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1 Activation of GATA3 was analysed by electrophoretic mobility shift assay.
2 2 and AIL5 promoter regions was confirmed by electrophoretic mobility shift assay.
3 is significantly higher than for dsDNA in an electrophoretic mobility shift assay.
4 stable, sequence-specific RNA binding in an electrophoretic mobility shift assay.
5 by chromatin immunoprecipitation (ChIP) and electrophoretic mobility shift assay.
6 the bhuA promoter region was observed in an electrophoretic mobility shift assay.
7 raction of DNA and protein was determined by electrophoretic mobility shift assay.
8 th purified NAC to produce a shifted band by electrophoretic mobility shift assay.
9 d with nonmethylated G or A alleles in a gel electrophoretic mobility shift assay.
10 oscopy, relative viscosity measurements, and electrophoretic mobility shift assay.
11 assessed for binding of nuclear proteins by electrophoretic mobility shift assay.
12 ofluorescence, subcellular fractionation and electrophoretic mobility shift assay.
13 nation was used; DNA binding was analyzed by electrophoretic mobility shift assay.
14 NFkB activation was measured by electrophoretic mobility shift assay.
15 he optimal N/P ratio was 50 as determined by electrophoretic mobility shift assay.
16 nt using flow cytometry, transactivation and electrophoretic mobility shift assays.
17 recombinant PenR and PntR were identified by electrophoretic mobility shift assays.
18 itative reverse transcriptase (qRT)-PCR, and electrophoretic mobility shift assays.
19 ased affinity for the biotin operator DNA in electrophoretic mobility shift assays.
20 ement (NRE) at this location by ChIP-seq and electrophoretic mobility shift assays.
21 anning and tested using light scattering and electrophoretic mobility shift assays.
22 urified CepR bound to each of these sites in electrophoretic mobility shift assays.
23 la BabR binds to the virB promoter region in electrophoretic mobility shift assays.
24 , hpd, and dhcA promoters in vitro by use of electrophoretic mobility shift assays.
25 somal target integration sequences (attB) in electrophoretic mobility shift assays.
26 inding specificities, which was confirmed by electrophoretic mobility shift assays.
27 pla3 using chromatin immunoprecipitation and electrophoretic mobility shift assays.
28 red for 12 of these promoters by competition electrophoretic mobility shift assays.
29 ormed a more stable complex with Bdnf RNA in electrophoretic mobility shift assays.
30 to the cydAB promoter DNA was analyzed using electrophoretic mobility shift assays.
31 scription factor binding was investigated by electrophoretic mobility shift assays.
37 ctor such as U2AF65, as determined by an RNA electrophoretic mobility shift assay and a chromatin imm
38 mplex to the PUMA promoter was identified by electrophoretic mobility shift assay and a chromatin imm
39 the snRNA duplex during di-snRNP assembly by electrophoretic mobility shift assay and accompanying co
40 erse ERalpha-bound loci were validated using electrophoretic mobility shift assay and ChIP-polymerase
43 Promoter activation studies combined with electrophoretic mobility shift assay and chromatin immun
46 1 to HIF-1alpha promoter was corroborated by electrophoretic mobility shift assay and chromatin immun
49 appaB transcription factor were validated by electrophoretic mobility shift assay and chromatin immun
50 tative STAT-binding sequence at -476 nt, and electrophoretic mobility shift assay and chromatin immun
52 lement in the AOC1 promoter was confirmed by electrophoretic mobility shift assay and chromatin immun
56 BI to directly activate ODO1, as revealed by electrophoretic mobility shift assay and yeast one-hybri
57 promoter and quantified it using competitive electrophoretic mobility shift assays and chromatin immu
58 ple sites in the promoter was verified using electrophoretic mobility shift assays and chromatin immu
59 Assays for ubiquitin conjugation include electrophoretic mobility shift assays and detection of e
