ライフサイエンスコーパス: element-binding protein

1 the transcription factor CREB (cAMP-response element binding protein).

2 e transcription factor CREB (cAMP-responsive element binding protein).

3 regulator CPEB3 (cytoplasmic polyadenylation element-binding protein).

4 f ERK1/2, pCREB [phospho-CREB (cAMP response element-binding protein)].

5 expression, as a SERT distal polyadenylation element binding protein.

6 ibosomal S6 kinase, c-Jun, and cAMP response element binding protein.

7 activation of Creb3l3/cyclic AMP-responsive element-binding protein.

8 athway involving Akt1/Akt2 and cAMP response element-binding protein.

9 on of the transcription factor cAMP response element-binding protein.

10 iption factors, especially sterol-regulatory element binding proteins.

11 inactivation of tristetraprolin, two AU-rich element-binding proteins.

12 pithelial cells, cytoplasmic polyadenylation element binding protein 1 (CPEB1) mediates the apical lo

13 ter through association with cAMP responsive element binding protein 1 (CREB)/CREB binding protein (C

14 AMP1-dependent activation of cAMP-responsive element binding protein 1 (CREB1) and SP7 (also known as

15 nteraction between Mesp1 and cAMP-responsive element binding protein 1 (Creb1) in vitro and in vivo.

16 lucocorticoid receptor (GR), cAMP responsive element binding protein 1 (CREB1), peroxisome proliferat

17 pogenesis that results from increased sterol element binding protein 1 (SREBP-1) activity.

18 r the transcription factor sterol regulatory element binding protein 1 (SREBP-1) and its transcriptio

19 domain containing 1C), RREB1 (Ras responsive element binding protein 1), and SEC24C (SEC24 family mem

20 RREB1 (RAS-responsive element binding protein 1), identified as a component of

21 through the activation of sterol regulatory element binding protein 1, which is mediated by the indu

22 ls through interference with cAMP responsive element binding protein 1-mediated transcriptional regul

23 The transcription factor cyclic AMP-response element-binding protein 1 (CREB1) controls approximately

24 hms, we identified cyclin J and far upstream element-binding protein 1 (FUBP1) as novel miR-16 target

25 replaced by its human homolog Ras-responsive element-binding protein 1 (RREB-1).

26 timulates lipogenesis in a sterol regulatory element-binding protein 1 (SREBP-1)-dependent manner.

27 eficiency inhibits hepatic sterol regulatory element-binding protein 1 (SREBP-1, ADD1) processing.

28 on 16 of the gene encoding sterol regulatory element-binding protein 1 (Srebp1), an activator of LC-P

29 Sterol regulatory element-binding protein 1 (SREBP1), when presented in it

30 n turn directly suppresses sterol regulatory element-binding protein 1 (SREBP1)-directed transcriptio

31 and activity and defined a sterol regulatory element-binding protein 1 (SREBP1)-mediated pathway thro

32 in cell number), increased sterol regulatory element-binding protein 1 expression (about 3-fold incre

33 synthetic promoters containing cAMP response element-binding protein 1(CREB1)-specific cAMP-responsiv

34 l factor CREB1 (cyclic AMP [cAMP]-responsive element-binding protein 1) had the strongest increase in

35 nd report the binding of FUBP1 (Far Upstream element-Binding Protein 1) to these elements and the rol

36 ch included higher nuclear sterol regulatory element-binding protein 1, higher Srepb2 messenger RNA (

37 nd duodenal homeobox 1 and sterol regulatory element-binding protein 1, together leading to poor proi

38 CU's transcriptional regulator cAMP response element-binding protein 1.

39 r-activated receptor gamma/sterol regulatory element-binding protein 1/CD36 in hepatocytes from high

40 f wild-type mice, SREBP-1 (sterol regulatory element binding protein-1) activates the same promoter i

41 Sterol regulatory element-binding protein-1 (SREBP-1) is a key transcripti

42 nositide 3 kinase (PI3K)/Akt/sterol response element-binding protein-1 (SREBP-1) signaling pathway in

43 -bound precursor forms of sterol-regulatory, element-binding protein-1 and -2, transcription factors

44 ls increased expression of sterol regulatory element binding protein 1c (SREBP-1c) and its downstream

45 simulating the increase in sterol-regulatory element binding protein 1c (SREBP-1c) seen in NAFLD pati

46 insulinemia, increased liver sterol response element binding protein 1c mRNA, or obesity.

