戻る
「早戻しボタン」を押すと検索画面に戻ります。

今後説明を表示しない

[OK]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 tive immunohistochemistry) and functionally (electroretinography).
2 provement of retinal function (assessed with electroretinography).
3 n of Akita and control mice was evaluated by electroretinography.
4 kinetic and static perimetry, and full-field electroretinography.
5 lted in functional deficits as determined by electroretinography.
6 fundus ophthalmoscopy, dark adaptometry, and electroretinography.
7 r degeneration was measured by histology and electroretinography.
8 static contrast sensitivity test; and global electroretinography.
9  cone survival was assessed by histology and electroretinography.
10              Retinal function was assayed by electroretinography.
11 escued photoreceptor function as measured by electroretinography.
12 inal histology up to 24 months of age and by electroretinography.
13 ull-field electroretinography and multifocal electroretinography.
14   Rescue of visual function was confirmed by electroretinography.
15 T) or DKO mice, as assessed by histology and electroretinography.
16 ctrometry, and degeneration by histology and electroretinography.
17 ncluding ophthalmoscopy and full-field flash electroretinography.
18 nohistochemistry, Western blot analysis, and electroretinography.
19 l function was characterized with the use of electroretinography.
20 t hypomorphic and Ret conditional mice using electroretinography.
21 ) measures of central retinal thickness, and electroretinography.
22 isual function in these mice was analyzed by electroretinography.
23 reserves RGC activity as measured by pattern electroretinography.
24 nction of USH2A patients was quantified with electroretinography.
25            Neural function was assessed with electroretinography.
26 t time of the N95 wave on results of pattern electroretinography.
27 es of mRDH11 disruption were investigated by electroretinography.
28 munohistochemistry, electron microscopy, and electroretinography.
29 us photography, fluorescein angiography, and electroretinography.
30 ant visual loss in treated abcr(-/-) mice by electroretinography.
31  promoted functional recovery as assessed by electroretinography.
32 the physiology of the retina was analyzed by electroretinography.
33             Dark adaptation was monitored by electroretinography.
34            Retinal function was evaluated by electroretinography.
35 tion of outer nuclear layer thickness and by electroretinography.
36 Dawley rats, and recovery was measured using electroretinography.
37 optical coherence tomography angiography and electroretinography.
38  glaucoma as well as cone-rod dysfunction on electroretinography.
39 imaging, and visual function was assessed by electroretinography.
40 omography, kinetic visual field testing, and electroretinography.
41 erence tomography, and multifocal or pattern electroretinography.
42 ed peripheral retinal function by multifocal electroretinography.
43 icity was evaluated by clinical findings and electroretinography: 30-Hz flicker responses were compar
44 thanol, and retinal function was assessed by electroretinography 72 hours after the initial dose of m
45 ticularly in areas with decreased multifocal electroretinography amplitude.
46           BEST1 mutations, imaging findings, electroretinography amplitudes, and implicit times.
47 ptor outer nuclear layer (ONL) thickness and electroretinography amplitudes.
48 ty, biomicroscopy, color fundus photography, electroretinography analysis, and visual-evoked potentia
49 tion and visual sensitivity were examined by electroretinography and an active avoidance behavioral t
50                                   Multifocal electroretinography and chromatic/achromatic contrast se
51 on retinal function and neurodegeneration by electroretinography and detailed morphology.
52  static perimetry, full-field and multifocal electroretinography and electro-oculography.
53 3 expression, mice were analyzed by scotopic electroretinography and fluorescein angiography.
54                                              Electroretinography and gene expression analyses suggest
55                                   Full-field electroretinography and histologic examination were used
56       Photoreceptor survival was assessed by electroretinography and histology.
57 rvival time, visual acuity decline rate, and electroretinography and imaging findings.
58 er retinal function as measured, in vivo, by electroretinography and manganese-enhanced MRI.
59 mprovement observed in this case, multifocal electroretinography and microperimetry indicate that sub
60 and cone function was analyzed by full-field electroretinography and multifocal electroretinography.
