ライフサイエンスコーパス: electroretinogram

1 .9 +/- 5.6 to 45.8 +/- 15.2 muV for photopic electroretinogram.

2 eld photopic negative response (PhNR) of the electroretinogram.

3 asis for cone dystrophy with supernormal rod electroretinogram.

4 s and decreased retinal function measured by electroretinogram.

5 that was reflected in the slow decay of the electroretinogram.

6 ing S- and M-opsins and a preserved photopic electroretinogram.

7 g in abnormal visual function as measured by electroretinogram.

8 ear identical derived rod responses from the electroretinogram.

9 tern blot analysis, histologic analysis, and electroretinogram.

10 unction can be assessed using the multifocal electroretinogram.

11 electro-oculogram light rise, and a reduced electroretinogram.

12 syndrome (SARDS) exhibiting a nonrecordable electroretinogram.

13 and increased the amplitude of the photopic electroretinogram.

14 pharmacological treatment through multifocal-electroretinogram.

15 d increased a-wave amplitude of the scotopic electroretinogram.

16 ement of retinal function as measured by the electroretinogram.

17 educed amplitudes and prolonged latencies in electroretinograms.

18 s to estimates of rod function from in vitro electroretinograms.

19 ones, concomitant with reduced cone-mediated electroretinograms.

20 y ranges from structural defects to abnormal electroretinograms.

21 plete loss of visual function as measured by electroretinograms.

22 compromised maximal voltage response in the electroretinograms.

23 rvation of cone function based upon photopic electroretinograms.

24 aturated fatty acids and severely attenuated electroretinograms.

25 change in the a and b waves of bright-flash electroretinograms.

26 at reported for visual fields and full-field electroretinograms.

27 Analysis of the electroretinogram a-wave indicates that rescued rod cell

28 and Rs1-KO mice have early reduction in the electroretinogram a-wave.

29 ifference in amplitudes or implicit times of electroretinogram a-waves, b-waves, and oscillatory pote

30 Several studies have shown that the pattern electroretinogram, a direct, objective method of measuri

31 Electroretinogram abnormalities observed in b-wave and o

32 electron microscopy, rhodopsin measurement, electroretinogram activity, and visual acuity, indicatin

33 Retinal function measured by electroretinogram after light exposure was also delayed

34 te stimulus presentation reduces the pattern electroretinogram amplitude and increases optic nerve bl

35 mplitudes and implicit times, and multifocal electroretinogram amplitude distribution.

36 USH2A patients lose visual field and cone electroretinogram amplitude faster than patients with RH

37 hout the study duration and exhibited higher electroretinogram amplitude, thicker photoreceptor layer

38 ite test light), and 30-Hz (cone) full-field electroretinogram amplitude.

39 y, 7.0% for visual field area, and 13.2% for electroretinogram amplitude.

40 has sometimes yielded improvement in pattern electroretinogram amplitude.

41 nd tritan thresholds, pattern and full-field electroretinogram amplitudes and implicit times, and mul

42 nths of pioglitazone significantly increased electroretinogram amplitudes in type 2 diabetic obese ra

43 The electroretinogram amplitudes of double heterozygotes are

44 onsyndromic USH2A patients had 30 Hz-flicker electroretinogram amplitudes that were significantly hig

45 isplay scattered OS disorganization, reduced electroretinogram amplitudes, and progressive photorecep

46 tor apoptosis and to a sustained increase in electroretinogram amplitudes.

47 Electroretinogram analyses revealed reduced cone light r

48 Electroretinogram analyses showed decreased B-wave and o

49 function was evaluated 8 weeks later by the electroretinogram and compared with photoreceptor cell l

50 While case 1 had an undetectable electroretinogram and features supporting a diagnosis of

51 9.4 +/- 4.6 to 57.6 +/- 8.8 muV for scotopic electroretinogram and from 10.9 +/- 5.6 to 45.8 +/- 15.2

52 umbers of degenerate capillaries, as well as electroretinogram and heart morphology.

53 A standard electroretinogram and intraocular pressure measurements

54 litazone's effects on retinal function using electroretinogram and markers of apoptosis.

