalp electroencephalogram and local field potentials from dee

38 oth had uneventful perinatal courses, normal electroencephalogram and magnetic resonance imaging scan

39 (5) Ancillary studies (electroencephalogram and radionuclide cerebral blood flo

40 5) Ancillary studies (electroencephalogram and radionuclide cerebral blood flo

41 The number of epileptiform discharges in the electroencephalogram and the number of clinical seizures

42 llations measured by local field potentials, electroencephalograms and magnetoencephalograms exhibit

43 s were assessed using 5-minute resting-state electroencephalograms and parallel electrocardiograms.

44 Mean HEP amplitudes in resting-state electroencephalograms and their correlation with self-re

45 and subthalamic nucleus along with cortical electroencephalograms and were compared to recordings fr

46 Clinical, electroencephalogram, and ECG data for each of their sei

47 pilepsy localization from seizure semiology, electroencephalogram, and magnetic resonance imaging.

48 in responses were recorded with high-density electroencephalogram, and sources of event-related poten

49 tivity and prolonged propagation time on the electroencephalogram, and the absence of metabolic dysfu

50 unction of pyramidal cell activity, with the electroencephalogram approximated by the sum of populati

51 Electroencephalograms are recorded while subjects say 'a

52 erized by spike-wave discharges (SWD) in the electroencephalogram, arises from aberrations within the

53 tions in membrane potential, which appear in electroencephalograms as slow wave activity (SWA) of <4

54 gativity (ERN), a negative deflection in the electroencephalogram associated with error commission.

55 evelops during SRSE despite seizure control (electroencephalogram background suppression with anesthe

56 nt effects on state consolidation and/or the electroencephalogram but had no effect on total wake.

57 by video observation of each animal and the electroencephalogram by an automated seizure detection p

58 Furthermore, we found that electroencephalogram cascades are related to blood oxyge

59 r 2 are suspected to be epileptogenic and if electroencephalogram changes are equivocal or discordant

60 Gaining a better understanding of sleep and electroencephalogram changes in patients with Huntington

61 Electroencephalogram characterization revealed auditory

62 mesial temporal lobe seizures based on scalp electroencephalogram coherence features, lends weight to

63 istic regression classifiers that used scalp electroencephalogram coherence properties as input featu

64 hol dependence and the beta frequency of the electroencephalogram, combined with biological evidence

65 ave a corresponding electrographic change on electroencephalogram consistent with hyperekplexia.

66 alized epilepsy and frequent absences, using electroencephalogram-correlated functional magnetic reso

67 relationship between cascades of activity in electroencephalogram data, cognitive state, and reaction

68 bispectral index monitor was used to capture electroencephalogram data.

69 Neither hypoxia nor electroencephalogram-defined arousals alone increased ar

70 M sleep time (r = 0.77; p < 0.001) and total electroencephalogram delta power (r = 0.79; p < 0.001) b

71 c acid receptor antagonist, reduces cortical electroencephalogram delta power and transiently inhibit

72 chanisms, and the low-frequency power in the electroencephalogram (delta power) during non-rapid eye

73 hypothesis that a protocol incorporating the electroencephalogram-derived bispectral index (BIS) is s

74 We compared distributions of electroencephalogram-derived cascades to reference power

75 make no recommendation concerning the use of electroencephalogram-derived parameters as a measure of

76 A detailed history, physical examination, electroencephalogram, developmental evaluation, Autism D

77 Electroencephalograms did not show the "typical" appeara

78 Here we studied sleep and electroencephalogram disturbances in a transgenic mouse

79 by analyzing event-related potentials in the electroencephalogram during a Go/NoGo task.

80 closely parallel those observed in the human electroencephalogram during propofol-induced unconscious

81 by measuring the level of randomness in the electroencephalogram during the prestimulus baseline per

82 e occipitotemporal regions bilaterally using electroencephalograms during several visual recognition

83 edominately display bilaterally synchronized electroencephalogram (EEG) activity during slow-wave sle

84 k, many mouse models for AD exhibit abnormal electroencephalogram (EEG) activity in addition to the e

85 of arousal: sleep and wakefulness, cortical electroencephalogram (EEG) activity, acetylcholine (ACh)

86 burst latency relative to sleep with stable electroencephalogram (EEG) and breathing (1.313 +/- 0.03

87 medullary reticular formation, and implanted electroencephalogram (EEG) and ECG recording electrodes.