47 insulinemia, increased liver sterol response element binding protein 1c, and obesity.

48 Sterol regulatory element-binding protein 1c (SREBP-1c) is a central regul

49 n in hepatic expression of sterol regulatory element-binding protein 1c (SREBP-1c), acetyl-CoA carbox

50 egulates the expression of sterol regulatory element-binding protein 1c (SREBP-1c), leading to increa

51 with the LXR target genes, steroid receptor element-binding protein 1c and ATP binding cassette tran

52 d the expression of mature sterol regulatory element-binding protein 1c and fatty acid synthase.

53 ased in WT mice, including sterol regulatory element-binding protein 1c target gene fatty-acid syntha

54 nding protein) or SREBP1c (sterol regulatory element-binding protein 1c) regulation of Mstn expressio

55 se Acc1 phosphorylation, and sterol response element-binding protein 1c-dependent gene expression.

56 ed up-regulated PGC-1beta, sterol regulatory element-binding proteins 1c and 2, acetyl-CoA carboxylas

57 X receptor (LXR)-mediated sterol regulatory element binding protein-1c (SREBP-1c) gene expression.

58 consensus sequence in the sterol regulatory-element binding protein-1c (Srebp-1c) promoter.

59 hich induced expression of sterol regulatory element binding protein-1c (SREBP-1c), resulting in indi

60 tor-gamma (PPAR-gamma) and sterol regulatory element binding protein-1c in WAT accounted for the phen

61 D-1 protein, activation of sterol regulatory element-binding protein-1c (SREBP-1), accumulation of ce

62 ption of the gene encoding sterol regulatory element-binding protein-1c (SREBP-1c).

63 Cytoplasmic polyadenylation element binding protein 2 (CPEB2) is an RNA-binding prot

64 tial equation model of the sterol regulatory element binding protein 2 (SREBP-2) cholesterol genetic

65 ular endothelial cells via sterol regulatory element binding protein 2 (SREBP2).

66 hibiting the activation of sterol regulatory-element binding protein 2 and downregulating low-density

67 lational regulator cytosolic polyadenylation element-binding protein 2 (CPEB2) has two isoforms, CPEB

68 nds to the W-boxes of DEHYDRATION-RESPONSIVE ELEMENT-BINDING PROTEIN 2 (GhDREB2), which encodes a deh

69 e have previously identified iron-responsive element-binding protein 2 (IRP2) as an important COPD su

70 within the genes encoding sterol regulatory element-binding protein 2 (SREBP-2) and SREBP-1, respect

71 ynthesis by a reduction in sterol regulatory element-binding protein 2 (SREBP2)-regulated transcripti

72 ream transcriptional repressor cAMP response element-binding protein 2 in SNs.

73 n-vivo models suggest that sterol regulatory element-binding protein 2 is a key molecule in aggravati

74 Sterol regulatory element-binding protein 2, a master regulator in cholest

75 take of cholesterol (human sterol-regulatory element-binding protein 2, human 3-hydroxy-3-methylgluta

76 esis through activation of sterol regulatory element-binding protein-2 (SREBP-2).

77 neurotrophins activate the sterol regulatory element-binding protein-2 (SREBP2) that regulates genes

78 ER stress can activate the sterol regulatory element-binding protein-2 (SREBP2), an ER-localized tran

79 w a strong increase in the sterol-regulatory element-binding protein-2 yet are unable to maintain nor

80 ein partners, DREB2A (dehydration-responsive element-binding protein 2A), ANAC013, and ANAC046, consi

81 Luman/cAMP response element binding protein 3 is an endoplasmic reticulum (E

82 eolytic processing of CREB3L1 (cAMP response element-binding protein 3-like 1), a membrane-bound tran

83 Cyclic AMP-responsive element-binding protein 3-like 3, hepatocyte specific (C

84 show that the mRNA encoding cAMP responsive element-binding protein-3 like-1 (CREB3L1), a transcript

85 ic adenosine monophosphate (cAMP) responsive-element-binding protein, a crucial mediator in long-term

86 The ABA Binding Factor/ABA-Responsive Element Binding Proteins (ABF/AREB) subfamily of bZIP-ty

87 tes leptin transcription via cAMP-responsive element binding protein activation (CREB activation) and

88 AGE increases sterol regulatory element binding protein activity, which leads to an incr

89 iated by inhibition of carbohydrate response element binding protein activity.