55 Vision was assessed using the electroretinogram and optokinetic response and retinal m

56 resulting in complete absence of a photopic electroretinogram and progressive cone degeneration.

57 Electroretinogram and visual evoked potential tests show

58 Electroretinograms and electro-oculograms were recorded

59 Electroretinograms and histology showed no evidence of d

60 Safety was evaluated by performing electroretinograms and histology.

61 To measure changes in rod physiology, electroretinograms and intracellular Ca(2+) recording we

62 attenuation of visual function with abnormal electroretinograms and reduced retinal rhodopsin levels.

63 dly disrupts retinal function as assessed by electroretinograms and vision as assessed by optomotor b

64 Intraocular pressure (IOP), pattern electroretinogram, and optical coherence tomography meas

65 ity, automated visual field testing, pattern electroretinogram, and spectral-domain optical coherence

66 onal data were obtained using the full-field electroretinogram, and static or kinetic perimetry.

67 receptor potential (ERP), a component of the electroretinogram arising from photoisomerization-induce

68 The pattern electroretinogram assesses retinal ganglion cell functio

69 All eyes underwent standard full-field electroretinogram at baseline and 8 weeks after surgery.

70 ight adaptation, as evidenced by an impaired electroretinogram b-wave from cones, whereas a dopamine

71 The loss of the electroretinogram b-wave in these mice suggests a severe

72 Retinal function measured by the electroretinogram b-wave threshold declined >100-fold fr

73 RGS11 produced delays in the ON-BPC-derived electroretinogram b-wave, but no changes in the photorec

74 photopsias, and a selective reduction of the electroretinogram b-wave.

75 ice is associated with an attenuation of the electroretinogram b-wave.

76 dystroglycan was sufficient to attenuate the electroretinogram b-wave.

77 nsmission as indicated by a reduction in the electroretinogram b-wave.

78 sing homozygous Nmnat1(V9M) mutant mice, the electroretinogram becomes undetectable and the pupillary

79 oducible and reversible changes in the flash electroretinogram between daylight and mel-low.

80 Multifocal electroretinogram can also help in early screening of fo

81 ce restricted peripheral vision and scotopic electroretinogram confirmed the diagnosis of retinitis p

82 as indicated by the loss of ON transients in electroretinograms, consistent with a neurotransmission

83 ice showed nonprogressive b-wave deficits on electroretinograms, consistent with compromised BP cell

84 while Prph2Y/+ animals exhibited a cone-rod electroretinogram defect, Prph2Y/+/Rom1-/- animals displ

85 racteristic of RPE and choroidal defects and electroretinogram defects.

86 Electroretinograms demonstrated that affected mice were

87 Electroretinograms demonstrated that the organ responds

88 nd/or macular pigment mottling, and abnormal electroretinograms demonstrating mixed cone and rod dysf

89 retinal function, measured by the multifocal electroretinogram, differs between males and females wit

90 ch as early visual impairment as assessed by electroretinogram, disorganization of lamination and api

91 The multifocal pattern electroretinogram does not seem to have an advantage ove

92 g with retinal function as determined by the electroretinogram (ERG) a- and b-waves.

93 As a group, CSNB1 patients have a normal electroretinogram (ERG) a-wave, indicative of photorecep

94 d and cone function declines, accompanied by electroretinogram (ERG) abnormalities.

95 erited retinal disorders with characteristic electroretinogram (ERG) abnormalities.

96 rea (V4e white test light) and in full-field electroretinogram (ERG) amplitudes to 0.5- and 30-Hz whi

97 expression, endogenous retinoid levels, and electroretinogram (ERG) analyses were performed on Lrat(

98 Morphometric and electroretinogram (ERG) analyses were used to assess the

99 Microperimetry 1 (MP-1) mapping and electroretinogram (ERG) analysis were performed on the p

100 uter nuclear layer (ONL) and functionally by electroretinogram (ERG) analysis, 5 to 7 days after expo

101 Recordings of the electroretinogram (ERG) and spiking activity of single r

102 n the etiology of psychiatric disorders, and electroretinogram (ERG) anomalies have been reported in

103 Electroretinogram (ERG) anomalies occur in patients with

104 ess, in which the a- and b-wave responses of electroretinogram (ERG) are abolished.

