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1 of adult NOD-SCID mice (n = 23) by transient elevation of intraocular pressure.
2 n fail to up-regulate Mac-2 at the MTZ after elevation of intraocular pressure.
3    Ischemia to the rat retina was induced by elevation of intraocular pressure.
4 ficantly influences the iris disease and the elevation of intraocular pressure.
5   Rats were subjected to retinal ischemia by elevation of intraocular pressure.
6 ion cells that is usually associated with an elevation of intraocular pressure.
7 atory efficacy with greatly reduced risk for elevation of intraocular pressure.
8  the trabecular meshwork cells, resulting in elevation of intraocular pressure.
9  ischemia, and 3 weeks following the chronic elevation of intraocular pressure.
10 nt of the response mechanism to pathological elevations of intraocular pressure.
11 regulated and deleterious effects, including elevation of intraocular pressure and development of gla
12  Retinal IR injury was induced by unilateral elevation of intraocular pressure and the level of Hmgb1
13 ecular meshwork of one eye to induce chronic elevation of intraocular pressure and weekly imaging con
14                                              Elevation of intraocular pressure causes decrease in RNF
15 V) plate is a common cause for postoperative elevation of intraocular pressure, especially in childre
16 f retinal ganglion cells following transient elevation of intraocular pressure (experimental ischaemi
17 study was to examine the effect that chronic elevation of intraocular pressure has on the intrinsic a
18 gested that this therapy may cause sustained elevation of intraocular pressure (IOP) and may potentia
19 ree weeks after the operation; the circadian elevation of intraocular pressure (IOP) at 2 hours into
20                                              Elevation of intraocular pressure (IOP) causes nonunifor
21 f retinal mRNA expression after experimental elevation of intraocular pressure (IOP) in a rat glaucom
22 utflow across the ciliary muscle could cause elevation of intraocular pressure (IOP) in patients with
23                                An endogenous elevation of intraocular pressure (IOP) occurs at night
24 phenylephrine for retinal examination, acute elevation of intraocular pressure (IOP) was observed in
25                                    Prolonged elevation of intraocular pressure (IOP) was produced in
26 ith topical ocular 0.1% dexamethasone led to elevation of intraocular pressure (IOP), functional and
27 as a potential fibrotic agent that can cause elevation of intraocular pressure (IOP).
28 odegenerative disorder that usually involves elevation of intraocular pressure (IOP).
29 etinal ganglion cells (RGC) was generated by elevation of intraocular pressure (IOP).
30 ation of retinal proteins after experimental elevation of intraocular pressure (IOP).
31 tic nerve atrophy subsequent to a pathologic elevation of intraocular pressure (IOP).
32 n Myoc/Tigr mRNA levels in eye tissues after elevation of intraocular pressure or optic nerve transec
33                                     Stepwise elevations of intraocular pressure resulted in comparabl
34 C1 facilitated Muller gliosis induced by the elevation of intraocular pressure, suggesting that TRPC
35              Tg mice demonstrated a moderate elevation of intraocular pressure, the loss of approxima
36 chemic damage produced by a 45- to 60-minute elevation of intraocular pressure to 120 mm Hg.
37 r study, the whole process from experimental elevation of intraocular pressure to tissue processing a
38    Ischemic injury was induced by unilateral elevation of intraocular pressure via direct corneal can
39 egulatory mechanisms in response to stepwise elevations of intraocular pressure was studied.
40      Transgenic mice demonstrated a moderate elevation of intraocular pressure, which was more pronou

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