90 ral kinases to inhibit carbohydrate response element-binding protein-alpha and -beta.

91 n of cyclic adenosine monophosphate response element binding protein and activating transcription fac

92 ding to the phosphorylation of cAMP response element binding protein and, consequently, SMC rediffere

93 f the transcription factor cellular response element-binding protein and an increase in brain-derived

94 s C1q-activated phosphorylated cAMP-response element-binding protein and AP-1, two transcription fact

95 s mediated by hepatic cholesterol responsive element-binding protein and featured portal/lobular infl

96 ng the transcription factors cAMP-responsive element-binding protein and forkhead box O.

97 g to cyclic adenosine monophosphate response element-binding protein and the induction of CD25, CD69,

98 by transcription factor CREB (cAMP-response element-binding protein) and silencing of CREB resulted

99 APETALA2/ethylene-responsive element binding protein (AP2/EREBP) transcription factor

100 PKG1 and cAMP response element-binding protein are involved in the signal trans

101 AU-rich element-binding proteins (ARE-BPs) offer post-transcript

102 on via inhibition of carbohydrate-responsive element-binding protein-beta, pyruvate kinase L, SCD-1,

103 or 1alpha (PGC1alpha), cyclic AMP-responsive element binding protein binding protein (CBP), steroid r

104 CBP (CREB (cAMP responsive element binding protein) binding protein (CREBBP)) and P

105 Overexpression of the p300/cAMP-response element-binding protein-binding protein (CBP) potentiate

106 ment of the coregulators cyclic-AMP response element-binding protein-binding protein and nuclear core

107 inished, thereby enabling p300/cAMP response element-binding protein-binding protein to acetylate ERa

108 alpha, and HATs (p300 and p300/cAMP response element-binding protein-binding protein-associated facto

109 luding activator protein 1 and cAMP response element-binding protein, both of which were elevated ove

110 reduction in the activation of cAMP response element-binding protein, but not the activation of Ca(2+

111 n of cyclic adenosine monophosphate response element binding protein (CBP), p300, and Cbp/p300-intera

112 uced expression of the cyclic AMP-responsive element-binding protein (CBP), which as part of the p300

113 se-responsive factor Carbohydrate-Responsive Element Binding Protein (ChREBP) and GLP-1 secretion by

114 Carbohydrate responsive element binding protein (ChREBP) is central for de novo

115 Carbohydrate-response element binding protein (ChREBP) plays a key role in reg

116 educes the activity of carbohydrate response element binding protein (ChREBP), a cellular hexose-phos

117 lleagues show that the carbohydrate response element-binding protein (ChREBP) coordinates an adaptive

118 egulated expression of carbohydrate response element-binding protein (ChREBP) in cells containing AR-

119 The carbohydrate-response element-binding protein (ChREBP) is a glucose-responsive

120 Carbohydrate-responsive element-binding protein (ChREBP) is a glucose-sensing tr

121 he expression of the carbohydrate-responsive element-binding protein (ChREBP) was decreased in adipos

122 we hypothesized that carbohydrate-responsive element-binding protein (ChREBP), a transcriptional acti

123 tor alpha (PPARalpha), carbohydrate response element-binding protein (ChREBP), and cAMP response elem

124 fficient to suppress carbohydrate-responsive-element-binding protein (ChREBP, a master lipogenic regu

125 tic glucose production through cAMP-response element-binding protein co-activators, we depleted these

126 Cytoplasmic polyadenylation element binding protein (CPEB)3 is a sequence-specific R

127 n of VEGF by the cytoplasmic polyadenylation element-binding proteins CPEB1 and CPEB4 during developm

128 sed cAMP signaling and reduced cAMP-response-element binding protein (CREB) activation, a critical pa

129 protein (MAP) kinase and cyclic-AMP-response element binding protein (CREB) activation.