62 inity binding site for BldD, as was shown by electrophoretic mobility shift assays and DNase I footpr
65 ssed mutant proteins, p.Q65X and p.Q119X, by electrophoretic mobility shift assays and immunoblot ana
68 of these genes by VqsM has been confirmed by electrophoretic mobility shift assays and quantitative r
69 of TALE DNA recognition, using quantitative electrophoretic mobility shift assays and reporter gene
72 NER pathways on these lesions, we performed electrophoretic mobility-shift assays and chromatin immu
76 ction, immunoblotting, immunohistochemistry, electrophoretic mobility shift assay, and luciferase rep
77 AR RNA interaction using UV melting studies, electrophoretic mobility shift assay, and RNase A footpr
78 ion of the dsbD promoter was demonstrated by electrophoretic mobility shift assay, and the MisR bindi
80 izing cell-based luciferase reporter assays, electrophoretic mobility shift assays, and chromatin imm
82 ing activity both in vitro, as determined by electrophoretic mobility shift assays, and in cells, as
83 experimentally validated, both in vitro, by electrophoretic mobility shift assays, and in vivo, by c
84 The WRKY70 binding site was defined using electrophoretic mobility shift assays, and its importanc
85 Quantitative chromatin immunoprecipitation, electrophoretic mobility shift assays, and luciferase re
87 , microscopy, chromatin immunoprecipitation, electrophoretic mobility shift assays, and VE-cadherin-l
88 udy describes a sensitive acetyl transferase electrophoretic mobility shift assay applicable both for
91 mount of metal ion to produce a shift in the electrophoretic mobility shift assay, but unlike the wil
93 technology, gene reporter luciferase assay, electrophoretic mobility shift assay, chromatin immunopr
96 ent, which chromatin immunoprecipitation and electrophoretic mobility shift assays confirm are bound
102 tivation experiments, mutation analyses, and electrophoretic mobility shift assays demonstrate that t
121 g approach was independently validated by an electrophoretic mobility shift assay demonstrating hMutS
129 n vitro using luciferase reporter assays and electrophoretic mobility shift assay (EMSA) analysis.
133 to a 45-bp binding site as determined by an electrophoretic mobility shift assay (EMSA) and DNase I
136 In this study, circular dichroism (CD) and electrophoretic mobility shift assay (EMSA) experiments
137 ion (ChIP) assay for our in vivo studies and electrophoretic mobility shift assay (EMSA) for our in v
141 dict transcription factor binding sites, and electrophoretic mobility shift assay (EMSA) was used to
143 oid site cleaving enzyme-1 (BACE1) genes for electrophoretic mobility shift assay (EMSA) with differe
144 mntH promoter region were demonstrated in an electrophoretic mobility shift assay (EMSA), and a Mur b
146 d A-261T, using transfection/cotransfection, electrophoretic mobility shift assay (EMSA), and chromat
148 2 also regulates the Bdnf gene, we performed electrophoretic mobility shift assay (EMSA), chromatin i
150 CcrB DNA binding, as assessed in vitro by electrophoretic mobility shift assay (EMSA), revealed th
151 to viral terminal repeat DNA as assessed by electrophoretic mobility shift assay (EMSA), the mutatio
156 Chromatin immunoprecipitation (ChIP) and electrophoretic mobility shift assays (EMSA) experiments
158 OLOGY/PRINCIPAL FINDINGS: Luciferase assays, electrophoretic mobility shift assays (EMSA), and RNA ex
163 analyzed for DNA binding with the use of an electrophoretic mobility-shift assay (EMSA) and confocal
166 ted by susceptibility and efflux assays, and electrophoretic mobility shift assays (EMSAs) confirmed
168 We describe a platform for high-throughput electrophoretic mobility shift assays (EMSAs) for identi
170 es and target antigen, we introduce affinity electrophoretic mobility shift assays (EMSAs) in a high-
176 transcription factor binding was analyzed by electrophoretic mobility shift assays (EMSAs) with Jurka
177 etween nmoA and nmoR, which was confirmed by electrophoretic mobility shift assays (EMSAs) with the p