88 continuous-valued neural recordings like the electroencephalogram (EEG) and local field potential (LF

89 itoring the patient's brain activity with an electroencephalogram (EEG) and manually titrating the an

90 ombined measures of cortical and hippocampal electroencephalogram (EEG) and neck muscle electromyogra

91 produced predominantly delta activity in the electroencephalogram (EEG) and sedation.

92 Electroencephalogram (EEG) approaches may provide import

93 al activity, because these components of the electroencephalogram (EEG) are sensitive to basal forebr

94 even though these conditions elicit maximal electroencephalogram (EEG) arousal.

95 Electroencephalogram (EEG) arousals also decreased in mi

96 s, and slow-wave sleep with interhemispheric electroencephalogram (EEG) asymmetry, resembling the uni

97 ental delay, and epileptiform abnormality on electroencephalogram (EEG) before withdrawal.

98 e highly synchronous across the scalp in the electroencephalogram (EEG) but have low spatial coherenc

99 However, electroencephalogram (EEG) changes in the theta-frequenc

100 Research on electroencephalogram (EEG) characteristics associated wi

101 Decreases in prefrontal electroencephalogram (EEG) cordance that are detectable

102 respond to sleep deprivation, the strongest electroencephalogram (EEG) correlate of sleep pressure,

103 the clinical, psychometric, and wake-/sleep-electroencephalogram (EEG) correlates of induced hyperam

104 the current study using spectral analysis of electroencephalogram (EEG) data during sleep.

105 ough multivariate classification analyses of electroencephalogram (EEG) data.

106 Electroencephalogram (EEG) delta wave power was decrease

107 ngle neurons before, during, and after ictal electroencephalogram (EEG) discharges.

108 increases in the delta-wave activity of the electroencephalogram (EEG) during NREMS, whereas EEG act

109 in alpha- and beta-range oscillations in the electroencephalogram (EEG) during observation of reachin

110 ance, brain biomarkers, and abnormalities in electroencephalogram (EEG) during the perioperative peri

111 In controls, the electroencephalogram (EEG) exhibited oscillatory activit

112 approach to study the effects of EMFs on the electroencephalogram (EEG) from rabbits.

113 Cholinergic systems are crucial in electroencephalogram (EEG) generation and regulation of

114 ate the acute effects of alcohol on cortical electroencephalogram (EEG) in adolescent (P36) and adult

115 amma-to-beta transition is seen in the human electroencephalogram (EEG) in response to novel auditory

116 blood flow (rCBF) and synchronization of the electroencephalogram (EEG) in the rat cerebral cortex el

117 The electroencephalogram (EEG) is a mainstay of clinical neu

118 Reduced background activity on electroencephalogram (EEG) is a sensitive marker of brai

119 Variation in resting electroencephalogram (EEG) is associated with common, co

120 The human electroencephalogram (EEG) is generated predominantly by

121 The signature of slow-wave sleep in the electroencephalogram (EEG) is large-amplitude fluctuatio

122 s, and seizure burden during 48 h continuous electroencephalogram (EEG) monitoring.

123 y review the principles underlying processed electroencephalogram (EEG) monitors and recent studies v

124 ent decline in the slow wave (delta, 1-4 Hz) electroencephalogram (EEG) of nonrapid eye movement (NRE

125 taneous, ChR2, or forepaw stimulation-evoked electroencephalogram (EEG) or local field potential (LFP

126 locked signals superimposed upon the ongoing electroencephalogram (EEG) or result from phase-alignmen

127 eration depends upon theta rhythm, a 6-10 Hz electroencephalogram (EEG) oscillation that is modulated

128 Abnormal resting state electroencephalogram (EEG) oscillations are reported in

129 ected by frontal-midline theta-band (4-8 Hz) electroencephalogram (EEG) oscillations, strengthen the

130 Fast beta (20-28 Hz) electroencephalogram (EEG) oscillatory activity may be a

131 l population are linked to variations in the electroencephalogram (EEG) over motor, pre-motor cortex

132 frog (Babina daunchina) using the optimized electroencephalogram (EEG) paradigm of mismatch negativi

133 EM sleep by an "activated," low-voltage fast electroencephalogram (EEG) paradoxically similar to that

134 es of unknown etiology with a characteristic electroencephalogram (EEG) pattern and developmental reg

135 ggered in real time as a function of ongoing electroencephalogram (EEG) phase.