130 increasing phosphorylation of CAMP response element binding protein (CREB) and PSD95 after TBI.

131 activated PERK phosphorylates CAMP response element binding protein (CREB) and PSD95 directly at the

132 ected with a dominant-negative cAMP response element binding protein (CREB) and was eliminated by mut

133 how that the activity of the cAMP responsive element binding protein (CREB) family of transcription f

134 the transcriptional regulator cAMP response element binding protein (CREB) in both mouse and human B

135 AMP and the phosphorylation of cAMP response element binding protein (CREB) in INS-1 cells.

136 rt, by activation of the cyclic AMP response element binding protein (Creb) in the aged hippocampus.

137 cAMP response element binding protein (CREB) is a key regulator of glu

138 cAMP-response element binding protein (CREB) is a nuclear transcriptio

139 The cyclic AMP response element binding protein (CREB) is a primary hub of activ

140 e have demonstrated that cyclic AMP response element binding protein (CREB) is constitutively activat

141 ctor cyclic adenosine monophosphate response element binding protein (CREB) is overexpressed in many

142 , TDCA significantly increased cAMP response element binding protein (CREB) phosphorylation in FLO-1

143 impair efficient TSH-dependent cAMP response element binding protein (CREB) phosphorylation.

144 ing to increased levels of the cAMP response element binding protein (CREB), a transcriptional factor

145 d protein kinase (p38 MAPK), cAMP-responsive element binding protein (CREB), and activating transcrip

146 e for the transcription factor cAMP response element binding protein (CREB), and we demonstrate that

147 iron-dependent activation of cAMP-responsive element binding protein (CREB), the transcription factor

148 racellular signaling molecule, cAMP-response element binding protein (CREB), which serves as a pivota

149 mutant S386/396E bound to the cAMP response element binding protein (CREB)-binding protein reveals t

150 arvation-inducible coactivator cAMP response element binding protein (CREB)-cAMP-regulated transcript

151 mic Ca(2+) elevation and cyclic AMP response element binding protein (CREB)-dependent transcription.

152 the cyclic adenosine monophosphate response element binding protein (CREB)-regulated transcription c

153 generates an unusual chimeric cAMP response element binding protein (CREB)-regulated transcriptional

154 w,Ind mice show changes on a cAMP-responsive element binding protein (CREB)-regulated transcriptional

155 ation and phosphorylation of cAMP responsive element binding protein (CREB).

156 iated phosphorylation of cyclic AMP response element binding protein (CREB).

157 cyclase, protein kinase A and cAMP response element binding protein (CREB).

158 of a persistent activation of cAMP-response element binding-protein (CREB) and C/EBPbeta expression.

159 d IL-10 resulted in epithelial cAMP response element-binding protein (CREB) activation and subsequent

160 by free fatty acid (FFA), and cAMP response element-binding protein (CREB) activation by glucagon, l

161 rk1/2 MAP-kinase signaling and cAMP response element-binding protein (CREB) activation in myeloid cel

162 e of cyclic adenosine monophosphate response element-binding protein (CREB) activity.

163 negative regulation of cAMP/calcium response element-binding protein (CREB) and CREB-activated gene e

164 pressant action: activation of cAMP response element-binding protein (CREB) and induction of brain-de

165 hanceosome consisting of cyclic AMP response element-binding protein (CREB) and nuclear factor Y (NF-

166 through the phosphorylation of cAMP response element-binding protein (CREB) and the dephosphorylation

167 ith higher levels of the cyclic-AMP-response-element-binding protein (CREB) are more likely to be rec

168 KA subsequently phosphorylated cAMP response element-binding protein (CREB) at Ser-133 to activate it

169 the phosphorylation of cAMP/calcium response element-binding protein (CREB) at serine 133 and CREB-me

170 For example, deficits in cAMP response element-binding protein (CREB) binding protein (CBP; an

171 h the BRCA1 promoter and the cAMP-responsive element-binding protein (CREB) complex, a regulator of B

