ライフサイエンスコーパス: element-binding protein

1 the transcription factor CREB (cAMP-response element binding protein).

2 e transcription factor CREB (cAMP-responsive element binding protein).

3 regulator CPEB3 (cytoplasmic polyadenylation element-binding protein).

4 f ERK1/2, pCREB [phospho-CREB (cAMP response element-binding protein)].

5 ibosomal S6 kinase, c-Jun, and cAMP response element binding protein.

6 on of the transcription factor cAMP response element-binding protein.

7 activation of Creb3l3/cyclic AMP-responsive element-binding protein.

8 proteins including the ERK and cAMP response element-binding protein.

9 athway involving Akt1/Akt2 and cAMP response element-binding protein.

10 iption factors, especially sterol-regulatory element binding proteins.

11 inactivation of tristetraprolin, two AU-rich element-binding proteins.

12 pithelial cells, cytoplasmic polyadenylation element binding protein 1 (CPEB1) mediates the apical lo

13 ter through association with cAMP responsive element binding protein 1 (CREB)/CREB binding protein (C

14 AMP1-dependent activation of cAMP-responsive element binding protein 1 (CREB1) and SP7 (also known as

15 nteraction between Mesp1 and cAMP-responsive element binding protein 1 (Creb1) in vitro and in vivo.

16 lucocorticoid receptor (GR), cAMP responsive element binding protein 1 (CREB1), peroxisome proliferat

17 s elegans homolog of mammalian cAMP response element binding protein 1 (CREB1)] isoforms on the abili

18 es (Su et al., 2020) identify RAS-responsive element binding protein 1 (RREB1) as a critical integrat

19 We identify RAS-responsive element binding protein 1 (RREB1), a RAS transcriptional

20 pogenesis that results from increased sterol element binding protein 1 (SREBP-1) activity.

21 r the transcription factor sterol regulatory element binding protein 1 (SREBP-1) and its transcriptio

22 domain containing 1C), RREB1 (Ras responsive element binding protein 1), and SEC24C (SEC24 family mem

23 through the activation of sterol regulatory element binding protein 1, which is mediated by the indu

24 ate the transcription factor cAMP-responsive element-binding protein 1 (CREB).

25 The transcription factor cyclic AMP-response element-binding protein 1 (CREB1) controls approximately

26 6) trimethyltransferase, and cAMP-responsive element-binding protein 1 (CREB1).

27 hms, we identified cyclin J and far upstream element-binding protein 1 (FUBP1) as novel miR-16 target

28 replaced by its human homolog Ras-responsive element-binding protein 1 (RREB-1).

29 o-Akt (Ser473), PPARalpha, sterol regulatory element-binding protein 1 (SREBP-1), fatty acid synthase

30 timulates lipogenesis in a sterol regulatory element-binding protein 1 (SREBP-1)-dependent manner.

31 eficiency inhibits hepatic sterol regulatory element-binding protein 1 (SREBP-1, ADD1) processing.

32 on 16 of the gene encoding sterol regulatory element-binding protein 1 (Srebp1), an activator of LC-P

33 Sterol regulatory element-binding protein 1 (SREBP1), when presented in it

34 SREBF1 encodes sterol regulatory element-binding protein 1 (SREBP1), which promotes the t

35 n turn directly suppresses sterol regulatory element-binding protein 1 (SREBP1)-directed transcriptio

36 se block the activation of sterol regulatory element-binding protein 1 (SREBP1)-mediated fatty acid s

37 and activity and defined a sterol regulatory element-binding protein 1 (SREBP1)-mediated pathway thro

38 ovo enhancer activation by sterol regulatory element-binding protein 1 (SREBP1).