61              Further investigations included electroretinography and neuroradiologic imaging.
62                                   Multifocal electroretinography and ocular coherence tomography are
63                              We showed using electroretinography and single cell recordings that the
64 tween photoreceptors and ON-BC neurons using electroretinography and single cell recordings.
65  mice were examined by scotopic and photopic electroretinography and then killed for biochemical and
66 phototransduction components was assessed by electroretinography and to couple to vertebrate transduc
67                                      In vivo electroretinography and transretinal recordings revealed
68             To delineate mechanisms, we used electroretinography and visual evoked potential recordin
69 times after infection, eyes were analyzed by electroretinography and were harvested for quantitation
70  used electrophysiological techniques (i.e., electroretinography) and molecular analyses, this work s
71 se of infection by biomicroscopy, histology, electroretinography, and bacterial and inflammatory cell
72 enetrating electrode arrays, multi-electrode electroretinography, and electromyography, are also viab
73  9 months of age using immunohistochemistry, electroretinography, and fluorescein angiography.
74 A), kinetic and static perimetry, full-field electroretinography, and fundus autofluorescence (FAF).
75 VF), dark-adapted absolute thresholds (DAT), electroretinography, and fundus photography.
76 t lamp examination, indirect ophthalmoscopy, electroretinography, and histologic examination.
77 ded toxicology (clinical examination, serial electroretinography, and histopathology) in normal rabbi
78 al retinal examination, noninvasive imaging, electroretinography, and histopathology/immunohistochemi
79   At P30, retinal function was measured with electroretinography, and morphologic preservation of the
80                 Slit lamp examination (SLE), electroretinography, and myeloperoxidase activity were p
81 ields, optical coherence tomography, pattern electroretinography, and neuro-ophthalmic examinations.
82 notype was characterized with psychophysics, electroretinography, and optical coherence tomography.
83 d safety were evaluated with ophthalmoscopy, electroretinography, and pathology.
84 s were analyzed by biomicroscopy, histology, electroretinography, and quantitation of bacteria and in
85 l recovery after ischemia was measured using electroretinography, and retinal histology was examined
86             Retinal function was assessed by electroretinography, and retinal morphological features
87             Retinal function was assessed by electroretinography, and retinal structure by light micr
88  photoreceptor function was assessed through electroretinography, and survival was documented in morp
89 e scanning laser ophthalmoscopy (AF-SLO) and electroretinography, and the extent of laser-induced CNV
90 nical ophthalmic examinations, neuroimaging, electroretinography, and the results of MMACHC mutation
91 -fluorescence, optical coherence tomography, electroretinography, and ultrasound.
92 fundus photography, fluorescein angiography, electroretinography, and visual evoked potentials were o
93 nfocal and electron microscopy, single-flash electroretinography, and whole-cell patch-clamp recordin
94             Visual acuity, retinal features, electroretinography, and whole-exome sequencing.
95  with fundus autofluorescence and multifocal electroretinography as indicated), will greatly minimize
96                                              Electroretinography assessed functional recovery after i
97 y histopathology, morphometric analysis, and electroretinography at P60.
98 erical equivalent refraction, visual fields, electroretinography B-wave amplitudes, and qualitative i
99                                           By electroretinography, b-wave amplitude was reduced by 75%
100                        Eyes were analyzed by electroretinography, bacterial quantitation, and antibio
101               Patients were also assessed by electroretinography, brain MRI and magnetic resonance sp
102                                              Electroretinography can be usually be obtained in infant
103 mination, visual acuity (VA), visual fields, electroretinography, color vision testing, and retinal i
104 r initial rate of loss of visual function by electroretinography, compared with eyes without these st
105  patients (aged 10-62 years) with full-field electroretinography-confirmed achromatopsia.