105 n optical coherence tomography (SD-OCT), and electroretinogram (ERG) assessment.

106 Electroretinogram (ERG) b-wave implicit time in young RG

107 In the Gbeta5(-/-) mouse, the electroretinogram (ERG) b-wave is absent, and the R7 sub

108 e impairment of night vision, absence of the electroretinogram (ERG) b-wave, and variable degrees of

109 Amplitude reduction of >50% in the relevant electroretinogram (ERG) component or a peak time shift o

110 mune retinopathy based on clinical features, electroretinogram (ERG) findings, and serum antiretinal

111 Goldmann perimetry, and full-field standard electroretinogram (ERG) from all patients were registere

112 s were classified on the basis of full-field electroretinogram (ERG) Fundus autofluorescence (FAF) an

113 wed nonspecific retinal inflammation, and an electroretinogram (ERG) illustrated decreased amplitude

114 the impact of KCNJ10 mutations on the human electroretinogram (ERG) in four unrelated patients with

115 The electroretinogram (ERG) is a non-invasive method used to

116 Electroretinogram (ERG) measurements were used to assay

117 and photopic negative response (PhNR) of the electroretinogram (ERG) noninvasively with an electrode

118 The electroretinogram (ERG) of the super p53 mouse exhibited

119 The electroretinogram (ERG) of TRPM1-deficient (TRPM1(-/-))

120 Multifocal electroretinogram (ERG) or VEP can provide an objective

121 Recordings of electroretinogram (ERG) oscillatory potentials and scoto

122 The visual symptoms and electroretinogram (ERG) phenotype characteristic of MAR

123 ouse, which lacks GPR179 and has a no b-wave electroretinogram (ERG) phenotype, to demonstrate that d

124 disrupts signal transduction and reveals an electroretinogram (ERG) phenotype.

125 To evaluate the diagnostic potential of the electroretinogram (ERG) photostress test and the focal c

126 Scotopic electroretinogram (ERG) recording was used to investigat

127 S males underwent ophthalmic examination and electroretinogram (ERG) recording.

128 (OCT), fundus autofluorescence imaging, and electroretinogram (ERG) recording.

129 The bright flash response in electroretinogram (ERG) recordings recovered quickly in

130 This was supported by electroretinogram (ERG) recordings which showed both the

131 nts of AIPL1 were studied by single-cell and electroretinogram (ERG) recordings.

132 Electroretinogram (ERG) response amplitudes were recorde

133 localization of cone opsin, loss of photopic electroretinogram (ERG) responses and loss of cone cells

134 Electroretinogram (ERG) responses to a 1.8-log-unit rang

135 Electroretinogram (ERG) responses were obtained from ten

136 Electroretinogram (ERG) responses were recorded in both

137 Cone electroretinogram (ERG) responses were reduced in Irbp(-

138 y biochemically, by recording single rod and electroretinogram (ERG) responses, by intracellular free

139 s photoreceptor survival but also suppresses electroretinogram (ERG) responses.

140 An electroretinogram (ERG) screen identified a mouse with a

141 The electroretinogram (ERG) undergoes parallel changes.

142 e known to contribute to the mammalian flash electroretinogram (ERG) via activity of third-order reti

143 An electroretinogram (ERG) was done at baseline and thereaf

144 The electroretinogram (ERG) was measured prior to ischemia a

145 A standard electroretinogram (ERG) was obtained before injection an

146 Electroretinogram (ERG) was recorded from MPS IIIB and w

147 group were investigated by monocular pattern electroretinogram (ERG), L&M (long and medium wavelength

148 assessed by restoration of the cone-mediated electroretinogram (ERG), optomotor responses, and cone o

149 d examining gene message and protein levels, electroretinogram (ERG), retinal morphology and ultrastr

150 ent in normal numbers, and the a-wave of the electroretinogram (ERG)--reflecting their physiological

151 , brain, and male reproductive organs and by electroretinogram (ERG)-based studies of the retina and

152 and luminance specific signals in the human electroretinogram (ERG).