172 ting transcriptional activator cAMP response element-binding protein (CREB) coordinately regulate the

173 denosine 3',5'-monophosphate (cAMP) response element-binding protein (CREB) family transcription fact

174 The transcription factor cAMP response element-binding protein (CREB) has been implicated in th

175 nding protein (C/EBPbeta), and cAMP-response element-binding protein (CREB) have been involved in res

176 elays the dephosphorylation of cAMP response element-binding protein (CREB) in human neuroblastoma (S

177 ctor cyclic adenosine monophosphate response element-binding protein (CREB) in NSTEMI (p < 0.05 vs. c

178 ty of the transcription factor cAMP response element-binding protein (CREB) in young adult rodents fa

179 by the transcription factor cAMP-responsive element-binding protein (CREB) is essential for a wide r

180 tors, the phosphorylation of cAMP-responsive element-binding protein (CREB) is strongly decreased in

181 n under basal/nicotine-induced/cAMP-response element-binding protein (CREB) overexpressed conditions

182 via the protein kinase C (PKC)-cAMP-response element-binding protein (CREB) pathway.

183 olar PregS similarly activates cAMP response element-binding protein (CREB) phosphorylation (within 1

184 Cyclic AMP-response element-binding protein (CREB) plays key transcriptional

185 Cyclic-AMP response element-binding protein (CREB) signaling has a critical

186 ccompanied by maintenance of cAMP responsive element-binding protein (CREB) signaling in neurons rath

187 PKA-mediated induction of the cAMP response element-binding protein (CREB) signaling pathway.

188 genome sequencing) to identify cAMP response element-binding protein (CREB) targets following chronic

189 at HDAC2 is a direct target of cAMP response element-binding protein (CREB) that is activated by beta

190 dependent binding of FGFR1 and cAMP-response element-binding protein (CREB) to a conserved cAMP respo

191 kinase II (CaMKII) and Ca(2+)/cAMP response element-binding protein (CREB) transcription factor.

192 gets of protein kinase A (PKA)-cAMP response element-binding protein (CREB), a pathway activated by f

193 NA for a transcription factor, cAMP response element-binding protein (CREB), and by an inhibitor of i

194 horylation of a CaN substrate, cAMP response element-binding protein (CREB), in the brains of Rcan1 k

195 ownstream substrate of PKAc, cAMP-responsive element-binding protein (CREB), is inhibited in cells ex

196 Phosphorylated cAMP response element-binding protein (CREB), nuclear NFATc4, and the

197 However, phosphorylation of cAMP-response element-binding protein (CREB), the cAMP-regulated trans

198 scriptional coactivators cyclic-AMP response element-binding protein (CREB)-binding protein (CBP) and

199 show that high glucose induced cAMP response element-binding protein (CREB)-binding protein (CBP)-med

200 normal ryanodine receptor and cAMP response element-binding protein (CREB)-dependent gene expression

201 gon via a cAMP-dependent and cAMP-responsive element-binding protein (CREB)-dependent mechanism in pr

202 712 on memory formation and on cAMP response element-binding protein (CREB)-regulated genes in aged m

203 ostaglandin E2 was mediated by cAMP response element-binding protein (CREB).

204 reduced phosphorylation of the cAMP response element-binding protein (CREB).

205 nhanced the phosphorylation of cAMP response element-binding protein (CREB).

206 cade adenylyl cyclase-cAMP-PKA-cAMP response element-binding protein (CREB).

207 ivation and subsequent cyclic AMP-responsive element-binding protein (CREB)/CREB-regulated transcript

208 ted the transcription factor cAMP-responsive element binding protein (Creb1) and its transcriptional

209 let alpha cells by GS/cAMP/PKA/cAMP-response element-binding protein-dependent activation of PC1.