39 The expression of sterol regulatory element-binding protein 1 (Srebp1c), 3-hydroxy-3-methylg

40 in cell number), increased sterol regulatory element-binding protein 1 expression (about 3-fold incre

41 l factor CREB1 (cyclic AMP [cAMP]-responsive element-binding protein 1) had the strongest increase in

42 nd report the binding of FUBP1 (Far Upstream element-Binding Protein 1) to these elements and the rol

43 ch included higher nuclear sterol regulatory element-binding protein 1, higher Srepb2 messenger RNA (

44 CU's transcriptional regulator cAMP response element-binding protein 1.

45 r-activated receptor gamma/sterol regulatory element-binding protein 1/CD36 in hepatocytes from high

46 nositide 3 kinase (PI3K)/Akt/sterol response element-binding protein-1 (SREBP-1) signaling pathway in

47 ls increased expression of sterol regulatory element binding protein 1c (SREBP-1c) and its downstream

48 simulating the increase in sterol-regulatory element binding protein 1c (SREBP-1c) seen in NAFLD pati

49 insulinemia, increased liver sterol response element binding protein 1c mRNA, or obesity.

50 Sterol regulatory element-binding protein 1c (SREBP-1c) is a central regul

51 egulates the expression of sterol regulatory element-binding protein 1c (SREBP-1c), leading to increa

52 d the expression of mature sterol regulatory element-binding protein 1c and fatty acid synthase.

53 ased in WT mice, including sterol regulatory element-binding protein 1c target gene fatty-acid syntha

54 se Acc1 phosphorylation, and sterol response element-binding protein 1c-dependent gene expression.

55 X receptor (LXR)-mediated sterol regulatory element binding protein-1c (SREBP-1c) gene expression.

56 consensus sequence in the sterol regulatory-element binding protein-1c (Srebp-1c) promoter.

57 hich induced expression of sterol regulatory element binding protein-1c (SREBP-1c), resulting in indi

58 tor-gamma (PPAR-gamma) and sterol regulatory element binding protein-1c in WAT accounted for the phen

59 D-1 protein, activation of sterol regulatory element-binding protein-1c (SREBP-1), accumulation of ce

60 ption of the gene encoding sterol regulatory element-binding protein-1c (SREBP-1c).

61 Cytoplasmic polyadenylation element binding protein 2 (CPEB2) is an RNA-binding prot

62 omotes activity of hepatic sterol regulatory element binding protein 2 (SREBP2) through activation of

63 hibiting the activation of sterol regulatory-element binding protein 2 and downregulating low-density

64 Remarkably, overexpressing sterol regulatory element binding protein 2 in the spinal cord of normal m

65 SOD1(G93A) mice; levels of sterol regulatory element binding protein 2, a transcriptional regulator o

66 lational regulator cytosolic polyadenylation element-binding protein 2 (CPEB2) has two isoforms, CPEB

67 nds to the W-boxes of DEHYDRATION-RESPONSIVE ELEMENT-BINDING PROTEIN 2 (GhDREB2), which encodes a deh

68 e have previously identified iron-responsive element-binding protein 2 (IRP2) as an important COPD su

69 within the genes encoding sterol regulatory element-binding protein 2 (SREBP-2) and SREBP-1, respect

70 ynthesis by a reduction in sterol regulatory element-binding protein 2 (SREBP2)-regulated transcripti

71 -dependent protein ALAS2 and iron-responsive element-binding protein 2 expression and reduced porphyr

72 Furthermore, SREBP2 (sterol regulatory element-binding protein 2) protein expression and activa

73 neurotrophins activate the sterol regulatory element-binding protein-2 (SREBP2) that regulates genes

74 ER stress can activate the sterol regulatory element-binding protein-2 (SREBP2), an ER-localized tran

75 w a strong increase in the sterol-regulatory element-binding protein-2 yet are unable to maintain nor

76 ein partners, DREB2A (dehydration-responsive element-binding protein 2A), ANAC013, and ANAC046, consi

77 se 3 (TIMP3) and cytoplasmic polyadenylation element binding protein 3 (CPEB3) were identified as put

78 The cytoplasmic polyadenylation element-binding protein 3 (CPEB3) regulates the translat

79 kably, ceramide-induced RIP of cAMP response element-binding protein 3-like 1 (CREB3L1) also involves

80 ly induced substance (OASIS)/cAMP responsive element-binding protein 3-like 1 (CREB3l1), a CREB/ATF f

81 eolytic processing of CREB3L1 (cAMP response element-binding protein 3-like 1), a membrane-bound tran

