ライフサイエンスコーパス: embolic stroke

1 lity of life and is associated with risk for embolic stroke.

2 ) to control atrial fibrillation and prevent embolic stroke.

3 the LAA is believed to decrease the risk of embolic stroke.

4 they require tPA administration following an embolic stroke.

5 tly involved in vascular damage following an embolic stroke.

6 ion during stroke recovery in a rat model of embolic stroke.

7 ceptor antagonist, 7E3 F(ab')2, at 4 h after embolic stroke.

8 d delayed rht-PA treatment in a rat model of embolic stroke.

9 ound to be independent predictors of thrombo-embolic stroke.

10 ases its therapeutic window for treatment of embolic stroke.

11 heart failure, and atrial fibrillation with embolic stroke.

12 pericardiocentesis, and 1 patient with minor embolic stroke.

13 1 in cerebral microvascular remodeling after embolic stroke.

14 ion is associated with an increased risk for embolic stroke.

15 There were no major embolic strokes.

16 of mitral-valve prolapse among patients with embolic stroke (28 to 40 percent), especially among youn

17 cific WMH lesion pattern among patients with embolic stroke aetiology.

18 diac arrhythmia, is the most common cause of embolic stroke and death associated with heart failure.

19 rdiovascular morbidity and mortality through embolic stroke and heart failure.

20 Male Wistar rats were subjected to embolic stroke and received treatment via a femoral vein

21 es were related to the frequency of probable embolic stroke and to clinical and transthoracic echocar

22 Male Wistar rats were subjected to embolic stroke and treated with the combination of rtPA

23 In embolic stroke animals, positron emission tomographic-co

24 hort of consecutive patients presenting with embolic stroke at an academic hospital and tertiary refe

25 Administration of sildenafil to rats with embolic stroke enhances angiogenesis and selectively inc

26 competent 29-year-old male presented with an embolic stroke from an unusual primary cardiac lymphoma.

27 that include a reduction in the incidence of embolic stroke from cardiac, aortic and carotid sites, s

28 lasminogen activator, or both, 4 hours after embolic stroke improves the functional outcome and reduc

29 In a clinically relevant model of embolic stroke in rodents, we now show that administrati

30 emorrhage after transient brain ischemia and embolic stroke in rodents.

31 (NILT) improves behavioral outcome following embolic strokes in embolized rabbits and clinical rating

32 is more effective than aspirin in preventing embolic strokes in patients older than 75 years of age,

33 t improving behavioral performance following embolic strokes in rabbits.

34 onal cortical blood flow (RCBF) following an embolic stroke is beneficial to neurological outcome.

35 Embolic stroke is the most devastating consequence of at

36 ients with non-valvular atrial fibrillation, embolic stroke is thought to be associated with left atr

37 cerebral ischemia using a rabbit small clot embolic stroke model (RSCEM) using clinical rating score

38 used a modification of the rabbit small clot embolic stroke model (RSCEM), a multiple infarct ischemi

39 ical ATP content using the rabbit small clot embolic stroke model (RSCEM), the model originally used

40 infarct ischemia using the rabbit small clot embolic stroke model (RSCEM).

41 The rabbit large clot embolic stroke model has been used for over 23 years to

42 hemic damage in both size and severity in an embolic stroke model of rat with and without a therapeut

43 Lastly, we show that in the rabbit embolic stroke model, hemorrhages are adjacent to areas

44 a fully blinded and randomized manner in an embolic stroke model, we determined if CEPO would be use

45 s has not been demonstrated using an in vivo embolic stroke model.

46 In experimental embolic stroke models, MMP inhibitors decreased cerebral

47 7); arrhythmic sudden death (SD) (n=17); and embolic stroke (n=2).

48 ation in the chronic stage of a rat model of embolic stroke (n=6), and (ii) whether this process can

49 elated mortality events (0.64%/y), including embolic stroke (n=6), progressive heart failure or trans

50 tid crush injury (mural thrombosis model) or embolic stroke (occlusive thrombosis model) followed by

51 isease, leading to the recent formulation of embolic stroke of undetermined source as a distinct targ

52 , recent clinical trials have indicated that embolic stroke of undetermined source may often stem fro

53 ial cardiomyopathy may explain many cases of embolic stroke of undetermined source, and oral anticoag

54 ested for stroke prevention in patients with embolic stroke of undetermined source, including specifi

55 he prevention of thrombosis in patients with embolic stroke of unknown source, heart failure, coronar

56 Accordingly, we propose that embolic strokes of undetermined source are a therapeutic

57 l anticoagulants for secondary prevention of embolic strokes of undetermined source are warranted.

58 enosis (OR, 7.52; CI, 6.22-9.09; P < 0.001), embolic stroke (OR, 4.43; CI, 3.05-6.42; P < 0.001), hyp

59 cerebral perfusion is impaired directly (eg, embolic stroke) or indirectly (eg, raised intracranial p

60 with in-hospital death, nonfatal recurrence, embolic stroke, or delayed normalization of ejection fra

61 8 or more emboli per hour was predictive of embolic stroke (P = 0.0076).

62 ion burden as assessed on the Fazekas scale, embolic stroke pattern, infarct distribution and pertine

63 gnificant impact on clot trajectory and thus embolic stroke propensity through the left common caroti

64 o HT data measured histologically at 48 h in embolic stroke rats, the enhanced areas by Gd-DTPA at 24

65 s document provides the current views on (1) embolic/stroke risk, (2) ischemic/thrombotic cardiac ris

66 y cause of death in HCM virtually limited to embolic stroke, supporting a low threshold for initiatin

67 iated with a substantially increased risk of embolic stroke/TIA.

68 In conclusion, following an embolic stroke, TNFalpha administration increased the in

69 se middle cerebral occlusion (MCAO) model of embolic stroke to study neuronal degeneration following

70 with Gd-DTPA to detect HT in a rat model of embolic stroke treated with rtPA and a glycoprotein IIb/

71 The incidence of embolic stroke was higher in AFIB (n = 8) vs. no AFIB (n

72 Embolic stroke was induced in 8 male Wistar rats and mag

73 Acute ischemic or embolic stroke was seen in 7 patients (16%) with an over

74 imvastatin-induced neuroprotection following embolic strokes, we used pharmacological intervention wi

75 of sildenafil, male Wistar rats subjected to embolic stroke were treated with sildenafil (n=11) or sa

76 ch curvature is an important risk factor for embolic stroke which should be tested in future clinical

77 major complication of carotid angioplasty is embolic stroke, which may occur after balloon inflation

78 in a nonhuman primate model (n = 2) of acute embolic stroke without or with thrombolytic therapy.