136 al isolation on sleep, wakefulness and delta electroencephalogram (EEG) power during non-rapid eye mo

137 ergic neurons on the sleep-wake behavior and electroencephalogram (EEG) power spectrum using the phar

138 Clinical and demographic data, electroencephalogram (EEG) readings, and treatment respo

139 We have conducted noninvasive electroencephalogram (EEG) recording of the brain neuron

140 During adulthood, electroencephalogram (EEG) recordings are used to distin

141 in the cerebral cortex fire irregularly and electroencephalogram (EEG) recordings display low-amplit

142 e simultaneous human intrathalamic and scalp electroencephalogram (EEG) recordings from eight volunte

143 ng an array of 12 behavioral assessments and electroencephalogram (EEG) recordings on freely-moving m

144 Behavioral phenotyping combined with electroencephalogram (EEG) recordings revealed that asc-

145 study was to use oscillatory changes in the electroencephalogram (EEG) related to informative cue pr

146 ndings, somatosensory evoked potentials, and electroencephalogram (EEG) results were recorded.

147 Electroencephalogram (EEG) showed generalized spike and

148 The slow (<1 Hz) rhythm, the most important electroencephalogram (EEG) signature of non-rapid eye mo

149 Sleep spindles are characteristic electroencephalogram (EEG) signatures of stage 2 non-rap

150 investigating characteristics of individual electroencephalogram (EEG) slow waves in young and elder

151 sleep homeostasis: slow-wave sleep (SWS) and electroencephalogram (EEG) slow-wave activity in non-rap

152 is work uses an innovative method to analyze electroencephalogram (EEG) spectral frequencies within s

153 acological treatments in rats to study which electroencephalogram (EEG) spectral properties are assoc

154 Electroencephalogram (EEG) stands out as a highly transl

155 ation approach for the analysis of the human electroencephalogram (EEG) to decode choice outcomes in

156 attention has turned to assessments based on electroencephalogram (EEG) to evaluate subtle post-concu

157 We used portable electroencephalogram (EEG) to simultaneously record brai

158 Interictal electroencephalogram (EEG) was normal in all cases but 2

159 During session 2, an electroencephalogram (EEG) was recorded during cued reca

160 The electroencephalogram (EEG) was recorded from 19 scalp lo

161 The electroencephalogram (EEG) was recorded while human part

162 CA1 blood flow and electroencephalogram (EEG) were continuously recorded.

163 Dense multichannel recordings of scalp electroencephalogram (EEG) were obtained in the vicinity

164 of a neurological disorder, and an abnormal electroencephalogram (EEG) were significant factors in i

165 pe test), or seizure activity (measured with electroencephalogram (EEG)).

166 uring somatosensory evoked potentials (SEP), electroencephalogram (EEG), direct current (DC) potentia

167 f human activity, biological signals such as Electroencephalogram (EEG), Electrooculogram (EOG), Elec

168 rized by fast, desynchronized rhythms in the electroencephalogram (EEG), hippocampal theta activity,

169 s activation of the cortical and hippocampal electroencephalogram (EEG), rapid eye movements, and los

170 seizure origin through an analysis of ictal electroencephalogram (EEG), which is proven to be an eff

171 Non-invasive, electroencephalogram (EEG)-based brain-computer interfac

172 A recent investigation has revealed that electroencephalogram (EEG)-derived movement-related cort

173 istence of a slow wave sleep (SWS)-promoting/electroencephalogram (EEG)-synchronizing center in the m

174 ovel method of tagging memories in the human electroencephalogram (EEG).

175 tems approach to motivate an analysis of the electroencephalogram (EEG).

176 ain activity during sleep measured using the electroencephalogram (EEG).

177 est spontaneous waves observed in the normal electroencephalogram (EEG).

178 d activity in the low-frequency bands in the electroencephalogram (EEG).

179 ry 6-12 months until they had an isoelectric electroencephalogram (EEG, attesting to a vegetative sta

180 ated markers of consciousness extracted from electroencephalograms (EEG), we computed autonomic cardi

181 (PB) complex in regulating electrocortical (electroencephalogram [EEG]) and behavioral arousal: lesi

182 art rate, and respiratory rate) and comfort (electroencephalogram [EEG], Bizek Agitation Scale, and t

183 Electroencephalograms (EEGs) and/or local field potentia

184 The initial scalp electroencephalograms (EEGs) failed to detect seizure ac

185 e recorded local field potentials (LFPs) and electroencephalograms (EEGs) from contralateral cortex.

186 We recorded electroencephalograms (EEGs) from unaffected and early s

187 Recently, through study of electroencephalograms (EEGs) in humans and local field p

188 nied by epilepsy and/or clearly epileptiform electroencephalograms (EEGs).