82 Cyclic AMP-responsive element-binding protein 3-like 3, hepatocyte specific (C

83 Ethylene-responsive element binding protein, a member of the APETALA2/ethyle

84 ic adenosine monophosphate (cAMP) responsive-element-binding protein, a crucial mediator in long-term

85 culum calcium efflux, and of cAMP-responsive element-binding protein, a key transcription factor in h

86 e palmitoyltransferase 1a, sterol regulatory element-binding protein, acetyl coenzyme A carboxylase,

87 tes leptin transcription via cAMP-responsive element binding protein activation (CREB activation) and

88 S2 expression was dependent on cAMP Response Element Binding Protein activity and that this was essen

89 AGE increases sterol regulatory element binding protein activity, which leads to an incr

90 iated by inhibition of carbohydrate response element binding protein activity.

91 ral kinases to inhibit carbohydrate response element-binding protein-alpha and -beta.

92 n of cyclic adenosine monophosphate response element binding protein and activating transcription fac

93 ding to the phosphorylation of cAMP response element binding protein and, consequently, SMC rediffere

94 f the transcription factor cellular response element-binding protein and an increase in brain-derived

95 s mediated by hepatic cholesterol responsive element-binding protein and featured portal/lobular infl

96 g to cyclic adenosine monophosphate response element-binding protein and the induction of CD25, CD69,

97 by transcription factor CREB (cAMP-response element-binding protein) and silencing of CREB resulted

98 n factor 1 (SREBF1), carbohydrate-responsive element-binding protein, and hepatocyte nuclear factor 4

99 APETALA2/ethylene-responsive element binding protein (AP2/EREBP) transcription factor

100 AU-rich element-binding proteins (ARE-BPs) offer post-transcript

101 25 functions by interacting with purine rich element binding protein B (PURB), and associating with a

102 on via inhibition of carbohydrate-responsive element-binding protein-beta, pyruvate kinase L, SCD-1,

103 or 1alpha (PGC1alpha), cyclic AMP-responsive element binding protein binding protein (CBP), steroid r

104 CBP (CREB (cAMP responsive element binding protein) binding protein (CREBBP)) and P

105 alpha, and HATs (p300 and p300/cAMP response element-binding protein-binding protein-associated facto

106 ruited the epigenetic modifier cAMP-response element-binding protein-binding protein/p300 and thereby

107 reduction in the activation of cAMP response element-binding protein, but not the activation of Ca(2+

108 t signaling, through the ERK/cAMP-responsive element-binding protein/c-Jun pathway.

109 n of cyclic adenosine monophosphate response element binding protein (CBP), p300, and Cbp/p300-intera

110 uced expression of the cyclic AMP-responsive element-binding protein (CBP), which as part of the p300

111 rough ACLY to increase carbohydrate response element binding protein (ChREBP) activity, histone acety

112 se-responsive factor Carbohydrate-Responsive Element Binding Protein (ChREBP) and GLP-1 secretion by

113 Carbohydrate response element binding protein (ChREBP) is a key transcriptiona

114 Carbohydrate responsive element binding protein (ChREBP) is central for de novo

115 Carbohydrate-response element binding protein (ChREBP) plays a key role in reg

116 educes the activity of carbohydrate response element binding protein (ChREBP), a cellular hexose-phos

117 n that the activity of carbohydrate response element binding protein (ChREBP), a key regulator of gly

118 lleagues show that the carbohydrate response element-binding protein (ChREBP) coordinates an adaptive

119 egulated expression of carbohydrate response element-binding protein (ChREBP) in cells containing AR-

120 The carbohydrate-response element-binding protein (ChREBP) is a glucose-responsive

121 Carbohydrate-responsive element-binding protein (ChREBP) is a glucose-sensing tr

122 we hypothesized that carbohydrate-responsive element-binding protein (ChREBP), a transcriptional acti

123 tor alpha (PPARalpha), carbohydrate response element-binding protein (ChREBP), and cAMP response elem

124 sponse to glucose in a carbohydrate-response element-binding protein (ChREBP)-dependent manner.

125 in many cell types is carbohydrate response element-binding protein (ChREBP).

126 transcription factor, carbohydrate response element-binding protein (ChREBP).