106 , and reduced scotopic responses observed on electroretinography consistent with the CRD phenotype of
107                                   Full-field electroretinography demonstrated a reduced rod response
108                                   Multifocal electroretinography demonstrated normal amplitude and im
109                                              Electroretinography demonstrated that cone responses wer
110  also remain functional, albeit with reduced electroretinography (ERG) amplitudes typical of RetGC1-/
111                       To determine toxicity, electroretinography (ERG) amplitudes were measured in re
112 tinal tissue as indicated by TUNEL assay and electroretinography (ERG) analysis.
113                                              Electroretinography (ERG) and both chemical and histolog
114 Functional retinal changes were evaluated by electroretinography (ERG) and by morphologic and ultrast
115 me course of retinal dysfunction by scotopic electroretinography (ERG) and by quantitative morphology
116 rimetry, fluorescein angiography, full-field electroretinography (ERG) and electro-oculography (EOG),
117                                              Electroretinography (ERG) and intraocular pressure (IOP)
118                                              Electroretinography (ERG) and light microscopy were used
119 ete ophthalmic examination, full-field flash electroretinography (ERG) and multifocal ERG, light-adap
120 gating their visual capabilities by means of electroretinography (ERG) and patterned visual evoked po
121                                     Baseline electroretinography (ERG) and preretinal oxygen (Po(2))
122     Function was assessed with perimetry and electroretinography (ERG) and retinal structure with opt
123   Finally, anesthetized mice were studied by electroretinography (ERG) at different times after expos
124                                     Spectral electroretinography (ERG) demonstrated significant impro
125 ship among vector dose, visual function, and electroretinography (ERG) findings.
126         The mice were analyzed by full-field electroretinography (ERG) for light response.
127  microvilli area and an abnormal response on electroretinography (ERG) in UBE3D(+/-) heterozygous mic
128 chanism of confocal IOS, comparative IOS and electroretinography (ERG) measurements were made using n
129 icity was evaluated by clinical findings and electroretinography (ERG) on 244 evaluable injections in
130 munoblots, and cone function was analyzed by electroretinography (ERG) recordings.
131                                     Scotopic electroretinography (ERG) showed a diminished c-wave amp
132            At 7 days after exposure to light electroretinography (ERG) showed that minocycline signif
133                                      We used electroretinography (ERG) to evaluate retinal function a
134 cy of optical coherence tomography (OCT) and electroretinography (ERG) to monitor pathological and fu
135                                              Electroretinography (ERG) was performed before surgery a
136            Among mice followed for 8 months, electroretinography (ERG) was performed on both eyes bef
137                                              Electroretinography (ERG) was performed to evaluate reti
138                                              Electroretinography (ERG) was used to assess rod and con
139                                              Electroretinography (ERG) was used to evaluate the recov
140                                 Strobe flash electroretinography (ERG) was used to examine outer reti
141                Computerized pupillometry and electroretinography (ERG) were performed to assess optic
142 tions, blood screenings, vision testing, and electroretinography (ERG) were performed.
143  transmission electron microscopy (TEM), and electroretinography (ERG) were used to analyze 6 genotyp
144 testing, dark adaptation testing, full-field electroretinography (ERG), and electro-oculography (EOG)
145 handheld tonometer, indirect ophthalmoscopy, electroretinography (ERG), and histology.
146 of bacteria, organ function as determined by electroretinography (ERG), and histopathologic changes w
147 on were documented using fundus photography, electroretinography (ERG), and histopathology.
148 ry, fundus-guided microperimetry, full-field electroretinography (ERG), and multifocal ERG.
149 ry, fundus-guided microperimetry, full-field electroretinography (ERG), and multifocal ERG.
150    Functional abnormalities were assessed by electroretinography (ERG), and neurodegeneration was ass
151 netic perimetry, chromatic static perimetry, electroretinography (ERG), and optical coherence tomogra
152           Retinal function was measured with electroretinography (ERG), and relative content of selec
153 sponses to light were measured by full-field electroretinography (ERG), and retinal tissues were exam
154                          Visual acuity (VA), electroretinography (ERG), and spectral-domain optical c
155 nd electron microscopy, immunocytochemistry, electroretinography (ERG), and spectrophotometry.