189 electrodes, frontofrontal and frontoparietal electroencephalogram electrodes and then recorded sleep/

190 Cortical electroencephalogram, electromyogram, eye movement, hipp

191 ype mice were surgically implanted to record electroencephalogram, electromyogram, locomotor activity

192 our previous examination of these mice using electroencephalogram/electromyogram (EEG/EMG) monitoring

193 hat affect sleep and wakefulness by using an electroencephalogram/electromyogram-based screen of rand

194 The electroencephalogram equivalent of this appears to be a

195 ry epilepsy patients undergoing intracranial electroencephalogram evaluation.

196 Using high-density electroencephalogram/event-related potential (EEG/ERP) r

197 developed spontaneous and recurring abnormal electroencephalogram events consistent with progressive

198 utine clinical interpretation of these scalp electroencephalograms failed to identify any of the scal

199 essive diffuse sensory motor axonopathy, and electroencephalogram findings progressed from generalize

200 We recorded intracranial electroencephalogram from human cortical and hippocampal

201 ally, as the disease progressed, an abnormal electroencephalogram gamma activity (30-40 Hz) emerged i

202 erogeneous patterns, along with intracranial electroencephalogram gamma power changes, several minute

203 tral index (BIS), developed from a processed electroencephalogram, has been reported to decrease the

204 th a corresponding hypsarrhythmia pattern on electroencephalogram have never been reported.

205 ment or even replace the use of intracranial electroencephalogram (ICEEG), an invasive, costly proced

206 milarity analysis (RSA) [17] to intracranial electroencephalogram (iEEG) data from ten presurgical ep

207 a (30-80 Hz) oscillations occur in mammalian electroencephalogram in a manner that indicates cognitiv

208 aits such as the beta frequency of the human electroencephalogram in conjunction with DNA markers.

209 n of the progression of changes in sleep and electroencephalogram in Huntington's disease has never b

210 son of various drug regimens, utility of the electroencephalogram in patient monitoring, emerging dru

211 terns of stimulus-evoked phaselocking of the electroencephalogram in the gamma band (30-100 Hz).

212 The electroencephalogram is the standard method of diagnosis

213 e contingent negative variation (CNV) in the electroencephalogram, is the signature of the subjective

214 We recorded cortical electroencephalogram/local field potential (LFP) activit

215 ng, electroencephalogram or continuous video electroencephalogram, lumbar puncture, and genetic testi

216 ilepsy is localized through different modes (electroencephalogram, magnetic resonance imaging, etc) t

217 ene carriers suggest that alterations in the electroencephalogram may reflect underlying neuronal dys

218 everely decreased brain wave activity on the electroencephalogram, may be unintentionally induced by

219 Behavioural and electroencephalogram measures indicated that patients wi

220 Electrocardiogram, electroencephalogram, mixed venous oxygen saturation, te

221 omatic injector combined with 24-h video and electroencephalogram monitoring demonstrated significant

222 In patients treated with hypothermia, electroencephalogram monitoring during the first 24 hrs

223 Capnography and processed electroencephalogram monitoring have been described in s

224 Continuous electroencephalogram monitoring of Pcmt1-/- mice reveale

225 epsy unit in conjunction with 24-h video and electroencephalogram monitoring.

226 nt in 9 of 32 subjects (28%) with continuous electroencephalogram monitoring.

227 Electroencephalogram, neck electromyogram, blood pressur

228 from the mesial temporal lobe based on scalp electroencephalogram network connectivity measures.

229 hallmark will be a brief burst of gamma-band electroencephalogram noise when and where such a recogni

230 = 9), and akinetic mutism (n = 5); a typical electroencephalogram occurred only once.

231 cidence of epileptiform abnormalities in the electroencephalogram of people with focal epilepsy.

232 Laboratory work, neuroimaging, electroencephalogram or continuous video electroencephal

233 Although most studies have recorded electroencephalograms or spike activity, recent research

234 e of alpha (7-12 Hz) and theta (4-7 Hz) band electroencephalogram oscillations.

