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1 tive immunohistochemistry) and functionally (electroretinography).
2 provement of retinal function (assessed with electroretinography).
3 n of Akita and control mice was evaluated by electroretinography.
4 kinetic and static perimetry, and full-field electroretinography.
5 lted in functional deficits as determined by electroretinography.
6 fundus ophthalmoscopy, dark adaptometry, and electroretinography.
7 r degeneration was measured by histology and electroretinography.
8 static contrast sensitivity test; and global electroretinography.
9 cone survival was assessed by histology and electroretinography.
10 Retinal function was assayed by electroretinography.
11 escued photoreceptor function as measured by electroretinography.
12 inal histology up to 24 months of age and by electroretinography.
13 ull-field electroretinography and multifocal electroretinography.
14 Rescue of visual function was confirmed by electroretinography.
15 T) or DKO mice, as assessed by histology and electroretinography.
16 ctrometry, and degeneration by histology and electroretinography.
17 ncluding ophthalmoscopy and full-field flash electroretinography.
18 nohistochemistry, Western blot analysis, and electroretinography.
19 l function was characterized with the use of electroretinography.
20 t hypomorphic and Ret conditional mice using electroretinography.
21 ) measures of central retinal thickness, and electroretinography.
22 isual function in these mice was analyzed by electroretinography.
23 reserves RGC activity as measured by pattern electroretinography.
24 nction of USH2A patients was quantified with electroretinography.
25 Neural function was assessed with electroretinography.
26 t time of the N95 wave on results of pattern electroretinography.
27 es of mRDH11 disruption were investigated by electroretinography.
28 munohistochemistry, electron microscopy, and electroretinography.
29 us photography, fluorescein angiography, and electroretinography.
30 ant visual loss in treated abcr(-/-) mice by electroretinography.
31 promoted functional recovery as assessed by electroretinography.
32 the physiology of the retina was analyzed by electroretinography.
33 Dark adaptation was monitored by electroretinography.
34 Retinal function was evaluated by electroretinography.
35 tion of outer nuclear layer thickness and by electroretinography.
36 Dawley rats, and recovery was measured using electroretinography.
37 optical coherence tomography angiography and electroretinography.
38 glaucoma as well as cone-rod dysfunction on electroretinography.
39 imaging, and visual function was assessed by electroretinography.
40 omography, kinetic visual field testing, and electroretinography.
41 erence tomography, and multifocal or pattern electroretinography.
42 ed peripheral retinal function by multifocal electroretinography.
43 icity was evaluated by clinical findings and electroretinography: 30-Hz flicker responses were compar
44 thanol, and retinal function was assessed by electroretinography 72 hours after the initial dose of m
48 ty, biomicroscopy, color fundus photography, electroretinography analysis, and visual-evoked potentia
49 tion and visual sensitivity were examined by electroretinography and an active avoidance behavioral t
59 mprovement observed in this case, multifocal electroretinography and microperimetry indicate that sub
60 and cone function was analyzed by full-field electroretinography and multifocal electroretinography.
65 mice were examined by scotopic and photopic electroretinography and then killed for biochemical and
66 phototransduction components was assessed by electroretinography and to couple to vertebrate transduc
69 times after infection, eyes were analyzed by electroretinography and were harvested for quantitation
70 used electrophysiological techniques (i.e., electroretinography) and molecular analyses, this work s
71 se of infection by biomicroscopy, histology, electroretinography, and bacterial and inflammatory cell
72 enetrating electrode arrays, multi-electrode electroretinography, and electromyography, are also viab
74 A), kinetic and static perimetry, full-field electroretinography, and fundus autofluorescence (FAF).
77 ded toxicology (clinical examination, serial electroretinography, and histopathology) in normal rabbi
78 al retinal examination, noninvasive imaging, electroretinography, and histopathology/immunohistochemi
79 At P30, retinal function was measured with electroretinography, and morphologic preservation of the
81 ields, optical coherence tomography, pattern electroretinography, and neuro-ophthalmic examinations.
82 notype was characterized with psychophysics, electroretinography, and optical coherence tomography.