156 lowing injection by slit lamp biomicroscopy, electroretinography (ERG), bacterial and inflammatory ce
157 The outcome after ischemia was examined with electroretinography (ERG), by measuring retinal cell lay
158 tical coherence tomography (OCT), full-field electroretinography (ERG), electro-oculography (EOG), an
159  for 8 wk and evaluated by AGE fluorescence, electroretinography (ERG), electron microscopy, and micr
160 -LCA (n = 30; ages, 4-55) were studied using electroretinography (ERG), full-field stimulus testing (
161 d reduced retinal thickness were found using electroretinography (ERG), fundus photography (FP), fund
162                       Clinical examinations, electroretinography (ERG), histology, and bacterial quan
163                                              Electroretinography (ERG), histology, light microscopy,
164 ogy, transmission electron microscopy (TEM), electroretinography (ERG), immunohistochemistry, Western
165 rn electroretinography (PERG) and full-field electroretinography (ERG), incorporating international s
166 -) and wild-type (WT) mice were evaluated by electroretinography (ERG), lectin cytochemistry, and cor
167 ctural, and biochemical assessments included electroretinography (ERG), light and electron microscopi
168  age-matched control puppies were studied by electroretinography (ERG), light and electron microscopy
169  of the gene targeted mice were evaluated by electroretinography (ERG), light, and electron microscop
170 almic examinations were performed, including electroretinography (ERG), multifocal ERG (mfERG), perim
171 s autofluorescence (FAF) imaging, full-field electroretinography (ERG), multifocal ERG, and central v
172 -induced retinal degeneration using scotopic electroretinography (ERG), optical coherence tomography
173 n were determined at multiple time points by electroretinography (ERG), optical coherence tomography
174 ional examinations were performed, including electroretinography (ERG), optical coherence tomography
175                  Phenotype was assessed with electroretinography (ERG), optical coherence tomography,
176 almic examinations were performed, including electroretinography (ERG), perimetry, optical coherence
177 bset of the detected RP1 heterozygotes using electroretinography (ERG), psychophysics, and optical co
178 resonance imaging, full-field and multifocal electroretinography (ERG), visual evoked potentials (VEP
179 s measured by optokinetic tracking (OKT) and electroretinography (ERG).
180 ically, and retinal function was analysed by electroretinography (ERG).
181 of CSNB2 and RP were confirmed by full-field electroretinography (ERG).
182 tudied with optical coherence tomography and electroretinography (ERG).
183              Cone function was determined by electroretinography (ERG).
184 A), visual field sensitivity, and full-field electroretinography (ERG).
185 utant Ush1c, were analyzed by microscopy and electroretinography (ERG).
186 ve impact on visual function, as assessed by electroretinography (ERG).
187 er retinal layers as well as functionally by electroretinography (ERG).
188 e analyzed by optokinetic response (OKR) and electroretinography (ERG).
189 electron microscopy, immunofluorescence, and electroretinography (ERG).
190 ron microscopy, and single-flash and flicker electroretinography (ERG).
191 c perimetry, static chromatic perimetry, and electroretinography (ERG).
192 he function of the retina was evaluated with electroretinography (ERG).
193 to examine the function of this rhodopsin by electroretinography (ERG).
194 e rate of dark adaptation was analyzed using electroretinography (ERG).
195 etinal function was assessed by dark-adapted electroretinography (ERG).
196 AF), optical coherence tomography (OCT), and electroretinography (ERG).
197 oldmann visual fields (GVFs), and full-field electroretinography (ERG).
198 inal function was assessed by three forms of electroretinography (ERG): slow-sequence multifocal (mf)
199 ed microperimetry, full-field and multifocal electroretinography (ffERG and mfERG), spectral-domain o
200 retinal function was studied with full-field electroretinography (ffERG) from 3 months through 2 year
201 hthalmoscopy, fundus photography, full-field electroretinography (ffERG), Goldmann visual fields (VFs
202 almoscopy, Goldmann visual field, full-field electroretinography (ffERG), OCT, and FAF photography.
