ライフサイエンスコーパス: electrolyte abnormality

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1 , cardiac ischemia, catecholamine excess, or electrolyte abnormality.

2 care unit stay, and inotrope use; and fewer electrolyte abnormalities.

3 ng the risks of worsening renal function and electrolyte abnormalities.

4 al intravascular volume depletion and marked electrolyte abnormalities.

5 n, rasburicase, and management of associated electrolyte abnormalities.

6 ctions, portal vein thrombosis, or fluid and electrolyte abnormalities.

7 se featuring arterial hypotension along with electrolyte abnormalities.

8 ical assessment, cerebral oxygen saturation, electrolyte abnormalities, adverse events, survival, and

9 tral obesity, hyperglycaemia, dyslipidaemia, electrolyte abnormalities and hypertension.

10 , although patients need to be monitored for electrolyte abnormalities and late toxicities.

11 al ventilation and intensive care unit stay, electrolyte abnormalities and repletion doses, duration

12 Treatment directed at correction of electrolyte abnormalities and the underlying cause for t

13 y therapy for congestion, is associated with electrolyte abnormalities and worsening renal function.

14 ious adverse events of hypotension, syncope, electrolyte abnormalities, and acute kidney injury or fa

15 y cardiac disease, drug-positive urine test, electrolyte abnormalities, and changes in their antiretr

16 tients with abnormal cardiac substrate, with electrolyte abnormalities, and during drug initiation.

17 c suction, intravenous fluids, correction of electrolyte abnormalities, and observation.

18 ravascular volume depletion, shock, profound electrolyte abnormalities, and organ dysfunction.

19 Electrolyte abnormalities are prevalent in patients with

20 Fluid and electrolyte abnormalities are very common in patients wi

21 Both recovered full renal function with no electrolyte abnormalities at the time of discharge.

22 erious adverse events (hypotension, syncope, electrolyte abnormalities, bradycardia, or acute kidney

23 d heart failure (ADHF) can be complicated by electrolyte abnormalities, but the major focus has been

24 Adverse effects include electrolyte abnormalities, cardiac failure, bleeding dia

25 es improve along with graft villi formation, electrolyte abnormalities continue, to which FK 506-medi

26 Hyperkalemia is the most frequent electrolyte abnormality found in whole organ transplant

27 ications of hypothermia including shivering, electrolyte abnormalities, hemodynamic changes, arrhythm

28 1.23 [95% CI, 0.76-2.00]), 4.0% vs 2.7% for electrolyte abnormalities (HR, 1.51 [95% CI, 0.99-2.33])

29 n College of Cardiology Practice Guidelines, electrolyte abnormalities, including abnormal serum pota

30 isted primarily of reversible hematologic or electrolyte abnormalities, including neutropenic fever i

31 d intravenous fluid hydration, correction of electrolyte abnormalities, nutritional support, and crit

32 pendent predictors of early readmission were electrolyte abnormalities on the day of discharge (odds

33 without structural heart disease, metabolic/electrolyte abnormalities, or the long QT syndrome.

34 t was associated with a greater incidence of electrolyte abnormalities, particularly hypokalemia.

35 ent, major trauma, hypotension, metabolic or electrolyte abnormalities, renal insufficiency, sepsis,

36 IQR, 4-8] vs 4.0 [IQR, 3-6] days) and higher electrolyte abnormality scores (median, 6 [IQR, 4-7] vs

37 autosomal recessive disease that results in electrolyte abnormalities shortly after birth.

38 Hyperkalemia is a common electrolyte abnormality that may be difficult to manage

39 Pendrin-knockout mice show no fluid-electrolyte abnormalities under baseline conditions, alt

40 portional hazards regression analysis, serum electrolyte abnormality was an independent predictor of

41 e frequently on the cyclophosphamide arm and electrolyte abnormalities were more common on the CCNU r

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