235 Burst suppression is an electroencephalogram pattern that consists of a quasi-pe

236 atio, 7.11; 95% CI, 5.01-10.08), unfavorable electroencephalogram patterns (false-positive rate, 0.07

237 gical outcome of low-voltage and isoelectric electroencephalogram patterns 24 hrs after resuscitation

238 These electroencephalogram patterns differ from those of contr

239 In patients with favorable outcome, electroencephalogram patterns improved within 24 hours a

240 poor outcome of low-voltage and isoelectric electroencephalogram patterns was 68% (confidence interv

241 Visual classification of electroencephalogram patterns was performed in 5-minute

242 At 12 hours, normal or diffusely slowed electroencephalogram patterns were associated with good

243 rol without suppression-burst or isoelectric electroencephalogram predicted good functional recovery

244 We test the hypothesis that quantitative electroencephalogram (qEEG) can be used to objectively a

245 The electroencephalogram (quantified using temporal brain sy

246 Electroencephalogram recorded over the left occipital co

247 Z) performed an auditory oddball task during electroencephalogram recording before and after auditory

248 We used direct intracranial electroencephalogram recordings from human epilepsy pati

249 Using high-density electroencephalogram recordings in humans, we show here

250 Here, using electroencephalogram recordings of great frigatebirds (F

251 Electroencephalogram recordings of ongoing rhythmic brai

252 In vivo electroencephalogram recordings showed persistent absenc

253 Recent evidence from electroencephalogram recordings suggests that one crucia

254 urons using invasive electrode techniques or electroencephalogram recordings using less- or non-invas

255 Electroencephalogram recordings were obtained from encod

256 magnetic resonance imaging, eye-tracking and electroencephalogram recordings.

257 However, early electroencephalogram recovery and ischemic neuronal inju

258 Alpha- and beta-band activity in the electroencephalogram reflected the logarithm of the like

259 seizures vs two or fewer, 1.08, 1.05-1.11), electroencephalogram results (epileptiform abnormality v

260 Ictal electroencephalogram results correspond to human infanti

261 cephalographic abnormalities; one had normal electroencephalogram results.

262 ergic PPT neurons suppressed lower-frequency electroencephalogram rhythms during NREM sleep.

263 ical outcome showed continuous, diffuse slow electroencephalogram rhythms, whereas this was never obs

264 by using trial-by-trial oscillations in the electroencephalogram's alpha band (8-14 Hz) collected fr

265 Here, we show that, as seen in electroencephalograms, SE induced by the muscarinic agon

266 ity, as assessed by quantitative analysis of electroencephalogram signal, and ischemic neuronal injur

267 measures the depth of sedation by analyzing electroencephalogram signals from a cutaneous probe.

268 Recent evidence suggests that electroencephalogram slow wave activity during sleep ref

269 cortisol and prolactin, and reduced resting electroencephalogram spectral power.

270 Electroencephalogram spike-triggered averages also showe

271 ayed robust in vivo activity in a sleep-wake electroencephalogram (sw-EEG) assay consistent with mGlu

272 Here, we used a specially designed dual-electroencephalogram system and the conceptual framework

273 p to the concurrent cycle of the hippocampal electroencephalogram theta rhythm.

274 Devices using the electroencephalogram to estimate anesthetic depth have b

275 ng and phase coherence in the scalp-recorded electroencephalogram to examine the synchronization of n

276 of autistic regression with an epileptiform electroencephalogram to Landau-Kleffner syndrome.

277 This was followed by a change in the child's electroencephalogram to the chaotic pattern of hypsarrhy

278 MRCPs were derived from back-averaging the electroencephalogram to the movement.

279 at this knowledge can guide their use of the electroencephalogram to track more accurately the brain

280 Wireless electroencephalogram transmitters were implanted into 23

281 Magnetic resonance spectroscopy and electroencephalogram-triggered functional magnetic reson

282 The electroencephalogram was abnormal in 15 patients and cer

283 Electroencephalogram was measured while older and younge

284 Continuous electroencephalogram was recorded during the first 5 day

285 An electroencephalogram was recorded from scalp electrodes

286 performed a category decision task whilst an electroencephalogram was recorded.

287 ed at the natural rate of signing, while the electroencephalogram was recorded.

288 Utilization of processed electroencephalogram waveforms has the greatest potentia

289 neonatal epilepsy with suppression bursts on electroencephalogram, we have expanded the phenotypic sp

290 We found that sleep and electroencephalogram were already significantly disrupte

291 us functional magnetic resonance imaging and electroencephalogram were recorded.

292 Cortical electroencephalograms were recorded in freely moving rat

293 Electroencephalograms were recorded while humans listene

294 ormed a visuospatial memory task while their electroencephalograms were recorded.

295 In this retrospective analysis of electroencephalograms were to identify a surrogate bioma

296 A) in the nonrapid eye movement (NREM) sleep electroencephalogram, which increases in proportion to t

297 zures based upon quantitative changes in the electroencephalogram, which they hypothesized began well

298 However, they had an abnormal electroencephalogram with overt seizure observed in a su

299 tant mice show persistent, abnormal cortical electroencephalograms with prominent delta and theta fre

300 Electroencephalograms with simultaneous functional MRI a