203 oherence tomography (SD-OCT), and full-field electroretinography (ffERG).
204                                              Electroretinography findings support the more severe cli
205                                       Global electroretinography findings were normal.
206 ed on visual acuity, fundus photography, and electroretinography findings.
207  in the retina and choroid were evaluated by electroretinography, fluorescein angiography, light micr
208                                              Electroretinography from an additional group of normal m
209 t underwent a complete clinical examination, electroretinography (full field and pattern), visual evo
210 dus appearance, visual field, and full-field electroretinography, fundus autofluorescence, and optica
211 phthalmic examinations, including full-field electroretinography, fundus photography, fundus autofluo
212                                   Multifocal electroretinography had the greatest proportion of posit
213 went complete ocular examination, full-field electroretinography, handheld spectral-domain optical co
214  of endophthalmitis was graded by slit lamp, electroretinography, histological examinations, and dete
215 riologically and by slit lamp biomicroscopy, electroretinography, histology, and inflammatory cell en
216 infection, the EBE incidence was assessed by electroretinography, histology, bacterial counts, and my
217 d a phototransduction deficit as assessed by electroretinography; however, their photoreceptor struct
218                                   Histology, electroretinography, immunohistochemistry, Western blot
219 dysfunction was provided through multi-focal electroretinography in a subset of such patients.
220 nd maintained visual function as assessed by electroretinography in C57BL/6 mice.
221 unostaining, and measured light responses by electroretinography in mice with targeted disruptions of
222                                  The role of electroretinography in pediatric practice, and to offer
223 d retinal ganglion cell function by means of electroretinography in three patients with cerebral hemi
224 nction because DMOG normalizes the b-wave on electroretinography in wild-type mice.
225 l loss occurred in adult Mfsd2a KO mice, but electroretinography indicated visual function was normal
226 RKL mutations were studied clinically and by electroretinography, kinetic, and chromatic static perim
227 nifest retinal degeneration, as evidenced by electroretinography, light microscopy and pupillometry r
228 trauma and assessed by clinical examination, electroretinography, light microscopy, electron microsco
229 ed using ophthalmoscopy, fundus photography, electroretinography, light microscopy, immunocytochemist
230                                   Multifocal electroretinography may be effective in detecting functi
231  the N95 implicit time on results of pattern electroretinography (median, 98.6 milliseconds [95% CI,
232                  With appropriate technique, electroretinography methods could be made more widely av
233 coherence tomography (SD-OCT) and multifocal electroretinography (mfERG) along with visual fields.
234             Functional studies by multifocal electroretinography (mfERG) evaluated neurodysfunction,
235 l coherence tomography (OCT), and multifocal electroretinography (mfERG) were performed at baseline a
236 oherence tomography (SD-OCT), and multifocal electroretinography (mfERG) were performed at various in
237 ests consisting of visual fields, multifocal electroretinography (mfERG), and contrast sensitivity we
238 gnosis were followed up including multifocal electroretinography (mfERG), spectral-domain optical coh
239 llary RNFLT, RNFL retardance, and multifocal electroretinography (mfERG).
240 fundus autofluorescence (FAF), or multifocal electroretinography (mfERG).
241 y visual field (HVF) testing, and multifocal electroretinography (mfERG).
242 nt routine examination, including full-field electroretinography, microperimetry, and optical coheren
243 ons included electro-oculography, full-field electroretinography, multifocal electroretinography, spe
244  vision (ie, dark adaptometry, pupillometry, electroretinography, nystagmus, and ambulatory behaviour
245                                      In vivo electroretinography of Panx1 knockout mice indicated an
246 P measurements were collected daily, whereas electroretinography, optical coherence tomography, and w
247 hy, fundus autofluorescence imaging, sedated electroretinography, optical coherence tomography, genet
248 ce of drug toxicity by clinical examination, electroretinography, or histologic examination.