153 asuring outer nuclear layer thickness and by electroretinogram (ERG).

154 re central macular dysfunction on multifocal electroretinogram (ERG).

155 to a decline in visual acuity as detected by electroretinogram (ERG).

156 retinal light response was measured with the electroretinogram (ERG).

157 delayed falling phase of photopic b-wave of electroretinogram (ERG).

158 utcome was a change in the parameters of the electroretinogram (ERG).

159 involuntary eye movement, and nonrecordable electroretinogram (ERG).

160 ing Gng13(-/-) knockout (KO) mice, recording electroretinograms (ERG) and performing immunocytochemic

161 in the retina, and the scotopic and photopic electroretinograms (ERG) and retinal morphology in wild-

162 athy were recruited, and photopic full-field electroretinograms (ERG) were performed at baseline and

163 no adverse effects on the eye as assessed by electroretinograms (ERG), corneal and retinal tomography

164 sual acuity, constricted fields, and reduced electroretinograms (ERGs) 5 years before death correlate

165 These mice had normal fundi, electroretinograms (ERGs) and retinal histology at 6 mon

166 Weekly electroretinograms (ERGs) followed by retinal histology

167 The electroretinograms (ERGs) of both eyes from all rats wer

168 nction was assessed with serial dark-adapted electroretinograms (ERGs) optimized for detection of the

169 However, we find further variability in cone electroretinograms (ERGs) ranging from normal to absent

170 In addition, electroretinograms (ERGs) recorded in the same groups we

171 Electroretinograms (ERGs) were measured to assess retina

172 Dark- and light-adapted electroretinograms (ERGs) were obtained in three mouse m

173 Scotopic electroretinograms (ERGs) were performed 3, 7, and 14 da

174 Electroretinograms (ERGs) were performed before and afte

175 Whole-field electroretinograms (ERGs) were performed on dynein morph

176 Electroretinograms (ERGs) were performed on transplanted

177 Electroretinograms (ERGs) were recorded 7 weeks after th

178 Electroretinograms (ERGs) were recorded and the retinas

179 Full-field scotopic electroretinograms (ERGs) were recorded from 44 male hem

180 Full-field electroretinograms (ERGs) were recorded from wild-type (

181 Electroretinograms (ERGs) were recorded to measure outer

182 ual acuity loss early in life, nondetectable electroretinograms (ERGs), and little or no detectable v

183 On the basis of standard electroretinograms (ERGs), patients were diagnosed with

184 ver, each patient had recordable and similar electroretinograms (ERGs), which demonstrated absent con

185 common cause of inherited PD) on Drosophila electroretinograms (ERGs).

186 evaluated by retinal histologic analyses and electroretinograms (ERGs).

187 erturbed retinal function as demonstrated by electroretinograms (ERGs).

188 ith dark-adapted electroretinograms (monthly electroretinograms [ERGs]), and cataract formation with

189 The photopic flash electroretinogram (FERG) and visual evoked potential (FV

190 uded kinetic widefield perimetry, full-field electroretinogram (ffERG), and visual acuity (VA).

191 n kinetic visual field (GVF), and full-field electroretinogram (ffERG).

192 At end of the study, electroretinogram findings, retinal ganglion cell (RGC)

193 ng the development of ametropia based on the electroretinogram findings.

194 lides or through physiological testing using electroretinogram flicker photometry.

195 Renewed interest in the pattern electroretinogram for early detection of pre-perimetric

196 fect on the b-wave was confirmed by in vitro electroretinograms from the inner retina.

197 topic negative response of the diffuse flash electroretinogram has shown changes in glaucoma, but may

198 Unlike visual fields and electroretinograms, however, the repeat variability is l

199 e were no other causes of an electronegative electroretinogram identified in any of the affected pati

200 unction as determined by almost extinguished electroretinogram in 2 of 3 siblings.