84 s were analyzed by biomicroscopy, histology, electroretinography, and quantitation of bacteria and in
85 l recovery after ischemia was measured using electroretinography, and retinal histology was examined
88 photoreceptor function was assessed through electroretinography, and survival was documented in morp
89 e scanning laser ophthalmoscopy (AF-SLO) and electroretinography, and the extent of laser-induced CNV
90 nical ophthalmic examinations, neuroimaging, electroretinography, and the results of MMACHC mutation
92 fundus photography, fluorescein angiography, electroretinography, and visual evoked potentials were o
93 nfocal and electron microscopy, single-flash electroretinography, and whole-cell patch-clamp recordin
95 with fundus autofluorescence and multifocal electroretinography as indicated), will greatly minimize
98 erical equivalent refraction, visual fields, electroretinography B-wave amplitudes, and qualitative i
103 mination, visual acuity (VA), visual fields, electroretinography, color vision testing, and retinal i
104 r initial rate of loss of visual function by electroretinography, compared with eyes without these st
106 , and reduced scotopic responses observed on electroretinography consistent with the CRD phenotype of
110 also remain functional, albeit with reduced electroretinography (ERG) amplitudes typical of RetGC1-/
114 Functional retinal changes were evaluated by electroretinography (ERG) and by morphologic and ultrast
115 me course of retinal dysfunction by scotopic electroretinography (ERG) and by quantitative morphology
116 rimetry, fluorescein angiography, full-field electroretinography (ERG) and electro-oculography (EOG),
119 ete ophthalmic examination, full-field flash electroretinography (ERG) and multifocal ERG, light-adap
120 gating their visual capabilities by means of electroretinography (ERG) and patterned visual evoked po
122 Function was assessed with perimetry and electroretinography (ERG) and retinal structure with opt
123 Finally, anesthetized mice were studied by electroretinography (ERG) at different times after expos
127 microvilli area and an abnormal response on electroretinography (ERG) in UBE3D(+/-) heterozygous mic
128 chanism of confocal IOS, comparative IOS and electroretinography (ERG) measurements were made using n
129 icity was evaluated by clinical findings and electroretinography (ERG) on 244 evaluable injections in
134 cy of optical coherence tomography (OCT) and electroretinography (ERG) to monitor pathological and fu
143 transmission electron microscopy (TEM), and electroretinography (ERG) were used to analyze 6 genotyp
144 testing, dark adaptation testing, full-field electroretinography (ERG), and electro-oculography (EOG)
146 of bacteria, organ function as determined by electroretinography (ERG), and histopathologic changes w
150 Functional abnormalities were assessed by electroretinography (ERG), and neurodegeneration was ass
151 netic perimetry, chromatic static perimetry, electroretinography (ERG), and optical coherence tomogra
153 sponses to light were measured by full-field electroretinography (ERG), and retinal tissues were exam
156 lowing injection by slit lamp biomicroscopy, electroretinography (ERG), bacterial and inflammatory ce
157 The outcome after ischemia was examined with electroretinography (ERG), by measuring retinal cell lay
158 tical coherence tomography (OCT), full-field electroretinography (ERG), electro-oculography (EOG), an
159 for 8 wk and evaluated by AGE fluorescence, electroretinography (ERG), electron microscopy, and micr
160 -LCA (n = 30; ages, 4-55) were studied using electroretinography (ERG), full-field stimulus testing (
161 d reduced retinal thickness were found using electroretinography (ERG), fundus photography (FP), fund
164 ogy, transmission electron microscopy (TEM), electroretinography (ERG), immunohistochemistry, Western
165 rn electroretinography (PERG) and full-field electroretinography (ERG), incorporating international s
166 -) and wild-type (WT) mice were evaluated by electroretinography (ERG), lectin cytochemistry, and cor
167 ctural, and biochemical assessments included electroretinography (ERG), light and electron microscopi
168 age-matched control puppies were studied by electroretinography (ERG), light and electron microscopy
169 of the gene targeted mice were evaluated by electroretinography (ERG), light, and electron microscop
170 almic examinations were performed, including electroretinography (ERG), multifocal ERG (mfERG), perim
171 s autofluorescence (FAF) imaging, full-field electroretinography (ERG), multifocal ERG, and central v
172 -induced retinal degeneration using scotopic electroretinography (ERG), optical coherence tomography
173 n were determined at multiple time points by electroretinography (ERG), optical coherence tomography
174 ional examinations were performed, including electroretinography (ERG), optical coherence tomography
176 almic examinations were performed, including electroretinography (ERG), perimetry, optical coherence
177 bset of the detected RP1 heterozygotes using electroretinography (ERG), psychophysics, and optical co
178 resonance imaging, full-field and multifocal electroretinography (ERG), visual evoked potentials (VEP
198 inal function was assessed by three forms of electroretinography (ERG): slow-sequence multifocal (mf)
199 ed microperimetry, full-field and multifocal electroretinography (ffERG and mfERG), spectral-domain o
200 retinal function was studied with full-field electroretinography (ffERG) from 3 months through 2 year
201 hthalmoscopy, fundus photography, full-field electroretinography (ffERG), Goldmann visual fields (VFs
202 almoscopy, Goldmann visual field, full-field electroretinography (ffERG), OCT, and FAF photography.
