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1 of adult NOD-SCID mice (n = 23) by transient elevation of intraocular pressure.
2 n fail to up-regulate Mac-2 at the MTZ after elevation of intraocular pressure.
3 Ischemia to the rat retina was induced by elevation of intraocular pressure.
4 ficantly influences the iris disease and the elevation of intraocular pressure.
5 Rats were subjected to retinal ischemia by elevation of intraocular pressure.
6 ion cells that is usually associated with an elevation of intraocular pressure.
7 atory efficacy with greatly reduced risk for elevation of intraocular pressure.
8 the trabecular meshwork cells, resulting in elevation of intraocular pressure.
9 ischemia, and 3 weeks following the chronic elevation of intraocular pressure.
10 nt of the response mechanism to pathological elevations of intraocular pressure.
11 regulated and deleterious effects, including elevation of intraocular pressure and development of gla
12 Retinal IR injury was induced by unilateral elevation of intraocular pressure and the level of Hmgb1
13 ecular meshwork of one eye to induce chronic elevation of intraocular pressure and weekly imaging con
15 V) plate is a common cause for postoperative elevation of intraocular pressure, especially in childre
16 f retinal ganglion cells following transient elevation of intraocular pressure (experimental ischaemi
17 study was to examine the effect that chronic elevation of intraocular pressure has on the intrinsic a
18 gested that this therapy may cause sustained elevation of intraocular pressure (IOP) and may potentia
19 ree weeks after the operation; the circadian elevation of intraocular pressure (IOP) at 2 hours into
21 f retinal mRNA expression after experimental elevation of intraocular pressure (IOP) in a rat glaucom
22 utflow across the ciliary muscle could cause elevation of intraocular pressure (IOP) in patients with
24 phenylephrine for retinal examination, acute elevation of intraocular pressure (IOP) was observed in
26 ith topical ocular 0.1% dexamethasone led to elevation of intraocular pressure (IOP), functional and
32 n Myoc/Tigr mRNA levels in eye tissues after elevation of intraocular pressure or optic nerve transec
34 C1 facilitated Muller gliosis induced by the elevation of intraocular pressure, suggesting that TRPC
37 r study, the whole process from experimental elevation of intraocular pressure to tissue processing a
38 Ischemic injury was induced by unilateral elevation of intraocular pressure via direct corneal can
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