210 : p38-dependent growth arrest, cAMP response element-binding protein-dependent cell survival, and ERK

211 Inhibition of cAMP response element-binding protein-dependent signaling may particip

212 s and subsequent increase in cAMP responsive element binding protein DNA-binding activity and inducti

213 2, ribosomal S6 kinase 1, or cAMP responsive element binding protein DNA-binding activity prevented t

214 1 interacts with the adenylateuridylate-rich element-binding protein embryonic lethal abnormal vision

215 The far upstream element binding protein (FBP) and the FBP-interacting re

216 The transcription factor far upstream element binding protein (FBP) binds and activates the MY

217 Transcription factors of the far-upstream element-binding protein (FBP) family represent cellular

218 R) protein that transports sterol regulatory element-binding proteins from the ER to Golgi, where the

219 els of specific members of the cAMP Response Element Binding protein gene family.

220 d transcription factor cyclic-AMP-responsive-element-binding protein H (CREBH) but not by peroxisome

221 Here we reported that cyclic AMP-responsive element-binding protein H (CREBH) positively regulates m

222 nuclear transcription factor cAMP-responsive element-binding protein H (CREBH) processing.

223 ike) gene encoding the carbohydrate response element binding protein has been associated with lower t

224 riched transcription factor, cAMP-responsive element-binding protein hepatic-specific (CREBH), modula

225 Cyclic AMP-responsive element binding protein, hepatocyte specific (CREBH), is

226 -binding protein (ChREBP), and cAMP response element-binding protein, hepatocyte specific (CREBH).

227 cy reduced the activation of cAMP-responsive element binding protein-hepatocyte specific and peroxiso

228 Orb2 is a cytoplasmic polyadenylation element-binding protein homolog in Drosophila melanogast

229 in the efficient signaling to cAMP-response element-binding protein in neurons.

230 but not its paralog carbohydrate-responsive element-binding protein, is the predominant glucose-resp

231 eby impeding activation of sterol regulatory element-binding proteins (key regulators of lipid metabo

232 ng reduction in phosphorylated cAMP-response element binding protein levels in the NAcSh that could a

233 nflammatory cytokines, carbohydrate response element binding protein-mediated transcription, and micr

234 anscription factors CREB/CREM (cAMP response element-binding protein/modulator) is linked to atrial f

235 critical for ChREBP (carbohydrate-responsive element-binding protein) or SREBP1c (sterol regulatory e

236 s of their respective targets, cAMP response element-binding protein, p38, and extracellular signal-r

237 e cyclase --> cAMP --> PKA --> cAMP response element-binding protein pathway mediating cell survival

238 of Insig proteins and the sterol regulatory element-binding protein pathway.

239 ogram, including Ras and cyclic AMP response element-binding protein pathways and other Gata factors,

240 ced levels of phosphorylated cAMP-responsive element binding protein (pCREB) in the CA1s of a-FMHis-t

241 lower levels of phosphorylated cAMP response element-binding protein (pCREB), an activity-dependent t

242 s protein kinase A-dependent cAMP-responsive element-binding protein phosphorylation, the effect of c

243 ACEA-induced Galphas-dependent cAMP response element-binding protein phosphorylation.

244 creases in PKA activity, CREB (cAMP-response element-binding protein) phosphorylation, induction of p

245 fatostatin A, which blocks sterol regulatory element-binding protein proteolytic cleavage and activat

246 olipoprotien C3 (Apoc3), CREB (cAMP Response Element Binding Protein) Regulated Transcription Coactiv

247 ates the transcription factor, cAMP response element-binding protein, regulating miR-212, which targe

248 elevated STMN Ser-63 and CREB (cAMP-response element-binding protein) Ser-133 phosphorylation that wa

249 B (cyclic adenosine monophosphate-responsive element-binding protein) shut-off and nuclear accumulati

250 otein 32 kDa [DARPP-32], and cAMP responsive element binding protein signaling [CREB]).

251 magnitudes of hepatic carbohydrate response element binding protein signaling activation.