207 in the retina and choroid were evaluated by electroretinography, fluorescein angiography, light micr
209 t underwent a complete clinical examination, electroretinography (full field and pattern), visual evo
210 dus appearance, visual field, and full-field electroretinography, fundus autofluorescence, and optica
211 phthalmic examinations, including full-field electroretinography, fundus photography, fundus autofluo
213 went complete ocular examination, full-field electroretinography, handheld spectral-domain optical co
214 of endophthalmitis was graded by slit lamp, electroretinography, histological examinations, and dete
215 riologically and by slit lamp biomicroscopy, electroretinography, histology, and inflammatory cell en
216 infection, the EBE incidence was assessed by electroretinography, histology, bacterial counts, and my
217 d a phototransduction deficit as assessed by electroretinography; however, their photoreceptor struct
221 unostaining, and measured light responses by electroretinography in mice with targeted disruptions of
223 d retinal ganglion cell function by means of electroretinography in three patients with cerebral hemi
225 l loss occurred in adult Mfsd2a KO mice, but electroretinography indicated visual function was normal
226 RKL mutations were studied clinically and by electroretinography, kinetic, and chromatic static perim
227 nifest retinal degeneration, as evidenced by electroretinography, light microscopy and pupillometry r
228 trauma and assessed by clinical examination, electroretinography, light microscopy, electron microsco
229 ed using ophthalmoscopy, fundus photography, electroretinography, light microscopy, immunocytochemist
231 the N95 implicit time on results of pattern electroretinography (median, 98.6 milliseconds [95% CI,
233 coherence tomography (SD-OCT) and multifocal electroretinography (mfERG) along with visual fields.
235 l coherence tomography (OCT), and multifocal electroretinography (mfERG) were performed at baseline a
236 oherence tomography (SD-OCT), and multifocal electroretinography (mfERG) were performed at various in
237 ests consisting of visual fields, multifocal electroretinography (mfERG), and contrast sensitivity we
238 gnosis were followed up including multifocal electroretinography (mfERG), spectral-domain optical coh
242 nt routine examination, including full-field electroretinography, microperimetry, and optical coheren
243 ons included electro-oculography, full-field electroretinography, multifocal electroretinography, spe
244 vision (ie, dark adaptometry, pupillometry, electroretinography, nystagmus, and ambulatory behaviour
246 P measurements were collected daily, whereas electroretinography, optical coherence tomography, and w
247 hy, fundus autofluorescence imaging, sedated electroretinography, optical coherence tomography, genet
249 angiography evidence of active choroiditis, electroretinography parameters indicative of stable or w
252 analyses included transient pattern-reversal electroretinography (PERG) and full-field flash ERG, wit
253 Macular function was assessed with pattern electroretinography (PERG) to checkerboard stimuli of di
260 d b-wave amplitudes of scotopic and photopic electroretinography responses 4 months after diabetes in
262 mice, their scotopic, maximal, and photopic electroretinography responses were comparable to those o
265 clinical symptom of night blindness and the electroretinography results suggest a primary rod dysfun
266 nction as assessed by VA, visual fields, and electroretinography results; and retinal structural chan
267 omain optical coherence tomography (SD-OCT), electroretinography, retinal morphology, and visual reti
268 -/-) and Oa1(-/-) mice had normal results on electroretinography, retrograde labeling showed a signif
272 xons project to the superior colliculus, and electroretinography revealed no defect of adult visual f
275 ailed neuro-ophthalmic examination including electroretinography showed him to have a typical retinal
280 , full-field electroretinography, multifocal electroretinography, spectral-domain optical coherence t
282 proportion to the clinical exams, prompting electroretinography testing that revealed an electronega
286 y screens (such as histological analysis and electroretinography) to identify the subset of fish with
287 ography, fundus autofluorescence, multifocal electroretinography, visual fields) and classification o
291 dioactive microsphere blood flow method, and electroretinography was performed during the first 120 m
293 was performed to assess for cell death, and electroretinography was performed to assess function.
295 n levels of several phototransduction genes; electroretinography was used to assess quantitatively th
296 -retinylidene-N-retinylethanolamine content; electroretinography was used to measure phototransductio
298 ure (IOP) tonometry, fundus photography, and electroretinography were performed over multiple time po
300 us photography, fluorescein angiography, and electroretinography were used to evaluate retinal anatom
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