178 a combination of protein purification steps, electrophoretic mobility shift assays (EMSAs), and mass
179 as hexahistidine fusion proteins, and using electrophoretic mobility shift assays (EMSAs), the IHFal
185 omplex formed with infected cell extracts in electrophoretic mobility shift assay experiments, (iv) s
189 cant increase in ATF3 promoter activity, and electrophoretic mobility shift assay identified this reg
190 , deletion studies, mutagenesis studies, and electrophoretic mobility shift assays identified a PPARa
192 ants in this window for functional impact on electrophoretic mobility shift assay identifies rs806371
193 e lead SNP rs4888378; multiplexed competitor electrophoretic mobility shift assays implicated FOXA as
194 identified as high-affinity Oct-1 binding by electrophoretic mobility shift assay in vitro and chroma
196 complex with a 34mer double-stranded DNA in electrophoretic mobility-shift assays, independent of di
201 affinity and pattern of shifted complexes in electrophoretic mobility shift assays is responsive to t
202 shown by ChIP-assays, bind KLF16 in vivo In electrophoretic mobility shift assays, KLF16 binds speci
203 a cytokine secretion assay of IL-17, and by electrophoretic mobility shift assay of activating prote
206 In combination with mutational analysis and electrophoretic mobility shift assays, our results provi
207 itro binding studies utilizing a pulse-chase electrophoretic mobility shift assay protocol revealed t
209 se to DNAm changes upon modulation of HOTAIR Electrophoretic mobility shift assays provided further e
213 vo and in vitro by coimmunoprecipitation and electrophoretic mobility shift assays, respectively.
216 re also up-regulated in resistant plants and electrophoretic mobility shift assay revealed sequence-s
240 A bacterial one-hybrid system technique and electrophoretic mobility shift assays showed that AioR i
244 r of AAO3 in mesophyll cell protoplasts, and electrophoretic mobility shift assays showed that NAP ca
252 ers, but some of these interact with ABI4 in electrophoretic mobility shift assays, suggesting that s
255 Here we report fluorescence quenching and electrophoretic mobility shift assays that probe siRNA b
264 gth and truncated forms of CpsA were used in electrophoretic mobility shift assays to characterize th
265 ariants of bacteriophage lambda Cro and used electrophoretic mobility shift assays to compare binding
267 ylogenetic relations, in situ hybridization, electrophoretic mobility shift assays to determine bindi
268 mapping, data on DNase hypersensitivity, and electrophoretic mobility shift assays to study protein-D
270 ht mass spectrometry of a band excised after electrophoretic mobility shift assay using a ZTRE probe.
271 f detecting nuclear transcription factors by electrophoretic mobility shift assay using digoxigenin (
278 rcular dichroism, NMR, microcalorimetry, and electrophoretic mobility shift assay), we have character
279 me-linked immunosorbent assay (ELISA) and an electrophoretic mobility shift assay, we found that the
280 Using chromatin immunoprecipitation and electrophoretic mobility shift assay, we show that TH ha
282 From the results of in vitro translation and electrophoretic mobility shift assays, we demonstrate th
284 recipitation, site-directed mutagenesis, and electrophoretic mobility shift assays, we identified a G
290 Surface plasmon resonance diffraction and electrophoretic mobility shift assays were consistent wi
293 nsfection, promoter/reporter constructs, and electrophoretic mobility shift assays were used to deter
294 on citZ transcription were also reflected in electrophoretic mobility shift assays where CcpE bound t
295 ding motifs were investigated by competitive electrophoretic mobility shift assay, which revealed tha
296 veral transcription factor-binding sites and electrophoretic mobility shift assays with MCF-7 nuclear
299 ly as determined by DNase I footprinting and electrophoretic mobility shift assays, with some DNA-bin
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