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1 d VPP mice is consistent with histologic and electroretinographic abnormalities determined in previou
2                                        These electroretinographic abnormalities were correlated with
3  photopsia, minimal funduscopic changes, and electroretinographic abnormalities.
4                          All 14 patients had electroretinographic abnormalities.
5 ents displayed a complete absence of vision, electroretinographic amplitude, and PLR at low light int
6                                              Electroretinographic amplitudes (full-field flash ERG) f
7 ls of IGF-I showed progressive impairment of electroretinographic amplitudes up to complete loss of r
8          On the 21st day, all rats underwent electroretinographic analyses followed by collection of
9  resultant congenic mice was evaluated using electroretinographic analyses.
10                          Full-field scotopic electroretinographic analysis (ERG) was performed to mea
11                                         Both electroretinographic and anatomic methods were used to a
12                                              Electroretinographic and immunohistochemical analyses of
13 irculating antiretinal antibodies along with electroretinographic and visual field abnormalities.
14 terized by progressive visual loss, abnormal electroretinographic and visual field findings in the pr
15 type and monogenic controls by histological, electroretinographic, and biochemical analysis.
16 n decrease ischemic damage to the retina, by electroretinographic assessment of visual function and b
17       Unexpectedly, the rate of reduction in electroretinographic B-wave amplitude in eyes infected w
18 nd of each treatment in dark-adapted maximal electroretinographic b-wave amplitude.
19  of either the presence or the absence of an electroretinographic b-wave.
20 dent fundus lesions developed accompanied by electroretinographic changes consistent with defects in
21 mpany, Princeton, NJ) produces histologic or electroretinographic changes in the rabbit retina up to
22 development of systemic glucose intolerance, electroretinographic defects, or microvascular disease.
23                            Strikingly, these electroretinographic deficits are abrogated in a dose-de
24                                     However, electroretinographic dysfunction began at 6 months in hi
25                                              Electroretinographic (ERG) a-wave responses were recorde
26 inal ischemia, as measured by a cessation of electroretinographic (ERG) activity, was induced in anes
27 days after ischemic injury, morphometric and electroretinographic (ERG) analyses were used to assess
28                                              Electroretinographic (ERG) and morphometric analyses wer
29 ssessed by comparing full-field, white-flash electroretinographic (ERG) data obtained before and afte
30                                              Electroretinographic (ERG) methods were used to determin
31                                              Electroretinographic (ERG) methods were used to investig
32              To study the development of the electroretinographic (ERG) oscillatory potentials (OPs)
33 formed binocularly, using DTL electrodes and electroretinographic (ERG) protocols with flash strength
34  function and structure were evaluated using electroretinographic (ERG) recordings and immunohistoche
35                    In a longitudinal design, electroretinographic (ERG) records and digital fundus im
36 ology, we eliminated the rod contribution to electroretinographic (ERG) responses by generating doubl
37                                              Electroretinographic (ERG) responses in rats treated wit
38                                              Electroretinographic (ERG) responses of eyes injected wi
39  evaluate the function of the neural retina, electroretinographic (ERG) responses to full-field stimu
40                                 Rod isolated electroretinographic (ERG) responses to full-field stimu
41      Consistently, the scotopic and photopic electroretinographic (ERG) responses to single-flash sti
42 y, protein expression, retinoid content, and electroretinographic (ERG) responses were evaluated befo
43                                 Dark-adapted electroretinographic (ERG) responses were twofold greate
44 ull mutations of GRK1 (GRK1 -/-) cone-driven electroretinographic (ERG) responses, including an a-wav
45                           Psychophysical and electroretinographic (ERG) studies indicate that patient
46 hotoreceptor status was evaluated by various electroretinographic (ERG) techniques, retinoid analyses
47 immunoblot, immunohistochemistry, TUNEL, and electroretinographic (ERG) techniques.
48                                              Electroretinographic (ERG) thresholds of mutant fish are
49 al coherence tomography (SD-OCT), full-field electroretinographic (ERG), and color vision testing.
50                                  Histologic, electroretinographic (ERG), and molecular studies were p
51 deficiency (CNGA3-/- mice) were evaluated by electroretinographic (ERG), morphometric, and Western bl
52 opia, reduced central vision, nystagmus, and electroretinographic evidence of ON bipolar cell dysfunc
53           In contrast to human patients, our electroretinographic examinations of Pde6h(-/-) mice sug
54   To compare retinal function via full-field electroretinographic (ffERG) recordings in 6.5-year-old
55                                              Electroretinographic findings showed abnormalities in fo
56  clinical manifestations (including abnormal electroretinographic findings).
57 d had no overt ocular defects or decrease in electroretinographic function, up to P28.
58 uated clinically and with psychophysical and electroretinographic measurements of rod and cone functi
59 eterozygously was examined by histologic and electroretinographic measurements.
60                             There is neither electroretinographic nor histologic evidence of photorec
61                              No demonstrable electroretinographic or histologic changes occurred to s
62                                              Electroretinographic photoreceptor responses had equal r
63 to assess the physiological role of Panx1 by electroretinographic recordings and also to ensure the s
64                                 We conducted electroretinographic recordings from dark- and light-ada
65 lities in IRBP-/- mice were more severe, and electroretinographic recordings revealed a marked loss i
66  the rescue of cone function as indicated by electroretinographic recordings using natural noise stim
67                                    At 12 Hz, electroretinographic response amplitudes and phases prim
68 ction, detected as a deficit in the scotopic electroretinographic response, was improved in this tran
69 hanges in the RPE, and a deficit in scotopic electroretinographic response, which is reflective of im
70                                              Electroretinographic responses from treated eyes were mo
71 orrelated with the development of visual and electroretinographic responses in the animal.
72 ompleted before eye opening and the onset of electroretinographic responses on postnatal day 13 (P13)
73 RR was present, others may have dark-adapted electroretinographic responses that are within normal ra
74                                              Electroretinographic responses to brief ganzfeld flashes
75              Standard clinical 30-Hz flicker electroretinographic responses were also measured.
76                                              Electroretinographic responses were clearly measurable o
77   Retinal ganglion cell survival and pattern electroretinographic responses were compared in normal c
78 cant changes in ophthalmoscopic findings and electroretinographic responses were detected.
79 ht-adapted cone-isolated (Wratten 26 filter) electroretinographic responses were measured as a functi
80 ent, and all patients had severely decreased electroretinographic responses with predominant rod impa
81 -treated Ins2(Akita) mice exhibited impaired electroretinographic responses, characterized by reduced
82 atrophy and maintained near-normal levels of electroretinographic responses.
83 lecular biological, histological and flicker electroretinographic results have established that mice
84                     While the behavioral and electroretinographic results indicate that PLC beta 4 pl
85                                              Electroretinographic spectral sensitivity analysis showe
86 e in N1 and P1 peak amplitudes on multifocal electroretinographic testing, and change in serum omega-
87 ual acuity or retinal function by multifocal electroretinographic testing.
88  (P = .12) or retinal function by multifocal electroretinographic testing.
89  in temporal tuning (both psychophysical and electroretinographic) were found only within visual fiel

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