127 fficient to suppress carbohydrate-responsive-element-binding protein (ChREBP, a master lipogenic regu

128 n of VEGF by the cytoplasmic polyadenylation element-binding proteins CPEB1 and CPEB4 during developm

129 sed cAMP signaling and reduced cAMP-response-element binding protein (CREB) activation, a critical pa

130 increasing phosphorylation of CAMP response element binding protein (CREB) and PSD95 after TBI.

131 activated PERK phosphorylates CAMP response element binding protein (CREB) and PSD95 directly at the

132 n of the transcription factors cAMP-response element binding protein (CREB) and signal transducer and

133 ected with a dominant-negative cAMP response element binding protein (CREB) and was eliminated by mut

134 the transcriptional regulator cAMP response element binding protein (CREB) in both mouse and human B

135 AMP and the phosphorylation of cAMP response element binding protein (CREB) in INS-1 cells.

136 cAMP response element binding protein (CREB) is a key regulator of glu

137 cAMP-response element binding protein (CREB) is a nuclear transcriptio

138 The cyclic AMP response element binding protein (CREB) is a primary hub of activ

139 ctor cyclic adenosine monophosphate response element binding protein (CREB) is overexpressed in many

140 , TDCA significantly increased cAMP response element binding protein (CREB) phosphorylation in FLO-1

141 impair efficient TSH-dependent cAMP response element binding protein (CREB) phosphorylation.

142 signal-regulated kinase (ERK)-cAMP-response element binding protein (CREB) signaling, as well as DA

143 ing to increased levels of the cAMP response element binding protein (CREB), a transcriptional factor

144 d protein kinase (p38 MAPK), cAMP-responsive element binding protein (CREB), and activating transcrip

145 e for the transcription factor cAMP response element binding protein (CREB), and we demonstrate that

146 iron-dependent activation of cAMP-responsive element binding protein (CREB), the transcription factor

147 racellular signaling molecule, cAMP-response element binding protein (CREB), which serves as a pivota

148 mutant S386/396E bound to the cAMP response element binding protein (CREB)-binding protein reveals t

149 arvation-inducible coactivator cAMP response element binding protein (CREB)-cAMP-regulated transcript

150 mic Ca(2+) elevation and cyclic AMP response element binding protein (CREB)-dependent transcription.

151 r domain associated-1 protein, cAMP response element binding protein (CREB)-regulated transcription c

152 the cyclic adenosine monophosphate response element binding protein (CREB)-regulated transcription c

153 he transcription factor Ca(2+)/cAMP response element binding protein (CREB).

154 of a persistent activation of cAMP-response element binding-protein (CREB) and C/EBPbeta expression.

155 in neonatal mice and reversed cAMP response element-binding protein (CREB) activation and activity-r

156 d IL-10 resulted in epithelial cAMP response element-binding protein (CREB) activation and subsequent

157 by free fatty acid (FFA), and cAMP response element-binding protein (CREB) activation by glucagon, l

158 rk1/2 MAP-kinase signaling and cAMP response element-binding protein (CREB) activation in myeloid cel

159 e of cyclic adenosine monophosphate response element-binding protein (CREB) activity.

160 4-DS1, binding PTH-sensitive cAMP-responsive element-binding protein (CREB) and a cluster termed C24-

161 negative regulation of cAMP/calcium response element-binding protein (CREB) and CREB-activated gene e

162 The transcription factors, cAMP-response element-binding protein (CREB) and heat-shock factor-4a

163 pressant action: activation of cAMP response element-binding protein (CREB) and induction of brain-de

164 hanceosome consisting of cyclic AMP response element-binding protein (CREB) and nuclear factor Y (NF-

165 KA subsequently phosphorylated cAMP response element-binding protein (CREB) at Ser-133 to activate it

166 the phosphorylation of cAMP/calcium response element-binding protein (CREB) at serine 133 and CREB-me

167 For example, deficits in cAMP response element-binding protein (CREB) binding protein (CBP; an

168 denosine 3',5'-monophosphate (cAMP) response element-binding protein (CREB) family transcription fact

169 T) depended on activation of cAMP-responsive element-binding protein (CREB) for induction of Ralpha2

170 ctor cyclic adenosine monophosphate response element-binding protein (CREB) in NSTEMI (p < 0.05 vs. c