201 o sign of vitreous inflammation and abnormal electroretinogram in at least 1 eye), and a negative fam

202 recording electrodes; the maturation of the electroretinogram in preterm infants and in the first ye

203 t seem to have an advantage over the pattern electroretinogram in the early detection of glaucoma.

204 s the modifications needed for performing an electroretinogram in young children.

205 cal modifications for performing a pediatric electroretinogram, including the possible need for sedat

206 red at 4 weeks of age, although a full-field electroretinogram indicated a visual response was still

207 Pde6d(-/-) scotopic paired-flash electroretinograms indicated a delay in recovery of the

208 Differences in the electroretinograms, intraocular pressure, and histopatho

209 The pattern electroretinogram is a noninvasive, direct, objective me

210 The electroretinogram is an essential tool in the evaluation

211 The pattern electroretinogram may also optimize treatment strategies

212 Electroretinogram measurements of the frequency response

213 croscopy parameters, retina cell counts, and electroretinogram measurements.

214 ity (BCVA), foveal threshold, and multifocal electroretinogram (mfERG) amplitude and timing.

215 functional recovery evidenced by multifocal-electroretinogram (mfERG) and microperimetry (MP1) after

216 The multifocal electroretinogram (mfERG) can provide objective corrobor

217 nship between DE development and: multifocal electroretinogram (mfERG) implicit time (IT) Z-score, mf

218 Local first-order multifocal electroretinogram (mfERG) implicit time (K1-IT) delays h

219 Multifocal electroretinogram (mfERG) provides evidence of focal ret

220 l parameters of surgical success, multifocal electroretinogram (mfERG), and histopathologic analyses

221 charts, 10-2 visual fields (VFs), multifocal electroretinograms (mfERG), and spectral-domain optical

222 Multifocal electroretinograms (mfERGs) and psychophysical assessmen

223 Photopic and scotopic multifocal electroretinograms (mfERGs) were recorded.

224 sitivity, retinal function with dark-adapted electroretinograms (monthly electroretinograms [ERGs]),

225 Although the technique for performing an electroretinogram must be modified for young children, w

226 ematic forward genetic Drosophila screen for electroretinogram mutants lacking synaptic transients id

227 statistically significant differences in the electroretinograms obtained between the linezolid-inject

228 We found that the electroretinogram of Gbeta5-/- mice lacks the b-wave com

229 Contrary to expectation, electroretinograms of CFH(-/-).C3(-/-) mice displayed mo

230 at flash responses measured by trans-retinal electroretinogram or single-cell suction electrode recor

231 no statistically significant differences in electroretinograms, or intraocular pressure measurements

232 croscopy parameters, retina cell counts, and electroretinogram parameters were compared between the g

233 Variants of the electroretinogram (pattern ERG and long-pulse ERG) were

234 Flash electroretinograms performed at 2, 7, and 10 months of a

235 mice were longitudinally tested with pattern electroretinogram (PERG) and spectral-domain optical coh

236 eliability, and dynamic range of the pattern electroretinogram (PERG) as a tool to monitor progressiv

237 ical response of RGC was measured by pattern electroretinogram (PERG) in 43 C57BL/6J mice 4 to 6 mont

238 optical coherence tomography (OCT), pattern electroretinogram (PERG) or frequency-doubling technolog

239 IOP and pattern electroretinogram (PERG) were sequentially measured with

240 avioral abnormalities was made using pattern electroretinogram (PERG), magnetic resonance imaging (MR

241 d structure were evaluated using the pattern electroretinogram (pERG), spectral domain optical cohere

242 ical coherence tomography (OCT), and pattern electroretinogram (PERG).

243 The authors retrospectively analyzed pattern electroretinograms (PERG) recorded twice a year in 32 gl

244 Serial pattern electroretinograms (PERGs) and IOPs measures were obtain

245 Pattern-electroretinograms (PERGs) were obtained in response to

246 pamine in vision using electrophysiological (electroretinogram), psychophysical (optokinetic tracking

247 Electroretinograms recorded from larval zebrafish show l

248 p2 mutant but also functionally improved the electroretinogram recording (ERG).