249  angiography evidence of active choroiditis, electroretinography parameters indicative of stable or w
250                                     Finally, electroretinography performed on mice deficient for one
251                                      Pattern electroretinography (PERG) and full-field electroretinog
252 analyses included transient pattern-reversal electroretinography (PERG) and full-field flash ERG, wit
253   Macular function was assessed with pattern electroretinography (PERG) to checkerboard stimuli of di
254                   Patients underwent pattern electroretinography (PERG), optical coherence tomography
255                     One month later, pattern electroretinography (PERG), rate of ATP synthesis, gene
256                                              Electroretinography phenotype (cone-rod vs rod-cone dysf
257          Objective functional tests, such as electroretinography, provide an alternative to subjectiv
258 ice, and to offer suggestions for successful electroretinography recordings in children.
259 ischemia by histologic analyses and scotopic electroretinography, respectively.
260 d b-wave amplitudes of scotopic and photopic electroretinography responses 4 months after diabetes in
261                           The mean change in electroretinography responses was not significantly diff
262  mice, their scotopic, maximal, and photopic electroretinography responses were comparable to those o
263                                   Full-field electroretinography responses were extinguished in 50% o
264 l coherence tomography, and severely reduced electroretinography responses.
265  clinical symptom of night blindness and the electroretinography results suggest a primary rod dysfun
266 nction as assessed by VA, visual fields, and electroretinography results; and retinal structural chan
267 omain optical coherence tomography (SD-OCT), electroretinography, retinal morphology, and visual reti
268 -/-) and Oa1(-/-) mice had normal results on electroretinography, retrograde labeling showed a signif
269                                              Electroretinography revealed a cone-rod pattern of dysfu
270                                              Electroretinography revealed a rod-cone pattern of dysfu
271                          However, twin flash electroretinography revealed a slight delay in recovery
272 xons project to the superior colliculus, and electroretinography revealed no defect of adult visual f
273         Retinal function was evaluated using electroretinography (scotopic, photopic, and pattern).
274                                     Ganzfeld electroretinography showed faster recovery of retinal fu
275 ailed neuro-ophthalmic examination including electroretinography showed him to have a typical retinal
276                                              Electroretinography showed mutant mouse eyes had a selec
277                                Histology and electroretinography showed no cre-mediated RPE toxicity.
278                                              Electroretinography showed that scotopic and photopic re
279                    Rod function (measured by electroretinography) showed modest but statistically sig
280 , full-field electroretinography, multifocal electroretinography, spectral-domain optical coherence t
281                                              Electroretinography studies demonstrated a strong correl
282  proportion to the clinical exams, prompting electroretinography testing that revealed an electronega
283 arent to clinical examination and full-field electroretinography testing.
284            At P28, animals were evaluated by electroretinography; tissues were then harvested for bio
285                                Here, we used electroretinography to examine the functional role of BK
286 y screens (such as histological analysis and electroretinography) to identify the subset of fish with
287 ography, fundus autofluorescence, multifocal electroretinography, visual fields) and classification o
288                  The high temporal frequency electroretinography was determined mainly by the luminan
289          Simultaneous bilateral dark-adapted electroretinography was performed 2 weeks and 12 weeks a
290                                   Full-field electroretinography was performed binocularly, using DTL
291 dioactive microsphere blood flow method, and electroretinography was performed during the first 120 m
292                              After exposure, electroretinography was performed on mice dark adapted f
293  was performed to assess for cell death, and electroretinography was performed to assess function.
294                                   Multifocal electroretinography was shown to have a high sensitivity
295 n levels of several phototransduction genes; electroretinography was used to assess quantitatively th
296 -retinylidene-N-retinylethanolamine content; electroretinography was used to measure phototransductio
297                      Fundus photography, and electroretinography were performed in 12 patients, and o
298 ure (IOP) tonometry, fundus photography, and electroretinography were performed over multiple time po
299                               Microscopy and electroretinography were used to characterize transgenic
300 us photography, fluorescein angiography, and electroretinography were used to evaluate retinal anatom

WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。
 
Page Top