171 ty of the transcription factor cAMP response element-binding protein (CREB) in young adult rodents fa

172 by the transcription factor cAMP-responsive element-binding protein (CREB) is essential for a wide r

173 tors, the phosphorylation of cAMP-responsive element-binding protein (CREB) is strongly decreased in

174 n under basal/nicotine-induced/cAMP-response element-binding protein (CREB) overexpressed conditions

175 olar PregS similarly activates cAMP response element-binding protein (CREB) phosphorylation (within 1

176 Cyclic AMP-response element-binding protein (CREB) plays key transcriptional

177 Cyclic-AMP response element-binding protein (CREB) signaling has a critical

178 PKA-mediated induction of the cAMP response element-binding protein (CREB) signaling pathway.

179 5) characterized by increased cAMP response element-binding protein (CREB) signalling responses in a

180 at HDAC2 is a direct target of cAMP response element-binding protein (CREB) that is activated by beta

181 dependent binding of FGFR1 and cAMP-response element-binding protein (CREB) to a conserved cAMP respo

182 ge on the transcription factor cAMP response element-binding protein (CREB) to enhance the expression

183 We found that loss of the cAMP response element-binding protein (CREB) transcription factor sign

184 kinase II (CaMKII) and Ca(2+)/cAMP response element-binding protein (CREB) transcription factor.

185 gets of protein kinase A (PKA)-cAMP response element-binding protein (CREB), a pathway activated by f

186 anscription factor 3 (E2F3), CAMP-responsive element-binding protein (CREB), and brain-derived neurot

187 NA for a transcription factor, cAMP response element-binding protein (CREB), and by an inhibitor of i

188 uclear factor kappa B (NF-kB), cAMP response element-binding protein (CREB), and glucocorticoid recep

189 Phosphorylated cAMP response element-binding protein (CREB), nuclear NFATc4, and the

190 However, phosphorylation of cAMP-response element-binding protein (CREB), the cAMP-regulated trans

191 P increased phosphorylation of cAMP response element-binding protein (CREB), which, in turn, increase

192 scriptional coactivators cyclic-AMP response element-binding protein (CREB)-binding protein (CBP) and

193 show that high glucose induced cAMP response element-binding protein (CREB)-binding protein (CBP)-med

194 (MAPK) signaling, induction of cAMP response element-binding protein (CREB)-insulin receptor substrat

195 712 on memory formation and on cAMP response element-binding protein (CREB)-regulated genes in aged m

196 cade adenylyl cyclase-cAMP-PKA-cAMP response element-binding protein (CREB).

197 ostaglandin E2 was mediated by cAMP response element-binding protein (CREB).

198 reduced phosphorylation of the cAMP response element-binding protein (CREB).

199 nhanced the phosphorylation of cAMP response element-binding protein (CREB).

200 rough GPR160-dependent ERK and cAMP response element-binding protein (CREB).

201 ivation and subsequent cyclic AMP-responsive element-binding protein (CREB)/CREB-regulated transcript

202 ted the transcription factor cAMP-responsive element binding protein (Creb1) and its transcriptional

203 The transcription factor cAMP response element-binding protein (CREB1) has been shown to be inv

204 let alpha cells by GS/cAMP/PKA/cAMP-response element-binding protein-dependent activation of PC1.

205 : p38-dependent growth arrest, cAMP response element-binding protein-dependent cell survival, and ERK

206 Inhibition of cAMP response element-binding protein-dependent signaling may particip

207 s and subsequent increase in cAMP responsive element binding protein DNA-binding activity and inducti

208 2, ribosomal S6 kinase 1, or cAMP responsive element binding protein DNA-binding activity prevented t

209 1 interacts with the adenylateuridylate-rich element-binding protein embryonic lethal abnormal vision

210 The far upstream element binding protein (FBP) and the FBP-interacting re

211 The transcription factor far upstream element binding protein (FBP) binds and activates the MY

212 Transcription factors of the far-upstream element-binding protein (FBP) family represent cellular

213 R) protein that transports sterol regulatory element-binding proteins from the ER to Golgi, where the

214 els of specific members of the cAMP Response Element Binding protein gene family.

