ライフサイエンスコーパス: embryonal tumor

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1 after diagnosis and treatment of a pediatric embryonal tumor.

2 en younger than 3 years with newly diagnosed embryonal tumors.

3 r abnormalities, including increased risk of embryonal tumors.

4 kidney, consistent with a potential role in embryonal tumors.

5 sing functions that are disrupted in BWS and embryonal tumors.

6 the maternal allele in fetal brain and some embryonal tumors.

7 y have utility for this and other MYC-driven embryonal tumors.

8 ulloblastoma/primitive neuroectodermal tumor/embryonal tumor, 17 had malignant astrocytoma, nine had

9 Younger patients with group 1 paratesticular embryonal tumors and all patients with group 1/2 orbit o

10 or of unknown cause, is not associated with embryonal tumors and cells from these individuals show m

11 RIP13 or BUB1B mutations have a high risk of embryonal tumors, and here we show that their cells disp

12 Pediatric patients with CNS embryonal tumors are at high risk for treatment-related

13 Overall, patients with embryonal tumors benefited from intensive three-drug che

14 c tumors representing sarcomas, extracranial embryonal tumors, brain tumors, hematologic malignancies

15 ally invasive or metastatic tumors, in which embryonal tumor cells are EGFR-negative, while SCU cells

16 stnatal tissue overgrowth, increased risk of embryonal tumors during early childhood, and numerous vi

17 Conversely, at an older age, mice escaping embryonal tumor formation present with malignant gliomas

18 Genomic analysis shows that embryonal tumors have more structural and copy number va

19 region implicated in the etiology of several embryonal tumors, including Wilms tumor, and in Beckwith

20 et consistent with a time-limited window for embryonal tumor initiation.

21 unger than 3 years with primary intracranial embryonal tumors is now in progress.

22 ne abdominal wall defects, macroglossia, and embryonal tumors, is a model for understanding the relat

23 toma and neuroblastoma exemplify the current embryonal tumor model.

24 ryoid tumors (n = 19), 26% for patients with embryonal tumors (n = 313), and 5% for patients with alv

25 are highly aggressive, poorly differentiated embryonal tumors occurring predominantly in young childr

26 human tumors including rhabdomyosarcoma, an embryonal tumor of skeletal muscle origin.

27 and ErbB-4 in childhood medulloblastoma, an embryonal tumor of the cerebellar external granule cell

28 lopment and the medulloblastoma, a malignant embryonal tumor of the cerebellum, have proven especiall

29 oter hypermethylation in medulloblastoma, an embryonal tumor of the cerebellum.

30 selective radioimmunoconjugates specific for embryonal tumors of childhood are currently being active

31 Embryonal tumors of the CNS include, among others, medul

32 Analysis of the subset of patients with embryonal tumors showed similar results (P =.0008).

33 bear a genetic similarity to other infantile embryonal tumors such as hepatoblastomas.

34 rnally expressed imprinted genes involved in embryonal tumor suppression and the cancer-predisposing

35 subtransferable fragment (STF) harboring an embryonal tumor-suppressor gene and spanning about 2.5 M

36 rhabdomyosarcoma and are more effective for embryonal tumors than therapies used previously.

37 For patients with embryonal tumors, the estimated 5-year survival rate was

38 Medulloblastoma is an embryonal tumor thought to arise from the granule cell p

39 Hepatoblastoma is a rare embryonal tumor with unknown etiology.

40 Embryonal tumors with multilayered rosettes (ETMRs) are

41 Intraocular medulloepitheliomas and embryonal tumors with multilayered rosettes of the brain

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