252 first evidence that enhancing cAMP response element binding protein signaling can alleviate RTT phen

253 eviously unappreciated role of cAMP response element binding protein signaling in RTT pathogenesis, a

254 Consequently, the cAMP-->PKA-->cAMP response element-binding protein signaling axis is inhibited, the

255 -terminal Kinase (JNK) and Sterol Regulatory Element Binding Protein (SREBP) activity in neurons lead

256 Sterol regulatory element binding protein (SREBP) is a major transcription

257 to be an inhibitor of the sterol regulatory element binding protein (SREBP), a key regulator of chol

258 elate with the activity of sterol regulatory element binding protein (SREBP), the master regulator of

259 a and serum deprivation on sterol regulatory element-binding protein (SREBP) activity and the express

260 djusts gene expression and sterol regulatory element-binding protein (SREBP) activity in enterocytes

261 SCAP [sterol-regulatory element-binding protein (SREBP) cleavage-activating prot

262 romotes N-glycosylation of sterol regulatory element-binding protein (SREBP) cleavage-activating prot

263 e-specific inactivation of sterol regulatory element-binding protein (SREBP) cleavage-activating prot

264 Members of the sterol regulatory element-binding protein (SREBP) family of transcription

265 25-HC acts by antagonizing sterol response element-binding protein (SREBP) processing to reduce Il1

266 lized complex required for sterol regulatory element-binding protein (SREBP) transcription factor act

267 tightly controlled by the sterol regulatory element-binding protein (SREBP) transcription factor tha

268 Sterol regulatory element-binding protein (SREBP) transcription factors ar

269 The sterol regulatory element-binding protein (SREBP) transcription factors ha

270 The sterol regulatory element-binding protein (SREBP) transcription factors re

271 c growth of fungi requires sterol regulatory element-binding protein (SREBP) transcription factors, a

272 analogous to the mammalian sterol regulatory element-binding protein (SREBP)-1 and SREBP-2 transcript

273 and overactivation of the sterol regulatory element-binding protein (SREBP)-1-mediated lipogenic pro

274 located in introns of the sterol regulatory element-binding protein (SREBP)-encoding genes and contr

275 ipose metabolism including sterol regulatory element-binding protein (SREBP-1), insulin receptor (IR)

276 decrease in the level of the sterol response element-binding protein (SREBP-1).

277 ation of membrane-associated sterol response element-binding protein (SREBP-2).

278 leads to activation of the sterol regulatory element-binding proteins (SREBP1 and SREBP2) that are re

279 entified a pivotal role of sterol-regulatory element binding proteins (SREBPs), in the HCV-mediated s

280 liver X receptors (LXRs), sterol regulatory element-binding proteins (SREBPs) and the G protein-coup

281 have learned how it binds sterol regulatory element-binding proteins (SREBPs) and transports them fr

282 Sterol-regulatory element-binding proteins (SREBPs) are key transcription

283 omeostasis by transporting sterol regulatory element-binding proteins (SREBPs) from the ER to the Gol

284 Sterol regulatory element-binding proteins (SREBPs) in the fission yeast S

285 TORC1 is known to activate sterol regulatory element-binding proteins (SREBPs) including SREBP-2, a m

286 fatty acid synthase (FAS), sterol regulatory element-binding proteins (SREBPs) play a pivotal role in

287 formed a complex with the sterol regulatory element-binding proteins (SREBPs) through their C-termin

288 hibiting the processing of sterol regulatory element-binding proteins (SREBPs), transcription factors

289 inhibits the activation of sterol regulatory element-binding proteins (SREBPs).

290 ogenesis by activating the sterol regulatory element-binding proteins (SREBPs).

291 iptional program involving sterol regulatory element-binding proteins (SREBPs).

292 lex interact with the STOREKEEPER RELATED1/G-Element Binding Protein (STKR1) inside the plant cell nu

293 E FACTOR of the APETALA2/ethylene responsive element binding protein superfamily, as a DELLA interact

294 t signaling by hepatic carbohydrate response element binding protein that improves glucose tolerance

295 pressor forms, and activates cAMP-responsive element binding protein that in turn represses gli1 tran

296 membranes that transports sterol regulatory element-binding proteins to the Golgi complex for proteo

297 essed binding of phospho-CREB (cAMP response element binding protein) to Bdnf promoters in VTA, which

298 ontrolling activity of the sterol regulatory element-binding protein transcription factor Sre1.

299 mRNA stability via modulation of the AU-rich element-binding protein tristetraprolin (TTP).

300 s via phosphorylation of cyclic-AMP response element binding protein, which requires activation of es