215 d transcription factor cyclic-AMP-responsive-element-binding protein H (CREBH) but not by peroxisome

216 Here we reported that cyclic AMP-responsive element-binding protein H (CREBH) positively regulates m

217 nuclear transcription factor cAMP-responsive element-binding protein H (CREBH) processing.

218 riched transcription factor, cAMP-responsive element-binding protein hepatic-specific (CREBH), modula

219 sensing transcription factor cAMP-responsive element-binding protein, hepatic-specific (CREBH) functi

220 Cyclic AMP-responsive element binding protein, hepatocyte specific (CREBH), is

221 -binding protein (ChREBP), and cAMP response element-binding protein, hepatocyte specific (CREBH).

222 cy reduced the activation of cAMP-responsive element binding protein-hepatocyte specific and peroxiso

223 Orb2 is a cytoplasmic polyadenylation element-binding protein homolog in Drosophila melanogast

224 in the efficient signaling to cAMP-response element-binding protein in neurons.

225 but not its paralog carbohydrate-responsive element-binding protein, is the predominant glucose-resp

226 the cyclic adenosine monophosphate response element-binding protein, leading to early-onset cone pro

227 ng reduction in phosphorylated cAMP-response element binding protein levels in the NAcSh that could a

228 nflammatory cytokines, carbohydrate response element binding protein-mediated transcription, and micr

229 omes, thereby activating the sterol response element-binding protein-mediated cholesterol synthesis p

230 anscription factors CREB/CREM (cAMP response element-binding protein/modulator) is linked to atrial f

231 DE4D) gene expression, phospho-cAMP response element-binding protein (p-CREB), and cAMP response elem

232 s of their respective targets, cAMP response element-binding protein, p38, and extracellular signal-r

233 e cyclase --> cAMP --> PKA --> cAMP response element-binding protein pathway mediating cell survival

234 of Insig proteins and the sterol regulatory element-binding protein pathway.

235 ogram, including Ras and cyclic AMP response element-binding protein pathways and other Gata factors,

236 ced levels of phosphorylated cAMP-responsive element binding protein (pCREB) in the CA1s of a-FMHis-t

237 ng occupancy of phosphorylated cAMP response element-binding protein (pCREB) at two sites in the lept

238 ity-related proteins such as cAMP-responsive element binding protein phosphorylated at Ser133, calciu

239 s protein kinase A-dependent cAMP-responsive element-binding protein phosphorylation, the effect of c

240 ACEA-induced Galphas-dependent cAMP response element-binding protein phosphorylation.

241 fatostatin A, which blocks sterol regulatory element-binding protein proteolytic cleavage and activat

242 experiments demonstrated that cAMP response elements binding protein regulates the expression of PGC

243 ates the transcription factor, cAMP response element-binding protein, regulating miR-212, which targe

244 mediated nuclear transport of Ras-responsive element-binding protein (RREB1), which sustains Ras/ERK

245 elevated STMN Ser-63 and CREB (cAMP-response element-binding protein) Ser-133 phosphorylation that wa

246 B (cyclic adenosine monophosphate-responsive element-binding protein) shut-off and nuclear accumulati

247 otein 32 kDa [DARPP-32], and cAMP responsive element binding protein signaling [CREB]).

248 magnitudes of hepatic carbohydrate response element binding protein signaling activation.

249 first evidence that enhancing cAMP response element binding protein signaling can alleviate RTT phen

250 eviously unappreciated role of cAMP response element binding protein signaling in RTT pathogenesis, a

251 ive, leading to a reduction in cAMP response element binding protein signaling, but this molecular de

252 Consequently, the cAMP-->PKA-->cAMP response element-binding protein signaling axis is inhibited, the

253 -terminal Kinase (JNK) and Sterol Regulatory Element Binding Protein (SREBP) activity in neurons lead

254 get and lipogenesis factor Sterol regulatory element binding protein (SREBP) also resulted in GSC los

255 Sterol regulatory element binding protein (SREBP) cleavage-activating prot

256 Sterol regulatory element binding protein (SREBP) is a major transcription

257 al activity, activation of sterol regulatory element binding protein (SREBP) is both necessary and su

