ライフサイエンスコーパス: elementary body

1 e body, and an extracellular form called the elementary body.

2 tein) on the outer surface of the chlamydial elementary body.

3 noprecipitated with TARP from C. trachomatis elementary bodies.

4 only minimally packaged into the infectious elementary bodies.

5 ges that include the formation of infectious elementary bodies.

6 within these cells by generating infectious elementary bodies.

7 with DCs pulsed with inactivated chlamydial elementary bodies.

8 ere was a restricted production of infective elementary bodies.

9 s with no evidence of redifferentiation into elementary bodies.

10 gement of cell surface heparan sulfate by L2 elementary bodies.

11 lected late-stage proteins and transition to elementary bodies, a Chlamydia developmental form that i

12 condition is reversible, yielding infectious elementary bodies after removal of the inducers, includi

13 to lysosomes due to an intrinsic property of elementary bodies and (ii) an active modification of the

14 g of sedimented culture transport medium for elementary bodies and by TMA with 16S rRNA as a target.

15 cells infected with multiple C. trachomatis elementary bodies and cultivated at 32 degrees C for 24

16 l, leading to the uptake of fewer chlamydial elementary bodies and diminished turnover of EGFR.

17 IF) antibody titers to Chlamydia trachomatis elementary bodies and ELISA antibody to recombinant chla

18 icroscopy for the presence of C. trachomatis elementary bodies and for the presence and number of cel

19 mydia developmental transition to infectious elementary bodies and highlights the potential applicati

20 Here, in elementary bodies and in preparations of the outer membr

21 identified in purified Chlamydia trachomatis elementary bodies and in the chlamydial parasitophorous

22 dsF is concentrated in the outer membrane of elementary bodies and is surface exposed as a component

23 d imply that the cytotoxin is present in the elementary body and delivered to host cells very early d

24 Two Chlamydia morphoforms, elementary body and reticulate body, could be distinguis

25 ioning between the infectious, extracellular elementary body and the replicative, intracellular retic

26 ering the conversion of reticulate bodies to elementary bodies, and for translocation of lipid drople

27 e compared by testing C. pneumoniae purified elementary bodies, animal tissues, 228 peripheral blood

28 had increased CSF antibodies to C pneumoniae elementary body antigens as shown by enzyme-linked immun

29 lamydophila abortus or Chlamydophila pecorum elementary body antigens quantified antibodies against C

30 med by western blot assays of the CSF, using elementary body antigens.

31 noglobulin (Ig) reactivity with C pneumoniae elementary body antigens.

32 stence leads to attenuated production of new elementary bodies, appearance of morphologically aberran

33 In contrast to localization in RB, SEP in elementary bodies appears diffuse and irregular, suggest

34 The small size of infectious chlamydial elementary bodies (approximately 0.3 microm in diameter)

35 Elementary bodies are characterized by a condensed chrom

36 in synthesis inhibitors, vesicles containing elementary bodies are very slow to acquire lysosomal cha

37 42 h, they contained intermediate bodies and elementary bodies as well.

38 At 18 h after infection, only a few elementary bodies attached to cells were visible, as wer

39 hose of proteins identified in C. pneumoniae elementary bodies by matrix-assisted laser desorption io

40 ermined by diminished recovery of infectious elementary bodies, differed markedly among chlamydial st

41 -mer OmcB peptide that bound the surfaces of elementary bodies (EB) and by heparin-binding peptide cr

42 IgG antibodies to serovar D of chlamydia elementary bodies (EB) and IgG antibodies to CHSP60-1, C

43 We show that elementary bodies (EB) from two biovars of Chlamydia tra

44 t reduction in infectivity of C. trachomatis elementary bodies (EB) harvested from Desferal-exposed p

45 Serovar E elementary bodies (EB) metabolically radiolabeled with 3

46 A8-35 had a higher ratio of Abs to denatured elementary bodies (EB) over live EB, recognized more syn

47 her intranasally or intravaginally with live elementary bodies (EB).

48 lated outer membrane complexes of infectious elementary bodies (EB).

49 were immunized i.n. with C. trachomatis MoPn elementary bodies (EB).

50 matis standards ranging from 32 to 1,048,576 elementary bodies (EB)/ml of urine exhibited a linear co

51 e investigated immune responses to Chlamydia elementary body (EB) and 3 genotypically variant heat sh

52 rized by an infectious cell type known as an elementary body (EB) and an intracellular replicative fo

53 tional forms during its developmental cycle: elementary body (EB) and reticulate body (RB).

54 n a regulated fashion between the infectious elementary body (EB) and the replicative reticulate body

55 two functional and morphological forms: the elementary body (EB) and the reticulate body (RB).