249 ETDRS chart), MP-1 microperimetry, and focal electroretinogram recording (fERG).

250 al function as shown by light microscopy and electroretinogram recording, respectively.

251 mination, retinal thickness measurement, and electroretinogram recording.

252 t of functional roles in the photoreceptors, electroretinogram recordings demonstrate impaired respon

253 Electroretinogram recordings from PCP2-null mice showed

254 tion in NCKX2-deficient (Nckx2(-/-)) mice by electroretinogram recordings revealed normal photopic b-

255 Consistently, electroretinogram recordings show age-progressive loss o

256 Electroretinogram recordings under scotopic conditions s

257 s but display reduced "on-off" transients in electroretinogram recordings, indicating a failure to ev

258 rget cones and rescue both the cone-mediated electroretinogram response and visual acuity in the Gnat

259 Multifocal electroretinogram response did not improve, yet peaks we

260 xyretinal chromophore but failed to evoke an electroretinogram response from fly photoreceptors.

261 ular toxicity, second cancer development and electroretinogram response were all evaluated.

262 restored rod morphology and the rod-derived electroretinogram response, but cone photoreceptors were

263 ght-blindness models with an electronegative electroretinogram response, which is also characteristic

264 With the noninvasive pattern electroretinogram, response abnormalities have been dete

265 enously expressed rod or cone Talpha rescued electroretinogram responses (ERGs) in mice lacking funct

266 ubretinal delivery of PDE6alpha' rescues rod electroretinogram responses and preserves retinal struct

267 Scotopic and photopic electroretinogram responses declined progressively from

268 ent, one fourth of the mice showed increased electroretinogram responses in the transplanted eyes.

269 The electroretinogram responses of both rod and cone photore

270 se of vitreo-macular traction and multifocal electroretinogram responses showed a significant increas

271 However, cone electroretinogram responses were decreased by 40% at 6 m

272 Electroretinogram responses were recorded using conducti

273 e exhibited absence of scotopic and photopic electroretinogram responses, a phenotype that resembles

274 nd no statistically significant influence on electroretinogram responses, and used in conjunction wit

275 nmf223 homozygotes also have reduced electroretinogram responses, which are coupled histologi

276 h statistically significant reduction in the electroretinogram responses.

277 Electroretinograms revealed abnormal cone photoreceptor

278 Electroretinograms revealed photoreceptor dysfunction pr

279 Measures of retinal function, such as the electroretinogram, show that photoreceptor function is d

280 examination, dilated fundus examination, and electroretinograms showed no evidence of vitritis, uveit

281 Electroretinograms showed significantly decreased functi

282 Also, we present electroretinogram studies that have added to our underst

283 In the retina, similarly, an abnormal electroretinogram suggested reduced transmission at the

284 induced negative-masking behavior and normal electroretinogram, suggesting an intact retina light res

285 e was bilateral and in the context of normal electroretinograms therefore indicates generalized dysfu

286 and the size and density of RPE melanosomes, electroretinograms to study retinal function, and retrog

287 Small, but robust electroretinogram type responses are routinely detected

288 gatively affected in glaucoma, including the electroretinogram, visual evoked potential, visual spati

289 Electroretinogram was used to evaluate visual function.

290 ts in visual impairment assessed by abnormal electroretinogram waveforms.

291 Focal electroretinograms were recorded in response to a sinuso

292 Bilateral electroretinograms were recorded simultaneously before a

293 Electroretinograms were recorded, and the outer nuclear

294 Photopic and scotopic electroretinograms were reviewed.

295 of neurophysiological responses (multifocal electroretinogram) were decreased in all eccentricity ri

296 f retinal degeneration, and a non-recordable electroretinogram with negligible amplitudes in both eye

297 d photophobia, reduced amplitude of the cone electroretinogram with normal rod responses, normal fund

298 ng to abnormalities of scotopic and photopic electroretinograms with decreased b-wave amplitude as th

299 Compound 49b maintained a normal electroretinogram, with no changes in blood pressure, in

300 nd N1-P1 amplitudes from photopic multifocal electroretinograms within the central 45 degrees.