258 emistry, the overexpressed sterol regulatory element binding protein (SREBP) is shown to interact wit

259 to be an inhibitor of the sterol regulatory element binding protein (SREBP), a key regulator of chol

260 elate with the activity of sterol regulatory element binding protein (SREBP), the master regulator of

261 a and serum deprivation on sterol regulatory element-binding protein (SREBP) activity and the express

262 djusts gene expression and sterol regulatory element-binding protein (SREBP) activity in enterocytes

263 SCAP [sterol-regulatory element-binding protein (SREBP) cleavage-activating prot

264 romotes N-glycosylation of sterol regulatory element-binding protein (SREBP) cleavage-activating prot

265 e-specific inactivation of sterol regulatory element-binding protein (SREBP) cleavage-activating prot

266 Members of the sterol regulatory element-binding protein (SREBP) family of transcription

267 saminoglycan biosynthesis, sterol regulatory element-binding protein (SREBP) signaling, bone morphoge

268 lized complex required for sterol regulatory element-binding protein (SREBP) transcription factor act

269 The sterol regulatory element-binding protein (SREBP) transcription factors ha

270 The sterol regulatory element-binding protein (SREBP) transcription factors re

271 c growth of fungi requires sterol regulatory element-binding protein (SREBP) transcription factors, a

272 analogous to the mammalian sterol regulatory element-binding protein (SREBP)-1 and SREBP-2 transcript

273 and overactivation of the sterol regulatory element-binding protein (SREBP)-1-mediated lipogenic pro

274 located in introns of the sterol regulatory element-binding protein (SREBP)-encoding genes and contr

275 se (iNOS) and induction of sterol regulatory element-binding protein (SREBP).

276 ipose metabolism including sterol regulatory element-binding protein (SREBP-1), insulin receptor (IR)

277 as drug targets in NAFLD (sterol regulatory element-binding protein [SREBP], acetyl-CoA carboxylase

278 leads to activation of the sterol regulatory element-binding proteins (SREBP1 and SREBP2) that are re

279 entified a pivotal role of sterol-regulatory element binding proteins (SREBPs), in the HCV-mediated s

280 have learned how it binds sterol regulatory element-binding proteins (SREBPs) and transports them fr

281 The sterol regulatory element-binding proteins (SREBPs) are a family of transc

282 Sterol-regulatory element-binding proteins (SREBPs) are key transcription

283 tion and the activation of sterol regulatory element-binding proteins (SREBPs) has a central role in

284 Sterol regulatory element-binding proteins (SREBPs) in the fission yeast S

285 TORC1 is known to activate sterol regulatory element-binding proteins (SREBPs) including SREBP-2, a m

286 formed a complex with the sterol regulatory element-binding proteins (SREBPs) through their C-termin

287 homologue of the mammalian sterol regulatory element-binding proteins (SREBPs), BbSre1, was shown to

288 mal cells is maintained by sterol regulatory element-binding proteins (SREBPs), membrane-bound transc

289 inhibits the activation of sterol regulatory element-binding proteins (SREBPs).

290 ogenesis by activating the sterol regulatory element-binding proteins (SREBPs).

291 lex interact with the STOREKEEPER RELATED1/G-Element Binding Protein (STKR1) inside the plant cell nu

292 t signaling by hepatic carbohydrate response element binding protein that improves glucose tolerance

293 pressor forms, and activates cAMP-responsive element binding protein that in turn represses gli1 tran

294 ch is regulated by the carbohydrate-response element-binding protein through raised cellular intermed

295 membranes that transports sterol regulatory element-binding proteins to the Golgi complex for proteo

296 essed binding of phospho-CREB (cAMP response element binding protein) to Bdnf promoters in VTA, which

297 ontrolling activity of the sterol regulatory element-binding protein transcription factor Sre1.

298 rough the transcription factor cAMP response element binding protein-triggered transcriptional activa

299 mRNA stability via modulation of the AU-rich element-binding protein tristetraprolin (TTP).

300 uch as ATF-2, c-Jun, and CREB (cAMP response element-binding protein), was disrupted.