56 to those from the reference C. psittaci B577 elementary body (EB) ELISA and the Chlamydia complement

57 The infectious chlamydial elementary body (EB) is metabolically inactive yet posse

58 at low or undetectable levels at the time of elementary body (EB) to reticulate body conversion early

59 transition between two forms: the infectious elementary body (EB), and the rapidly dividing reticulat

60 g between two distinct forms: the infectious elementary body (EB), and the replicative but non-infect

61 acellular chlamydial infectious particle, or elementary body (EB), is enveloped by an intra- and inte

62 We used a Chlamydia trachomatis elementary body (EB)-based enzyme-linked immunosorbent a

63 ial cell-surface GAGs, as well as chlamydial elementary body (EB)-surface GAGs, were investigated.

64 not a function of infection by more than one elementary body (EB).

65 nvironmentally resistant cell type called an elementary body (EB).

66 Vaccines based on live attenuated Chlamydia elementary bodies (EBs) can cause disease in vaccinated

67 ajor outer membrane protein (MOMP) and whole elementary bodies (EBs) from the 4 predominant serovars

68 hasic developmental cycle whereby infectious elementary bodies (EBs) invade host epithelial cells and

69 The chromatin of chlamydial elementary bodies (EBs) is stabilized by proteins with s

70 t full-length CT153 (p91) was present in the elementary bodies (EBs) of 15 C. trachomatis reference s

71 lity to recognize the immunizing peptide and elementary bodies (EBs) of C. pneumoniae by enzyme-linke

72 Elementary bodies (EBs) of C. trachomatis, serovar E (BO

73 lfate receptor for binding C. trachomatis L2 elementary bodies (EBs) prior to entry into HeLa cells.

74 In this study, C pneumoniae elementary bodies (EBs) rapidly activated p44/p42 mitoge

75 ated the presence of a type III apparatus on elementary bodies (EBs) that might function early in inf

76 vity to formalin-fixed Chlamydia trachomatis elementary bodies (EBs) to address whether these associa

77 he susceptibilities of Chlamydia trachomatis elementary bodies (EBs) to human defensin HNP-2 and porc

78 entiation of infectious, metabolically inert elementary bodies (EBs) to larger, metabolically active

79 nifested by the transformation of infectious elementary bodies (EBs) to larger, non-infectious reticu

80 crophages at low MOIs but yielded few viable elementary bodies (EBs) when macrophages were infected a

81 ection (after 20 h) when RBs reorganize into elementary bodies (EBs), and is absent in infectious EBs

82 keleton in the internalization of chlamydial elementary bodies (EBs).

83 ed absorption studies with viable chlamydial elementary bodies (EBs).

84 attenuates the production of new, infectious elementary bodies (EBs).

85 rected at formalin-fixed purified chlamydial elementary bodies (EBs).

86 Using elementary body ELISA, we demonstrated approximately 1 i

87 Chlamydia trachomatis elementary body enzyme-linked immunosorbent assay (ELISA

88 t RAM can detect as few as 10 C. trachomatis elementary bodies in less than 2 h, comparable to result

89 esponse occurs both with the introduction of elementary bodies into the host and early replication of

90 fectious reticulate bodies (RBs) and back to elementary bodies, into a state of persistence.

91 d significantly decreased incorporation into elementary bodies of both C. trachomatis and Chlamydophi

92 tocol for the growth and harvest of purified elementary bodies of Chlamydia pneumoniae is presented.

93 , to label proteins in the outer membrane of elementary bodies of Chlamydia trachomatis LGV serovar L

94 ominent lipid and nucleic acid signals while elementary bodies revealed higher carbohydrate and prote

95 Western blot analyses of purified elementary bodies showed that 11 of the 21 Pmps were det

96 odies generated to the Chlamydia trachomatis elementary bodies stained Chlamydia-infected cells, but

97 infected with multiple Chlamydia trachomatis elementary bodies, they are internalized by endocytosis

98 etween infectious but metabolically inactive elementary bodies to metabolically active but noninfecti

99 Exposure of C. pneumoniae elementary bodies to periodate, but not elevated tempera

100 Conversion of C. trachomatis elementary bodies to reticulate bodies and cell division

101 Differentiation of elementary bodies to reticulate bodies is accompanied by

102 iption, as well as initial transformation of elementary bodies to reticulate bodies, were detected ea

103 the maturation of the reticulate body to the elementary body, TTS genes expressed in the later stages

104 ation of rCPAF and UV-inactivated chlamydial elementary bodies (UV-EB) against vaginal chlamydial cha

105 d antibodies targeting Chlamydia trachomatis elementary bodies was not associated with reduced endome

106 n PMNs were depleted, the number of released elementary bodies was significantly greater as determine

107 n a dose-dependent manner; were not toxic to elementary bodies; were cidal at a concentration of > or